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by Johnson, Toby and Gaunt, Tom R and Newhouse, Stephen J and Padmanabhan, Sandosh and Tomaszewski, Maciej and Kumari, Meena and Morris, Richard W and Tzoulaki, Ioanna and O'Brien, Eoin T and Poulter, Neil R and Sever, Peter and Shields, Denis C and Thom, Simon and Wannamethee, Sasiwarang G and Whincup, Peter H and Brown, Morris J and Connell, John M and Dobson, Richard J and Howard, Philip J and Mein, Charles A and Onipinla, Abiodun and Shaw-Hawkins, Sue and Zhang, Yun and Smith, George Davey and Day, Ian N.M and Lawlor, Debbie A and Goodall, Alison H and Fowkes, F. Gerald and Abecasis, Gonçalo R and Elliott, Paul and Gateva, Vesela and Braund, Peter S and Burton, Paul R and Nelson, Christopher P and Tobin, Martin D and van der Harst, Pim and Glorioso, Nicola and Neuvrith, Hani and Salvi, Erika and Staessen, Jan A and Stucchi, Andrea and Devos, Nabila and Jeunemaitre, Xavier and Plouin, Pierre-François and Tichet, Jean and Juhanson, Peeter and Org, Elin and Putku, Margus and Sõber, Siim and Veldre, Gudrun and Viigimaa, Margus and Levinsson, Anna and Rosengren, Annika and Thelle, Dag S and Hastie, Claire E and Hedner, Thomas and Lee, Wai K and Melander, Olle and Wahlstrand, Björn and Hardy, Rebecca and Wong, Andrew and Cooper, Jackie A and Palmen, Jutta and Chen, Li and Stewart, Alexandre F.R and Wells, George A and Westra, Harm-Jan and Wolfs, Marcel G.M and Clarke, Robert and Franzosi, Maria Grazia and Goel, Anuj and Hamsten, Anders and Lathrop, Mark and Peden, John F and Seedorf, Udo and Watkins, Hugh and Ouwehand, Willem H and Sambrook, Jennifer and Stephens, Jonathan and Casas, Juan-Pablo and Drenos, Fotios and Holmes, Michael V and Kivimaki, Mika and Shah, Tina and Shah, Sonia and Talmud, Philippa J and Whittaker, John and Wallace, Chris and Delles, Christian and Laan, Maris and Kuh, Diana and Humphries, Steve E and Nyberg, Fredrik and Cusi, Daniele and Roberts, Robert and Newton-Cheh, Christopher and Franke, Lude and Stanton, Alice V and Dominiczak, Anna F and Farrall, Martin and ... and Global BPgen Consortium and Cardiogenics Consortium and Institutionen för medicin, avdelningen för samhällsmedicin och folkhälsa and Institute of Medicine and Göteborgs universitet and Gothenburg University and Institutionen för medicin, avdelningen för invärtesmedicin and Sahlgrenska Academy and Sahlgrenska akademin and Institutionen för medicin, avdelningen för akut och kardiovaskulär medicin and Institute of Medicine, Department of Emergeny and Cardiovascular Medicine and Institute of Medicine, Department of Internal Medicine and Institute of Medicine, Department of Public Health and Community Medicine and Institutionen för medicin
The American Journal of Human Genetics, ISSN 0002-9297, 12/2011, Volume 89, Issue 6, pp. 688 - 700
Journal Article
Journal Article
Journal of Medical Genetics, ISSN 0022-2593, 2015, Volume 52, Issue 1, pp. 28 - 36
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, pp. e29951 - e29951
Background: It is well known that familial sick sinus syndrome (SSS) is caused by functional alterations of ion channels and gap junction. Limited information... 
SIGNAL-TRANSDUCTION | HEART | OVEREXPRESSION | CONDUCTION SYSTEM | HUMAN GENOME | AT RECEPTOR | MULTIDISCIPLINARY SCIENCES | HAPLOTYPE | BRADYCARDIA | HYPERPLASIA | ATRIAL-FIBRILLATION | Gene Frequency - genetics | Humans | Genetic Loci - genetics | Molecular Sequence Data | Male | Case-Control Studies | Sick Sinus Syndrome - diagnostic imaging | Haplotypes - genetics | DNA Mutational Analysis | Base Sequence | Ultrasonography | Electrocardiography | Heart Rate - physiology | Female | Transcription, Genetic | Electrophoretic Mobility Shift Assay | Sick Sinus Syndrome - genetics | Promoter Regions, Genetic | Genetic Predisposition to Disease | Genetic Association Studies | Gene Expression Regulation | Linkage Disequilibrium - genetics | Genetic Vectors - genetics | Hep G2 Cells | Angiotensinogen - genetics | Sick Sinus Syndrome - physiopathology | Polymorphism, Single Nucleotide - genetics | Aged | Medical research | Genes | Luciferase | Angiotensin | Medicine, Experimental | Genetic aspects | Gene expression | Sick sinus syndrome | Genetic polymorphisms | Haplotypes | Cardiac arrhythmia | Multiple sclerosis | Transcription | Arrhythmia | Pathogenesis | Genomes | Kinases | Assaying | Gene polymorphism | Gene sequencing | Proteins | Signal transduction | Sinuses | Renin | Sinus | Rodents | Angiotensinogen | Ion channels | Life sciences | Cardiology | Age | Deoxyribonucleic acid--DNA | Genotypes | Heart failure | Hypertension | Enzymes | Patients | Medicine | Hospitals | Electrophoretic mobility | Alzheimers disease | Polymorphism | Gap junctions | Nucleotides | angiotensinogen | Promoters | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Hypertension, ISSN 0194-911X, 05/2011, Volume 57, Issue 5, pp. 903 - 910
Journal Article
Experimental Physiology, ISSN 0958-0670, 01/2014, Volume 99, Issue 1, pp. 220 - 231
Journal Article