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Development, ISSN 0950-1991, 10/2002, Volume 129, Issue 19, pp. 4613 - 4625
We present here an analysis of cardiovascular and pharyngeal arch development in mouse embryos hypomorphic for Fgf8 . Previously, we have described the... 
Transposition | Arch artery | Fgf8 | Patterning | Mouse | Pharyngeal arch | Cardiovascular | Cell death | Double outlet right ventricle | 22q11 | DiGeaorge | Neural crest | MIGRATION | SIGNALING PATHWAYS | cell death | arch artery | SONIC HEDGEHOG | neural crest | FIRST BRANCHIAL ARCH | DEVELOPMENTAL BIOLOGY | cardiovascular | patterning | DiGeorge | DIGEORGE-SYNDROME REGION | transposition | mouse | double outlet right ventricle | GENE | pharyngeal arch | CHICK-EMBRYO | NEURAL CREST ABLATION | EXPRESSION | TRANSGENIC MICE | Heart Defects, Congenital - embryology | Cell Count | Heart - embryology | Fibroblast Growth Factors - genetics | Male | Helix-Loop-Helix Motifs | Transcription Factor AP-2 | Receptors, Retinoic Acid - genetics | Neural Crest - metabolism | Branchial Region - abnormalities | Cell Division | Body Patterning | Female | Cardiovascular Abnormalities - metabolism | Cardiovascular System - embryology | Basic Helix-Loop-Helix Transcription Factors | Fibroblast Growth Factors - physiology | Pulmonary Artery - embryology | Gene Expression | Neural Crest - cytology | Aorta, Thoracic - embryology | Zebrafish Proteins | Pulmonary Artery - abnormalities | Coronary Vessels - embryology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | T-Box Domain Proteins - genetics | Mice, Inbred ICR | Mice, Knockout | Animals | Neural Crest - embryology | Biomarkers | Heart Defects, Congenital - metabolism | Mice | Apoptosis | Branchial Region - embryology | Cell Movement | Fibroblast Growth Factor 8
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2013, Volume 123, Issue 12, pp. 5052 - 5060
Children conceived by assisted reproductive technologies (ART) display a level of vascular dysfunction similar to that seen in children of mothers with... 
PATERNAL METHYLATION | PULMONARY | MEDICINE, RESEARCH & EXPERIMENTAL | EPIGENETIC INHERITANCE | DNA METHYLATION | NITRIC-OXIDE SYNTHASE | INSULIN-RESISTANCE | MOUSE | DISEASE | IMPRINTED GENE | ASSISTED REPRODUCTIVE TECHNOLOGIES | Butyrates - pharmacology | Nitric Oxide Synthase Type III - biosynthesis | Male | Vascular Resistance - physiology | Hypertension - etiology | DNA Methylation | Cardiovascular Abnormalities - etiology | Gene Expression Regulation, Developmental | Butyrates - therapeutic use | Fertilization in Vitro - methods | Female | Nitric Oxide Synthase Type III - physiology | Vasodilation - physiology | Aorta - enzymology | Ovulation Induction - adverse effects | Endothelium, Vascular - embryology | Fertilization in Vitro - adverse effects | Promoter Regions, Genetic | Disease Susceptibility | Down-Regulation | Endothelium, Vascular - physiopathology | Cardiovascular Abnormalities - embryology | Longevity | Nitric Oxide Synthase Type III - genetics | Hypertension - physiopathology | Gene Expression Regulation, Enzymologic | Diet, Atherogenic | Animals | Hypertension - embryology | Histone Deacetylase Inhibitors - pharmacology | Histone Deacetylase Inhibitors - therapeutic use | Mice | Nitric Oxide - metabolism | Infants (Newborn) | Blood circulation disorders | Research | Risk factors | Reproductive technology | Diseases | Studies | Blood pressure | Rodents | Metabolic disorders
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2016, Volume 311, Issue 5, pp. H1150 - H1159
Cardiac neural crest cell (CNCC) ablation creates congenital heart defects (CHDs) that resemble those observed in many syndromes with craniofacial and cardiac... 
