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1. Full Text p21-Activated Kinases 1 and 3 Control Brain Size through Coordinating Neuronal Complexity and Synaptic Properties
by Wayne Huang and Zikai Zhou and Suhail Asrar and Mark Henkelman and Wei Xie and Zhengping Jia
Molecular and Cellular Biology, ISSN 0270-7306, 02/2011, Volume 31, Issue 3, pp. 388 - 403
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Neurons - pathology | Axons - enzymology | Synaptic Transmission - physiology | Brain - anatomy & histology | Neuroglia - pathology | Cell Count | Actins - metabolism | Brain - enzymology | Neurons - cytology | Hyperkinesis - physiopathology | Brain - growth & development | Synapses - pathology | Actin Depolymerizing Factors - metabolism | Behavior, Animal | Neuronal Plasticity - physiology | Anxiety - complications | Hyperkinesis - enzymology | Animals, Newborn | Hyperkinesis - complications | p21-Activated Kinases - genetics | Dendritic Spines - enzymology | Neuroglia - enzymology | p21-Activated Kinases - deficiency | Anxiety - physiopathology | Organ Size | Synapses - enzymology | p21-Activated Kinases - metabolism | Anxiety - enzymology | Animals | Axons - pathology | Dendritic Spines - pathology | Neurons - enzymology | Brain - pathology | Mice | Memory - physiology | Index Medicus
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2. Full Text Diet-induced obesity progressively alters cognition, anxiety-like behavior and lipopolysaccharide-induced depressive-like behavior: Focus on brain indoleamine 2,3-dioxygenase activation
by André, Caroline and Dinel, Anne-Laure and Ferreira, Guillaume and Layé, Sophie and Castanon, Nathalie
Brain, behavior, and immunity, ISSN 0889-1591, 2014, Volume 41, Issue 1, pp. 10 - 21
Psychiatry | Allergy and Immunology | Obesity | Cytokines | Lipopolysaccharide | Indoleamine 2,3-dioxygenase | Depression | Inflammation | Mice | Anxiety | Hippocampus | Spatial memory | Neurosciences | Immunology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Obesity - psychology | Endotoxins | Brain - enzymology | Male | Neuroimmunomodulation - physiology | Behavior, Animal | Depression - etiology | Cognition Disorders - etiology | Obesity - etiology | Hormones - blood | Cytokines - genetics | Cognition Disorders - psychology | Cytokines - blood | Cognition Disorders - enzymology | Memory Disorders - enzymology | Memory Disorders - psychology | Nerve Tissue Proteins - physiology | Mice, Inbred C57BL | Anxiety - psychology | Gene Expression Regulation | Obesity - physiopathology | Anxiety - enzymology | Diet, Western - adverse effects | Depression - enzymology | Animals | Maze Learning | Memory Disorders - etiology | Depression - psychology | Obesity - enzymology | Enzyme Activation | Anxiety - etiology | Cytokines - biosynthesis | Indoleamine-Pyrrole 2,3,-Dioxygenase - physiology | Type 2 diabetes | Tryptophan | Cognition | Mitogens | Depression, Mental | Index Medicus | Life Sciences
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3. Full Text Metformin protects the brain against ischemia/reperfusion injury through PI3K/Akt1/JNK3 signaling pathways in rats
by Ge, Xu-Hua and Zhu, Guo-Ji and Geng, De-Qin and Zhang, Han-Zhi and He, Juan-Mei and Guo, Ai-Zhen and Ma, Lin-Lin and Yu, De-Hua
Physiology & behavior, ISSN 0031-9384, 03/2017, Volume 170, pp. 115 - 123
Metformin | Akt | Ischemia/reperfusion | Caspase-3 | JNK3 | Psychology, Biological | Social Sciences | Life Sciences & Biomedicine | Behavioral Sciences | Psychology | Science & Technology | Apoptosis - drug effects | Caspase 3 - metabolism | Reperfusion Injury - drug therapy | Male | Anxiety - drug therapy | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | CA1 Region, Hippocampal - drug effects | Brain Ischemia - enzymology | CA1 Region, Hippocampal - pathology | Neuroprotective Agents - pharmacology | Cognition Disorders - etiology | Pyramidal Cells - drug effects | Reperfusion Injury - complications | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Brain Ischemia - complications | Cognition Disorders - enzymology | CA1 Region, Hippocampal - enzymology | Maze Learning - physiology | Reperfusion Injury - pathology | Reperfusion Injury - enzymology | Metformin - pharmacology | Cognition Disorders - pathology | Enzyme Inhibitors - pharmacology | Pyramidal Cells - enzymology | Cognition Disorders - drug therapy | Maze Learning - drug effects | Anxiety - enzymology | Rats, Sprague-Dawley | Anxiety - pathology | Mitogen-Activated Protein Kinase 10 - metabolism | Animals | Signal Transduction - drug effects | Brain Ischemia - drug therapy | Brain Ischemia - pathology | Pyramidal Cells - pathology | Apoptosis - physiology | Anxiety - etiology | Brain | Brain damage | Neurons | Index Medicus
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4. Full Text Therapeutic potential of monoacylglycerol lipase inhibitors
by Mulvihill, Melinda M and Nomura, Daniel K
Life sciences (1973), ISSN 0024-3205, 03/2013, Volume 92, Issue 8-9, pp. 492 - 497
Monoacylglycerol lipase | Endocannabinoid | Prostaglandin | Eicosanoid | mgll | Neurodegenerative disease | MAGL | Inflammation | Cannabinoid | JZL184 | Cancer | Pharmacology & Pharmacy | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Monoacylglycerol Lipases - antagonists & inhibitors | Humans | Monoacylglycerol Lipases - physiology | Enzyme Inhibitors - pharmacology | Neoplasms - enzymology | Substance-Related Disorders - enzymology | Anxiety - drug therapy | Anxiety - enzymology | Enzyme Inhibitors - therapeutic use | Neoplasms - drug therapy | Animals | Pain - enzymology | Inflammation - drug therapy | Substance-Related Disorders - drug therapy | Inflammation - enzymology | COX-2 inhibitors | Nervous system diseases | Analysis | Marijuana | Lipase | Health aspects | Fatty acids | Index Medicus | monoacylglycerol lipase | endocannabinoid | eicosanoid | neurodegenerative disease | prostaglandin | inflammation | cannabinoid | cancer
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5. Full Text Rosmarinic Acid Protects Rat Hippocampal Neurons from Cerebral Ischemia/Reperfusion Injury via the Akt/JNK3/Caspase-3 Signaling Pathway
by Zhang, Min and Yan, Hui and Li, Sumei and Yang, Jun
Brain research, ISSN 0006-8993, 2016, Volume 1657, pp. 9 - 15
Neurology | Rosmarinic acid | Cerebral ischemia | Akt | Caspase-3 | JNK3 | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Neurons - pathology | Apoptosis - drug effects | Caspase 3 - metabolism | Reperfusion Injury - drug therapy | Male | Anxiety - drug therapy | CA1 Region, Hippocampal - drug effects | Depsides - pharmacology | Brain Ischemia - enzymology | CA1 Region, Hippocampal - pathology | Neuroprotective Agents - pharmacology | Phosphorylation - drug effects | Neurons - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Cell Survival - physiology | Disease Models, Animal | Cognition Disorders - enzymology | CA1 Region, Hippocampal - enzymology | Cell Survival - drug effects | Maze Learning - physiology | Reperfusion Injury - pathology | Reperfusion Injury - enzymology | Cognition Disorders - pathology | Random Allocation | Reperfusion Injury - psychology | Cognition Disorders - drug therapy | Maze Learning - drug effects | Anxiety - enzymology | Cinnamates - pharmacology | Rats, Sprague-Dawley | Anxiety - pathology | Mitogen-Activated Protein Kinase 10 - metabolism | Animals | Brain Ischemia - drug therapy | Neurons - enzymology | Brain Ischemia - pathology | Brain Ischemia - psychology | Apoptosis - physiology | Neurons | Brain | Brain damage | Cell death | Injuries | Index Medicus
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6. Full Text Alterations in neuronal control of body weight and anxiety behavior by glutathione peroxidase 4 deficiency
by Schriever, Sonja C and Zimprich, Annemarie and Pfuhlmann, Katrin and Baumann, Peter and Giesert, Florian and Klaus, Valentina and Kabra, Dhiraj G and Hafen, Ulrich and Romanov, Artem and Tschöp, Matthias H and Wurst, Wolfgang and Conrad, Marcus and Hölter, Sabine M and Vogt Weisenhorn, Daniela and Pfluger, Paul T
