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Cell stem cell, ISSN 1934-5909, 2015, Volume 16, Issue 1, pp. 51 - 66
Mesenchymal stem cells (MSCs) reside in the perivascular niche of many organs, including kidney, lung, liver, and heart, although their roles in these tissues... 
REGULATOR | ORIGIN | SONIC HEDGEHOG | PATHWAY | BONE-MARROW NICHE | MYOFIBROBLASTS | NG2 PROTEOGLYCAN | EXPRESSION | MESENCHYMAL STEM-CELLS | GROWTH FACTOR-AA | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Organ Specificity - drug effects | Diphtheria Toxin - pharmacology | Neovascularization, Physiologic - drug effects | Pericytes - drug effects | Blood Vessels - metabolism | Blood Vessels - pathology | Humans | Fibrosis - metabolism | Cell Lineage - drug effects | Myofibroblasts - metabolism | Mesenchymal Stromal Cells - cytology | Mesenchymal Stromal Cells - ultrastructure | Kruppel-Like Transcription Factors - metabolism | Pericytes - pathology | Bone Marrow Cells - drug effects | Colony-Forming Units Assay | Aorta - physiopathology | Homeostasis - drug effects | Heart Ventricles - pathology | Receptor, Platelet-Derived Growth Factor beta - metabolism | Mesenchymal Stromal Cells - drug effects | Endothelial Cells - metabolism | Aorta - drug effects | Pericytes - metabolism | Cells, Cultured | Proteoglycans - metabolism | Antigens - metabolism | Aorta - pathology | Myofibroblasts - cytology | Animals | Heart Ventricles - physiopathology | Cell Differentiation - drug effects | Endothelial Cells - cytology | Blood Vessels - drug effects | Mice | Stem Cell Niche - drug effects | Zinc Finger Protein GLI1 | Fibrosis - pathology | Bone Marrow Cells - metabolism | Endothelial Cells - drug effects | Heart Ventricles - drug effects
Journal Article
Journal of translational medicine, ISSN 1479-5876, 2019, Volume 17, Issue 1, pp. 127 - 14
...) especially in those with HF at baseline. These might indicate a potent direct cardioprotective effect, which is currently incompletely understood... 
Sodium-glucose cotransporter-2 inhibitor | Cardioprotection | Canagliflozin | Myocardial ischemia-reperfusion injury | MEDICINE, RESEARCH & EXPERIMENTAL | CARDIOVASCULAR OUTCOMES | SGLT2 INHIBITORS | PHOSPHORYLATION | KINASE | CARDIOMYOCYTES | HEART | PRESSURE | AMPK | ARTERY | Apoptosis - drug effects | Canagliflozin - pharmacology | Myocardial Reperfusion Injury - complications | Systole - drug effects | Male | Aldehydes - metabolism | Liver - physiopathology | Cardiotonic Agents - therapeutic use | Liver - drug effects | Canagliflozin - therapeutic use | Diastole - drug effects | Aorta - physiopathology | Myocardial Reperfusion Injury - drug therapy | Phosphorylation - drug effects | Kidney - physiopathology | Biomarkers - metabolism | Endothelium - pathology | Glycosuria - physiopathology | Kidney - drug effects | Aorta - drug effects | Ventricular Function, Left - drug effects | Cardiotonic Agents - pharmacology | Rats, Sprague-Dawley | Glycosuria - complications | Myocardial Reperfusion Injury - physiopathology | Endothelium - physiopathology | Aorta - pathology | Endothelium - drug effects | Animals | Signal Transduction - drug effects | Oxidative Stress - drug effects | Vasodilation - drug effects | Blood Glucose - metabolism | Myocardial Reperfusion Injury - prevention & control | Nitrosative Stress - drug effects | Heart failure | Diabetics | Rats as laboratory animals | Analysis | Clinical trials | Research | Drug therapy | Health aspects | Occlusion | Myocardial infarction | Heart | Oxidative stress | Phosphorylation | Drug delivery systems | Intravenous administration | Heart attacks | Bax protein | 4-Hydroxynonenal | Bcl-2 protein | AKT protein | Myocardial ischemia | mRNA | Size determination | Kinases | Vasodilation | Proteins | Reperfusion | Ischemia | Rodents | Calcium-binding protein | Aorta | Heart diseases | Injury analysis | Medical research | Adenosine monophosphate | AMP | Diabetes mellitus | Coronary artery | Gene expression | Nitric-oxide synthase | Endothelium | Sodium | Protein kinase | Adenosine kinase | Nitric oxide | Ventricle | Diabetes | Laboratory animals | Apoptosis | Myocardial ischemia–reperfusion injury | Sodium–glucose cotransporter-2 inhibitor
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 509, Issue 7502, pp. 641 - 644
Despite the introduction of antiproliferative drug-eluting stents, coronary heart disease remains the leading cause of death in the United States(1... 
