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rna, messenger - metabolism (81) 81
pharmacology & pharmacy (80) 80
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p38 mitogen-activated protein kinases - metabolism (79) 79
reconsolidation (76) 76
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hippocampus - physiology (73) 73
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physiology (59) 59
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1. Full Text Mechanisms of Locomotor Sensitization to Drugs of Abuse in a Two-Injection Protocol
by Valjent, Emmanuel and Bertran-Gonzalez, Jesus and Aubier, Benjamin and Greengard, Paul and Hervé, Denis and Girault, Jean-Antoine
Neuropsychopharmacology, ISSN 0893-133X, 01/2010, Volume 35, Issue 2, pp. 401 - 415
A single exposure to psychostimulants or morphine is sufficient to induce persistent locomotor sensitization, as well as neurochemical and electrophysiological... 
NMDA | Morphine | Cocaine | Dopamine | Protein synthesis inhibitor | GBR12783 | cocaine | PSYCHIATRY | BEHAVIORAL SENSITIZATION | COCAINE SENSITIZATION | PROTEIN-SYNTHESIS | NEUROSCIENCES | morphine | VENTRAL TEGMENTAL AREA | NUCLEUS-ACCUMBENS | DOPAMINE NEURONS | SIGNALING PATHWAY | SINGLE EXPOSURE | NMDA-RECEPTORS | PHARMACOLOGY & PHARMACY | dopamine | protein synthesis inhibitor | UPTAKE INHIBITOR GBR12783 | Motor Activity - physiology | Dopamine and cAMP-Regulated Phosphoprotein 32 - genetics | Motor Activity - drug effects | Male | Green Fluorescent Proteins - genetics | Extracellular Signal-Regulated MAP Kinases - metabolism | Corpus Striatum - metabolism | Substance-Related Disorders - genetics | Point Mutation - genetics | Time Factors | Threonine - genetics | Raclopride - pharmacology | Protein Synthesis Inhibitors - pharmacology | Disease Models, Animal | Receptors, Dopamine D1 - antagonists & inhibitors | Dopamine Agonists - pharmacology | Drug Administration Routes | Dopamine Uptake Inhibitors - pharmacology | Drug Administration Schedule | Morphine - pharmacology | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Mice, Transgenic | Excitatory Amino Acid Antagonists - pharmacology | Maze Learning - drug effects | Substance-Related Disorders - physiopathology | Gene Expression Regulation - drug effects | Cocaine - pharmacology | Motor Activity - genetics | Animals | Analysis of Variance | Dopamine Antagonists - pharmacology | Corpus Striatum - drug effects | Anisomycin - pharmacology | Mice | Narcotics - pharmacology | Dizocilpine Maleate - pharmacology | Receptors, Dopamine D1 - genetics | Original
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2. Full Text Curcumin Modulates Efflux Mediated by Yeast ABC Multidrug Transporters and Is Synergistic with Antifungals
by Monika Sharma and Raman Manoharlal and Suneet Shukla and Nidhi Puri and Tulika Prasad and Suresh V. Ambudkar and Rajendra Prasad
Antimicrobial Agents and Chemotherapy, ISSN 0066-4804, 08/2009, Volume 53, Issue 8, pp. 3256 - 3265
Classifications Services AAC Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit... 
RESISTANCE PROTEIN-1 ABCC1 | CDR1P | CANDIDA-ALBICANS | TURMERIC POWDER | HUMAN PATHOGENIC YEAST | AZOLE RESISTANCE | MICROBIOLOGY | PHARMACOLOGY & PHARMACY | DRUG-RESISTANCE | SACCHAROMYCES-CEREVISIAE | UP-REGULATION | P-GLYCOPROTEIN ABCB1 | Antifungal Agents - pharmacology | Fluconazole - pharmacology | Methotrexate - metabolism | Curcumin - pharmacology | Enzyme Inhibitors - pharmacology | Saccharomyces cerevisiae - drug effects | Candida albicans - metabolism | Pyrimidines - pharmacology | Rhodamines - metabolism | Cycloheximide - pharmacology | Itraconazole - pharmacology | Ketoconazole - pharmacology | Drug Synergism | Saccharomyces cerevisiae - metabolism | Triazoles - pharmacology | Miconazole - pharmacology | Voriconazole | Anisomycin - pharmacology | Biological Transport - drug effects | Multidrug Resistance-Associated Proteins - metabolism | Index Medicus | Susceptibility
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3. Full Text Dynamic Translational and Proteasomal Regulation of Fragile X Mental Retardation Protein Controls mGluR-Dependent Long-Term Depression
by Hou, Lingfei and Antion, Marcia D and Hu, Daoying and Spencer, Corinne M and Paylor, Richard and Klann, Eric
Neuron, ISSN 0896-6273, 2006, Volume 51, Issue 4, pp. 441 - 454
Genetic deletion of fragile X mental retardation protein (FMRP) has been shown to enhance mGluR-dependent long-term depression (LTD). Herein, we demonstrate... 