Cardiac cushion | Cardiac neural crest | Optical coherence tomography | Cardiac valve | Cardiac function | PERSISTENT TRUNCUS ARTERIOSUS | CELLS | DEFECTS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | optical coherence tomography | OUTFLOW TRACT | cardiac cushion | EPIGENETIC FACTOR | CONGENITAL HEART-DISEASE | EMBRYONIC HEART | cardiac neural crest | CHICK-EMBRYO | PERIPHERAL VASCULAR DISEASE | cardiac function | cardiac valve | EXPOSURE | Aorta - abnormalities | Heart Defects, Congenital - embryology | Heart - embryology | Laser Therapy | Pulmonary Artery - diagnostic imaging | Heart Defects, Congenital - diagnostic imaging | Endocardial Cushion Defects - embryology | Heart Valves - embryology | Endocardial Cushion Defects - diagnostic imaging | Pulmonary Artery - embryology | Aorta - diagnostic imaging | Tomography, Optical Coherence | Fetal Alcohol Spectrum Disorders | Aorta - embryology | Neural Crest - surgery | Organ Size | Pulmonary Artery - abnormalities | Quail | Heart Valves - abnormalities | Heart - diagnostic imaging | Phenotype | Animals | Neural Crest - embryology | Embryo, Nonmammalian | Heart Valves - diagnostic imaging | Heart | Ablation (Surgery) | Care and treatment | Surgery | Congenital heart disease | Health aspects | Methods | Blood flow | Integrative Cardiovascular Physiology and Pathophysiology
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 06/2010, Volume 30, Issue 6, pp. 1118 - 1126
OBJECTIVE—Regulation of vascular smooth muscle (VSM) proliferation and contractile differentiation is an important factor in vascular development and... 
Molecular biology | Contractile proteins | Vascular biology | Vascular muscle | molecular biology | ACTIVATION | PHENOTYPE | vascular biology | ACTIN DYNAMICS | PROLIFERATION | DICER | vascular muscle | MOUSE DEVELOPMENT | NEOINTIMAL LESION FORMATION | EMBRYONIC STEM-CELLS | SERUM RESPONSE FACTOR | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | contractile proteins | Endoribonucleases - genetics | Cell Proliferation | Muscle, Smooth, Vascular - metabolism | Transcriptional Activation | Actins - metabolism | Male | MicroRNAs - metabolism | Aorta - metabolism | Muscle, Smooth, Vascular - physiopathology | Muscle, Smooth, Vascular - embryology | Cell Differentiation - genetics | Vasodilation - genetics | Transfection | Gene Expression Regulation, Developmental | DEAD-box RNA Helicases - deficiency | Trans-Activators - genetics | Hemorrhage - embryology | Endoribonucleases - deficiency | Nuclear Proteins - genetics | Microfilament Proteins - genetics | Vasoconstriction - genetics | Liver Diseases - embryology | Hemorrhage - genetics | Liver Diseases - genetics | Umbilical Arteries - pathology | Muscle, Smooth, Vascular - ultrastructure | Cells, Cultured | Umbilical Arteries - embryology | Aorta - embryology | Genotype | Nuclear Proteins - metabolism | Hemorrhage - metabolism | Gestational Age | Mice, Knockout | Aorta - pathology | Muscle Proteins - genetics | DEAD-box RNA Helicases - genetics | Embryo Loss | Phenotype | Stress Fibers - metabolism | Animals | Ribonuclease III | Muscle Development - genetics | Trans-Activators - metabolism | Mice | Liver Diseases - metabolism | Integrases - genetics | Umbilical Arteries - metabolism
Journal Article
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 07/2011, Volume 26, Issue 7, pp. 1543 - 1553
Vascular calcifications can occur in the elderly and in patients suffering from various diseases. Interestingly, depending on the pathology, different regions... 