APOPTOSIS | CELLS | MECHANISMS | MULTIDISCIPLINARY SCIENCES | MITOCHONDRIA | DISEASE | Iliac Artery - pathology | Protein-Serine-Threonine Kinases - deficiency | Constriction, Pathologic - pathology | Mitochondria, Heart - metabolism | Iliac Artery - drug effects | Apoptosis - drug effects | Humans | Myocytes, Smooth Muscle - pathology | Iliac Artery - injuries | Arteries - injuries | Secondary Prevention | Mitochondria, Heart - drug effects | Angioplasty, Balloon - adverse effects | Membrane Potential, Mitochondrial - drug effects | Dichloroacetic Acid - therapeutic use | Gene Knockdown Techniques | Swine | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Hyperplasia - drug therapy | Mammary Arteries - drug effects | Myocytes, Smooth Muscle - drug effects | Coronary Vessels - injuries | Disease Models, Animal | Constriction, Pathologic - prevention & control | Rabbits | Coronary Vessels - drug effects | Coronary Vessels - pathology | Aorta - drug effects | Protein-Serine-Threonine Kinases - genetics | Rats | Mammary Arteries - injuries | Arteries - drug effects | Aorta - injuries | Arteries - pathology | Enzyme Activation - drug effects | Tunica Intima - injuries | Aorta - pathology | Hyperplasia - pathology | Tunica Intima - drug effects | Animals | Tunica Intima - pathology | Mammary Arteries - pathology | Dichloroacetic Acid - pharmacology | Cell Proliferation - drug effects | Stents - adverse effects | Complications and side effects | Patient outcomes | Genetic aspects | Research | Coronary heart disease | Health aspects | Risk factors | Stent (Surgery) | Apoptosis | Permeability | Angioplasty | Rodents | Binding sites | Veins & arteries | Index Medicus
Journal Article
Journal of the American Heart Association, ISSN 2047-9980, 09/2016, Volume 5, Issue 9, p. n/a
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2007, Volume 293, Issue 4, pp. 2210 - 2218
Targeting cannabinoid-2 (CB2) receptors with selective agonists may represent a novel therapeutic avenue in various inflammatory diseases, but the mechanisms by which CB2 activation exerts its anti-inflammatory effects... 