HUMDISEASE | MOLNEURO | SYSTEM | MESSENGER-RNAS | SYNAPSES | UBIQUITIN | FMR1 | MOUSE MODEL | DEGRADATION | GLUTAMATE-RECEPTOR ACTIVATION | HIPPOCAMPAL AREA CA1 | SYNAPTIC PLASTICITY | NEUROSCIENCES | Glycine - analogs & derivatives | Microtubule-Associated Proteins - metabolism | Protein Biosynthesis - physiology | Receptors, Metabotropic Glutamate - physiology | Male | Fragile X Mental Retardation Protein - metabolism | Long-Term Synaptic Depression - drug effects | RNA, Messenger - metabolism | Dose-Response Relationship, Drug | Drug Interactions | Protein Synthesis Inhibitors - pharmacology | Animals, Newborn | Long-Term Synaptic Depression - physiology | Blotting, Western - methods | Fluorescent Antibody Technique - methods | Excitatory Amino Acid Antagonists - pharmacology | Mice, Knockout | Leupeptins - pharmacology | Methoxyhydroxyphenylglycol - analogs & derivatives | Methoxyhydroxyphenylglycol - pharmacology | Animals | Glycine - pharmacology | Signal Transduction - drug effects | Models, Biological | Cysteine Proteinase Inhibitors - pharmacology | Benzoates - pharmacology | Anisomycin - pharmacology | Protein Biosynthesis - drug effects | Fragile X Mental Retardation Protein - genetics | Mice | Pyridines - pharmacology | Proteasome Endopeptidase Complex - metabolism | In Vitro Techniques | Ubiquitin | Physiological aspects | Neurosciences | Proteolysis | Mental retardation | Depression, Mental | Proteins | Studies | Software | Protein synthesis | Experiments
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4. Full Text Nitric oxide is necessary for labilization of a consolidated context memory during reconsolidation in terrestrial snails
by Balaban, Pavel M and Roshchin, Matvey and Timoshenko, Alia K and Gainutdinov, Khalil L and Bogodvid, Tatiana K and Muranova, Lyudmila N and Zuzina, Alena B and Korshunova, Tatiana A
European Journal of Neuroscience, ISSN 0953-816X, 09/2014, Volume 40, Issue 6, pp. 2963 - 2970
Nitric oxide ( NO ) is known to be involved in associative memory formation. We investigated the influence of blocking NO function on the reconsolidation of... 
Helix lucorum | learning | animal model | behavior | invertebrates | Learning | Animal model | Behavior | Invertebrates | LONG-TERM-MEMORY | PROTEIN-SYNTHESIS | NEUROSCIENCES | RETRIEVAL | CRAB CHASMAGNATHUS | S-NITROSYLATION | REACTIVATION | EXTINCTION | APLYSIA | PLASTICITY | FOOD | Free Radical Scavengers - pharmacology | Memory - drug effects | NG-Nitroarginine Methyl Ester - pharmacology | Motor Activity - physiology | Helix (Snails) - physiology | Enzyme Inhibitors - pharmacology | Motor Activity - drug effects | Nitric Oxide Synthase - antagonists & inhibitors | Imidazoles - pharmacology | Helix (Snails) - drug effects | Indazoles - pharmacology | Nitroarginine - pharmacology | Animals | Time Factors | Benzoates - pharmacology | Anisomycin - pharmacology | Nitric Oxide Synthase - metabolism | Nitric Oxide Donors - pharmacology | Protein Synthesis Inhibitors - pharmacology | Nitric Oxide - metabolism | Electroshock | S-Nitroso-N-Acetylpenicillamine - pharmacology | Memory - physiology | Nitric oxide | Analysis
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5. Full Text Developmental Switch in Synaptic Mechanisms of Hippocampal Metabotropic Glutamate Receptor-Dependent Long-Term Depression
by Nosyreva, Elena D and Huber, Kimberly M
Journal of Neuroscience, ISSN 0270-6474, 03/2005, Volume 25, Issue 11, pp. 2992 - 3001
The presynaptic and postsynaptic properties of synapses change over the course of postnatal development. Therefore, synaptic plasticity mechanisms would be... 
