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American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2007, Volume 293, Issue 4, pp. 2210 - 2218
Targeting cannabinoid-2 (CB2) receptors with selective agonists may represent a novel therapeutic avenue in various inflammatory diseases, but the mechanisms... 
Adhesion molecules | Inflammation | RhoA | Endothelial activation | C-REACTIVE PROTEIN | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | MOLECULE EXPRESSION | ATHEROSCLEROSIS | NECROSIS-FACTOR-ALPHA | KNOCKOUT MICE | endothelial activation | CANNABINOID RECEPTORS | inflammation | adhesion molecules | NITRIC-OXIDE | THERAPEUTIC TARGETS | PERIPHERAL VASCULAR DISEASE | POTENTIAL ROLE | CB2 RECEPTOR ACTIVATION | Inflammation - chemically induced | Leukocyte Rolling - drug effects | Tumor Necrosis Factor-alpha - metabolism | Receptor, Cannabinoid, CB2 - agonists | Humans | Male | Monocytes - metabolism | NF-kappa B - metabolism | rhoA GTP-Binding Protein - metabolism | Aorta - metabolism | RNA, Messenger - metabolism | Receptor, Cannabinoid, CB2 - genetics | Coronary Vessels - metabolism | Lipopolysaccharides | Dose-Response Relationship, Drug | Inflammation - metabolism | Anti-Inflammatory Agents - therapeutic use | Chemokine CCL2 - metabolism | Disease Models, Animal | Coronary Vessels - drug effects | Anti-Inflammatory Agents - pharmacology | Endothelial Cells - metabolism | Aorta - drug effects | Mice, Inbred C57BL | Cells, Cultured | Cannabinoids - pharmacology | Monocytes - drug effects | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Cannabinoids - therapeutic use | Intercellular Adhesion Molecule-1 - metabolism | Animals | Signal Transduction - drug effects | Inflammation - prevention & control | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Endothelial Cells - drug effects
Journal Article
Journal of Vascular Surgery, ISSN 0741-5214, 2015, Volume 64, Issue 5, pp. 1444 - 1449
Journal Article
The FASEB journal, ISSN 0892-6638, 11/2005, Volume 19, Issue 13, pp. 1890 - 1892
ABSTRACTWe investigated a possible beneficial role for bilirubin, one of the products of heme degradation by the cytoprotective enzyme heme oxygenase‐1 in... 
nitric oxide | oxygen free radicals | OXIDATIVE STRESS | SUPEROXIDE | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | INDUCTION | BILIVERDIN | CELL BIOLOGY | SYNTHASE EXPRESSION | ANTIOXIDANT | IN-VIVO | BIOLOGY | HEME OXYGENASE-1 | SERUM BILIRUBIN | Nitric Oxide Synthase Type II - biosynthesis | Bilirubin - metabolism | Antioxidants - metabolism | Kidney - enzymology | Malondialdehyde - chemistry | NADPH Oxidases - metabolism | Male | Aorta - metabolism | Kidney - metabolism | Time Factors | Myocardium - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Interleukin-6 - metabolism | Glutathione Peroxidase - metabolism | Free Radicals | Down-Regulation | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Rats | Rats, Sprague-Dawley | Blotting, Western | Nitrates - metabolism | Macrophages - metabolism | Rats, Gunn | Models, Biological | Lipopolysaccharides - chemistry | Mice | Nitric Oxide Synthase - metabolism | Blood Pressure | Jaundice - pathology | Tumor Necrosis Factor-alpha - metabolism | Nitric Oxide Synthase Type II - chemistry | Nitrites - metabolism | Shock, Septic - prevention & control | Oxygen - metabolism | Shock, Septic - metabolism | Heme - chemistry | Escherichia coli - metabolism | Shock, Septic - drug therapy | Interleukin-10 - metabolism | Aorta - enzymology | Superoxide Dismutase - metabolism | Cell Line | Bilirubin - chemistry | Models, Statistical | Homozygote | Myocardium - enzymology | Animals | Nitric Oxide - metabolism
Journal Article
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 12/2015, Volume 35, Issue 12, pp. 2605 - 2616
OBJECTIVE—Kawasaki disease (KD) is the most common cause of acute vasculitis and acquired cardiac disease among US children. We have previously shown that both... 
