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81. KRAS and BRAF mutations induce anoikis resistance and characteristic 3D phenotypes in Caco-2 cells
by Patankar, Madhura and Eskelinen, Sinikka and Tuomisto, Anne and Makinen, Markus J and Karttunen, Tuomo J
Molecular medicine reports, ISSN 1791-2997, 11/2019, Volume 20, Issue 5, pp. 4634 - 4644
Oncology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Caco-2 Cells | Phenotype | Anoikis - genetics | Proto-Oncogene Proteins B-raf - genetics | Proto-Oncogene Proteins p21(ras) - genetics | Colorectal Neoplasms - genetics | Humans | Gene Expression Regulation, Neoplastic | Cell Line, Tumor | Mutation | Biomarkers, Tumor | Colorectal Neoplasms - metabolism
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82. Full Text The AMPK stress response pathway mediates anoikis resistance through inhibition of mTOR and suppression of protein synthesis
by Ng, T.L and Leprivier, G and Robertson, M.D and Chow, C and Martin, M.J and Laderoute, K.R and Davicioni, E and Triche, T.J and Sorensen, P.H.B
Cell death and differentiation, ISSN 1350-9047, 03/2012, Volume 19, Issue 3, pp. 501 - 510
anoikis | protein translation | AMPK | mTORC1 | cellular stress | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | NIH 3T3 Cells | AMP-Activated Protein Kinases - metabolism | Protein Biosynthesis | TOR Serine-Threonine Kinases - metabolism | Multiprotein Complexes | Embryo, Mammalian - metabolism | Mechanistic Target of Rapamycin Complex 1 | Mice, Knockout | Proteins - genetics | TOR Serine-Threonine Kinases - antagonists & inhibitors | Animals | Proteins - metabolism | TOR Serine-Threonine Kinases - genetics | Embryo, Mammalian - cytology | Cell Line, Tumor | Anoikis - physiology | Fibroblasts - cytology | Mice | AMP-Activated Protein Kinases - genetics | Cell Line, Transformed | Fibroblasts - metabolism | Index Medicus | Original Paper
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83. Full Text Cellular and metabolic functions for autophagy in cancer cells
by Kenific, Candia M and Debnath, Jayanta
Trends in cell biology, ISSN 0962-8924, 2014, Volume 25, Issue 1, pp. 37 - 45
Pathology | unfolded protein response | anoikis | metabolism | cancer | autophagy | invasion | Anoikis | Metabolism | Autophagy | Invasion | Cancer | Unfolded protein response | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Neoplasms - metabolism | Autophagy - physiology | Metabolism - physiology | Carcinogenesis - metabolism | Disease Progression | Carcinogenesis - pathology | Animals | Signal Transduction - physiology | Mice | Neoplasms - physiopathology | Neoplasms - pathology | Unfolded Protein Response - physiology | Disease Models, Animal | Index Medicus
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84. Full Text Diindolylmethane-mediated Gli1 protein suppression induces anoikis in ovarian cancer cells in vitro and blocks tumor formation ability in vivo
by Kandala, Prabodh K and Srivastava, Sanjay K
The Journal of biological chemistry, ISSN 0021-9258, 08/2012, Volume 287, Issue 34, pp. 28745 - 28754
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Ovarian Neoplasms - pathology | Hedgehog Proteins - metabolism | Embryo, Mammalian - metabolism | Ovarian Neoplasms - genetics | Hedgehog Proteins - genetics | Veratrum Alkaloids - pharmacology | Female | Indoles - pharmacology | Ovarian Neoplasms - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Ovarian Neoplasms - drug therapy | Gene Expression Regulation, Neoplastic - genetics | Fibroblasts - metabolism | Kruppel-Like Transcription Factors - biosynthesis | Embryo, Mammalian - pathology | Anoikis - genetics | Fibroblasts - pathology | Mice, Knockout | Drug Resistance, Neoplasm - genetics | Poly(ADP-ribose) Polymerases - metabolism | Animals | Poly(ADP-ribose) Polymerases - genetics | Cell Line, Tumor | Anoikis - drug effects | Mice | Zinc Finger Protein GLI1 | Kruppel-Like Transcription Factors - genetics | Drug Resistance, Neoplasm - drug effects | Index Medicus | Gene Regulation | Ovarian Cancer | Hedgehog | Signal Transduction | Anoikis | Metastasis | Tumor Model | Gli1 | Adhesion | Apoptosis
