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Journal Article
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Nature communications, ISSN 2041-1723, 04/2019, Volume 10, Issue 1, pp. 1916 - 1916
Potentiation of neutrophil extracellular trap (NET) release is one mechanism by which antiphospholipid antibodies (aPL Abs... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Antibodies, Antiphospholipid - blood | Immunoglobulin G - blood | Extracellular Traps - metabolism | Humans | Male | Extracellular Traps - immunology | Venous Thrombosis - immunology | Adenosine A2 Receptor Agonists - pharmacology | Dipyridamole - pharmacology | Extracellular Traps - drug effects | Receptor, Adenosine A2A - immunology | Antiphospholipid Syndrome - genetics | Receptor, Adenosine A2A - genetics | Cyclic AMP - metabolism | Disease Models, Animal | Phenethylamines - pharmacology | Signal Transduction | Venous Thrombosis - drug therapy | Adenosine - immunology | Antiphospholipid Syndrome - drug therapy | Mice, Inbred C57BL | Neutrophils - drug effects | Gene Expression Regulation | Neutrophils - immunology | Vena Cava, Inferior - metabolism | Adenosine - pharmacology | Vena Cava, Inferior - immunology | Antiphospholipid Syndrome - immunology | Fibrinolytic Agents - pharmacology | Vena Cava, Inferior - drug effects | Animals | Antiphospholipid Syndrome - pathology | Adenosine - analogs & derivatives | Cyclic AMP - immunology | Adenosine - metabolism | Mice | Venous Thrombosis - genetics | Venous Thrombosis - pathology | Protein kinase A | Potentiation | Adenosine | Animal models | Neutrophils | Dipyridamole | Antibodies | Cyclic AMP | Leukocytes (neutrophilic) | Inflammation | Kinases | Antiphospholipid antibodies | Thrombosis | Receptors | Thromboembolism | Antiphospholipid syndrome | Index Medicus
Journal Article