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Kidney International, ISSN 0085-2538, 12/2013, Volume 84, Issue 6, pp. 1129 - 1144
The TGF-β/Smad3 pathway plays a major role in tissue fibrosis, but the precise mechanisms are not fully understood. Here we identified microRNA miR-433 as an... 
miR-433 | renal fibrosis | TGF-β signaling | microRNA therapy | KIDNEY-DISEASE | TGF-BETA | DIABETIC-NEPHROPATHY | MESENCHYMAL TRANSITION | TGF-beta signaling | GROWTH-FACTOR-BETA | TARGETED DISRUPTION | POLYAMINE DEPLETION | UNILATERAL URETERAL OBSTRUCTION | EPITHELIAL-CELLS | TUBULOINTERSTITIAL FIBROSIS | UROLOGY & NEPHROLOGY | Up-Regulation | Ureteral Obstruction - complications | Kidney - pathology | Transforming Growth Factor beta1 - metabolism | Male | MicroRNAs - metabolism | Smad3 Protein - metabolism | Mice, 129 Strain | Anti-Glomerular Basement Membrane Disease - metabolism | Doxorubicin | Kidney Diseases - genetics | Smad3 Protein - deficiency | Smad3 Protein - genetics | Kidney - metabolism | Transfection | RNA Interference | Time Factors | Smad2 Protein - genetics | Anti-Glomerular Basement Membrane Disease - genetics | Smad7 Protein - genetics | Binding Sites | Disease Models, Animal | Kidney Diseases - metabolism | Smad7 Protein - metabolism | Cell Line | Promoter Regions, Genetic | Signal Transduction | Kidney Diseases - pathology | Kidney Diseases - prevention & control | Anti-Glomerular Basement Membrane Disease - pathology | Mice, Inbred C57BL | Smad2 Protein - metabolism | Rats | Smad2 Protein - deficiency | Transforming Growth Factor beta1 - genetics | Mice, Knockout | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Fibrosis | Mice | MicroRNAs - genetics | Kidney Diseases - etiology
Journal Article
Kidney International, ISSN 0085-2538, 11/2014, Volume 86, Issue 5, pp. 932 - 942
Journal Article
Kidney International, ISSN 0085-2538, 03/2018, Volume 93, Issue 3, pp. 626 - 642
A key feature of glomerular diseases such as crescentic glomerulonephritis and focal segmental glomerulosclerosis is the activation, migration and... 
cell migration | parietal epithelial cells | collapsing FSGS | crescentic glomerulonephritis | CD44 | I COLLAGEN | BOWMANS CAPSULE | PARIETAL EPITHELIAL-CELLS | NEPHROPATHY | MURINE MODEL | NEPHRITIS | UROLOGY & NEPHROLOGY | COLLAGEN MESSENGER-RNA | MICE | EXPRESSION | PODOCYTE REGENERATION | Cell Proliferation | Epithelial Cells - metabolism | Glomerulosclerosis, Focal Segmental - genetics | Anti-Glomerular Basement Membrane Disease - metabolism | Anti-Glomerular Basement Membrane Disease - immunology | Thy-1 Antigens - genetics | Hyaluronan Receptors - metabolism | Anti-Glomerular Basement Membrane Disease - genetics | Kidney Glomerulus - metabolism | Albuminuria - metabolism | Granulocytes - metabolism | Extracellular Matrix Proteins - metabolism | Macrophages - immunology | Disease Models, Animal | Genetic Predisposition to Disease | Signal Transduction | Thy-1 Antigens - immunology | Anti-Glomerular Basement Membrane Disease - pathology | Mice, Inbred C57BL | Cells, Cultured | Epithelial Cells - pathology | Kidney Glomerulus - pathology | Hyaluronan Receptors - genetics | Glomerulosclerosis, Focal Segmental - metabolism | Mice, Knockout | Albuminuria - genetics | Thy-1 Antigens - metabolism | Autoantibodies - immunology | Macrophages - metabolism | Phenotype | Animals | Glomerulosclerosis, Focal Segmental - immunology | Epithelial Cells - immunology | Albuminuria - immunology | Hyaluronan Receptors - immunology | Kidney Glomerulus - immunology | Glomerulosclerosis, Focal Segmental - pathology | Granulocytes - immunology | Cell Movement | Cell proliferation | Glomerulonephritis | Animal models | Kidneys | Leukocyte migration | Epithelial cells | Pathogenesis | Histology | Macrophages | Cell growth | Granulocytes | CD44 antigen | Rodents | Glomerulus | Leukocytes (granulocytic) | Cell migration
Journal Article
Journal of Immunology, ISSN 0022-1767, 06/2015, Volume 194, Issue 12, pp. 5713 - 5724
Journal Article
Science China Life Sciences, ISSN 1674-7305, 12/2016, Volume 59, Issue 12, pp. 1282 - 1289
Cell-mediated autoimmunity, particularly that involving autoreactive T cells, participates in mediating anti-glomerular basement membrane (GBM) disease.... 
