Articles

Search Articles

Advanced search
Help

Catalogue

Articles

Databases

X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (930) 930
Publication (170) 170
Book Review (7) 7
Book Chapter (6) 6
Conference Proceeding (3) 3
Dissertation (2) 2
Government Document (2) 2
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (672) 672
anoikis (661) 661
index medicus (479) 479
apoptosis (433) 433
animals (410) 410
cell biology (300) 300
cell line, tumor (274) 274
anoikis - physiology (263) 263
oncology (255) 255
biochemistry & molecular biology (252) 252
mice (218) 218
metastasis (215) 215
expression (213) 213
cancer (206) 206
female (182) 182
signal transduction (177) 177
activation (155) 155
survival (138) 138
cell adhesion (125) 125
phosphorylation (120) 120
cell survival (115) 115
anoikis - drug effects (110) 110
apoptosis - physiology (107) 107
growth (107) 107
cell proliferation (101) 101
cells, cultured (100) 100
cell line (99) 99
male (96) 96
focal adhesion kinase (93) 93
adhesion (92) 92
article (92) 92
integrins (92) 92
proteins (90) 90
extracellular-matrix (88) 88
research (84) 84
down-regulation (82) 82
cell adhesion - physiology (81) 81
tumors (81) 81
anoikis resistance (80) 80
breast cancer (77) 77
gene expression (75) 75
gene expression regulation, neoplastic (74) 74
signal transduction - physiology (74) 74
breast neoplasms - pathology (72) 72
neoplasm metastasis (69) 69
induced apoptosis (67) 67
up-regulation (67) 67
transfection (65) 65
blotting, western (64) 64
metastases (64) 64
invasion (63) 63
rats (63) 63
cell death (62) 62
extracellular matrix (62) 62
extracellular matrix - metabolism (62) 62
e-cadherin (61) 61
kinases (61) 61
research article (61) 61
breast-cancer (60) 60
analysis (59) 59
physiology (59) 59
stem cells (59) 59
cells (58) 58
development and progression (58) 58
cancer cells (57) 57
caspases - metabolism (57) 57
epithelial-cells (57) 57
medicine (57) 57
phosphatidylinositol 3-kinases - metabolism (57) 57
tumor cells, cultured (57) 57
anoikis - genetics (56) 56
epithelial-mesenchymal transition (56) 56
protein (56) 56
breast neoplasms - metabolism (54) 54
cadherins - metabolism (54) 54
proto-oncogene proteins c-akt - metabolism (54) 54
enzyme activation (53) 53
neoplasm invasiveness (53) 53
apoptosis - drug effects (52) 52
cell movement (51) 51
epithelial cells - metabolism (51) 51
inhibition (51) 51
pathway (51) 51
death (50) 50
physiological aspects (50) 50
immunohistochemistry (49) 49
mice, nude (49) 49
cell adhesion - drug effects (48) 48
cell survival - physiology (48) 48
genetic aspects (48) 48
health aspects (47) 47
proliferation (47) 47
carcinoma (46) 46
cell survival - drug effects (46) 46
genetics & heredity (46) 46
kinase (46) 46
multidisciplinary sciences (46) 46
proto-oncogene proteins - metabolism (46) 46
cell adhesion & migration (45) 45
autophagy (44) 44
more...
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (913) 913
Chinese (8) 8
French (5) 5
Japanese (5) 5
Czech (1) 1
Hebrew (1) 1
Polish (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


1. Full Text P14ARF interacts with the focal adhesion kinase and protects cells from anoikis
by Vivo, M and Fontana, R and Ranieri, M and Capasso, G and Angrisano, T and Pollice, A and Calabrò, V and La Mantia, G
Oncogene, ISSN 0950-9232, 08/2017, Volume 36, Issue 34, pp. 4913 - 4928
The ARF protein functions as an important sensor of hyper-proliferative stimuli restricting cell proliferation through both p53-dependent and - independent... 