CA1 | AMPA receptor endocytosis | Protein synthesis | Long-term depression | Hippocampus | Metabotropic glutamate receptor | Age Factors | Rats, Long-Evans | Receptors, Metabotropic Glutamate - metabolism | Benzoxazines | Amino Acids - pharmacology | Drug Interactions | Receptor, Cannabinoid, CB1 - agonists | Reaction Time - drug effects | Neurons - metabolism | Protein Synthesis Inhibitors - pharmacology | Electric Stimulation - methods | Hippocampus - growth & development | Xanthenes - pharmacology | Pyrazoles - pharmacology | Animals, Newborn | Long-Term Synaptic Depression - physiology | Synapses - physiology | Morpholines - pharmacology | Rats | Excitatory Amino Acid Antagonists - pharmacology | Reaction Time - physiology | Hippocampus - cytology | Cycloheximide - pharmacology | Naphthalenes - pharmacology | Hippocampus - metabolism | Methoxyhydroxyphenylglycol - analogs & derivatives | Methoxyhydroxyphenylglycol - pharmacology | Animals | Anisomycin - pharmacology | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Dizocilpine Maleate - pharmacology | In Vitro Techniques | Cellular | hippocampus | metabotropic glutamate receptor | long-term depression | protein synthesis | Molecular
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6. Full Text Role of Hippocampal Cav1.2 Ca2+ Channels in NMDA Receptor-Independent Synaptic Plasticity and Spatial Memory
by Moosmang, Sven and Haider, Nicole and Klugbauer, Norbert and Adelsberger, Helmuth and Langwieser, Nicolas and Muller, Jochen and Stiess, Michael and Marais, Else and Schulla, Verena and Lacinova, Lubica and Goebbels, Sandra and Nave, Klaus-Armin and Storm, Daniel R and Hofmann, Franz and Kleppisch, Thomas
Journal of Neuroscience, ISSN 0270-6474, 10/2005, Volume 25, Issue 43, pp. 9883 - 9892
Current knowledge about the molecular mechanisms of NMDA receptor (NMDAR)-independent long-term potentiation (LTP) in the hippocampus and its function for... 
Learning | NMDA receptor independent | Calcium channels | 1.2 | Memory | Synaptic plasticity | Tetraethylammonium - pharmacology | Nitriles - pharmacology | 2-Amino-5-phosphonovalerate - pharmacology | Neuronal Plasticity - drug effects | Membrane Potentials - radiation effects | Patch-Clamp Techniques - methods | Behavior, Animal | Dose-Response Relationship, Drug | Drug Interactions | Neuronal Plasticity - physiology | Time Factors | Pyramidal Cells - physiology | Pyramidal Cells - drug effects | Protein Synthesis Inhibitors - pharmacology | Pyramidal Cells - radiation effects | Electric Stimulation - methods | Potassium Channel Blockers - pharmacology | Membrane Potentials - drug effects | Butadienes - pharmacology | Nerve Tissue Proteins - physiology | Neuronal Plasticity - radiation effects | Fluorescent Antibody Technique - methods | Enzyme Inhibitors - pharmacology | Spatial Behavior - physiology | Calcium Channels, L-Type - physiology | Excitatory Amino Acid Antagonists - pharmacology | Hippocampus - cytology | Membrane Potentials - physiology | Nerve Tissue Proteins - drug effects | Receptors, N-Methyl-D-Aspartate - physiology | Mice, Knockout | Gene Expression Regulation - drug effects | Animals | Calcium Channels, L-Type - deficiency | Anisomycin - pharmacology | Mice | Hippocampus - physiology | Dose-Response Relationship, Radiation | Memory - physiology | Cellular | memory | calcium channels | learning | synaptic plasticity | Molecular | Cav1.2
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7. Full Text Morphine reduces mouse microglial engulfment induced by lipopolysaccharide and interferon-γ via δ opioid receptor and p38 mitogen-activated protein kinase
by Ryu, Jung-Hee and Do, Sang-Hwan and Han, Sung-Hee and Zuo, Zhiyi
Neurological Research, ISSN 0161-6412, 07/2018, Volume 40, Issue 7, pp. 602 - 608
Objective To investigate the effects of morphine on microglial phagocytosis during neuroinflammation. Methods C8-B4 mouse microglial cells were exposed to... 
