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Blood, ISSN 0006-4971, 02/2015, Volume 125, Issue 9, pp. 1367 - 1376
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, pp. e0169648 - e0169648
The structural maintenance of chromosome 5/6 complex (Smc5/6) is a restriction factor that represses hepatitis B virus (HBV) transcription. HBV counters this... 
CELLS | NUCLEAR-BODIES | HEPATITIS-B-VIRUS | REPLICATION | DNA | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | EPIGENETIC REGULATION | COMPONENTS | BINDING | HUMANIZED MICE | Autoantigens - metabolism | Hepatitis B - metabolism | Antigens, Nuclear - metabolism | Humans | Hepatitis B - virology | Male | Hepatocytes - metabolism | Autoantigens - genetics | Hepatitis B - immunology | Hepatocytes - cytology | Trans-Activators - genetics | Cell Cycle Proteins - genetics | Promyelocytic Leukemia Protein - metabolism | Nuclear Proteins - genetics | Cytokines - genetics | Hepatitis B virus - immunology | Promyelocytic Leukemia Protein - genetics | Cytokines - metabolism | Cell Cycle Proteins - metabolism | Cells, Cultured | Chromosomal Proteins, Non-Histone | Nuclear Proteins - metabolism | Mice, SCID | Immunity, Innate - immunology | Animals | Antigens, Nuclear - genetics | Virus Replication | Trans-Activators - metabolism | Mice | Immune response | Analysis | Genetic aspects | Hepatitis B virus | Research | Genetic transcription | Hepatitis B | Cell culture | HBX protein | Pathogenesis | Viruses | Infections | Genomes | Degradation | Proteins | Hepatitis | Hepatology | Localization | Bioinformatics | Chromosomes | Deoxyribonucleic acid--DNA | Antigens | Cytokines | Chromosome 5 | Gene expression | Ribonucleic acid--RNA | Immune systems | Hepatocytes | Interferon | Kinetics | X protein | Index Medicus | RNA | Deoxyribonucleic acid | Ribonucleic acid
Journal Article
PLoS Genetics, ISSN 1553-7390, 06/2008, Volume 4, Issue 6, pp. e1000110 - e1000110
Journal Article
Journal Article
Nature Cell Biology, ISSN 1465-7392, 03/2012, Volume 14, Issue 3, pp. 311 - 317
Mitotic spindle positioning by cortical pulling forces(1) defines the cell division axis and location(2), which is critical for proper cell division and... 
ACTIVATION | PROTEIN | MECHANISM | KINETOCHORE | IMPORTIN-BETA | MITOTIC SPINDLE | SEGREGATION | ASYMMETRIC CELL-DIVISION | GRADIENT | RAN | CELL BIOLOGY | NIH 3T3 Cells | ran GTP-Binding Protein - genetics | Microtubule-Associated Proteins - genetics | Antigens, Nuclear - metabolism | Microtubule-Associated Proteins - metabolism | Humans | Molecular Sequence Data | Green Fluorescent Proteins - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Centrioles - physiology | Spindle Apparatus - metabolism | RNA Interference | Cell Cycle Proteins - genetics | Chromosome Segregation - physiology | Spindle Apparatus - physiology | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Cytoplasmic Dyneins - metabolism | Dyneins - metabolism | Proto-Oncogene Proteins - metabolism | Amino Acid Sequence | Green Fluorescent Proteins - metabolism | Cell Cycle Proteins - metabolism | Cytoplasmic Dyneins - genetics | Protein-Serine-Threonine Kinases - genetics | Dynactin Complex | Nuclear Matrix-Associated Proteins - metabolism | Proto-Oncogene Proteins - genetics | ran GTP-Binding Protein - metabolism | Blotting, Western | Animals | Antigens, Nuclear - genetics | Mitosis - physiology | Nuclear Matrix-Associated Proteins - genetics | Models, Biological | Protein Binding | Signal Transduction - physiology | Mice | Centrioles - metabolism | Dyneins - genetics | HeLa Cells | Microscopy, Fluorescence | Spindle (Cell division) | Physiological aspects | Cell division | Research | Chromosomes | Dynein | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 07/2012, Volume 14, Issue 7, pp. 753 - 763
It is becoming clear that interconnected functional gene networks, rather than individual genes, govern stem cell self-renewal and differentiation. To identify... 
