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Science Translational Medicine, ISSN 1946-6234, 12/2015, Volume 7, Issue 318, pp. 318ra200 - 318ra200
Autoantibodies to components of apoptotic cells, such as anti-perlecan antibodies, contribute to rejection in organ transplant recipients. However, mechanisms... 
SOLID-ORGAN TRANSPLANTATION | MEDICINE, RESEARCH & EXPERIMENTAL | ACUTE KIDNEY INJURY | SYSTEMIC-LUPUS-ERYTHEMATOSUS | CORONARY-ARTERY DISEASE | EXTRACELLULAR VESICLES | ANTIVIMENTIN ANTIBODIES | PERLECAN FRAGMENT LG3 | NON-HLA ANTIBODIES | MESENCHYMAL STEM-CELLS | CHRONIC ALLOGRAFT NEPHROPATHY | CELL BIOLOGY | Cell-Derived Microparticles - enzymology | Humans | Muscle, Smooth, Vascular - immunology | Immunity, Humoral | Ischemia - immunology | Time Factors | Peptide Fragments - immunology | Exosomes - immunology | Acute Kidney Injury - immunology | Proteomics - methods | Graft Rejection - pathology | Proteasome Endopeptidase Complex - immunology | Ischemia - pathology | Aorta - transplantation | Disease Models, Animal | Biomarkers - metabolism | Acute Kidney Injury - pathology | Myocytes, Smooth Muscle - enzymology | Rats | Aorta - immunology | Acute Kidney Injury - enzymology | Aorta - pathology | Human Umbilical Vein Endothelial Cells - enzymology | Exosomes - enzymology | Human Umbilical Vein Endothelial Cells - pathology | Mice, Inbred BALB C | Graft Rejection - immunology | Proteasome Endopeptidase Complex - metabolism | Muscle, Smooth, Vascular - enzymology | Ischemia - enzymology | Myocytes, Smooth Muscle - pathology | Human Umbilical Vein Endothelial Cells - immunology | Exosomes - pathology | Autoantibodies - biosynthesis | Allografts | Cell-Derived Microparticles - pathology | Kidney Tubules, Proximal - enzymology | Aorta - enzymology | Graft Rejection - enzymology | Kidney Tubules, Proximal - immunology | Kidney Tubules, Proximal - pathology | Peptide Fragments - metabolism | Mice, Inbred C57BL | Cells, Cultured | Heparan Sulfate Proteoglycans - immunology | Autoantibodies - immunology | Muscle, Smooth, Vascular - pathology | Animals | Apoptosis - immunology | Cell-Derived Microparticles - immunology | Heparan Sulfate Proteoglycans - metabolism | Myocytes, Smooth Muscle - immunology | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 11/2012, Volume 32, Issue 11, pp. 2580 - 2588
OBJECTIVE—Vascular calcification significantly increases cardiovascular morbidity and mortality. We recently reported that the deficiency of cartilage... 