Adhesion molecules | Inflammation | RhoA | Endothelial activation | C-REACTIVE PROTEIN | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | MOLECULE EXPRESSION | ATHEROSCLEROSIS | NECROSIS-FACTOR-ALPHA | KNOCKOUT MICE | endothelial activation | CANNABINOID RECEPTORS | inflammation | adhesion molecules | NITRIC-OXIDE | THERAPEUTIC TARGETS | PERIPHERAL VASCULAR DISEASE | POTENTIAL ROLE | CB2 RECEPTOR ACTIVATION | Inflammation - chemically induced | Leukocyte Rolling - drug effects | Tumor Necrosis Factor-alpha - metabolism | Receptor, Cannabinoid, CB2 - agonists | Humans | Male | Monocytes - metabolism | NF-kappa B - metabolism | rhoA GTP-Binding Protein - metabolism | Aorta - metabolism | RNA, Messenger - metabolism | Receptor, Cannabinoid, CB2 - genetics | Coronary Vessels - metabolism | Lipopolysaccharides | Dose-Response Relationship, Drug | Inflammation - metabolism | Anti-Inflammatory Agents - therapeutic use | Chemokine CCL2 - metabolism | Disease Models, Animal | Coronary Vessels - drug effects | Anti-Inflammatory Agents - pharmacology | Endothelial Cells - metabolism | Aorta - drug effects | Mice, Inbred C57BL | Cells, Cultured | Cannabinoids - pharmacology | Monocytes - drug effects | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Cannabinoids - therapeutic use | Intercellular Adhesion Molecule-1 - metabolism | Animals | Signal Transduction - drug effects | Inflammation - prevention & control | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Endothelial Cells - drug effects
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 8, p. e104020
Hypertension is considered as a low-grade inflammatory disease, with adaptive immunity being an important mediator of this pathology. TLR4 may have a role in... 
OXIDATIVE STRESS | ACTIVATION | IMMUNE-SYSTEM | ENDOTHELIAL DYSFUNCTION | RESISTANCE ARTERIES | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | LIPOPOLYSACCHARIDE | SMOOTH-MUSCLE-CELLS | EXPRESSION | BLOOD-PRESSURE | Reactive Oxygen Species - metabolism | Rats, Wistar | NADPH Oxidases - metabolism | Endothelium, Vascular - drug effects | Male | Heart Rate - drug effects | Phenylephrine - pharmacology | Superoxides - metabolism | Blood Pressure - drug effects | Aorta - physiopathology | Hypertension - genetics | Toll-Like Receptor 4 - antagonists & inhibitors | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Rats, Inbred SHR | Angiotensin II - pharmacology | Acetylcholine - pharmacology | Aorta - drug effects | Endothelium, Vascular - physiopathology | Toll-Like Receptor 4 - genetics | Antioxidants - pharmacology | Toll-Like Receptor 4 - metabolism | Vasoconstriction - drug effects | Hypertension - pathology | Hypertension - physiopathology | Aorta - pathology | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Cell Movement - drug effects | Muscle, Smooth, Vascular - pathology | Animals | Endothelium, Vascular - pathology | Cell Proliferation - drug effects | Oxidative Stress - drug effects | Vasodilation - drug effects | In Vitro Techniques | Potentiation | Oxidative stress | Reactive oxygen species | Biomedical research | Immunoglobulin G | Smooth muscle | Innate immunity | mRNA | Activation | Biochemistry | Immunity | Experiments | Contraction | NAD(P)H oxidase | Ethics | Renin | Catalase | Rodents | Atherosclerosis | Phenylephrine | Toll-like receptors | Aorta | Physiology | Blood pressure | Angiotensin II | Heart diseases | Hypertension | Heparan sulfate | Oxygen | Muscles | Rats | Superoxide | Inflammation | Adaptive immunity | TLR4 protein | Yang, Cindy | Endothelium | Heart rate | NAD | Pathology | Angiotensin | Acetylcholine | Cardiovascular diseases
Journal Article
American Journal of Hypertension, ISSN 0895-7061, 2017, Volume 30, Issue 4, pp. 389 - 399
BACKGROUND We investigated the influence of salt overconsumption on the functionality of the RhoA/Rho-associated kinase (ROCK) pathway and calcium regulation... 