STEM-CELLS | CHROMATIN | TISSUE | KERATINOCYTES | GENETIC INTERACTION | PHD FINGER | SKIN | P63 | EXPRESSION | MICE LACKING | CELL BIOLOGY | Antigens, Nuclear - metabolism | Epigenesis, Genetic | Humans | Multiprotein Complexes | Integrins - genetics | Gene Regulatory Networks | RNA, Messenger - metabolism | Stem Cells - metabolism | Integrins - metabolism | DNA-Binding Proteins - metabolism | Cell Differentiation - genetics | Transfection | RNA Interference | Tumor Suppressor Proteins - genetics | CCAAT-Enhancer-Binding Proteins - genetics | Stem Cell Niche - genetics | CCAAT-Enhancer-Binding Proteins - metabolism | Cell Adhesion - genetics | Tumor Suppressor Proteins - metabolism | Chromosomal Proteins, Non-Histone - metabolism | Cells, Cultured | Gene Expression Regulation | Adenosine Triphosphatases - metabolism | Chromatin Assembly and Disassembly | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Enhancer of Zeste Homolog 2 Protein | Chromosomal Proteins, Non-Histone - genetics | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Polycomb Repressive Complex 2 | Antigens, Nuclear - genetics | Keratinocytes - metabolism | Bayes Theorem | Adenosine Triphosphatases - genetics | Models, Genetic | Cluster Analysis | Epidermis - cytology | Physiological aspects | Epigenetic inheritance | Genetic aspects | Research | Cell differentiation | Stem cells | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2015, Volume 112, Issue 27, pp. E3495 - E3504
Damage repair mechanisms at transcriptionally active sites during the G0/G1 phase are largely unknown. To elucidate these mechanisms, we introduced genome... 
Dna damage | Recombination | CSB | Transcription | RNA polymerase II | COMPLEX | recombination | PROTEIN | transcription | DNA damage | HUMAN-CELLS | MULTIDISCIPLINARY SCIENCES | DOUBLE-STRAND BREAKS | PREFERENTIAL REPAIR | CHROMOSOMES | 8-OXOGUANINE | GENE | DEGRADATION | Cell Cycle - genetics | Antigens, Nuclear - metabolism | Humans | DNA Repair Enzymes - genetics | Homologous Recombination | RNA - genetics | Cockayne Syndrome - pathology | DNA-Binding Proteins - metabolism | Poly-ADP-Ribose Binding Proteins | RNA Interference | Rad52 DNA Repair and Recombination Protein - metabolism | Replication Protein A - genetics | DNA Repair Enzymes - metabolism | HEK293 Cells | Cell Cycle Proteins - genetics | Transcription, Genetic | G1 Phase - genetics | Nuclear Proteins - genetics | DNA Helicases - genetics | RNA - metabolism | Rad51 Recombinase - metabolism | Rad51 Recombinase - genetics | Cockayne Syndrome - metabolism | Cell Cycle Proteins - metabolism | Cells, Cultured | Nuclear Proteins - metabolism | Replication Protein A - metabolism | DNA-Binding Proteins - genetics | Blotting, Western | Rad52 DNA Repair and Recombination Protein - genetics | DNA Helicases - metabolism | Microscopy, Confocal | Antigens, Nuclear - genetics | Resting Phase, Cell Cycle - genetics | Ku Autoantigen | DNA Repair | Cockayne Syndrome - genetics | Cell Line, Tumor | Models, Genetic | DNA Damage | HeLa Cells | Physiological aspects | Observations | Genetic recombination | RNA polymerases | Proteins | Zebrafish | Ribonucleic acid--RNA | Neurodegeneration | DNA repair | Index Medicus | Biological Sciences | PNAS Plus
Journal Article
Blood, ISSN 0006-4971, 02/2013, Volume 121, Issue 6, pp. 1028 - 1038
Journal Article
European Urology, ISSN 0302-2838, 2017, Volume 72, Issue 6, pp. 952 - 959
Abstract Background Molecular characterization of nonmuscle invasive bladder cancer (NMIBC) may provide a biologic rationale for treatment response and novel... 
Urology | Bacillus Calmette-Guérin | DNA damage repair | Nonmuscle invasive bladder cancer | Targeted therapy | Genomics | Immunotherapy | AT-Rich interaction domain 1A | SURVIVAL | UROTHELIAL CARCINOMA | INACTIVATION | Bacillus Calmette-Guerin | ANEUPLOIDY | STAG2 | HIGH-RISK | URINE | UROLOGY & NEPHROLOGY | GRADE | MUTATIONS | PROMOTER | Class I Phosphatidylinositol 3-Kinases - genetics | Receptor, ErbB-2 - genetics | Exons | Humans | Middle Aged | Male | DNA Repair - genetics | Molecular Targeted Therapy | Histone Demethylases - genetics | Tumor Suppressor Protein p53 - genetics | DNA Copy Number Variations | Neoplasm Grading | Telomerase - genetics | Urinary Bladder Neoplasms - genetics | Urinary Bladder Neoplasms - pathology | Aged, 80 and over | DNA Damage - genetics | Adult | Female | Nuclear Proteins - genetics | Receptor, Fibroblast Growth Factor, Type 3 - genetics | Neoplasm Invasiveness | Kaplan-Meier Estimate | Proportional Hazards Models | Gene Fusion | Transcription Factors - genetics | Cystectomy | Antigens, Nuclear - genetics | Antineoplastic Agents, Immunological - therapeutic use | Urinary Bladder Neoplasms - therapy | Biomarkers | Neoplasm Recurrence, Local - genetics | Aged | High-Throughput Nucleotide Sequencing | BCG Vaccine - therapeutic use | Mutation | Neoplasm Staging | Bladder cancer | Health aspects | Analysis | Index Medicus
Journal Article