Smooth muscle cell | Calcification | Animal model of human disease remodeling | MicroRNA | Protease | animal model of human disease remodeling | CELLS | smooth muscle cell | PATHOBIOLOGY | MECHANISMS | OLIGOMERIC MATRIX PROTEIN | protease | PHOSPHATE | IN-VITRO | ROLES | ARTERIAL CALCIFICATION | ANEURYSM DEVELOPMENT | microRNA | PERIPHERAL VASCULAR DISEASE | calcification | CHRONIC KIDNEY-DISEASE | HEMATOLOGY | Up-Regulation | Carotid Artery Diseases - chemically induced | Humans | MicroRNAs - metabolism | Matrilin Proteins | Nephrectomy | RNA Interference | Time Factors | Carotid Artery Diseases - pathology | Transcription, Genetic | 3' Untranslated Regions | Real-Time Polymerase Chain Reaction | Organ Culture Techniques | Disease Models, Animal | Uremia - enzymology | Myocytes, Smooth Muscle - enzymology | Down-Regulation | Computational Biology | Rats | Vascular Calcification - pathology | Rats, Sprague-Dawley | Kidney Failure, Chronic - genetics | ADAM Proteins - metabolism | Aortic Diseases - genetics | Carotid Artery Diseases - enzymology | Carotid Artery, Common - enzymology | Carotid Artery Diseases - genetics | Kidney Failure, Chronic - complications | ADAMTS7 Protein | Carotid Artery, Common - pathology | ADAM Proteins - genetics | Muscle, Smooth, Vascular - enzymology | Vascular Calcification - genetics | Glycoproteins - metabolism | Myocytes, Smooth Muscle - pathology | Aortic Diseases - chemically induced | Transfection | Uremia - pathology | Female | Vascular Calcification - enzymology | Aortic Diseases - enzymology | Aorta, Abdominal - pathology | Kidney Failure, Chronic - enzymology | Extracellular Matrix Proteins - metabolism | Aortic Diseases - pathology | Promoter Regions, Genetic | Calcium Chloride | Cartilage Oligomeric Matrix Protein | Cells, Cultured | Aorta, Abdominal - enzymology | Gene Expression Regulation, Enzymologic | Muscle, Smooth, Vascular - pathology | Animals | Vascular Calcification - chemically induced | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 11/2016, Volume 37, Issue 2, pp. 328 - 340
OBJECTIVE—The calcium composition of atherosclerotic plaque is thought to be associated with increased risk for cardiovascular events, but whether plaque... 
atherosclerosis | coronary artery disease | macrophages | inflammation | biomarkers | C-REACTIVE PROTEIN | CORONARY-ARTERY CALCIUM | CARDIOVASCULAR EVENTS | VASCULAR CALCIFICATION | VULNERABLE PATIENT | RISK-ASSESSMENT STRATEGIES | COMPUTED-TOMOGRAPHY | IN-VIVO | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | NF-KAPPA-B | Up-Regulation | Prognosis | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Humans | Myocytes, Smooth Muscle - pathology | rac GTP-Binding Proteins - deficiency | rac GTP-Binding Proteins - metabolism | Male | Coronary Artery Disease - enzymology | Atherosclerosis - enzymology | Transfection | Interleukin-1beta - metabolism | rac GTP-Binding Proteins - genetics | Coronary Artery Disease - pathology | Inflammation Mediators - metabolism | Aortic Diseases - prevention & control | Vascular Calcification - mortality | Female | Vascular Calcification - enzymology | Aortic Diseases - enzymology | Interleukin 1 Receptor Antagonist Protein - pharmacology | Aorta - enzymology | Aortic Diseases - pathology | Atherosclerosis - pathology | Coronary Vessels - pathology | Genetic Predisposition to Disease | Coronary Vessels - enzymology | Macrophages - pathology | Signal Transduction | Myocytes, Smooth Muscle - enzymology | Mice, Inbred C57BL | Cells, Cultured | Neuropeptides - metabolism | Coronary Artery Disease - mortality | Plaque, Atherosclerotic | Vascular Calcification - pathology | Macrophages - enzymology | Mice, Knockout | Aorta - pathology | Aortic Diseases - genetics | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Apolipoproteins E - genetics | Muscle, Smooth, Vascular - enzymology | Atherosclerosis - prevention & control | rac1 GTP-Binding Protein - metabolism | Index Medicus
Journal Article
Journal of the American Heart Association, ISSN 2047-9980, 02/2016, Volume 5, Issue 2, p. n/a
Background-The choline-derived metabolite trimethylamine N-oxide (TMAO) has been demonstrated to contribute to atherosclerosis and is associated with coronary... 