DAP-KINASE | SURVIVAL | APOPTOSIS | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | CANCER | CELL BIOLOGY | ONCOLOGY | MOTOR COMPLEX | IN-VIVO | GENETICS & HEREDITY | ARF TUMOR-SUPPRESSOR | PROGRESSION | Phosphorylation - physiology | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Humans | Cell Proliferation - physiology | Death-Associated Protein Kinases - metabolism | MCF-7 Cells | Cell Adhesion - physiology | Focal Adhesions - metabolism | Cell Line, Tumor | Cytoskeleton - metabolism | Signal Transduction - physiology | Anoikis - physiology | Cytoskeleton - physiology | Apoptosis - physiology | HeLa Cells | Focal Adhesions - physiology | Tumor Suppressor Protein p14ARF - metabolism | Cell Survival - physiology | Research | Tumor proteins | Protein kinases | Apoptosis | Cell proliferation | Tumor cells | Death-associated protein kinase | p53 Protein | Polymerization | Kinases | Cell adhesion & migration | ARF protein | Proteins | Cell growth | Actin | Cytoskeleton | Anoikis | Focal adhesion kinase | Cancer | Original
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
2. Full Text A Twist-Snail Axis Critical for TrkB-Induced Epithelial-Mesenchymal Transition-Like Transformation, Anoikis Resistance, and Metastasis
by Marjon A. Smit and Thomas R. Geiger and Ji-Ying Song and Inna Gitelman and Daniel S. Peeper
Molecular and Cellular Biology, ISSN 0270-7306, 07/2009, Volume 29, Issue 13, pp. 3722 - 3737
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
BREAST-CANCER CELLS | TARGETED DISRUPTION | TUMOR PROGRESSION | E-CADHERIN REPRESSOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | GASTROINTESTINAL-TRACT | TRANSCRIPTION FACTOR SNAIL | NEUROTROPHIC RECEPTOR TRKB | EXTRACELLULAR-MATRIX | KINASE INHIBITORS | PROMOTES METASTASIS | CELL BIOLOGY | Cell Line | Cadherins - metabolism | Humans | Rats | Receptor, trkB - genetics | Mesoderm - cytology | Transcription Factors - genetics | Cell Movement - physiology | Transcription Factors - metabolism | Epithelial Cells - physiology | Neoplasm Metastasis | Animals | Twist-Related Protein 1 - genetics | Mesoderm - physiology | Signal Transduction - physiology | Anoikis - physiology | Brain-Derived Neurotrophic Factor - metabolism | Epithelium - physiology | Mice | Twist-Related Protein 1 - metabolism | Receptor, trkB - metabolism | Snail Family Transcription Factors | Morphogenesis - physiology
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
3. Full Text FAK engages multiple pathways to maintain survival of fibroblasts and epithelia - Differential roles for paxillin and p130Cas
by Zouq, Nadia K and Keeble, James A and Lindsay, Jennefer and Valentijn, Anthony J and Zhang, Lu and Mills, Deborah and Turner, Christopher E and Streuli, Charles H and Gilmore, Andrew P
Journal of Cell Science, ISSN 0021-9533, 02/2009, Volume 122, Issue 3, pp. 357 - 367
Different cell types interpret their distinct extracellular matrix (ECM) environments to bring about specific cell fate decisions, and can differentiate or... 
Epithelial cells | Anoikis | Focal adhesion kinase | SIGNALING PATHWAYS | TYROSINE PHOSPHORYLATION | TERMINAL DOMAIN | JUN NH2-TERMINAL KINASE | TUMOR-CELLS | HUMAN BREAST-CANCER | CELL-SURVIVAL | CELL BIOLOGY | FOCAL-ADHESION KINASE | EXTRACELLULAR-MATRIX | BINDS PAXILLIN | Cell Line | Focal Adhesion Kinase 1 - genetics | Epithelial Cells - metabolism | Fibroblasts - physiology | Extracellular Matrix - metabolism | Focal Adhesion Kinase 1 - physiology | Epithelial Cells - physiology | Animals | Crk-Associated Substrate Protein - physiology | Signal Transduction - physiology | Anoikis - physiology | Mice | Apoptosis - physiology | Cell Survival - physiology | Paxillin - physiology | Fibroblasts - metabolism
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
4. Full Text Pyruvate uptake is increased in highly invasive ovarian cancer cells under anoikis conditions for anaplerosis, mitochondrial function, and migration
by Christine A. Caneba and Nadège Bellance and Lifeng Yang and Lisa Pabst and Deepak Nagrath
American Journal of Physiology - Endocrinology And Metabolism, ISSN 0193-1849, 10/2012, Volume 303, Issue 8, pp. 1036 - 1052
Anoikis resistance, or the ability for cells to live detached from the extracellular matrix, is a property of epithelial cancers. The “Warburg effect,” or the... 