microglia | Engulfment | morphine | CELLS | PHAGOCYTOSIS | MACROPHAGES | MAPK | NEUROSCIENCES | CLINICAL NEUROLOGY | Animals, Newborn | Receptors, Opioid, delta - metabolism | Microglia - drug effects | Morphine - pharmacology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Analgesics, Opioid - pharmacology | Imidazoles - pharmacology | Dose-Response Relationship, Drug | Animals | Enzyme Activators | Naloxone - pharmacology | Narcotic Antagonists - pharmacology | Lipopolysaccharides - pharmacology | Anisomycin - pharmacology | Mice | Pyridines - pharmacology | p38 Mitogen-Activated Protein Kinases - metabolism | Interferon-gamma - pharmacology | opioid effect
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8. Full Text A cell‐based screening assay to identify pharmaceutical compounds that enhance the regenerative quality of corneal repair
by Gordon, Gabriel M and LaGier, Adriana J and Ponchel, Corinne and Bauskar, Aditi and Itakura, Tatsuo and Jeong, Shinwu and Patel, Nitin and Fini, M. Elizabeth
Wound Repair and Regeneration, ISSN 1067-1927, 01/2016, Volume 24, Issue 1, pp. 89 - 99
The goal of this study was to develop and validate a simple but quantitative cell‐based assay to identify compounds that might be used pharmaceutically to give... 
MEDICINE, RESEARCH & EXPERIMENTAL | SURGERY | ACTIVATED PROTEIN-KINASE | DRUG DISCOVERY | SURGICAL OUTCOMES | HEPATIC-FIBROSIS | CELL BIOLOGY | DERMATOLOGY | EPITHELIAL-CELLS | GENE-EXPRESSION | INFLAMMATORY RESPONSE | MYOFIBROBLAST DIFFERENTIATION | REPORTER GENE | TRANSGENIC MICE | Lovastatin - pharmacology | Epithelium, Corneal - cytology | Humans | Actins - drug effects | Dichlororibofuranosylbenzimidazole - pharmacology | Actins - genetics | Tubercidin - pharmacology | Cornea - drug effects | Transforming Growth Factor beta2 - drug effects | Epithelium, Corneal - drug effects | Cornea - cytology | Protein Synthesis Inhibitors - pharmacology | Thiazolidines - pharmacology | Wound Healing - drug effects | Cell Line | Rabbits | Enzyme Inhibitors - pharmacology | Corneal Keratocytes - drug effects | Imidazoles - pharmacology | Tyrphostins - pharmacology | Bridged Bicyclo Compounds, Heterocyclic - pharmacology | Regeneration - drug effects | Animals | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Corneal Keratocytes - cytology | Cytological Techniques | Anisomycin - pharmacology | Pyridines - pharmacology | Transforming Growth Factor beta2 - genetics | Tubercidin - analogs & derivatives
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9. Full Text Hippocampal Protein Kinase C Signaling Mediates the Short-Term Memory Impairment Induced by Delta9-Tetrahydrocannabinol
by Busquets-Garcia, Arnau and Gomis-González, Maria and Salgado-Mendialduá, Victòria and Galera-López, Lorena and Puighermanal, Emma and Martín-Garcia, Elena and Maldonado, Rafael and Ozaita, Andrés
Neuropsychopharmacology, ISSN 0893-133X, 04/2018, Volume 43, Issue 5, pp. 1021 - 1031
Cannabis affects cognitive performance through the activation of the endocannabinoid system, and the molecular mechanisms involved in this process are poorly... 
