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1. Full Text Fibrinogen as a key regulator of inflammation in disease
by Davalos, Dimitrios and Davalos, Dimitrios and Akassoglou, Katerina and Akassoglou, Katerina
Seminars in immunopathology, ISSN 1863-2297, 1/2012, Volume 34, Issue 1, pp. 43 - 62
Autoimmunity | Complement receptor 3 | Multiple sclerosis | Stroke | Alzheimer’s disease | Internal Medicine | Macrophages | Blood brain barrier | Inflammatory disease | Microglia | Biomedicine | Immunology | Rheumatoid arthritis | CD11b/CD18 | Atherosclerosis | Plasminogen | Anticoagulant therapy | Alzheimer's disease | Pathology | Life Sciences & Biomedicine | Science & Technology | Colitis - genetics | Humans | Brain Injuries - metabolism | Arthritis, Rheumatoid - metabolism | Bacterial Infections - genetics | Inflammation - metabolism | Blood Coagulation - immunology | Muscular Dystrophy, Duchenne - immunology | Alzheimer Disease - immunology | Fibrinogen - immunology | Thromboplastin - immunology | Pulmonary Fibrosis - immunology | Vascular Diseases - genetics | Brain Injuries - genetics | Thrombin - genetics | Kidney Diseases - immunology | Arthritis, Rheumatoid - genetics | Neoplasms - immunology | Colitis - metabolism | Muscular Dystrophy, Duchenne - genetics | Bacterial Infections - metabolism | Neoplasms - metabolism | Pulmonary Fibrosis - genetics | Spinal Cord Injuries - genetics | Kidney Diseases - genetics | Stroke - genetics | Brain Injuries - immunology | Neoplasms - genetics | Bacterial Infections - immunology | Pulmonary Fibrosis - metabolism | Colitis - immunology | Thrombin - immunology | Stroke - immunology | Kidney Diseases - metabolism | Multiple Sclerosis - metabolism | Vascular Diseases - immunology | Spinal Cord Injuries - metabolism | Multiple Sclerosis - genetics | Inflammation - immunology | Fibrinogen - genetics | Stroke - metabolism | Animals | Thromboplastin - genetics | Alzheimer Disease - metabolism | Fibrinogen - metabolism | Inflammation - genetics | Multiple Sclerosis - immunology | Blood Coagulation - genetics | Spinal Cord Injuries - immunology | Muscular Dystrophy, Duchenne - metabolism | Thrombin - metabolism | Thromboplastin - metabolism | Alzheimer Disease - genetics | Arthritis, Rheumatoid - immunology | Vascular Diseases - metabolism | Nervous system diseases | Bacterial infections | Oncology, Experimental | Thrombin | Inflammation | Anticoagulants (Medicine) | Research | Rheumatoid factor | Fibrin | Fibrinogen | Genetic research | Colitis | Cancer | Index Medicus | Traumatic brain injury | Coagulation | Tissue factor | hemostasis | Spinal cord injury | Inflammatory diseases | Kidney | Signal transduction | Duchenne's muscular dystrophy | Neurodegenerative diseases | Therapeutic applications | Thrombosis | Coagulation factors | Infection | Molecular modelling | Fibrosis | Brain injury
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2. Full Text Hypothesis for potential pathogenesis of SARS-CoV-2 infection-a review of immune changes in patients with viral pneumonia
by Lin, Ling and Lu, Lianfeng and Cao, Wei and Li, Taisheng
Emerging microbes & infections, ISSN 2222-1751, 01/2020, Volume 9, Issue 1, pp. 727 - 732
COVID-19 | pathogenesis | IVIg | SARS-CoV-2 | anticoagulation | Immunology | Life Sciences & Biomedicine | Microbiology | Science & Technology | Betacoronavirus - pathogenicity | SARS Virus - immunology | Pandemics | Coronavirus Infections - drug therapy | Humans | Pneumonia, Viral - drug therapy | Severe Acute Respiratory Syndrome - immunology | Antibodies, Viral - blood | Heparin, Low-Molecular-Weight - therapeutic use | Middle East Respiratory Syndrome Coronavirus - immunology | Influenza A Virus, H1N1 Subtype | Cytokines - immunology | Cytokines - metabolism | Pneumonia, Viral - therapy | Anticoagulants - therapeutic use | Coronavirus Infections - immunology | Pneumonia, Viral - immunology | Animals | B-Lymphocytes - immunology | Coronavirus Infections - virology | Immunoglobulins, Intravenous - therapeutic use | Coronavirus Infections - therapy | Antibodies, Viral - immunology | T-Lymphocytes - immunology | Mice | Pneumonia, Viral - virology | Immunologic Factors - therapeutic use | Influenza, Human - immunology | Index Medicus