Bioener-getics | Cancer migration and anoikis | Metabolomics | Oxidative phosphorylation | Warburg effect | PHYSIOLOGY | ACID | KINASE | TUMOR-CELLS | oxidative phosphorylation | cancer migration and anoikis | LIVER-CANCER | GLYCOLYSIS | bioenergetics | METABOLISM | metabolomics | PROSTATE-CANCER | LACTATE | ENDOCRINOLOGY & METABOLISM | GROWTH IN-VIVO | EXPRESSION | Humans | Ovarian Neoplasms - pathology | Oxidative Phosphorylation | Adenosine Triphosphate - biosynthesis | Oxygen Consumption - physiology | Cell Movement - physiology | Amino Acids - metabolism | Culture Media | Neoplasm Invasiveness - pathology | Protein Footprinting | Cell Line, Tumor | Pyruvic Acid - metabolism | Female | Indicators and Reagents | Anoikis - physiology | Citric Acid Cycle - physiology | Neovascularization, Pathologic - metabolism | Ovarian Neoplasms - metabolism | Wound Healing - physiology | Kinetics | Energy Metabolism - physiology | Mitochondria - physiology | Cell Survival - physiology | Pyruvates | Cancer cells | Physiological aspects | Development and progression | Research | Health aspects | Anaplerosis | Cell migration | Ovarian cancer
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
5. Full Text c-Met Overexpression Contributes to the Acquired Apoptotic Resistance of Nonadherent Ovarian Cancer Cells through a Cross Talk Mediated by Phosphatidylinositol 3-Kinase and Extracellular Signal-Regulated Kinase 1/2
by Tang, Maggie K.S and Zhou, Hong Y and Yam, Judy W.P and Wong, Alice S.T
Neoplasia, ISSN 1476-5586, 02/2010, Volume 12, Issue 2, pp. 128,IN5 - 138,IN5
Ovarian cancer is the most lethal gynecologic cancer mainly because of widespread peritoneal dissemination and malignant ascites. Key to this is the capacity... 
CARCINOMA-CELLS | FACTOR-RECEPTOR | ACTIVATED PROTEIN-KINASE | ONCOLOGY | RAS | HEPATOCYTE GROWTH-FACTOR | ENDOTHELIAL-CELLS | GENE-EXPRESSION | MELANOMA-CELLS | PHOSPHOINOSITIDE 3-KINASE | HGF RECEPTOR | Proto-Oncogene Proteins c-met - metabolism | Paclitaxel - pharmacology | Humans | ras Proteins - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Receptor Cross-Talk - physiology | Female | Anoikis - physiology | Antineoplastic Agents - pharmacology | Ovarian Neoplasms - metabolism | In Situ Nick-End Labeling | Cisplatin - pharmacology | Enzyme Activation - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Hepatocyte Growth Factor - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Cell Line, Tumor | Anoikis - drug effects | Signal Transduction - physiology | Drug Resistance, Neoplasm - physiology | Enzyme Activation - physiology | Mitogen-Activated Protein Kinase 1 - metabolism | Receptor Cross-Talk - drug effects
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
6. Full Text Mutant B-RAF mediates resistance to anoikis via Bad and Bim
by Boisvert-Adamo, K and Aplin, A.E
Oncogene, ISSN 0950-9232, 05/2008, Volume 27, Issue 23, pp. 3301 - 3312
Normal cells undergo anoikis when they lose adhesion to or encounter an inappropriate extracellular matrix. By contrast, oncogenic signaling in tumor cells... 