dendritic cells | interleukin-1 | mucocutaneous lymph node syndrome | MyD88 | Endothelial cells | protein | endothelial cells | ELEMENT-BINDING PROTEIN-2 | ACTIVATION | RECEPTOR | CORONARY-ARTERY ANEURYSMS | NLRP3 INFLAMMASOME | POLYMORPHISM | ACUTE-PHASE | INTRAVENOUS IMMUNOGLOBULIN | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | ASSOCIATION | Lactobacillus casei | Mucocutaneous Lymph Node Syndrome - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Stromal Cells - pathology | Caspase 1 - metabolism | Interleukin-1alpha - metabolism | Aortitis - chemically induced | Aorta - metabolism | Coronary Vessels - metabolism | Cell Wall | DNA-Binding Proteins - metabolism | Receptors, Interleukin-1 Type I - metabolism | Interleukin-1beta - metabolism | Bone Marrow Transplantation | Coronary Artery Disease - pathology | Aortitis - pathology | Dendritic Cells - metabolism | Disease Models, Animal | Mucocutaneous Lymph Node Syndrome - pathology | Coronary Vessels - pathology | Signal Transduction | Coronary Artery Disease - chemically induced | Endothelial Cells - metabolism | Mucocutaneous Lymph Node Syndrome - chemically induced | Coronary Artery Disease - metabolism | Mice, Inbred C57BL | Stromal Cells - metabolism | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Aortitis - genetics | DNA-Binding Proteins - genetics | Transplantation Chimera | Mice, Knockout | Aorta - pathology | Mucocutaneous Lymph Node Syndrome - genetics | Aortitis - metabolism | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Coronary Artery Disease - genetics | Receptors, Interleukin-1 Type I - genetics | Myeloid Differentiation Factor 88 - metabolism | Bone Marrow Cells - metabolism | coronary artery vasculitis | Kawasaki disease | IL-1
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2007, Volume 27, Issue 12, pp. 2589 - 2596
OBJECTIVE—Aortic calcification is prevalent in type II diabetes (T2DM), enhancing morbidity and tracking metabolic syndrome parameters. Ldlr mice fed high-fat... 
Aortic calcification | Diabetes | TNF-α | Metabolic syndrome | Wnt | TNF-alpha | BONE MORPHOGENETIC PROTEIN-2 | MATRIX GLA PROTEIN | VASCULAR CALCIFICATION | aortic calcification | DEFICIENT MICE | metabolic syndrome | INFLAMMATION | IN-VIVO | ARTERIAL CALCIFICATION | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | DIFFERENTIATION | HEMATOLOGY | diabetes | EXPRESSION | Inflammation - pathology | Fibroblasts - enzymology | Homeodomain Proteins - metabolism | Tumor Necrosis Factor-alpha - blood | Tumor Necrosis Factor-alpha - genetics | Antibodies, Monoclonal - therapeutic use | Male | Aorta - metabolism | Haptoglobins - metabolism | RNA, Messenger - metabolism | Wnt Proteins - metabolism | Aortic Diseases - metabolism | Bone Morphogenetic Proteins - metabolism | Inflammation - metabolism | Calcinosis - etiology | Dietary Fats - administration & dosage | Anti-Inflammatory Agents - therapeutic use | Aortic Diseases - prevention & control | Disease Models, Animal | Fibroblasts - metabolism | Anti-Inflammatory Agents - pharmacology | Antibodies, Monoclonal - pharmacology | Receptors, LDL - metabolism | Mice, Transgenic | Mice, Knockout | Aorta - pathology | Muscle Proteins - genetics | Signal Transduction - drug effects | Wnt3 Protein | Calcinosis - prevention & control | Inflammation - prevention & control | Mice | Diabetes Mellitus, Type 2 - pathology | Calcinosis - pathology | Wnt3A Protein | Tumor Necrosis Factor-alpha - metabolism | Alkaline Phosphatase | Diabetes Mellitus, Type 2 - metabolism | DNA-Binding Proteins - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Wnt Proteins - genetics | Infliximab | Receptors, LDL - deficiency | Calcinosis - metabolism | Microfilament Proteins - genetics | Aorta - enzymology | Diabetes Mellitus, Type 2 - complications | Aortic Diseases - pathology | Promoter Regions, Genetic | Receptors, LDL - genetics | Bone Morphogenetic Protein 2 | Mice, Inbred C57BL | Cells, Cultured | DNA-Binding Proteins - genetics | Homeodomain Proteins - genetics | Animals | Diabetes Mellitus, Type 2 - chemically induced | Transforming Growth Factor beta - metabolism | Aortic Diseases - etiology | Tumor Necrosis Factor-alpha - antagonists & inhibitors
Journal Article
Atherosclerosis, ISSN 0021-9150, 2015, Volume 239, Issue 2, pp. 393 - 400
Abstract Objective The pathogenic events responsible for accelerated atherosclerosis in patients with chronic renal failure (CRF) are poorly understood. Here... 