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3. Full Text Cationic antimicrobial peptides in psoriatic skin cooperate to break innate tolerance to self‐DNA
by Lande, Roberto and Chamilos, Georgios and Ganguly, Dipyaman and Demaria, Olivier and Frasca, Loredana and Durr, Sophie and Conrad, Curdin and Schröder, Jens and Gilliet, Michel
European journal of immunology, ISSN 0014-2980, 01/2015, Volume 45, Issue 1, pp. 203 - 213
Plasmacytoid DC | Toll‐like receptor | Skin | Psoriasis | Antimicrobial peptides | Toll-like receptor | Life Sciences & Biomedicine | Immunology | Science & Technology | Cathelicidins - immunology | Langerhans Cells - pathology | Dendritic Cells - immunology | Humans | Toll-Like Receptor 9 - genetics | Dendritic Cells - pathology | Cathelicidins - genetics | Muramidase - immunology | Toll-Like Receptor 9 - immunology | Psoriasis - genetics | Psoriasis - pathology | Langerhans Cells - immunology | T-Lymphocytes - pathology | Skin - pathology | Skin - immunology | Psoriasis - immunology | beta-Defensins - genetics | DNA - immunology | Signal Transduction | Antimicrobial Cationic Peptides | Muramidase - genetics | Gene Expression Regulation | Self Tolerance | DNA - genetics | T-Lymphocytes - immunology | beta-Defensins - immunology | Glycosaminoglycans | Peptides | Dendritic cells | Analysis | DNA | Anticoagulants (Medicine) | T cells | Lysozyme | Antimicrobial agents | Deoxyribonucleic acid--DNA | Immune system | Index Medicus
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4. Full Text A novel Kunitz protein with proposed dual function from Eudiplozoon nipponicum (Monogenea) impairs haemostasis and action of complement in vitro
by Jedličková, Lucie and Dvořák, Jan and Hrachovinová, Ingrid and Ulrychová, Lenka and Kašný, Martin and Mikeš, Libor
International journal for parasitology, ISSN 0020-7519, 04/2019, Volume 49, Issue 5, pp. 337 - 346
Helminth | Factor X | Kunitz | Haematophagy | Peptidase inhibitor | Anticoagulant | Diplozoidae | Plasmin | Life Sciences & Biomedicine | Parasitology | Science & Technology | Fibrinolysin - immunology | Factor Xa Inhibitors - chemistry | Fish Diseases - blood | Helminth Proteins - chemistry | Complement System Proteins - immunology | Trematode Infections - blood | Fish Diseases - parasitology | Trematoda - genetics | Trematode Infections - veterinary | Anticoagulants - chemistry | Hemostasis | Enzyme Inhibitors - chemistry | Fish Diseases - immunology | Antifibrinolytic Agents - chemistry | Host-Parasite Interactions | Carps - parasitology | Factor Xa Inhibitors - immunology | Amino Acid Sequence | Factor Xa - immunology | Antifibrinolytic Agents - immunology | Plasma Kallikrein - antagonists & inhibitors | Enzyme Inhibitors - immunology | Helminth Proteins - genetics | Carps - immunology | Trematoda - immunology | Sequence Alignment | Animals | Anticoagulants - immunology | Helminth Proteins - immunology | Trematoda - chemistry | Trematode Infections - immunology | Carps - blood | Plasma Kallikrein - immunology | Trematode Infections - parasitology | Kallikrein | Snakes | Index Medicus
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5. Full Text PF4/heparin-antibody complex induces monocyte tissue factor expression and release of tissue factor positive microparticles by activation of FcγRI
by Kasthuri, Raj S and Glover, Sam L and Jonas, William and McEachron, Troy and Pawlinski, Rafal and Arepally, Gowthami M and Key, Nigel S and Mackman, Nigel
Blood, ISSN 0006-4971, 06/2012, Volume 119, Issue 22, pp. 5285 - 5293