atherosclerosis | cardiovascular disease | inflammation | endothelial cell | nuclear factor‐κB signaling | vascular smooth muscle cell | leukocyte adhesion | trimethylamine N‐oxide | Endothelial cell | Trimethylamine N-oxide | Nuclear factor-κB signaling | Atherosclerosis | Cardiovascular disease | Leukocyte adhesion | Vascular smooth muscle cell | Inflammation | MICROBIAL-METABOLISM | CARDIAC & CARDIOVASCULAR SYSTEMS | HYDROSTATIC-PRESSURE | HEART-FAILURE | ADHESION MOLECULES | trimethylamine N-oxide | HUMAN ENDOTHELIAL-CELLS | L-CARNITINE | LYSOPHOSPHATIDIC ACID | CARDIOVASCULAR-DISEASE | PHOSPHATIDYLCHOLINE | nuclear factor-kappa B signaling | Atherosclerosis - genetics | Coculture Techniques | Humans | Myocytes, Smooth Muscle - pathology | Aortitis - enzymology | NF-kappa B - metabolism | Aortitis - chemically induced | Leukocytes - enzymology | Atherosclerosis - enzymology | Receptors, LDL - deficiency | Female | Aortitis - pathology | Myocytes, Smooth Muscle - drug effects | Aorta - enzymology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Receptors, LDL - genetics | Atherosclerosis - pathology | Genetic Predisposition to Disease | Myocytes, Smooth Muscle - enzymology | Aorta - drug effects | Atherosclerosis - chemically induced | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Aortitis - genetics | Cell Adhesion - drug effects | Mice, Knockout | Aorta - pathology | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Signal Transduction - drug effects | Choline | Leukocytes - drug effects | Enzyme Activation | Methylamines - toxicity | Endothelial Cells - pathology | Endothelial Cells - enzymology | Muscle, Smooth, Vascular - enzymology | Endothelial Cells - drug effects | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
Journal Article
Clinical and Experimental Pharmacology and Physiology, ISSN 0305-1870, 10/2008, Volume 35, Issue 10, pp. 1156 - 1163
1 Fabry disease results from an X‐linked mutation in the lysosomal α‐galactosidase A (Gla) gene. Defective Gla results in multi‐organ accumulation of neutral... 
Fabry disease | endothelium | globotriaosylceramide | α‐galactosidase A | Globotriaosylceramide | α-galactosidase A | Endothelium | PHYSIOLOGY | REACTIVITY | AORTA | FABRY-DISEASE | DEPENDENT CONTRACTIONS | MODEL | SMALL MESENTERIC-ARTERIES | DEFICIENCY | GLYCOSPHINGOLIPIDS | C-GAMMA ACTIVITY | alpha-galactosidase A | PHARMACOLOGY & PHARMACY | MICE | alpha-Galactosidase - genetics | Vascular Diseases - pathology | Blood Pressure - genetics | Fabry Disease - genetics | Male | Fabry Disease - enzymology | alpha-Galactosidase - physiology | Endothelium, Vascular - enzymology | Fabry Disease - pathology | Cell Membrane - pathology | Blood Pressure - physiology | Cell Membrane - metabolism | Aorta, Thoracic - pathology | Disease Models, Animal | Mice, Inbred C57BL | Aorta, Thoracic - metabolism | Vascular Diseases - genetics | Vascular Diseases - enzymology | Mice, Knockout | Aorta, Thoracic - enzymology | Cell Membrane - enzymology | Animals | Endothelial Cells - cytology | Endothelium, Vascular - metabolism | Glycosphingolipids - metabolism | Endothelium, Vascular - pathology | Mice | Endothelial Cells - pathology | In Vitro Techniques | Endothelial Cells - enzymology | Vascular Diseases, pathology | Aorta, Thoracic, pathology | Endothelial Cells, cytology | Endothelium, Vascular, metabolism | Cell Membrane, enzymology | Aorta, Thoracic, metabolism | Endothelial Cells, pathology | alpha-Galactosidase, genetics | Glycosphingolipids, metabolism | Endothelial Cells, enzymology | Endothelium, Vascular, enzymology | Endothelium, Vascular, pathology | Cell Membrane, pathology | Blood Pressure, physiology | Cell Membrane, metabolism | Vascular Diseases, enzymology | Fabry Disease, pathology | Fabry Disease, enzymology | Aorta, Thoracic, enzymology | Blood Pressure, genetics | alpha-Galactosidase, physiology | Vascular Diseases, genetics | Fabry Disease, genetics | Index Medicus
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2015, Volume 35, Issue 3, pp. 547 - 557
OBJECTIVE—Atherosclerosis, an inflammatory disease of arterial vessel walls, requires migration and matrix metalloproteinase (MMP)-9–dependent invasion of... 