Neuroscience, ISSN 0306-4522, 2017, Volume 357, pp. 241 - 254
Neurology | antioxidant | DJ-1 | lipid peroxidation | obesity | Parkinson’s disease | hypothalamus | Parkinson's disease | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Motor Activity - physiology | Dopaminergic Neurons - pathology | Oxidative Stress - physiology | Male | Protein Deglycase DJ-1 - genetics | Diet, High-Fat | Female | Dietary Sucrose | Glutathione Peroxidase - deficiency | Hypothalamus - immunology | Glutathione Peroxidase - metabolism | Dopaminergic Neurons - immunology | Protein Deglycase DJ-1 - metabolism | Mice, Inbred C57BL | Behavior, Animal - physiology | Parkinsonian Disorders - enzymology | Hypothalamus - pathology | Sex Characteristics | Anxiety - enzymology | Glutathione Peroxidase - genetics | Anxiety - pathology | Mice, Knockout | Obesity - pathology | Anxiety - immunology | Dopaminergic Neurons - enzymology | Animals | Parkinsonian Disorders - pathology | Hypothalamus - enzymology | Adiposity - physiology | Obesity - enzymology | Body Weight - physiology | Parkinsonian Disorders - immunology | Antioxidants | Obesity | Neurons | Body weight | Physiological aspects | Peroxidase | Intermedin | Index Medicus
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7. Full Text Using c-Jun to identify fear extinction learning-specific patterns of neural activity that are affected by single prolonged stress
by Knox, Dayan and Stanfield, Briana R and Staib, Jennifer M and David, Nina P and DePietro, Thomas and Chamness, Marisa and Schneider, Elizabeth K and Keller, Samantha M and Lawless, Caroline
Behavioural brain research, ISSN 0166-4328, 04/2018, Volume 341, pp. 189 - 197
PTSD | Fear | Anxiety | C-Jun | Extinction | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Behavioral Sciences | Science & Technology | Immunohistochemistry | Neurons - pathology | Basolateral Nuclear Complex - enzymology | Conditioning (Psychology) - physiology | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Hippocampus - pathology | Fear - physiology | Rats, Sprague-Dawley | Prefrontal Cortex - pathology | Stress, Psychological - pathology | Animals | Basolateral Nuclear Complex - pathology | Neurons - enzymology | Hippocampus - enzymology | Prefrontal Cortex - enzymology | Stress, Psychological - enzymology | Extinction, Psychological - physiology | Index Medicus | anxiety | c-Jun | extinction | fear
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8. Full Text The GCs-SGK1-ATP Signaling Pathway in Spinal Astrocytes Underlied Presurgical Anxiety-Induced Postsurgical Hyperalgesia
by Zhang, ZuoXia and Wu, Hao and Liu, Yue and Gu, XiaoPing and Zhang, Wei and Ma, ZhengLiang
Anesthesia and analgesia, ISSN 0003-2999, 08/2018, Volume 129, Issue 4, pp. 1163 - 1169
Life Sciences & Biomedicine | Anesthesiology | Science & Technology | Pain, Postoperative - etiology | Tumor Necrosis Factor-alpha - metabolism | Corticosterone - pharmacology | Spinal Cord - drug effects | Male | Astrocytes - enzymology | Immediate-Early Proteins - metabolism | Interleukin-1beta - metabolism | Adenosine Triphosphate - metabolism | Anxiety - complications | Pain Threshold - drug effects | Protein-Serine-Threonine Kinases - metabolism | Second Messenger Systems | Surgical Procedures, Operative - adverse effects | Disease Models, Animal | Astrocytes - drug effects | Pain, Postoperative - enzymology | Spinal Cord - enzymology | Anxiety - physiopathology | Anxiety - enzymology | Rats, Sprague-Dawley | Hyperalgesia - physiopathology | Animals | Hyperalgesia - etiology | Corticosterone - metabolism | Spinal Cord - physiopathology | Surgical Procedures, Operative - psychology | Pain, Postoperative - physiopathology | Hyperalgesia - enzymology | Index Medicus | Abridged Index Medicus
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