anti-GBM disease | kidney injury | CD8 | T cell | IL-17 | CRESCENTIC GLOMERULONEPHRITIS | DENDRITIC CELLS | PROGNOSIS | IDENTIFICATION | PREVENTS | PODOCYTES | ANTIBODY-INDUCED GLOMERULONEPHRITIS | BIOLOGY | EXPERIMENTAL AUTOIMMUNE GLOMERULONEPHRITIS | NEPHRITIS | ANTIGEN | Autoimmunity | Forkhead Transcription Factors - immunology | Immunohistochemistry | Antigens, CD - immunology | Autoantibodies - metabolism | Kidney - pathology | Humans | Interleukin-17 - immunology | Kidney - immunology | Male | Anti-Glomerular Basement Membrane Disease - metabolism | Antigens, CD - metabolism | Young Adult | Kidney - metabolism | Anti-Glomerular Basement Membrane Disease - immunology | T-Lymphocytes - metabolism | Forkhead Transcription Factors - metabolism | Immunoglobulin G - immunology | CD8-Positive T-Lymphocytes - metabolism | Kidney Glomerulus - metabolism | Adult | Female | Antigens, Differentiation, Myelomonocytic - immunology | Macrophages - immunology | Kidney Failure, Chronic - immunology | Enzyme-Linked Immunosorbent Assay | Kidney Glomerulus - pathology | CD3 Complex - metabolism | Kidney Failure, Chronic - metabolism | Interleukin-17 - metabolism | Autoantibodies - immunology | Macrophages - metabolism | Antigens, Differentiation, Myelomonocytic - metabolism | Adolescent | CD3 Complex - immunology | T-Lymphocytes - immunology | Kidney Glomerulus - immunology | CD8-Positive T-Lymphocytes - immunology | Immunoglobulin G - metabolism
Journal Article
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 1931-857X, 04/2010, Volume 298, Issue 4, pp. 870 - 882
Macrophages are heterogeneous immune cell populations that include classically activated and alternatively activated (M2) macrophages. We examined the... 
Angiotensin II type 1 receptor blocker | KIDNEY-DISEASE | PHYSIOLOGY | ANGIOTENSIN-ALDOSTERONE SYSTEM | WKY RATS | PPAR-GAMMA ACTIVATION | TYPE-1 RECEPTOR | angiotensin II type 1 receptor blocker | TELMISARTAN | INFLAMMATION | GLOMERULAR CAPILLARY REPAIR | UROLOGY & NEPHROLOGY | RENAL INJURY | MONONUCLEAR PHAGOCYTES | Inflammation - pathology | Tetrazoles - pharmacology | Angiotensin Receptor Antagonists | Rats, Inbred WKY | Male | Anti-Glomerular Basement Membrane Disease - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | CD4-Positive T-Lymphocytes - physiology | Dose-Response Relationship, Drug | Inflammation - metabolism | Anti-Glomerular Basement Membrane Disease - immunology | Kidney Glomerulus - metabolism | Blood Pressure - drug effects | Cytokines - genetics | Disease Models, Animal | Macrophages - physiology | Cytokines - metabolism | Anti-Glomerular Basement Membrane Disease - pathology | Kidney Glomerulus - drug effects | Gene Expression Regulation - physiology | Rats | Imidazoles - pharmacology | Kidney Glomerulus - pathology | CD8-Positive T-Lymphocytes - physiology | Gene Expression Regulation - drug effects | Animals | CD8-Positive T-Lymphocytes - drug effects | Macrophages - drug effects | CD4-Positive T-Lymphocytes - drug effects | Glomerulonephritis | Care and treatment | Research | Cytokines | Macrophages | Health aspects
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 1931-857X, 03/2010, Volume 298, Issue 3, pp. 702 - 711
Studies have shown that certain cells of the glomerular tuft begin to express proteins considered unique to other cell types upon injury. Little is known about... 
Glomerulonephritis | FSGS | Phenotype | Membranous nephropathy | Differentiation | phenotype | PROGENITOR CELLS | CRESCENTIC GLOMERULONEPHRITIS | PHYSIOLOGY | differentiation | DIFFERENTIAL EXPRESSION | ACTIN | NEPHRIN | MOUSE | PROLIFERATION | membranous nephropathy | TO-MESENCHYMAL TRANSITION | EXPERIMENTAL MEMBRANOUS NEPHROPATHY | UROLOGY & NEPHROLOGY | glomerulonephritis | FOCAL SEGMENTAL GLOMERULOSCLEROSIS | Immunohistochemistry | Cell Proliferation | Epithelial Cells - metabolism | Transforming Growth Factor beta1 - metabolism | Antibodies | Male | Anti-Glomerular Basement Membrane Disease - metabolism | Glomerulonephritis - metabolism | Doxorubicin | Glomerulonephritis, Membranous - metabolism | Heymann Nephritis Antigenic Complex - immunology | Anti-Glomerular Basement Membrane Disease - immunology | Time Factors | Glomerulonephritis, Membranous - immunology | Glomerulosclerosis, Focal Segmental - chemically induced | Biomarkers - metabolism | Autoantibodies | Podocytes - metabolism | Anti-Glomerular Basement Membrane Disease - pathology | Glomerulonephritis, Membranous - pathology | Glomerulonephritis - genetics | Rats | Epithelial Cells - pathology | Mice, Transgenic | Transforming Growth Factor beta1 - genetics | Podocytes - pathology | Rats, Sprague-Dawley | Glomerulosclerosis, Focal Segmental - metabolism | Animals | Proteins - metabolism | Glomerulonephritis - pathology | Mice | Mice, Inbred BALB C | Glomerulosclerosis, Focal Segmental - pathology | Cellular proteins | Epithelial cells | Physiological aspects | Parietal cells | Development and progression | Genetic aspects | Cell differentiation | Gene expression | Properties
Journal Article