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85. Full Text P14ARF interacts with the focal adhesion kinase and protects cells from anoikis
by Vivo, M and Fontana, R and Ranieri, M and Capasso, G and Angrisano, T and Pollice, A and Calabrò, V and La Mantia, G
Oncogene, ISSN 0950-9232, 08/2017, Volume 36, Issue 34, pp. 4913 - 4928
Biochemistry & Molecular Biology | Oncology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Phosphorylation - physiology | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Humans | Cell Proliferation - physiology | Death-Associated Protein Kinases - metabolism | MCF-7 Cells | Cell Adhesion - physiology | Focal Adhesions - metabolism | Cell Line, Tumor | Cytoskeleton - metabolism | Signal Transduction - physiology | Anoikis - physiology | Cytoskeleton - physiology | Apoptosis - physiology | HeLa Cells | Focal Adhesions - physiology | Tumor Suppressor Protein p14ARF - metabolism | Cell Survival - physiology | Research | Tumor proteins | Protein kinases | Apoptosis | Cell proliferation | Tumor cells | Death-associated protein kinase | p53 Protein | Polymerization | Kinases | Cell adhesion & migration | ARF protein | Proteins | Cell growth | Actin | Cytoskeleton | Anoikis | Focal adhesion kinase | Cancer | Index Medicus | Original
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86. Full Text Cell surface adhesion molecules and adhesion-initiated signaling: Understanding of anoikis resistance mechanisms and therapeutic opportunities
by Zhong, Xiaoling and Rescorla, Frederick J
Cellular signalling, ISSN 0898-6568, 2012, Volume 24, Issue 2, pp. 393 - 401
Multicellular aggregate | Cell-matrix adhesion | Anoikis resistance | Cancer metastasis | Intercellular adhesion | Survival signaling | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Neoplasms - metabolism | Cell Adhesion Molecules - genetics | Signal Transduction | Anoikis - genetics | Humans | Gene Expression Regulation, Neoplastic | Cell Communication | Cell Survival - genetics | Extracellular Matrix - metabolism | Receptors, Cell Surface - metabolism | Molecular Targeted Therapy | Cell Adhesion | Cell Adhesion Molecules - metabolism | Extracellular Matrix - genetics | Cell-Matrix Junctions - genetics | Neoplasm Metastasis | Animals | Neoplasms - genetics | Cell-Matrix Junctions - metabolism | Mice | Neoplasms - pathology | Receptors, Cell Surface - genetics | Health aspects | Metastasis | Index Medicus
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87. Full Text The chemopreventive isothiocyanate sulforaphane reduces anoikis resistance and anchorage-independent growth in non-small cell human lung cancer cells
by Tsai, Ju-Ying and Tsai, Shin-Han and Wu, Chin-Chung
Toxicology and applied pharmacology, ISSN 0041-008X, 01/2019, Volume 362, pp. 116 - 124
Anoikis | Anchorage-independent growth | Sulforaphane | p53 | Pharmacology & Pharmacy | Toxicology | Life Sciences & Biomedicine | Science & Technology | Lung Neoplasms - genetics | Lung Neoplasms - drug therapy | Carcinoma, Non-Small-Cell Lung - genetics | Humans | Cell Line, Tumor | Anoikis - drug effects | Carcinoma, Non-Small-Cell Lung - drug therapy | Anticarcinogenic Agents - pharmacology | Isothiocyanates - pharmacology | Tumor Suppressor Protein p53 - genetics | Prevention | Medical colleges | Growth | Cancer cells | Metastasis | Tumor proteins | Lung cancer, Non-small cell | Cancer | Index Medicus