B-RAF | Bad | Bim | Melanoma | Apoptosis | MULTIPLE-MYELOMA | CYCLIN D1 | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | apoptosis | melanoma | DEATH | CELL BIOLOGY | EPITHELIAL-CELLS | ONCOLOGY | PATHWAY | GENETICS & HEREDITY | SURVIVAL SIGNALS | MELANOMA-CELLS | BCL-2 FAMILY-MEMBERS | Phosphorylation | Proto-Oncogene Proteins B-raf - physiology | Humans | Gene Expression Regulation, Neoplastic | Bcl-2-Like Protein 11 | Melanoma - genetics | Membrane Proteins - physiology | bcl-Associated Death Protein - physiology | bcl-Associated Death Protein - metabolism | Membrane Proteins - metabolism | Tumor Cells, Cultured | Proto-Oncogene Proteins - metabolism | bcl-Associated Death Protein - genetics | Cell Adhesion - genetics | Anoikis - genetics | RNA, Small Interfering - pharmacology | Mutant Proteins - physiology | Apoptosis Regulatory Proteins - metabolism | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Cell Adhesion - physiology | Proto-Oncogene Proteins B-raf - genetics | Proto-Oncogene Proteins - physiology | MAP Kinase Kinase Kinases - physiology | Apoptosis Regulatory Proteins - physiology | Signal Transduction - physiology | Proteasome Endopeptidase Complex - metabolism | Signal transduction | Genetics | Mutation | Cellular biology | Skin cancer | Cell adhesion & migration
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
7. Full Text Calcium and cell death mechanisms: A perspective from the cell death community
by Zhivotovsky, Boris and Orrenius, Sten
Cell Calcium, ISSN 0143-4160, 2011, Volume 50, Issue 3, pp. 211 - 221
Abstract Research during the past several decades has provided convincing evidence for a crucial role of the Ca2+ ion in cell signaling. Hence, intracellular... 
Advanced Basic Science | Cell death mechanisms | Calcium | Toxicity | Anoikis | Autophagy | AIF | Phagocytosis | Apoptosis | Necrosis | APOPTOSIS-INDUCING FACTOR | BECLIN 1 | CALPAIN | RECEPTOR | MEDIATED CLEAVAGE | CELL BIOLOGY | MITOCHONDRIAL PERMEABILITY TRANSITION | REGULATES APOPTOSIS | ENDOPLASMIC-RETICULUM | THYMOCYTE APOPTOSIS | Humans | Phagocytosis - physiology | Endoplasmic Reticulum - metabolism | Mitochondria - metabolism | Mammals | Animals | Calcium - physiology | Cell Death - physiology | Anoikis - physiology | Apoptosis - physiology | Calcium Signaling | Apoptosis Inducing Factor - metabolism | Physiological aspects | Cell death | Biochemistry
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
8. Full Text Loss of Sertoli-Germ Cell Adhesion Determines the Rapid Germ Cell Elimination During the Seasonal Regression of the Seminiferous Epithelium of the Large Hairy Armadillo Chaetophractus villosus
by Luaces, JP and Rossi, LF and Sciurano, RB and Rebuzzini, P and Merico, V and Zuccotti, M and Merani, MS and Garagna, S
BIOLOGY OF REPRODUCTION, ISSN 0006-3363, 03/2014, Volume 90, Issue 3, p. 48
The armadillo Chaetophractus villosus is a seasonal breeder whose seminiferous epithelium undergoes rapid regression with massive germ cell loss, leaving the... 