Calcium signaling | Hypertension | Calcium sensitization | High-salt | Vascular smooth muscle cell | Blood pressure | Vascular dysfunction | blood pressure | RHO-ASSOCIATED KINASE | TRANSLOCATION | SENSITIVE RATS | ACTIVATION | PROTEIN-KINASE | calcium signaling | VASCULAR SMOOTH-MUSCLE | BLOOD-PRESSURE | high-salt | calcium sensitization | vascular dysfunction | CONTRACTION | PERIPHERAL VASCULAR DISEASE | vascular smooth muscle cell | hypertension | MYOSIN PHOSPHATASE | Rats, Wistar | Sodium Chloride, Dietary - pharmacology | Calcium - metabolism | Male | Mesenteric Arteries - drug effects | Ryanodine Receptor Calcium Release Channel - metabolism | Aorta - metabolism | Stromal Interaction Molecule 1 - drug effects | Sarcoplasmic Reticulum Calcium-Transporting ATPases - drug effects | Inositol 1,4,5-Trisphosphate Receptors - drug effects | Phenylephrine - pharmacology | rho-Associated Kinases - metabolism | Inositol 1,4,5-Trisphosphate Receptors - metabolism | rho-Associated Kinases - drug effects | Phosphorylation - drug effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | ORAI1 Protein - drug effects | Amides - pharmacology | Mesenteric Arteries - cytology | ORAI1 Protein - metabolism | Vasoconstrictor Agents - pharmacology | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Aorta - drug effects | Enzyme Inhibitors - pharmacology | Rats | Ryanodine Receptor Calcium Release Channel - drug effects | Mesenteric Arteries - metabolism | Vasoconstriction - drug effects | rho GTP-Binding Proteins - drug effects | Stromal Interaction Molecule 1 - metabolism | Protein Phosphatase 1 - metabolism | Animals | Protein Phosphatase 1 - drug effects | Signal Transduction - drug effects | rho GTP-Binding Proteins - metabolism | Pyridines - pharmacology | Aorta - cytology | Physiological aspects | Salt | Research | Health aspects | Rattus | Blood Vessels | Original
Journal Article
Endocrinology, ISSN 0013-7227, 10/2017, Volume 158, Issue 10, pp. 3592 - 3604
...)-4 inhibitors exert cardioprotective effects, we examined the hypothesis that DPP-4 inhibition with saxagliptin (Saxa... 
MYOCARDIAL FIBROSIS | CIKS ACT1 | INFLAMMATION | HEART-FAILURE | FEMALE MICE | ENDOCRINOLOGY & METABOLISM | RECEPTOR EXPRESSION | PRESERVED EJECTION FRACTION | SENSITIVE HYPERTENSIVE-RATS | DIET-INDUCED OBESITY | CD8(+) T-CELLS | Dipeptidyl Peptidase 4 - drug effects | Dipeptidyl Peptidase 4 - metabolism | Gene Expression - drug effects | Male | Vasoconstrictor Agents - toxicity | Dipeptidyl-Peptidase IV Inhibitors - pharmacology | CD8 Antigens - drug effects | Blood Pressure - drug effects | Diastole - drug effects | Angiotensin II - toxicity | Adamantane - analogs & derivatives | Heart - physiopathology | Adamantane - pharmacology | Echocardiography | Fibrosis - chemically induced | Signal Transduction | Aorta - drug effects | Dipeptides - pharmacology | Inflammation | Interleukin-17 - metabolism | CD8 Antigens - metabolism | Macrophages - metabolism | Animals | Proto-Oncogene Proteins c-jun - metabolism | Lymphocytes - drug effects | Vascular Stiffness - drug effects | Cardiomegaly - chemically induced | Heart - drug effects | Macrophages - drug effects | Mice | Adaptor Proteins, Signal Transducing - drug effects | Adaptor Proteins, Signal Transducing - metabolism | Proto-Oncogene Proteins c-jun - drug effects | CD4-Positive T-Lymphocytes - drug effects | Interleukin-18 - metabolism | Heart | Flow cytometry | Transcription factors | CD8 antigen | Interleukin | Lymphocytes T | mRNA | Activation | Leukocytes | Aldosterone | Macrophages | CD45 antigen | Cell activation | Lymphocytes | CD44 antigen | Rodents | Aorta | Blood pressure | Inhibition | Angiotensin II | Hypertension | CD11c antigen | CD11b antigen | c-Jun protein | Stiffening | Gene expression | Ribonucleic acid--RNA | Coronary artery disease | Interleukin 18 | CD4 antigen | White blood cells | Cytometry | Signaling | Gene flow | Fibrosis | Angiotensin | Infiltration
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2016, Volume 36, Issue 8, pp. 1534 - 1548
OBJECTIVE—Drug-eluting coronary stents reduce restenosis rate and late lumen loss compared with bare-metal stents... 