Hyperphosphatemia | Alkaline phosphatase | Smooth muscle cells | Matrix Gla protein | Vascular calcifications | Neural crest | EXPERIMENTAL CANINE ATHEROSCLEROSIS | SMOOTH MUSCLE CELLS | MATRIX GLA PROTEIN | VASCULAR CALCIFICATIONS | PATTERNS | ALKALINE PHOSPHATASE | FATE | CORONARY-ARTERIES | CARTILAGE | HYPERPHOSPHATEMIA | AORTIC HOMOGRAFTS | DISEASE | MINERALIZATION | ENDOCRINOLOGY & METABOLISM | MICE | NEURAL CREST | Extracellular Matrix Proteins - deficiency | Neural Crest - pathology | Aging - drug effects | Alkaline Phosphatase - metabolism | Mesoderm - drug effects | Myocytes, Smooth Muscle - pathology | Aorta, Abdominal - drug effects | Muscle, Smooth, Vascular - embryology | Aorta, Abdominal - pathology | Aorta, Thoracic - drug effects | Calcinosis - metabolism | Myocytes, Smooth Muscle - drug effects | Aorta, Abdominal - metabolism | Myocytes, Smooth Muscle - metabolism | Phosphates - pharmacology | Aorta, Thoracic - pathology | Extracellular Matrix Proteins - metabolism | Calcium-Binding Proteins - metabolism | Alkaline Phosphatase - genetics | Mesoderm - embryology | Cells, Cultured | Aorta, Thoracic - metabolism | Calcium-Binding Proteins - deficiency | Aging - pathology | Neural Crest - drug effects | Muscle, Smooth, Vascular - pathology | Animals | Neural Crest - embryology | Calcinosis - embryology | Mice | Kinetics | Calcinosis - pathology | Mesoderm - pathology | Life Sciences | Morphogenesis | Human health and pathology | Biochemistry, Molecular Biology | Development Biology | Molecular biology | Cellular Biology | Embryology and Organogenesis | Cardiology and cardiovascular system | Cell Behavior
Journal Article
Publication / Carnegie Institution of Washington, Volume no. 277, 47-110, 3 leaves of plates
Book
Circulation Research, ISSN 0009-7330, 01/2009, Volume 104, Issue 1, pp. 19 - 31
Reentry is the main mechanism of life-threatening ventricular arrhythmias, including ventricular fibrillation and tachycardia. Its occurrence depends on the... 
Ventricular arrhythmias | Connexin | Cardiac development | Second heart field | RVOT | CURRENT I-KR | GAP-JUNCTION CHANNELS | CARDIAC & CARDIOVASCULAR SYSTEMS | REPOLARIZING K+ CURRENTS | CONDUCTION SYSTEM-DEVELOPMENT | INTERVENTRICULAR SEPTUM FORMATION | second heart field | BRUGADA-SYNDROME | ventricular arrhythmias | CONGENITAL HEART-DISEASE | PERIPHERAL VASCULAR DISEASE | HOLT-ORAM-SYNDROME | HEMATOLOGY | cardiac development | TRANSCRIPTION FACTOR | connexin | CARDIAC IMPULSE PROPAGATION | Brugada Syndrome - genetics | Connexins - biosynthesis | Brugada Syndrome - physiopathology | Humans | Heart Ventricles - embryology | Ion Channels - genetics | Arrhythmogenic Right Ventricular Dysplasia - physiopathology | Action Potentials | Arrhythmogenic Right Ventricular Dysplasia - genetics | Heart Conduction System - embryology | Gene Expression Regulation, Developmental | Tachycardia, Ventricular - genetics | Ventricular Fibrillation - genetics | Ventricular Fibrillation - physiopathology | Transcription, Genetic | Ventricular Fibrillation - embryology | Aorta - physiopathology | Fetal Heart - metabolism | Pulmonary Artery - embryology | Neural Crest - cytology | Ion Channels - biosynthesis | Aorta - embryology | Connexins - genetics | Pulmonary Artery - physiopathology | Mammals | Genetic Heterogeneity | Phenotype | Tachycardia, Ventricular - physiopathology | Animals | Gap Junctions - physiology | Heart Conduction System - physiopathology | Heart Ventricles - physiopathology | Myocytes, Cardiac - metabolism | Myocytes, Cardiac - classification | Tachycardia, Ventricular - embryology
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 11/2017, Volume 214, Issue 11, pp. 3347 - 3360
Journal Article