SPERMATIDS | phagocytosis | TESTIS REGRESSION | LOCALIZATION | testosterone | apoptosis | nectin-3 | IBERIAN MOLE | PROLIFERATION | anoikis | APOPTOTIC SPERMATOGENIC CELLS | REPRODUCTIVE BIOLOGY | N-cadherin and beta-catenin | DISRUPTION | adhesion molecules | Cadm1 | IN-VIVO | JUNCTIONS | spermatogenesis | seasonal reproduction | intermediate filaments | Immunohistochemistry | In Situ Nick-End Labeling | Meiosis - physiology | Microscopy, Electron, Transmission | Cadherins - metabolism | Cytochromes c - metabolism | Caspase 3 - metabolism | Phagocytosis - physiology | Male | Cell Adhesion Molecules - metabolism | Germ Cells - physiology | beta Catenin - metabolism | Nectins | Sertoli Cells - physiology | Animals | Cell Adhesion - physiology | Armadillos - physiology | Intercellular Junctions - physiology | Seminiferous Epithelium - physiology | Apoptosis - physiology | Seasons
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
9. Full Text EMT as the ultimate survival mechanism of cancer cells
by Tiwari, Neha and Gheldof, Alexander and Tatari, Marianthi and Christofori, Gerhard
Seminars in Cancer Biology, ISSN 1044-579X, 2012, Volume 22, Issue 3, pp. 194 - 207
Abstract Epithelial cancers make up the vast majority of cancer types and, during the transition from benign adenoma to malignant carcinoma and metastasis,... 
Hematology, Oncology and Palliative Medicine | Signaling | Tumorigenesis | Metastasis | EMT | Cancer | E-CADHERIN EXPRESSION | REPRESSES E-CADHERIN | TRANSCRIPTION FACTOR SNAIL | METASTATIC PROSTATE-CANCER | RADIATION-INDUCED APOPTOSIS | EPITHELIAL-MESENCHYMAL TRANSITION | REGULATES E-CADHERIN | GROWTH-FACTOR RECEPTOR | ONCOLOGY | EMBRYONIC STEM-CELLS | NF-KAPPA-B | Transcription Factors - physiology | Epithelial-Mesenchymal Transition - physiology | Humans | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | Cadherins - physiology | Cell-Matrix Junctions - pathology | Epithelial-Mesenchymal Transition - genetics | Cell Transformation, Neoplastic - metabolism | Disease Progression | Neoplasm Metastasis - genetics | Cell-Matrix Junctions - genetics | Neoplastic Stem Cells - metabolism | Cell Transformation, Neoplastic - genetics | Neoplasm Metastasis - pathology | Neoplasm Metastasis - physiopathology | Neoplastic Stem Cells - pathology | Signal Transduction - physiology | Cadherins - genetics | Cell Differentiation - physiology | Genes | Cancer cells | Stem cells
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
10. Full Text Integrins and cell-fate determination
by Streuli, Charles H
Journal of Cell Science, ISSN 0021-9533, 01/2009, Volume 122, Issue 2, pp. 171 - 177
All cellular processes are determined by adhesive interactions between cells and their local microenvironment. Integrins, which constitute one class of... 
Mitosis | Integrin signalling | Growth factor receptors | Proliferation | Adhesome | Adhesion | Mitotic spindle | Adhesion complex | Cell fate | Metaphase | Cell cycle | Stem cells | Anoikis | Differentiation | Apoptosis | STEM-CELLS | REGULATES SPINDLE ORIENTATION | MAMMARY EPITHELIUM | CELL BIOLOGY | MEDIATED ADHESION | BETA-1-INTEGRIN DELETION | GROWTH | EXTRACELLULAR-MATRIX | FOCAL ADHESION KINASE | Cell Division - physiology | Animals | Cell Proliferation | Cell Adhesion - physiology | Humans | Cell Polarity - physiology | Cytoskeletal Proteins - metabolism | Apoptosis - physiology | Spindle Apparatus - physiology | Cell Differentiation - physiology | Integrins - metabolism | Receptors, Growth Factor - metabolism
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
11. Full Text Cell detachment activates the Hippo pathway via cytoskeleton reorganization to induce anoikis
by Zhao, Bin and Li, Li and Wang, Lloyd and Wang, Cun-Yu and Yu, Jindan and Guan, Kun-Liang
Genes and Development, ISSN 0890-9369, 01/2012, Volume 26, Issue 1, pp. 54 - 68
Cell attachment to the extracellular matrix (ECM) is crucial to cell physiology such as polarity, motility, and proliferation. In normal cells, loss of... 
