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Science, ISSN 0036-8075, 4/2011, Volume 332, Issue 6027, pp. 361 - 365
Angiotensin II (AngII) mediates progression of aortic aneurysm, but the relative contribution of its type 1 (AT1) and type 2 (AT2) receptors remains unknown.... 
Connective tissues | Receptors | Root growth | Medical treatment | REPORTS | Aneurysms | Placebos | Aorta | Mice | Aortic aneurysm | Marfan syndrome | PATHOGENESIS | ACTIVATION | MECHANISM | MULTIDISCIPLINARY SCIENCES | MARFAN-SYNDROME | MOUSE MODEL | GROWTH | SMOOTH-MUSCLE-CELLS | BLOCKADE | CONTRIBUTES | EXPRESSION | Aortic Aneurysm - metabolism | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Aortic Rupture - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Enalapril - therapeutic use | MAP Kinase Signaling System | Marfan Syndrome - drug therapy | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Aortic Rupture - prevention & control | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Aortic Aneurysm - pathology | Aortic Aneurysm - prevention & control | Disease Models, Animal | Receptor, Angiotensin, Type 2 - genetics | Angiotensin II - metabolism | Signal Transduction | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Losartan - pharmacology | Aortic Rupture - pathology | Disease Progression | Mice, Knockout | Animals | Enalapril - pharmacology | Receptor, Angiotensin, Type 2 - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Aortic Aneurysm - drug therapy | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Aortic aneurysms | Development and progression | Genetic aspects | Health aspects | Angiotensin | Signal transduction | Peptides | Cellular biology | Coronary vessels | Rodents
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 12/2008, Volume 118, Issue 24, pp. 2523 - 2532
Journal Article
Kidney International, ISSN 0085-2538, 07/2014, Volume 86, Issue 1, pp. 75 - 85
Fibrosis is a hallmark of chronic kidney disease, for which there is currently no effective cure. The hormone relaxin is emerging as an effective antifibrotic... 
Smad2 | kidney disease | TGF-β | fibrosis | relaxin | AT2 receptor | KIDNEY-DISEASE | NITRIC-OXIDE SYNTHASE | PROTEIN-KINASE | RESONANCE ENERGY-TRANSFER | FIBROBLASTS | TGF-beta | GROWTH-FACTOR-BETA | AT RECEPTORS | IN-VIVO | UROLOGY & NEPHROLOGY | HYPERTENSIVE-RATS | HORMONE RELAXIN | Relaxin - metabolism | Kidney - pathology | Receptors, G-Protein-Coupled - metabolism | Transforming Growth Factor beta1 - antagonists & inhibitors | Humans | Protein Multimerization | Male | Receptors, Peptide - chemistry | Angiotensin II Type 2 Receptor Blockers - pharmacology | Relaxin - pharmacology | Renal Insufficiency, Chronic - drug therapy | Myofibroblasts - metabolism | Renal Insufficiency, Chronic - metabolism | Kidney - metabolism | Myofibroblasts - pathology | Receptor, Angiotensin, Type 2 - genetics | Kidney - drug effects | Cells, Cultured | Rats | Imidazoles - pharmacology | Recombinant Proteins - pharmacology | Myofibroblasts - drug effects | Rats, Sprague-Dawley | Disease Progression | Mice, Knockout | Renal Insufficiency, Chronic - pathology | Animals | MAP Kinase Signaling System - drug effects | Receptor, Angiotensin, Type 2 - metabolism | Signal Transduction - drug effects | Fibrosis | Mice | Pyridines - pharmacology | Receptor, Angiotensin, Type 2 - deficiency | Receptors, G-Protein-Coupled - chemistry | Receptors, Peptide - metabolism
Journal Article
Hypertension, ISSN 0194-911X, 02/2012, Volume 59, Issue 2 Suppl 1, pp. 485 - 492
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 08/2018, Volume 115, Issue 34, p. E8057
The type I collagenopathies are a group of heterogeneous connective tissue disorders, that are caused by mutations in the genes encoding type I collagen and... 
Collagen (type I) | Neuropathology | Genes | Variability | Disorders | Collagens | Clinical trials | Neuropathy | Macrophages | Connective tissues | Biomedical materials | Pain | Pain management | Biocompatibility | Angiotensin II | Phenotypes | Medical treatment | Phenotypic variations | Zebrafish | Pharmacology | Angiotensin | Osteogenesis imperfecta | Diagnostic systems | Mutation | ACE inhibitors
Journal Article
Journal Article
Journal Article
Journal Article
Molecular Medicine, ISSN 1076-1551, 2015, Volume 21, Issue 1, pp. 626 - 636
Journal Article
Journal Article