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micrornas - genetics (543) 543
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Nature Communications, ISSN 2041-1723, 07/2017, Volume 8, Issue 1, p. 16003
Journal Article
Molecular Therapy, ISSN 1525-0016, 02/2018, Volume 26, Issue 2, pp. 593 - 605
Journal Article
STEM CELLS, ISSN 1066-5099, 11/2016, Volume 34, Issue 11, pp. 2721 - 2732
Prenatal folic acid (FA) supplementation prevents neural tube defects. Folate receptor alpha (FRα) is critical for embryonic development, including neural... 
Oct4 | Sox2 | Enhancer/promoters | Folate receptor alpha | miR‐138 | Multipotency | Klf4 | miR‐let‐7 | Cranial neural crest cells | miR-138 | miR-let-7 | SURVIVAL | TUBE | TRANSCRIPTIONAL NETWORK | MOUSE | PAX3 | PLURIPOTENCY | MICRORNAS | FOLIC-ACID | CELL & TISSUE ENGINEERING | CELL BIOLOGY | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | EMBRYONIC STEM-CELLS | TARGET GENE | HEMATOLOGY | RNA, Small Interfering - genetics | RNA-Binding Proteins - genetics | Folic Acid - pharmacology | MicroRNAs - antagonists & inhibitors | Transcriptional Activation | MicroRNAs - metabolism | Neural Stem Cells - cytology | SOXB1 Transcription Factors - agonists | Neural Crest - metabolism | SOXB1 Transcription Factors - metabolism | Octamer Transcription Factor-3 - genetics | SOXB1 Transcription Factors - genetics | p300-CBP Transcription Factors - genetics | Gene Expression Regulation, Developmental | Kruppel-Like Transcription Factors - agonists | Kruppel-Like Transcription Factors - metabolism | PAX3 Transcription Factor - genetics | Female | Promoter Regions, Genetic | Neural Crest - cytology | Ribonuclease III - genetics | Ribonuclease III - metabolism | Signal Transduction | Neural Stem Cells - drug effects | PAX3 Transcription Factor - deficiency | Folate Receptor 1 - antagonists & inhibitors | Transcription Factors - genetics | Mice, Knockout | Neural Crest - drug effects | Octamer Transcription Factor-3 - agonists | Transcription Factors - metabolism | p300-CBP Transcription Factors - metabolism | Animals | Histones - genetics | Folate Receptor 1 - genetics | Folate Receptor 1 - metabolism | Octamer Transcription Factor-3 - metabolism | Folic Acid - metabolism | Protein Binding | Mice | MicroRNAs - genetics | Histones - metabolism | Antagomirs - metabolism | Antagomirs - genetics | Kruppel-Like Transcription Factors - genetics | Neural Stem Cells - metabolism | RNA-Binding Proteins - metabolism | RNA, Small Interfering - metabolism | Genes | Cell proliferation | Chromatin | Immunoprecipitation | Oct-4 protein | Effector cells | Event-related potentials | Nuclei | Folic acid | Defects | Embryogenesis | KLF4 protein | Coding | Supplementation | Prenatal experience | Neural tube defects | MiRNA | Neural crest | Promoters | Embryonic growth stage | Polymerase chain reaction | Gene silencing | Stem cells | Neural stem cells | Skull
Journal Article
Cell Death and Disease, ISSN 2041-4889, 2017, Volume 8, Issue 6, pp. e2849 - e2849
Lin28A is a highly conserved RNA-binding protein that concurs to control the balance between stemness and differentiation in several tissue lineages. Here, we... 
RNA-BINDING PROTEINS | MESSENGER-RNAS | SIGNATURES | ACUTE LYMPHOBLASTIC-LEUKEMIA | HEMATOPOIETIC STEM-CELLS | GENE-EXPRESSION | CLASSIFICATION | REGULATORS | MICRORNA EXPRESSION | CANCER | CELL BIOLOGY | RNA-Binding Proteins - genetics | Annexin A1 - genetics | Antigens, CD34 - metabolism | MicroRNAs - antagonists & inhibitors | Early Growth Response Protein 2 - genetics | Humans | Leukemia, Myeloid, Acute - metabolism | MicroRNAs - metabolism | Lentivirus - metabolism | Tristetraprolin - metabolism | Cell Cycle Checkpoints - genetics | Lentivirus - genetics | Cell Differentiation | Myeloid Progenitor Cells - metabolism | Genetic Vectors - chemistry | Signal Transduction | Leukemia, Myeloid, Acute - pathology | Genetic Vectors - metabolism | Hematopoiesis - genetics | Myeloid Progenitor Cells - pathology | Gene Expression Regulation, Leukemic | Tristetraprolin - genetics | Cell Line, Tumor | Annexin A1 - metabolism | Early Growth Response Protein 2 - metabolism | MicroRNAs - genetics | Primary Cell Culture | Antagomirs - metabolism | Antagomirs - genetics | Antigens, CD34 - genetics | RNA-Binding Proteins - metabolism | Leukemia, Myeloid, Acute - genetics | CD34 antigen | Cell proliferation | Myeloid leukemia | Leukemia | Blast | Macrophages | Ribonucleic acid--RNA | Promyeloid leukemia | RNA-binding protein | Transfection | MicroRNAs | Classification | Cell lines | Stem cells | Cell cycle | Protein expression | Egr-2 protein | Acute myeloid leukemia | Blast cells | Acute promyeloid leukemia | Original
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2010, Volume 107, Issue 1, pp. 264 - 269
MicroRNA (miRNAs) are negative regulators of gene expression and can function as tumor suppressors or oncogenes. Expression patterns of miRNAs and their role... 
Tumor cell line | Liver cancer | Cell growth | MicroRNA | Liver | Cell lines | Stem cells | Hepatocellular carcinoma | Signatures | Tumors | Mouse model | AntagomiRs | Hepatocarcinogenesis | DDIT4 | MIR-34A | mouse model | MULTIDISCIPLINARY SCIENCES | CELL LINES | PROLIFERATION | MECHANISMS | hepatocarcinogenesis | IDENTIFICATION | antagomiRs | CANCER | GENE | microRNA | TUMOR-SUPPRESSOR | HUMAN HEPATOCELLULAR-CARCINOMA | MICRORNA EXPRESSION | Liver - pathology | Humans | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | Gene Expression Profiling | Fibrosis - metabolism | Tumor Suppressor Protein p53 - genetics | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Microarray Analysis | Carcinoma, Hepatocellular - genetics | Liver Neoplasms - pathology | Diagnosis, Differential | Fibrosis - genetics | Reproducibility of Results | Liver Neoplasms - genetics | Tumor Suppressor Protein p53 - metabolism | Transcription Factors - genetics | Mice, Knockout | Transcription Factors - metabolism | Animals | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | Liver - physiology | Mice | MicroRNAs - genetics | Fibrosis - pathology | Carcinoma, Hepatocellular - metabolism | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Development and progression | Genetic aspects | Research | Gene expression | Carcinogenesis | Risk factors | Biological Sciences
Journal Article
Journal Article
Journal of Biosciences, ISSN 0250-5991, 12/2018, Volume 43, Issue 5, pp. 985 - 1000
Retinal injury plays a leading role in the onset of visual impairment. Current forms of treatment are not able to ameliorate scarring, cell death and tissue... 
glial cell line-derived neurotrophic factor | retinal neurons | yttrium aluminium garnet | microRNA-216a | GDNF/GFRα1/RET signalling pathway | retinal injury | Brain-Derived Neurotrophic Factor - genetics | Cell Cycle - genetics | RNA, Small Interfering - genetics | Proto-Oncogene Proteins c-ret - metabolism | Retina - metabolism | Cell Proliferation | MicroRNAs - antagonists & inhibitors | Rats, Wistar | Ependymoglial Cells - metabolism | Glial Cell Line-Derived Neurotrophic Factor Receptors - metabolism | Male | MicroRNAs - metabolism | Retinal Degeneration - etiology | Vascular Endothelial Growth Factor A - metabolism | Glial Cell Line-Derived Neurotrophic Factor - antagonists & inhibitors | Vascular Endothelial Growth Factor A - genetics | Retinal Degeneration - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Glial Cell Line-Derived Neurotrophic Factor - metabolism | Oligoribonucleotides - metabolism | Brain-Derived Neurotrophic Factor - metabolism | MicroRNAs - agonists | Ependymoglial Cells - pathology | Signal Transduction | Retinal Degeneration - genetics | Gene Expression Regulation | Glial Cell Line-Derived Neurotrophic Factor - genetics | Proto-Oncogene Proteins c-fos - metabolism | Rats | Lasers, Solid-State - adverse effects | Glial Cell Line-Derived Neurotrophic Factor Receptors - genetics | Animals | Proto-Oncogene Proteins c-fos - genetics | MicroRNAs - genetics | Primary Cell Culture | Antagomirs - metabolism | Retinal Degeneration - pathology | Antagomirs - genetics | Proto-Oncogene Proteins c-bcl-2 - genetics | Retina - injuries | Apoptosis | Oligoribonucleotides - genetics | Proto-Oncogene Proteins c-ret - genetics | RNA, Small Interfering - metabolism | Immunohistochemistry | Brain | Bcl-2 protein | Aluminum | Labelling | Retina | Neuronal-glial interactions | Proteins | Signal transduction | Receptors | Cell growth | Ret protein | Lasers | Vascular endothelial growth factor | Growth factors | c-Fos protein | Visually handicapped people | Glial cell line-derived neurotrophic factor | MiRNA | Aluminium | Polymerase chain reaction | Scars | Injury prevention | Inhibitors | Yttrium | Cell proliferation | Flow cytometry | Bax protein | Transcription | Genes | Tissues | Garnet | Control | Semiconductor lasers | Cell cycle | Down-regulation | Injuries | G1 phase | Nucleotide sequence | Proliferation | Ribonucleic acid--RNA | DNA nucleotidylexotransferase | Regeneration | Signaling | YAG lasers | Brain-derived neurotrophic factor | Cell death | Cell number | MicroRNAs | Levels
Journal Article
Nature communications, ISSN 2041-1723, 11/2018, Volume 9, Issue 1, pp. 4847 - 8
Journal Article
Molecular Therapy, ISSN 1525-0016, 03/2018, Volume 26, Issue 3, pp. 718 - 729
Journal Article
Circulation, ISSN 0009-7322, 2011, Volume 124, Issue 6, pp. 720 - 730
BACKGROUND: Myocardial infarction leads to cardiac remodeling and development of heart failure. Insufficient myocardial capillary density after myocardial... 