Lats | Anoikis | YAP | Phosphorylation | Cancer metastasis | YES-ASSOCIATED PROTEIN | GROWTH-CONTROL | PROLIFERATION ARREST | DEVELOPMENTAL BIOLOGY | CELL BIOLOGY | anoikis | HEPATOCELLULAR-CARCINOMA | SIGNALING-PATHWAY | ALPHA-CATENIN | GENETICS & HEREDITY | YAP ONCOPROTEIN | TUMOR-SUPPRESSOR | CONTACT INHIBITION | phosphorylation | cancer metastasis | PROMOTES APOPTOSIS | Humans | Protein-Serine-Threonine Kinases - genetics | Nuclear Proteins - metabolism | Protein Transport | Cells - enzymology | Transcription Factors - metabolism | Animals | Cell Adhesion - physiology | Cell Line, Tumor | Cells - metabolism | Cytoskeleton - metabolism | Anoikis - physiology | Mice | HeLa Cells | Focal Adhesions - physiology | Enzyme Activation - physiology | Protein-Serine-Threonine Kinases - metabolism | Cells - cytology | Cancer cells | Cytoskeleton | Genetic aspects | Genetic transcription | Research | Metastasis | Apoptosis | Research Paper
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
12. Full Text Drosophila EGFR pathway coordinates stem cell proliferation and gut remodeling following infection
by Buchon, Nicolas and Broderick, Nichole A and Kuraishi, Takayuki and Lemaitre, Bruno
BMC Biology, ISSN 1741-7007, 12/2010, Volume 8, Issue 1, pp. 152 - 152
Background: Gut homeostasis is central to whole organism health, and its disruption is associated with a broad range of pathologies. Following damage, complex... 
HOMEOSTASIS | ACTIVATION | ANOIKIS | BIOLOGY | TISSUE-DAMAGE | SELF-RENEWAL | RECEPTOR | POSTERIOR MIDGUT | E-CADHERIN | BACTERIAL-INFECTION | DIFFERENTIATION | Intestinal Mucosa - metabolism | Intestines - metabolism | Stem Cells - metabolism | Drosophila - physiology | Intestinal Mucosa - growth & development | Receptor, Epidermal Growth Factor - metabolism | Muscle, Smooth - blood supply | Animals, Genetically Modified | Enterobacteriaceae Infections - metabolism | Intestines - growth & development | Enterobacteriaceae Infections - pathology | Intestinal Mucosa - microbiology | Muscle, Smooth - metabolism | Pectobacterium carotovorum - physiology | Enterobacteriaceae Infections - physiopathology | Drosophila - microbiology | Intestinal Mucosa - physiology | Animals | Intestines - microbiology | Signal Transduction - drug effects | Models, Biological | Morphogenesis - drug effects | Signal Transduction - physiology | Stem Cells - physiology | Cell Proliferation - drug effects | Drosophila - growth & development | Drosophila - metabolism | Epidermal Growth Factor - pharmacology | Intestines - physiopathology | Morphogenesis - physiology | Insects | Kinases | Bacterial infections
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
13. Full Text Cellular and metabolic functions for autophagy in cancer cells
by Kenific, Candia M and Debnath, Jayanta
Trends in Cell Biology, ISSN 0962-8924, 2014, Volume 25, Issue 1, pp. 37 - 45
Highlights • Autophagy sustains metabolic pathways required for aggressive tumor growth. • The unfolded protein response induces cytoprotective autophagy in... 
Pathology | unfolded protein response | anoikis | metabolism | cancer | autophagy | invasion | Anoikis | Metabolism | Autophagy | Invasion | Cancer | Unfolded protein response | ADVANCED SOLID TUMORS | EXTRACELLULAR-MATRIX DETACHMENT | PHOSPHATIDYLINOSITOL 3-KINASE | C-MYC | SELECTIVE AUTOPHAGY | P62 | CELL BIOLOGY | HYDROXYCHLOROQUINE | INHIBITION | PHASE-I TRIAL | LUNG-TUMORS | Neoplasms - metabolism | Autophagy - physiology | Metabolism - physiology | Carcinogenesis - metabolism | Disease Progression | Carcinogenesis - pathology | Animals | Signal Transduction - physiology | Mice | Neoplasms - physiopathology | Neoplasms - pathology | Unfolded Protein Response - physiology | Disease Models, Animal
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights