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Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2010, Volume 5, Issue 11, p. e14062
Background: The cancer stem cell theory hypothesizes that cancers are perpetuated by cancer stem cells (CSC) or tumor initiating cells (TIC) possessing... 
COLON-CANCER | INITIATING CELLS | POPULATION | MARKER | MULTIDISCIPLINARY SCIENCES | TUMOR | HYALURONAN | CHEMORESISTANCE | FLOW-CYTOMETRY | IDENTIFICATION | LINES | Immunohistochemistry | Humans | Lung Neoplasms - metabolism | Middle Aged | Glycoproteins - metabolism | Peptides - genetics | Immunoblotting | Lung Neoplasms - pathology | Male | Transplantation, Heterologous | Gene Expression Profiling | Antigens, CD - genetics | Antigens, CD - metabolism | Neoplasms, Experimental - pathology | Octamer Transcription Factor-3 - genetics | Flow Cytometry | Peptides - metabolism | Neoplastic Stem Cells - metabolism | Hyaluronan Receptors - metabolism | Neoplasms, Experimental - genetics | Neoplastic Stem Cells - pathology | Female | Nuclear Proteins - genetics | Repressor Proteins - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Glycoproteins - genetics | Lung Neoplasms - genetics | Proto-Oncogene Proteins - metabolism | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | AC133 Antigen | Hyaluronan Receptors - genetics | Animals | Polycomb Repressive Complex 1 | Mice, Nude | Octamer Transcription Factor-3 - metabolism | Cell Line, Tumor | Aged | Mice | Neoplasms, Experimental - metabolism | Squamous cell carcinoma | Lung cancer, Non-small cell | Analysis | Stem cells | Flow cytometry | Biotechnology | Laboratories | Heart surgery | Oct-4 protein | Lung cancer | Colorectal cancer | Stem cell transplantation | Proteins | Allografts | Epidermal growth factor | CD44 antigen | Xenografts | Cell cycle | CD34 antigen | Subpopulations | Cell survival | Tumor cells | Non-small cell lung carcinoma | Tumor cell lines | Cisplatin | Studies | Polymerase chain reaction | Pathology | Cytometry | Properties (attributes) | Medical prognosis | Biomarkers | In vivo methods and tests | Pluripotency | Tumors | Apoptosis | Cancer
Journal Article
Journal of Immunology, ISSN 0022-1767, 01/2014, Volume 192, Issue 2, pp. 623 - 629
.... Although the precise origin of the unchecked inflammatory response in obesity is unclear, it is known that overproduction of proinflammatory cytokines by innate immune cells affects metabolism... 
IMMUNE-SYSTEM | INFLAMMATION | DISEASE | RESISTANCE | FAT | AKT | IMMUNOLOGY | DIET-INDUCED OBESITY | KEY PLAYERS | ADIPOSE-TISSUE | CUTTING EDGE | Tumor Necrosis Factor-alpha - metabolism | Antigens, CD - immunology | T-Lymphocytes, Regulatory - metabolism | Epithelial Cells - metabolism | TOR Serine-Threonine Kinases - metabolism | Obesity - immunology | Insulin - immunology | Interferon-gamma - metabolism | Antigens, CD - metabolism | T-Lymphocytes, Regulatory - immunology | Epithelium - immunology | Signal Transduction - immunology | Inflammation - metabolism | Tumor Necrosis Factor-alpha - immunology | Interleukin-10 - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Receptor, Insulin - immunology | Macrophages - immunology | Hyperinsulinism - metabolism | Intra-Abdominal Fat - immunology | Transforming Growth Factor beta - immunology | Epithelium - metabolism | Mice, Inbred C57BL | Apyrase - metabolism | Cells, Cultured | Apyrase - immunology | CTLA-4 Antigen - metabolism | Inflammation - immunology | CTLA-4 Antigen - immunology | Intra-Abdominal Fat - metabolism | Obesity - metabolism | TOR Serine-Threonine Kinases - immunology | Insulin - metabolism | Macrophages - metabolism | Animals | Hyperinsulinism - immunology | Proto-Oncogene Proteins c-akt - immunology | Epithelial Cells - immunology | Interferon-gamma - immunology | Interleukin-10 - antagonists & inhibitors | Receptor, Insulin - metabolism | Mice | Interleukin-10 - immunology | Transforming Growth Factor beta - metabolism
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 08/2008, Volume 118, Issue 8, pp. 2908 - 2916
Journal Article
Hepatology (Baltimore, Md.), ISSN 0270-9139, 2012, Volume 56, Issue 6, pp. 2255 - 2267
Liver cirrhosis is a predominant risk factor for hepatocellular carcinoma (HCC). However, the mechanism underlying the progression from cirrhosis to HCC... 
PROGENITOR CELLS | STEM-CELLS | HEPATOCELLULAR-CARCINOMA | HEPATOCYTES | TGF-BETA | EPIGENETIC REGULATION | PTEN | SELF-RENEWAL | TUMORIGENICITY | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Rats, Wistar | TOR Serine-Threonine Kinases - metabolism | Humans | Glycoproteins - metabolism | Liver Neoplasms, Experimental - chemically induced | Male | MicroRNAs - metabolism | Proto-Oncogene Proteins c-akt - genetics | Antigens, CD - metabolism | Epithelial Cell Adhesion Molecule | Pluripotent Stem Cells - pathology | Liver Neoplasms, Experimental - metabolism | Peptides - metabolism | Neoplastic Stem Cells - metabolism | Diethylnitrosamine | Liver Cirrhosis - metabolism | Antigens, Neoplasm - metabolism | Biomarkers, Tumor - metabolism | Antigens, Differentiation - metabolism | Liver Neoplasms, Experimental - pathology | Proto-Oncogene Proteins c-akt - metabolism | STAT3 Transcription Factor - metabolism | Liver - metabolism | Mice, Inbred C57BL | PTEN Phosphohydrolase - metabolism | Rats | Mice, SCID | AC133 Antigen | Cell Adhesion Molecules - metabolism | Cell Transformation, Neoplastic - metabolism | Pluripotent Stem Cells - metabolism | Thy-1 Antigens - metabolism | Transforming Growth Factor beta - pharmacology | Animals | Pluripotent Stem Cells - drug effects | Liver Cirrhosis - pathology | Mice, Inbred NOD | Mice | Cell Transformation, Neoplastic - pathology | Transforming Growth Factor beta - metabolism | Proteins | Liver cancer | Liver cirrhosis | Hepatology
Journal Article
The Journal of Physiology, ISSN 0022-3751, 2004, Volume 556, Issue 3, pp. 983 - 1000
Muscular adaptation to physical exercise has previously been described as a repair process following tissue damage. Recently, evidence has been published to... 
INDUCED INJURY | PHYSIOLOGY | IMMUNOREACTIVITY | ECCENTRIC EXERCISE | REGENERATION | LEUKEMIA INHIBITORY FACTOR | DENERVATED HUMAN MUSCLE | SATELLITE CELLS | STRENGTH | FACTOR-I | EXPRESSION | NEUROSCIENCES | Immunohistochemistry | Granulocytes - cytology | Cytokines - analysis | Humans | Middle Aged | DNA-Binding Proteins - analysis | Ki-67 Antigen - metabolism | Male | Muscle, Skeletal - metabolism | Proteins - analysis | Pain - metabolism | fas Receptor - metabolism | Fascia - chemistry | Oxygen Consumption - physiology | fas Receptor - analysis | Antigens, CD - metabolism | Antigens, CD - analysis | Running - physiology | C-Reactive Protein - metabolism | Aryl Hydrocarbon Receptor Nuclear Translocator | CD11b Antigen - analysis | Receptors, Cytokine - metabolism | Testosterone - blood | C-Reactive Protein - analysis | Hormones - blood | Leukocytes - chemistry | Interleukin-6 - metabolism | Lymphocytes - metabolism | CD56 Antigen - analysis | Lymphocytes - cytology | Insulin-Like Growth Factor I - analysis | Muscle, Skeletal - physiology | CD3 Complex - metabolism | Leukemia Inhibitory Factor Receptor alpha Subunit | Regression Analysis | Pain - physiopathology | Receptors, Cytokine - analysis | Adolescent | Antigens, Differentiation, Myelomonocytic - analysis | Creatine Kinase - metabolism | Transcription Factors - analysis | Leukocytes - metabolism | Insulin-Like Growth Factor I - metabolism | Receptors, OSM-LIF | Interleukin-6 - analysis | Monocytes - cytology | Receptors, Cell Surface - analysis | Monocytes - metabolism | Receptors, Aryl Hydrocarbon - analysis | DNA-Binding Proteins - metabolism | Testosterone - metabolism | Flow Cytometry | Interleukin-6 - blood | Exercise Test - methods | Muscle, Skeletal - chemistry | Fascia - metabolism | Heart Rate - physiology | Receptors, Aryl Hydrocarbon - metabolism | Adult | Female | Leukocyte Count | Leukemia Inhibitory Factor | Isometric Contraction - physiology | Cytokines - blood | Leukocytes - cytology | CD56 Antigen - metabolism | Creatine Kinase - blood | Cytokines - metabolism | Receptors, Cell Surface - metabolism | Transcription Factors - metabolism | CD3 Complex - analysis | Ki-67 Antigen - analysis | Proteins - metabolism | Hormones - metabolism | Antigens, Differentiation, Myelomonocytic - metabolism | CD11b Antigen - metabolism | Growth Substances - metabolism | Pain - diagnosis | Research Papers
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 2009, Volume 325, Issue 5936, pp. 83 - 87
Type IV pili mediate the initial interaction of many bacterial pathogens with their host cells. In Neisseria meningitidis, the causative agent of cerebrospinal... 
Intercellular junctions | Cell lines | Actins | Small interfering RNA | Bacteria | Cadherins | Reports | Bacterial adhesion | Infections | Blood brain barrier | Endothelial cells | CDC42 | ADHESION | HUMAN EPITHELIAL-CELLS | PATHOGENIC NEISSERIA | TWITCHING MOTILITY | MULTIDISCIPLINARY SCIENCES | NEISSERIA-MENINGITIDIS | BARRIER | PAR3 | RECEPTOR | ADHERENS | Cell Polarity | Brain - microbiology | Cadherins - metabolism | Humans | cdc42 GTP-Binding Protein - metabolism | Intercellular Junctions - microbiology | Phosphoproteins - metabolism | Blood-Brain Barrier - microbiology | Antigens, CD - metabolism | Catenins | Endothelial Cells - microbiology | Endothelium, Vascular - ultrastructure | Brain - blood supply | Protein Kinase C - metabolism | Membrane Proteins - metabolism | Neisseria meningitidis - physiology | Neisseria meningitidis - pathogenicity | Cell Line | Brain - cytology | Endothelial Cells - metabolism | Cell Cycle Proteins - metabolism | Intercellular Junctions - metabolism | Cell Adhesion Molecules - metabolism | Blood-Brain Barrier - metabolism | Endothelium, Vascular - microbiology | Bacterial Adhesion | Endothelium, Vascular - metabolism | Fimbriae, Bacterial - physiology | Adaptor Proteins, Signal Transducing - metabolism | Intercellular Junctions - ultrastructure | Measurement | Polarity (Biology) | Neisseria meningitidis | Properties | Health aspects | Endothelium | Brain | Meningitis | Eukaryotes | Cell adhesion & migration | Fimbriae, Bacterial | Endothelial Cells | Cell Adhesion Molecules | Intercellular Junctions | Membrane Proteins | Life Sciences | Microbiology and Parasitology | Adaptor Proteins, Signal Transducing | Phosphoproteins | cdc42 GTP-Binding Protein | Endothelium, Vascular | Cell Cycle Proteins | Antigens, CD | Protein Kinase C | Blood-Brain Barrier | cytology | meningitis | microbiology | physiology | ultrastructure | blood brain barrier | pathogenicity | blood supply | metabolism
Journal Article
Scientific Reports, ISSN 2045-2322, 11/2016, Volume 6, Issue 1, p. 37953
Interactions between endothelial selectins and the leukocyte counter-receptor PSGL1 mediates leukocyte recruitment to inflammation sites. PSGL1 is highly... 
CELLS | ADHESION | E-SELECTIN LIGAND-1 | PROTEIN | MULTIDISCIPLINARY SCIENCES | GLYCOPROTEIN LIGAND | P-SELECTIN | NEUTROPHILS | SIALIC-ACID | CD33-RELATED SIGLECS | EXPRESSION | Inflammation - chemically induced | Inflammation - pathology | Leukocytes, Mononuclear - metabolism | Membrane Glycoproteins - metabolism | P-Selectin - metabolism | Humans | Antigens, Differentiation, Myelomonocytic - pharmacology | Antigens, CD - genetics | Antigens, CD - metabolism | Lectins - metabolism | Inflammation - drug therapy | Female | Protein Interaction Domains and Motifs | Disease Models, Animal | Leukocytes, Mononuclear - drug effects | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Solubility | E-Selectin - metabolism | Lectins - pharmacology | Tumor Necrosis Factor-alpha - toxicity | Antigens, Differentiation, Myelomonocytic - genetics | Membrane Glycoproteins - genetics | Animals | Antigens, Differentiation, Myelomonocytic - metabolism | Leukocyte Rolling - physiology | Antigens, CD - pharmacology | Lectins - genetics | Antiinflammatory agents | Calcium | Perfusion | Rodents | Selectins | Peripheral blood mononuclear cells | Ligands | Leukocytes (mononuclear) | Inflammation | CD162 antigen | Leukocyte rolling | P-selectin | Membrane Glycoproteins/genetics | E-Selectin/metabolism | P-Selectin/metabolism | Antigens, CD/metabolism | Membrane Glycoproteins/metabolism | Life Sciences | Antigens, CD/pharmacology | Lectins/pharmacology | Leukocyte Rolling/physiology | Inflammation/pathology | Lectins/genetics | Leukocytes, Mononuclear/metabolism | Leukocytes, Mononuclear/drug effects | Inflammation/chemically induced | Antigens, CD/genetics | Anti-Inflammatory Agents/pharmacology | Antigens, Differentiation, Myelomonocytic/metabolism | Inflammation/drug therapy | Antigens, Differentiation, Myelomonocytic/pharmacology | Antigens, Differentiation, Myelomonocytic/genetics | Lectins/metabolism | Tumor Necrosis Factor-alpha/toxicity
Journal Article