microRNAs | heart failure | antagomir | angiogenesis | gene expression | myocardial infarction | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | ZEBRAFISH | HEART-FAILURE | DOWN-REGULATION | KAPPA-B | HUMAN ENDOTHELIAL-CELLS | CONTROLS ANGIOGENESIS | CARDIAC-HYPERTROPHY | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | NEOVASCULARIZATION | HEMATOLOGY | Myocardial Infarction - genetics | Neovascularization, Physiologic - drug effects | MicroRNAs - antagonists & inhibitors | Capillaries - pathology | Apoptosis - drug effects | Male | Gene Expression Profiling | Zebrafish - embryology | Cell Hypoxia | Oligoribonucleotides - pharmacology | Heme Oxygenase-1 - genetics | GATA2 Transcription Factor - biosynthesis | Ventricular Remodeling | Laminin | RNA Interference | Myocardial Infarction - physiopathology | Drug Evaluation, Preclinical | Spheroids, Cellular | Heart Failure - etiology | GATA2 Transcription Factor - genetics | Cells, Cultured - drug effects | Zebrafish Proteins - biosynthesis | Heme Oxygenase-1 - biosynthesis | Proteoglycans | p21-Activated Kinases - genetics | Endothelial Cells - metabolism | Neovascularization, Physiologic - genetics | Mice, Inbred C57BL | RNA, Small Interfering - pharmacology | p21-Activated Kinases - biosynthesis | Arterioles - pathology | Myocardial Infarction - complications | Animals | RNA, Small Interfering - therapeutic use | Collagen | Cells, Cultured - metabolism | Mice | MicroRNAs - genetics | MicroRNAs - physiology | Zebrafish Proteins - genetics | Endothelial Cells - pathology | Drug Combinations | Heart failure | MicroRNA | Patient outcomes | Physiological aspects | Development and progression | Genetic aspects | Neovascularization | Research | Heart attack
Journal Article
Journal Article
Nucleic Acids Research, ISSN 0305-1048, 02/2018, Volume 46, Issue 3, pp. 1124 - 1138
Abstract Phospholipids, such as 1-palmitoyl-2-arachidonoyl-sn-glycero-3-phosphocholine (PAPC), are the major components of cell membranes. Their exposure to... 
CELLS | ANGIOGENESIS | VEGF | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | TRANSCRIPTION | GENE-EXPRESSION | MECHANISMS | STRESS | MICROPARTICLES CORRELATE | DIFFERENTIAL EXPRESSION ANALYSIS | MicroRNAs - antagonists & inhibitors | Reactive Oxygen Species - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Humans | MicroRNAs - metabolism | Gene Expression Profiling | Vascular Endothelial Growth Factor A - metabolism | Phosphatidylcholines - metabolism | Vascular Endothelial Growth Factor A - genetics | Glycolysis - drug effects | Glycolysis - genetics | Kruppel-Like Transcription Factors - metabolism | Human Umbilical Vein Endothelial Cells - cytology | NF-E2-Related Factor 2 - genetics | Forkhead Box Protein O1 - metabolism | Human Umbilical Vein Endothelial Cells - drug effects | Oxidation-Reduction | Signal Transduction | Phosphatidylcholines - pharmacology | Gene Expression Regulation | Phosphatidylcholines - chemistry | Proto-Oncogene Proteins c-myc - metabolism | Phosphofructokinase-2 - metabolism | Sequence Analysis, RNA | NF-E2-Related Factor 2 - metabolism | Phosphofructokinase-2 - genetics | Cell Proliferation - drug effects | MicroRNAs - genetics | Proto-Oncogene Proteins c-myc - genetics | Primary Cell Culture | Antagomirs - metabolism | Antagomirs - genetics | Forkhead Box Protein O1 - genetics | Kruppel-Like Transcription Factors - genetics | Gene regulation, Chromatin and Epigenetics
Journal Article