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Nature Medicine, ISSN 1078-8956, 09/2007, Volume 13, Issue 9, pp. 1102 - 1107
Journal Article
Neuropharmacology, ISSN 0028-3908, 07/2014, Volume 82, pp. 161 - 173
Previous work has suggested that activation of mGlu5 receptor augments NMDA receptor function and thereby may constitute a rational approach addressing... 
Schizophrenia | Positive allosteric modulator | Neurotoxicity | Metabotropic glutamate receptor | Antipsychotic activity | METABOTROPIC GLUTAMATE-RECEPTOR | PROTEIN-KINASE-C | NEGATIVE SYMPTOMS | FRAGILE-X | NEUROSCIENCES | PREFRONTAL CORTEX | D-SERINE | PHARMACOLOGY & PHARMACY | LONG-TERM POTENTIATION | BINDING-SITES | ACUTE STRESS | METHYL-D-ASPARTATE | Antipsychotic Agents - toxicity | Neurons - pathology | Cricetulus | Rats, Wistar | Benzamides - pharmacokinetics | Allosteric Regulation | Humans | Male | Excitatory Amino Acid Agents - pharmacokinetics | Mice, 129 Strain | Necrosis - pathology | Excitatory Amino Acid Agents - chemistry | Oxadiazoles - toxicity | Receptor, Metabotropic Glutamate 5 - genetics | Necrosis - physiopathology | Neurotoxicity Syndromes - pathology | Neurons - physiology | Female | Cell Death - drug effects | Neurons - drug effects | Oxadiazoles - chemistry | Oxadiazoles - pharmacokinetics | Benzamides - chemistry | CHO Cells | Benzamides - toxicity | Brain - physiopathology | Cells, Cultured | Antipsychotic Agents - pharmacokinetics | Receptor, Metabotropic Glutamate 5 - metabolism | Rats, Sprague-Dawley | Mice, Knockout | Brain - drug effects | Excitatory Amino Acid Agents - toxicity | Animals | Antipsychotic Agents - chemistry | Cell Death - physiology | Neurotoxicity Syndromes - physiopathology | Brain - pathology | Methyl aspartate | Antipsychotic drugs | Glutamate | Index Medicus
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2010, Volume 107, Issue 33, pp. 14845 - 14850
The direct and indirect pathways of the basal ganglia have been proposed to oppositely regulate locomotion and differentially contribute to pathological... 
Locomotion | Receptors | Basal ganglia | Neurons | Catalepsy | Antipsychotic agents | Cocaine | Behavioral neuroscience | Genotypes | Dyskinesia | DARPP-32 | Striatopallidal | Striatonigral | Locomotor behavior | basal ganglia | DOPA-INDUCED DYSKINESIA | NEUROTRANSMISSION | MULTIDISCIPLINARY SCIENCES | COCAINE | MODEL | striatopallidal | STRIATOPALLIDAL NEURONS | locomotor behavior | MICE | striatonigral | D1 RECEPTOR | PARKINSONS-DISEASE | Immunohistochemistry | Motor Activity - physiology | Dyskinesia, Drug-Induced - physiopathology | Dopamine and cAMP-Regulated Phosphoprotein 32 - genetics | Motor Activity - drug effects | Male | Neurons - cytology | Green Fluorescent Proteins - genetics | Corpus Striatum - cytology | Corpus Striatum - metabolism | Catalepsy - physiopathology | Neurons - classification | Neuronal Plasticity - physiology | Catalepsy - chemically induced | Long-Term Potentiation - physiology | Female | Neurons - metabolism | Dyskinesia, Drug-Induced - etiology | Green Fluorescent Proteins - metabolism | Synaptic Potentials - physiology | Dopamine Uptake Inhibitors - pharmacology | Mice, Inbred C57BL | Mice, Transgenic | Levodopa - toxicity | Dopamine and cAMP-Regulated Phosphoprotein 32 - metabolism | Mice, Knockout | Cocaine - pharmacology | Animals | Dopamine and cAMP-Regulated Phosphoprotein 32 - physiology | Fluorescent Antibody Technique | Dopamine Agents - toxicity | Mice | Haloperidol - toxicity | Human locomotion | Health aspects | Motor ability | Drugs | Animal models | Neuroleptics | Parkinson's disease | Neurodegenerative diseases | Haloperidol | spiny neurons | Signal transduction | Clonal deletion | Neostriatum | DARPP-32 protein | Levodopa | Locomotor activity | Movement disorders | Biological Sciences
Journal Article
Genetics, ISSN 0016-6731, 05/2016, Volume 203, Issue 1, pp. 599 - 609
Journal Article
Bioorganic & Medicinal Chemistry Letters, ISSN 0960-894X, 11/2015, Volume 25, Issue 22, pp. 5299 - 5305
Journal Article
Chemico-Biological Interactions, ISSN 0009-2797, 08/2016, Volume 255, Issue C, pp. 63 - 73
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 02/2005, Volume 25, Issue 8, pp. 2157 - 2165
Regulator of G-protein signaling 9-2 (RGS9-2), a member of the RGS family of G GTPase accelerating proteins, is expressed specifically in the striatum, which... 
DEP domain | Striatum | Antipsychotic | RGS9 | dopamine receptor | Dyskinesia | Antipsychotic Agents - toxicity | RGS Proteins - genetics | Reserpine - toxicity | Sulpiride - pharmacology | Antiparkinson Agents - pharmacology | Dyskinesia, Drug-Induced - physiopathology | Humans | Recombinant Fusion Proteins - physiology | Antiparkinson Agents - toxicity | RGS Proteins - physiology | Dopamine Agents - therapeutic use | Receptors, Dopamine D2 - metabolism | Antiparkinson Agents - therapeutic use | Transfection | Apomorphine - pharmacology | Dopamine - physiology | Dopamine Agents - pharmacology | RGS Proteins - deficiency | Dopamine Antagonists - toxicity | Reserpine - pharmacology | Female | Haloperidol - pharmacology | Receptors, G-Protein-Coupled - physiology | Protein Structure, Tertiary | Corpus Striatum - physiopathology | Parkinson Disease - physiopathology | Receptors, Dopamine D2 - genetics | Mice, Knockout | Protein Interaction Mapping | Patch-Clamp Techniques | Animals | Movement Disorders - physiopathology | Dopamine Antagonists - pharmacology | 2,3,4,5-Tetrahydro-7,8-dihydroxy-1-phenyl-1H-3-benzazepine - pharmacology | Corpus Striatum - drug effects | Mice | Quinpirole - pharmacology | Antipsychotic Agents - pharmacology | Movement Disorders - genetics | Haloperidol - toxicity | Subcellular Fractions - chemistry | Receptors, Dopamine D1 - genetics | striatum | Neurobiology of Disease | antipsychotic | dyskinesia | D2 dopamine receptor
Journal Article
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