blood platelets | endothelial cells | peroxisome proliferator-activated receptors | vascular smooth muscle cells | coronary in-stent restenosis | Recurrence | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Glucose Transporter Type 1 - metabolism | Aorta - metabolism | Angioplasty, Balloon - adverse effects | Aortic Diseases - metabolism | Neointima | Time Factors | Aortic Diseases - prevention & control | Myocytes, Smooth Muscle - drug effects | PPAR delta - genetics | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Atherosclerosis - pathology | Drug-Eluting Stents | Human Umbilical Vein Endothelial Cells - drug effects | Coronary Artery Disease - metabolism | Rats | Rats, Sprague-Dawley | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Cell Movement - drug effects | Thrombosis - pathology | Thrombosis - metabolism | Coronary Artery Disease - therapy | Signal Transduction - drug effects | Blood Platelets - metabolism | PPAR delta - agonists | PPAR delta - metabolism | Energy Metabolism - drug effects | Steroids - administration & dosage | Muscle, Smooth, Vascular - metabolism | Myocytes, Smooth Muscle - pathology | Dose-Response Relationship, Drug | Transfection | Coronary Artery Disease - pathology | Thrombosis - prevention & control | Blood Platelets - drug effects | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Aortic Diseases - pathology | Carotid Artery Injuries - pathology | Cardiovascular Agents - administration & dosage | PPAR delta - deficiency | Aorta - drug effects | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Re-Epithelialization - drug effects | Angioplasty, Balloon - instrumentation | Muscle, Smooth, Vascular - pathology | Animals | Glucose Transporter Type 1 - genetics | Thrombosis - etiology | Cell Proliferation - drug effects | Platelet Activation - drug effects | Atherosclerosis - prevention & control
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 12/2016, Volume 311, Issue 6, pp. H1360 - H1366
Journal Article
Carcinogenesis (New York), ISSN 1460-2180, 2009, Volume 31, Issue 3, pp. 481 - 488
...) has become an attractive target for anti-angiogenesis treatment. However, the side effects associated with most anti-VEGF agents limit their chronic use... 
PROSTATE | DIETARY ANTHOCYANIDIN | INSULIN-RECEPTOR | POLYPHENOLS | ONCOLOGY | MECHANISMS | TUMOR ANGIOGENESIS | GRAPE SEED EXTRACT | SUPPRESSION | CANCER | ENDOTHELIAL GROWTH-FACTOR | Biflavonoids - pharmacology | Neovascularization, Physiologic - drug effects | Plant Extracts - pharmacology | Cells, Cultured - cytology | Humans | Male | Cell Line, Tumor - drug effects | Cell Line, Tumor - transplantation | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Laminin | Proanthocyanidins - pharmacology | Protein Processing, Post-Translational - drug effects | Cattle | Female | Aorta, Thoracic - drug effects | Phosphorylation - drug effects | Catechin - pharmacology | Cells, Cultured - drug effects | Proteoglycans | Cell Line, Tumor - enzymology | Cinnamomum zeylanicum - chemistry | Angiogenesis Inhibitors - pharmacology | Plant Extracts - isolation & purification | Mice, SCID | Chick Embryo | Cell Division - drug effects | Cells, Cultured - enzymology | Xenograft Model Antitumor Assays | Cell Movement - drug effects | Biflavonoids - isolation & purification | Animals | Signal Transduction - drug effects | Breast Neoplasms - pathology | Endothelial Cells - cytology | Collagen | Catechin - isolation & purification | Morphogenesis - drug effects | Angiogenesis Inhibitors - isolation & purification | Mice | Endothelial Cells - enzymology | Drug Combinations | Proanthocyanidins - isolation & purification | Endothelial Cells - drug effects | Index Medicus | Cancer Prevention
Journal Article