Life Sciences & Biomedicine | Hematology | Science & Technology | Hematologic and hematopoietic diseases | Biological and medical sciences | Medical sciences | Autoantibodies - blood | Humans | Platelet Factor 4 - immunology | Heparin - therapeutic use | Male | Monocytes - metabolism | Monocytes - immunology | Extracellular Signal-Regulated MAP Kinases - metabolism | Heparin - adverse effects | MAP Kinase Signaling System - immunology | Extracellular Signal-Regulated MAP Kinases - immunology | Heparin - pharmacokinetics | Receptors, IgG - metabolism | Female | Cell-Derived Microparticles - metabolism | Thromboplastin - biosynthesis | Thromboplastin - immunology | Gene Expression Regulation - immunology | Thrombocytopenia - immunology | Thrombosis - immunology | Anticoagulants - therapeutic use | Thrombocytopenia - chemically induced | Receptors, IgG - immunology | Anticoagulants - adverse effects | Thrombocytopenia - blood | Gene Expression Regulation - drug effects | Autoantibodies - immunology | Heparin - immunology | Anticoagulants - immunology | MAP Kinase Signaling System - drug effects | Cell-Derived Microparticles - immunology | Anticoagulants - pharmacokinetics | Platelet Factor 4 - blood | Thrombosis - drug therapy | Index Medicus | Abridged Index Medicus | Thrombosis and Hemostasis
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6. Full Text The procoagulant and proinflammatory plasma contact system
by Renné, Thomas and Renné, Thomas
Seminars in immunopathology, ISSN 1863-2297, 1/2012, Volume 34, Issue 1, pp. 31 - 41
Leakage | Plasma | Edema | Biomedicine | Immunology | Hereditary angioedema | Bradykinin | Internal Medicine | Factor XII | Thrombosis | Pathology | Life Sciences & Biomedicine | Science & Technology | Factor XII - immunology | Kallikreins - immunology | Blood Platelets - immunology | Humans | Platelet Activation - immunology | Hypersensitivity - immunology | Kinins - genetics | Bacterial Infections - genetics | Blood Coagulation - immunology | Bacterial Infections - immunology | Hypersensitivity - genetics | Factor XII - genetics | Kallikreins - genetics | Plasma - immunology | Thrombosis - immunology | Mice, Transgenic | Inflammation - immunology | Angioedemas, Hereditary - immunology | Animals | Inflammation - genetics | Blood Coagulation - genetics | Thrombosis - genetics | Mice | Kinins - immunology | Angioedemas, Hereditary - genetics | Anticoagulants (Medicine) | Glycosaminoglycans | Bacterial infections | Proteases | Health aspects | Genetically modified organisms | Index Medicus | Pathogens | Proteinase | Coagulation | polyphosphates | Data processing | Allergic diseases | Inflammation | Inflammatory diseases | Infection | angioedema | Reviews | Genetic engineering | Heparin | Platelets | Coagulation factor XII | Medicin och hälsovetenskap
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7. Full Text Toll-like receptor and antiphospholipid mediated thrombosis: in vivo studies
by Vega-Ostertag, Mariano E and Raschi, Elena and Liu, Xiaowei and Romay-Penabad, Zurina and De Micheli, Valeria and Galli, Monica and Moia, Marco and Tincani, Angela and Borghi, Maria Orietta and Nguyen-Oghalai, Tracy and Meroni, Pier Luigi
Annals of the Rheumatic Diseases, ISSN 0003-4967, 10/2007, Volume 66, Issue 10, pp. 1327 - 1333
Life Sciences & Biomedicine | Rheumatology | Science & Technology | Cardiology. Vascular system | Hematologic and hematopoietic diseases | Sarcoidosis. Granulomatous diseases of unproved etiology. Connective tissue diseases. Elastic tissue diseases. Vasculitis | Diseases of the peripheral vessels. Diseases of the vena cava. Miscellaneous | Biological and medical sciences | Medical sciences | Blood and lymphatic vessels | Platelet diseases and coagulopathies | Antibodies, Anticardiolipin - immunology | Thrombosis - complications | Humans | Antibodies, Antiphospholipid - immunology | Thrombosis - immunology | Toll-Like Receptor 4 - genetics | Male | Toll-Like Receptor 4 - immunology | Antiphospholipid Syndrome - complications | Mice, Inbred C3H | Antiphospholipid Syndrome - immunology | Cell Adhesion - immunology | Endothelial Cells - immunology | Leukocytes - immunology | Thrombophilia - immunology | Animals | Anticoagulants - immunology | Immunoglobulin G - immunology | Adult | Female | Polymorphism, Genetic - genetics | Mice | beta 2-Glycoprotein I - immunology | Thromboplastin - immunology | Physiological aspects | Research | Antiphospholipid antibodies | Health aspects | Thrombosis | Blood clot | Antiphospholipid syndrome | Proteins | Studies | Immunoglobulins | Cytokines | Genes | Laboratory animals | Index Medicus | Extended Report
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