PSYCHIATRY | BIDIRECTIONAL SYNAPTIC PLASTICITY | NEUROSCIENCES | NEUROGRANIN | AXON TERMINALS | CALMODULIN | IN-VIVO | DELTA-TETRAHYDROCANNABINOL | PHARMACOLOGY & PHARMACY | CANNABINOIDS | MODULATION | BRAIN | CB1 RECEPTORS | Dronabinol - antagonists & inhibitors | Sirolimus - analogs & derivatives | Microinjections | Male | Memory, Short-Term - drug effects | Hippocampus - drug effects | Rimonabant - pharmacology | Sirolimus - pharmacology | Dose-Response Relationship, Drug | Quinoxalines - pharmacology | Animals | Drug Interactions | Piperidines - pharmacology | Signal Transduction - drug effects | Isoenzymes - metabolism | Neurogranin - metabolism | Protein Kinase C - metabolism | Anisomycin - pharmacology | Hippocampus - enzymology | Mice | Phosphorylation - drug effects | Dizocilpine Maleate - pharmacology | Dronabinol - pharmacology | Phenols - pharmacology | TOR protein | Phosphorylation | Protein kinase C | Memory | Cognitive ability | Neurogranin | Impairment | Rapamycin | Hydrophobicity | Kinases | Object recognition | Proteins | Signaling | Cannabis | Molecular modelling | Inhibitors | Calcium-binding protein | Isoforms | Long term memory | Pretreatment | Short term memory | Tetrahydrocannabinol | Hippocampus | Calmodulin | Proteïnes quinases | Cànnabis | Memòria | Sistema endocannabinoid | Original
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10. Full Text β-adrenergic receptor activation facilitates induction of a protein synthesis-dependent late phase of long-term potentiation
by Gelinas, Jennifer N and Nguyen, Peter V
Journal of Neuroscience, ISSN 0270-6474, 03/2005, Volume 25, Issue 13, pp. 3294 - 3303
Long-term potentiation (LTP) is activity-dependent enhancement of synaptic strength that can critically regulate long-term memory storage. Like memory, LTP... 
LTP | Protein synthesis | Neuromodulation | Memory | Hippocampus | Noradrenergic receptors | INDUCED LTP | memory | hippocampus | noradrenergic receptors | protein synthesis | HIPPOCAMPAL CA1 REGION | SYNAPTIC PLASTICITY | AREA CA1 | NEUROSCIENCES | neuromodulation | DENDRITIC SPINES | IN-VITRO | SPATIAL MEMORY | LOW-FREQUENCY STIMULATION | DENTATE GYRUS | REQUIRING CAMP | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Adrenergic beta-Agonists - pharmacology | Receptors, Adrenergic, beta - physiology | Hippocampus - cytology | Long-Term Potentiation - radiation effects | Adrenergic beta-Antagonists - pharmacology | Propranolol - pharmacology | Animals | Drug Interactions | Isoproterenol - pharmacology | Neurons - physiology | Long-Term Potentiation - physiology | Anisomycin - pharmacology | Isoproterenol - analogs & derivatives | Flavonoids - pharmacology | Mice | Protein Synthesis Inhibitors - pharmacology | In Vitro Techniques | Neurons - drug effects | Neurons - radiation effects | Dactinomycin - pharmacology | Dose-Response Relationship, Radiation | Electric Stimulation - methods | Cellular | Molecular
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11. Full Text Synergistic requirements for the induction of dopaminergic D1/D5-receptor-mediated LTP in hippocampal slices of rat CA1 in vitro
by Navakkode, Sheeja and Sajikumar, Sreedharan and Frey, Julietta Uta
Neuropharmacology, ISSN 0028-3908, 2007, Volume 52, Issue 7, pp. 1547 - 1554
Dopaminergic D1/D5-receptor-mediated processes are important for certain forms of memory and its cellular model, i.e. hippocampal long-term potentiation (LTP)... 