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88. Full Text Inhibition of PI3K pathway using BKM120 intensified the chemo-sensitivity of breast cancer cells to arsenic trioxide (ATO)
by Alipour, Faranak and Riyahi, Niknam and Safaroghli-Azar, Ava and Sari, Soyar and Zandi, Zahra and Bashash, Davood
The international journal of biochemistry & cell biology, ISSN 1357-2725, 11/2019, Volume 116, pp. 105615 - 105615
Arsenic trioxide (ATO) | Breast cancer | PI3K signaling pathway | BKM120 (Buparlisib) | CAL-101 (Idelalisib) | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Epithelial Cells - metabolism | Epithelial Cells - drug effects | Humans | Gene Expression Regulation, Neoplastic | Phosphatidylinositol 3-Kinases - metabolism | DNA-Binding Proteins - metabolism | Telomerase - genetics | MCF-7 Cells | Forkhead Transcription Factors - metabolism | Survivin - genetics | Female | Telomerase - metabolism | Antineoplastic Agents - pharmacology | Arsenic Trioxide - pharmacology | Survivin - metabolism | Cell Line | Forkhead Transcription Factors - agonists | Signal Transduction | Anoikis - genetics | Phosphatidylserines - metabolism | Morpholines - pharmacology | Epithelial Cells - pathology | Survivin - antagonists & inhibitors | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Forkhead Transcription Factors - genetics | G1 Phase Cell Cycle Checkpoints - genetics | Drug Synergism | Phosphatidylinositol 3-Kinases - genetics | Transcription Factors - metabolism | Aminopyridines - pharmacology | Anoikis - drug effects | Cell Proliferation - drug effects | Telomerase - antagonists & inhibitors | G1 Phase Cell Cycle Checkpoints - drug effects | Drug Combinations | Index Medicus
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89. Full Text Visualization and targeting of LGR5 + human colon cancer stem cells
by Shimokawa, Mariko and Ohta, Yuki and Nishikori, Shingo and Matano, Mami and Takano, Ai and Fujii, Masayuki and Date, Shoichi and Sugimoto, Shinya and Kanai, Takanori and Sato, Toshiro
Nature (London), ISSN 0028-0836, 05/2017, Volume 545, Issue 7653, pp. 187 - 192
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Colonic Neoplasms - genetics | Cell Proliferation | Receptors, G-Protein-Coupled - metabolism | Neoplastic Stem Cells - drug effects | Colonic Neoplasms - drug therapy | Humans | Male | Molecular Targeted Therapy | Organoids - transplantation | Colonic Neoplasms - metabolism | Keratin-20 - genetics | Cell Tracking | Cell Self Renewal | Neoplastic Stem Cells - metabolism | Organoids - metabolism | Neoplastic Stem Cells - pathology | Cell Differentiation | Organoids - pathology | Neoplastic Stem Cells - transplantation | Gene Knock-In Techniques | Xenograft Model Antitumor Assays | Cell Lineage | Animals | Colonic Neoplasms - pathology | Mice | Receptors, G-Protein-Coupled - genetics | Keratin-20 - metabolism | Prevention | Colon cancer | Health aspects | Cancer cells | Cell culture | Surgical implants | Leukemia | Colorectal carcinoma | Colorectal cancer | Fluorescence | Smooth muscle | Transplantation | Genomes | Drug resistance | Assaying | Fragmentation | Metastases | Heterogeneity | Interleukin 4 | Allografts | Organoids | Intestine | Actin | Cell cycle | Plasticity | Libraries | Lesions | Medical research | Kidneys | Preservation | Probability theory | Color | Regrowth | Histology | Regression analysis | Gene expression | Ablation | Chemotherapy | Image analysis | Detachment | Nuclear fuels | Lymphocytes B | Microscopy | Stem cells | Alleles | Light microscopy | Anoikis | Genetic engineering | Gene mapping | Differentiation | Tumors | Index Medicus
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