Inflammation | Migration and invasion protein | Atherosclerosis | Serum-glucocorticoid regulated kinase | atherosclerosis | migration and invasion protein | APOE-NULL MICE | ATHEROSCLEROTIC PLAQUES | INCREASED EXPRESSION | PATHOGENIC T(H)17 CELLS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | NEOINTIMA FORMATION | MATRIX METALLOPROTEINASE-3 | inflammation | MUSCLE-CELL MIGRATION | PERIPHERAL VASCULAR DISEASE | serum-glucocorticoid regulated kinase | HEMATOLOGY | NF-KAPPA-B | E-DEFICIENT MICE | Inflammation - pathology | Protein-Serine-Threonine Kinases - deficiency | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Humans | Male | Immediate-Early Proteins - metabolism | Matrix Metalloproteinase 9 - metabolism | Carotid Artery Diseases - pathology | Matrix Metalloproteinase 9 - genetics | Peritonitis - genetics | Carotid Arteries - enzymology | Immediate-Early Proteins - deficiency | Transcription, Genetic | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Atherosclerosis - pathology | Macrophages - pathology | NF-kappa B p50 Subunit - metabolism | Signal Transduction | Matrix Metalloproteinase 2 - metabolism | Plaque, Atherosclerotic | Macrophages - enzymology | Mice, Knockout | Aorta - pathology | Aortic Diseases - genetics | Peritonitis - enzymology | Immediate-Early Proteins - genetics | Carotid Artery Diseases - enzymology | Carotid Artery Diseases - genetics | Mutation | Inflammation - enzymology | Vascular Remodeling | I-kappa B Proteins - metabolism | Atherosclerosis - enzymology | Transfection | I-kappa B Kinase - metabolism | Thioglycolates | Aortic Diseases - enzymology | Active Transport, Cell Nucleus | Aorta - enzymology | Aortic Diseases - pathology | Cell Line | Mice, Inbred C57BL | Protein-Serine-Threonine Kinases - genetics | Peritonitis - chemically induced | Chemotaxis | Gene Expression Regulation, Enzymologic | Animals | Apolipoproteins E - genetics | Inflammation - genetics | Carotid Arteries - pathology | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2006, Volume 26, Issue 7, pp. 1579 - 1585
OBJECTIVE—To study the distribution of group V secretory phospholipase A2 (sPLA2) in human and mouse lesions and compare its expression by human vascular... 
Atherogenesis | Phospholipase | Lipoprotein-retention | Inflammation | Proteoglycans | Blood Vessels - pathology | Humans | Phospholipases A - metabolism | Phospholipases A2 | RNA, Messenger - metabolism | Lipopolysaccharide Receptors - analysis | Arteries - metabolism | Atherosclerosis - enzymology | Drug Interactions | Carotid Artery Diseases - pathology | Isoenzymes - metabolism | Blood Vessels - enzymology | Phospholipases A - pharmacology | Aorta - enzymology | Macrophages - immunology | Atherosclerosis - pathology | Isoenzymes - genetics | Blood - drug effects | Enzyme Induction | Proteoglycans - metabolism | Recombinant Proteins - pharmacology | Macrophages - enzymology | Immunohistochemistry - methods | Group II Phospholipases A2 | Animals | Diet | Phospholipases A - genetics | Carotid Artery Diseases - enzymology | Staining and Labeling | Isoenzymes - pharmacology | Mice | Lipoproteins - drug effects | Index Medicus | Macrophages/enzymology/immunology | RNA | Recombinant Proteins/pharmacology | MEDICIN OCH HÄLSOVETENSKAP | Carotid Artery Diseases/enzymology/pathology | Aorta/enzymology | Immunohistochemistry/methods | Blood/drug effects | Blood Vessels/enzymology/pathology | Lipoproteins/drug effects | Phospholipases A/genetics/metabolism/pharmacology | MEDICAL AND HEALTH SCIENCES | Antigens | Proteoglycans/metabolism | Isoenzymes/genetics/metabolism/pharmacology | CD14/analysis | Arteries/metabolism | Messenger/metabolism | Atherosclerosis/enzymology/pathology
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2012, Volume 287, Issue 19, pp. 15966 - 15980
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2017, Volume 37, Issue 4, pp. 694 - 706
OBJECTIVE—Increasing evidence suggests that contractile dysfunction in smooth muscle cells (SMCs) plays a critical role in aortic biomechanical dysfunction and... 
Caspase 1 | Glyburide | Inflammasomes | Contractile proteins | Proteolysis | Aortic aneurysm | OXIDATIVE STRESS | ACTIVATION | glyburide | inflammasomes | FOCUS | INJURY | caspase 1 | DELETION | aortic aneurysm | PERIPHERAL VASCULAR DISEASE | MICE | MUTATIONS | ISCHEMIA-REPERFUSION | HEMATOLOGY | proteolysis | contractile proteins | Aneurysm, Dissecting - genetics | Aortic Aneurysm, Abdominal - prevention & control | Inflammasomes - metabolism | Humans | Middle Aged | Caspase 1 - metabolism | Male | Mice, 129 Strain | Aorta, Abdominal - drug effects | Aneurysm, Dissecting - physiopathology | Glyburide - pharmacology | Inflammasomes - antagonists & inhibitors | Myocytes, Smooth Muscle - drug effects | Aneurysm, Dissecting - prevention & control | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Genetic Predisposition to Disease | Myocytes, Smooth Muscle - enzymology | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Vasoconstriction - drug effects | Aortic Aneurysm, Thoracic - physiopathology | Mice, Knockout | Aorta, Thoracic - enzymology | Biomechanical Phenomena | Phenotype | Aortic Aneurysm, Abdominal - enzymology | Aortic Aneurysm, Abdominal - physiopathology | Caspase 1 - deficiency | Muscle, Smooth, Vascular - enzymology | Aortic Aneurysm, Thoracic - genetics | Aortic Aneurysm, Thoracic - prevention & control | Myocytes, Smooth Muscle - pathology | Muscle, Smooth, Vascular - physiopathology | NLR Family, Pyrin Domain-Containing 3 Protein - genetics | Aneurysm, Dissecting - enzymology | Muscle Proteins - metabolism | Female | Aorta, Abdominal - pathology | Aorta, Thoracic - drug effects | Angiotensin II | Aorta, Abdominal - physiopathology | Aorta, Thoracic - pathology | Aortic Aneurysm, Thoracic - enzymology | Mice, Inbred C57BL | Cells, Cultured | Aortic Aneurysm, Abdominal - genetics | Aorta, Thoracic - physiopathology | Aorta, Abdominal - enzymology | Inflammasomes - genetics | NLR Family, Pyrin Domain-Containing 3 Protein - deficiency | Muscle, Smooth, Vascular - pathology | Animals | Caspase 1 - genetics | Aged | Index Medicus | aneurysm | cardiovascular diseases | contractility | vessels
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2013, Volume 33, Issue 4, pp. 777 - 784
OBJECTIVE—To analyze the role of toll-like receptor 4 in modulating metabolism and endothelial function. APPROACH AND RESULTS—Type 2 diabetic mice with mutated... 
systolic arterial blood pressure | endothelium-derived contracting factor | nitric oxide | proinflammatory cytokines | obesity | OXIDATIVE STRESS | ACTIVATION | MACROPHAGES | VASCULAR SMOOTH-MUSCLE | SYNTHASE | INSULIN-RESISTANCE | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | NADH/NADPH OXIDASE | HYPERTENSION | HEMATOLOGY | INNATE IMMUNITY | Mesenteric Arteries - physiopathology | Oxidative Stress | Receptors, Leptin - genetics | NADPH Oxidases - metabolism | Endothelium, Vascular - drug effects | RNA, Messenger - metabolism | Toll-Like Receptor 4 - deficiency | Carotid Arteries - enzymology | NADPH Oxidases - genetics | Disease Models, Animal | NADH, NADPH Oxidoreductases - genetics | Down-Regulation | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Toll-Like Receptor 4 - genetics | Obesity - physiopathology | Toll-Like Receptor 4 - metabolism | Vasoconstriction - drug effects | Mice, Knockout | Receptors, Leptin - deficiency | 6-Ketoprostaglandin F1 alpha - metabolism | Lipopolysaccharides - pharmacology | Mice | NADH, NADPH Oxidoreductases - antagonists & inhibitors | Mutation | Vasodilation - drug effects | Mesenteric Arteries - enzymology | Muscle, Smooth, Vascular - enzymology | Phosphorylation | Diabetes Mellitus, Type 2 - genetics | Endothelium, Vascular - enzymology | Muscle, Smooth, Vascular - physiopathology | Obesity - genetics | Dose-Response Relationship, Drug | Superoxides - metabolism | Membrane Proteins - metabolism | Aorta - physiopathology | Nitric Oxide Synthase Type III - metabolism | NADH, NADPH Oxidoreductases - metabolism | Aorta - enzymology | Vasodilator Agents - pharmacology | Mice, Inbred C57BL | Diabetes Mellitus, Type 2 - enzymology | Endothelium, Vascular - physiopathology | NADPH Oxidase 4 | Mice, Inbred C3H | NADPH Oxidase 1 | Animals | Diabetes Mellitus, Type 2 - physiopathology | Carotid Arteries - physiopathology | Obesity - enzymology | Cyclooxygenase 1 - metabolism | Nitric Oxide - metabolism | Index Medicus
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 04/2015, Volume 35, Issue 4, pp. 948 - 959
Objective-Activation of liver X receptor (LXR) inhibits atherosclerosis but induces hypertriglyceridemia. In vitro, it has been shown that mitogen-activated... 
atherosclerosis | foam cells | hypertriglyceridemia | lipogenesis | extracellular signal-regulated map kinases | liver X receptor | TRIGLYCERIDE | HOMEOSTASIS | PROTEIN-KINASE | CELL ACTIVATION | REVERSE CHOLESTEROL TRANSPORT | DISEASE | PERIPHERAL VASCULAR DISEASE | LIGAND | HEMATOLOGY | EXPRESSION | LIVER-X-RECEPTOR | Apolipoproteins E - deficiency | Fatty Liver - pathology | Atherosclerosis - genetics | Humans | Male | Fatty Liver - chemically induced | Orphan Nuclear Receptors - metabolism | Liver - drug effects | Fatty Liver - enzymology | Aortic Diseases - prevention & control | Foam Cells - pathology | Disease Models, Animal | Chemical and Drug Induced Liver Injury - prevention & control | Atherosclerosis - pathology | Butadienes - pharmacology | Liver - metabolism | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Orphan Nuclear Receptors - agonists | Fatty Liver - prevention & control | Foam Cells - enzymology | Sulfonamides - pharmacology | Mice, Knockout | Aorta - pathology | Drug Synergism | Hydrocarbons, Fluorinated - toxicity | Aortic Diseases - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | Nitriles - pharmacology | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Hydrocarbons, Fluorinated - pharmacology | Hypertriglyceridemia - pathology | Foam Cells - drug effects | Atherosclerosis - enzymology | Liver X Receptors | Female | Aortic Diseases - enzymology | Chemical and Drug Induced Liver Injury - pathology | Drug Therapy, Combination | Aorta - enzymology | Chemical and Drug Induced Liver Injury - enzymology | Aortic Diseases - pathology | Aorta - drug effects | Mice, Inbred C57BL | Cholesterol - metabolism | Hep G2 Cells | Hypertriglyceridemia - enzymology | Hypertriglyceridemia - prevention & control | Hypertriglyceridemia - chemically induced | Animals | Apolipoproteins E - genetics | Sulfonamides - toxicity | Protein Kinase Inhibitors - pharmacology | Atherosclerosis - prevention & control | Index Medicus
Journal Article