Cellular memory formation | Heterosynaptic late-LTP | Protein synthesis-dependent LTP | Dopamine | Late-LTP | Hippocampus | protein synthesis-dependent LTP | STIMULATION | hippocampus | LATE-PHASE | INFORMATION | RECEPTOR | PROTEIN-SYNTHESIS | NEUROSCIENCES | REGION | MAINTENANCE | cellular memory formation | late-LTP | MEMORY | heterosynaptic late-LTP | PHARMACOLOGY & PHARMACY | LONG-TERM POTENTIATION | dopamine | EXPRESSION | Excitatory Postsynaptic Potentials - radiation effects | Rats, Wistar | 2-Amino-5-phosphonovalerate - pharmacology | Male | Hippocampus - drug effects | Excitatory Postsynaptic Potentials - drug effects | Excitatory Postsynaptic Potentials - physiology | Long-Term Potentiation - drug effects | Long-Term Potentiation - physiology | Protein Synthesis Inhibitors - pharmacology | Electric Stimulation - methods | Dopamine Agonists - pharmacology | Receptors, Dopamine D5 - physiology | Rats | Excitatory Amino Acid Antagonists - pharmacology | Benzazepines - pharmacology | Long-Term Potentiation - radiation effects | Drug Synergism | Animals | Dopamine Antagonists - pharmacology | Receptors, Dopamine D1 - physiology | Hippocampus - radiation effects | 2,3,4,5-Tetrahydro-7,8-dihydroxy-1-phenyl-1H-3-benzazepine - pharmacology | Anisomycin - pharmacology | Hippocampus - physiology | In Vitro Techniques
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12. Full Text GABA a receptor blockade enhances memory consolidation by increasing hippocampal BDNF levels
by Kim, Dong Hyun and Kim, Jong Min and Park, Se Jin and Cai, Mudan and Liu, Xiaotong and Lee, Seungheon and Shin, Chan Young and Ryu, Jong Hoon
Neuropsychopharmacology, ISSN 0893-133X, 01/2012, Volume 37, Issue 2, pp. 422 - 433
Memory consolidation is the process by which acquired information is converted to something concrete to be retrieved later. Here we examined a potential role... 
GABA | passive avoidance test | receptor | bicuculline methiodide | mature brain-derived neurotrophic factor | memory consolidation | NEURONAL-ACTIVITY | LONG-TERM-MEMORY | C-FOS | PSYCHIATRY | NEUROTROPHIC FACTOR | BASOLATERAL AMYGDALA | SYNAPTIC PLASTICITY | NEUROSCIENCES | STATE DEPENDENCY | GABA(A) receptor | IN-VIVO | PHARMACOLOGY & PHARMACY | RECOGNITION MEMORY | ONE-TRIAL AVOIDANCE | Memory - drug effects | Humans | Retention (Psychology) - drug effects | Carbazoles - administration & dosage | Male | Indole Alkaloids - administration & dosage | Retention (Psychology) - physiology | CA1 Region, Hippocampal - drug effects | GABA-A Receptor Antagonists - administration & dosage | Brain-Derived Neurotrophic Factor - pharmacology | Brain-Derived Neurotrophic Factor - physiology | Reaction Time - drug effects | CA1 Region, Hippocampal - metabolism | Anisomycin - administration & dosage | Brain-Derived Neurotrophic Factor - metabolism | Bicuculline - analogs & derivatives | Bicuculline - antagonists & inhibitors | GABA-A Receptor Antagonists - pharmacology | Microinjections | Avoidance Learning - physiology | Bicuculline - pharmacology | Recombinant Proteins - pharmacology | Mice, Inbred ICR | Recombinant Proteins - administration & dosage | Antibodies - pharmacology | Antibodies - administration & dosage | Bicuculline - administration & dosage | Brain-Derived Neurotrophic Factor - administration & dosage | Animals | Indole Alkaloids - pharmacology | Receptors, GABA-A - physiology | Anisomycin - pharmacology | Avoidance Learning - drug effects | Mice | Carbazoles - pharmacology | Memory - physiology
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13. Full Text Isoflurane attenuates mouse microglial engulfment induced by lipopolysaccharide and interferon-γ possibly by inhibition of p38 mitogen-activated protein kinase
by Ryu, Jung-Hee and Wang, Zhi and Fan, Dan and Han, Sung-Hee and Do, Sang-Hwan and Zuo, Zhiyi
NeuroReport, ISSN 0959-4965, 09/2016, Volume 27, Issue 14, pp. 1101 - 1105
Microglial engulfment is a basic function to clean up dead and injured cells and invaders, such as bacteria. This study was designed to assess the effects of... 
engulfment | isoflurane | microglia | p38 MAPK | Animals, Newborn | Calcium-Binding Proteins - metabolism | Microglia - metabolism | Isoflurane - pharmacology | Microglia - drug effects | Cells, Cultured | Anesthetics - pharmacology | Imidazoles - pharmacology | Animals | Cerebellum - cytology | Lipopolysaccharides - pharmacology | Anisomycin - pharmacology | Mice | Microfilament Proteins - metabolism | Pyridines - pharmacology | p38 Mitogen-Activated Protein Kinases - metabolism | Protein Synthesis Inhibitors - pharmacology | Interferon-gamma - pharmacology
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14. Full Text Activity-Dependent Long-Term Potentiation of Intrinsic Excitability in Hippocampal CA1 Pyramidal Neurons
by Xu, Jun and Kang, Ning and Jiang, Li and Nedergaard, Maiken and Kang, Jian
Journal of Neuroscience, ISSN 0270-6474, 02/2005, Volume 25, Issue 7, pp. 1750 - 1760
The efficiency of neural circuits is enhanced not only by increasing synaptic strength but also by increasing intrinsic excitability. In contrast to the... 
Intrinsic excitability | channel | CaM kinases | NMDA receptors | Protein synthesis | Long-term potentiation | ACTIVATED PROTEIN-KINASE | EPSP-SPIKE POTENTIATION | SYNAPTIC-TRANSMISSION | intrinsic excitability | BIDIRECTIONAL CHANGES | Na+ channel | protein synthesis | LTP | AREA CA1 | NEUROSCIENCES | LASTING POTENTIATION | SLICES | RAT CEREBELLUM | long-term potentiation | PLASTICITY | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Calcium - metabolism | Emetine - pharmacology | 2-Amino-5-phosphonovalerate - pharmacology | Potassium Channels - physiology | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | Sodium Channels - physiology | Pyramidal Cells - physiology | Egtazic Acid - analogs & derivatives | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Long-Term Potentiation - physiology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 | 4-Aminopyridine - pharmacology | Calcium-Calmodulin-Dependent Protein Kinases - physiology | Phosphinic Acids | Rats | Excitatory Amino Acid Antagonists - pharmacology | Sodium Channels - drug effects | Bicuculline - pharmacology | Potassium Channels - drug effects | Rats, Sprague-Dawley | Learning - physiology | Patch-Clamp Techniques | Animals | Egtazic Acid - pharmacology | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | Anisomycin - pharmacology | Propanolamines | Cellular | Molecular
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15. Full Text Evidence of VTA and LC control of protein synthesis required for the behavioral tagging process
by Moncada, Diego
Neurobiology of Learning and Memory, ISSN 1074-7427, 02/2017, Volume 138, pp. 226 - 237
Several works have shown that the formation of different long-term memories relies on a behavioral tagging process. In other words, to establish a lasting... 
Behavioral tagging | Dopamine | Locus coereleus | Ventral tegmental area | Noradrenaline | Memory | OBJECT RECOGNITION MEMORY | BETA-ADRENERGIC-RECEPTORS | HIPPOCAMPAL CA1 REGION | NEUROSCIENCES | PSYCHOLOGY | PSYCHOLOGY, MULTIDISCIPLINARY | SPATIAL-MEMORY | MEDIAL PREFRONTAL CORTEX | LOCUS-COERULEUS | BEHAVIORAL SCIENCES | LONG-TERM POTENTIATION | DOPAMINE-RECEPTORS | ASCENDING PROJECTIONS | Memory - drug effects | Muscimol - pharmacology | Rats, Wistar | Memory Consolidation - drug effects | Locus Coeruleus - drug effects | Protein Biosynthesis - physiology | Male | Recognition (Psychology) - physiology | Adrenergic beta-Antagonists - pharmacology | Propranolol - pharmacology | GABA-A Receptor Agonists - pharmacology | Behavior, Animal - drug effects | Neurons - metabolism | Protein Synthesis Inhibitors - pharmacology | Neurons - drug effects | Ventral Tegmental Area - drug effects | Rats | Behavior, Animal - physiology | Avoidance Learning - physiology | Benzazepines - pharmacology | Memory Consolidation - physiology | Animals | Recognition (Psychology) - drug effects | Dopamine Antagonists - pharmacology | Ventral Tegmental Area - metabolism | Anisomycin - pharmacology | Protein Biosynthesis - drug effects | Avoidance Learning - drug effects | Locus Coeruleus - metabolism | Memory - physiology | Protein biosynthesis | Proteins | Multiprocessing | Analysis | Neurophysiology
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