Cardiovascular | MnTBAP | chronic renal failure | UCP-1 | Endothelial cells | uncoupling protein 1 | Urea | O-linked-N-acetylglucosamine | manganese tetrakis (4-benzoic acid) porphyrin | CRF | ROS | reactive oxygen species | GlcNAc | Prostacyclin synthase | Reactive oxygen species | Chronic renal failure | Manganese tetrakis (4-benzoic acid) porphyrin | Uncoupling protein 1 | OXIDATIVE STRESS | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PREDICTOR | ATHEROSCLEROSIS | INSULIN-RESISTANCE | CARDIOVASCULAR-DISEASE | GENE-EXPRESSION | HIGH GLUCOSE | PERIPHERAL VASCULAR DISEASE | CHRONIC KIDNEY-DISEASE | Tumor Necrosis Factor-alpha - metabolism | Reactive Oxygen Species - metabolism | Oxidative Stress | Humans | Cytochrome P-450 Enzyme System - metabolism | Endothelium, Vascular - drug effects | Aorta - metabolism | Urea - chemistry | Antigens, CD - metabolism | Endoglin | Atherosclerosis - enzymology | Kidney Failure, Chronic - chemically induced | Chemokine CCL2 - metabolism | Endothelium - enzymology | Intramolecular Oxidoreductases - metabolism | Interleukin-6 - metabolism | Superoxide Dismutase - metabolism | Atherosclerosis - physiopathology | Endothelial Cells - metabolism | Mice, Inbred C57BL | Receptors, Cell Surface - metabolism | Random Allocation | Atherosclerosis - metabolism | Kidney Failure, Chronic - metabolism | Catalase - metabolism | Glyceraldehyde-3-Phosphate Dehydrogenase (Phosphorylating) - metabolism | Animals | Endothelium, Vascular - pathology | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Chronic kidney failure | Atherosclerosis | Endothelium
Journal Article
PloS one, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e68197
Free fatty acids (FFAs), elevated in metabolic syndrome and diabetes, play a crucial role in the development of atherosclerotic cardiovascular disease, and... 
SUPPLEMENTATION | ARTERY ENDOTHELIAL-CELLS | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | DISEASE | CALCIUM | CALCIFICATION | RISK | FREE FATTY-ACID | DEPENDENT ACTIVATION | EXPRESSION | Calcinosis - genetics | Carotid Arteries - metabolism | Osteopontin - genetics | Palmitic Acid - metabolism | Coenzyme A Ligases - genetics | Muscle, Smooth, Vascular - metabolism | Calcium - metabolism | Gene Expression - genetics | Homeodomain Proteins - metabolism | Humans | Eicosapentaenoic Acid - genetics | Myocytes, Smooth Muscle - pathology | NF-kappa B - metabolism | Osteopontin - metabolism | Aorta - metabolism | Promoter Regions, Genetic - genetics | Coenzyme A Ligases - metabolism | Cell Differentiation - genetics | Caspases - metabolism | Bone Morphogenetic Protein 2 - metabolism | Calcinosis - metabolism | Plaque, Atherosclerotic - pathology | Cell Differentiation - physiology | Myocytes, Smooth Muscle - metabolism | Bone Morphogenetic Protein 2 - genetics | Macrophages - pathology | Plaque, Atherosclerotic - metabolism | Caspases - genetics | Cells, Cultured | Eicosapentaenoic Acid - metabolism | Homeodomain Proteins - genetics | Aorta - pathology | Osteoblasts - pathology | Macrophages - metabolism | Muscle, Smooth, Vascular - pathology | NF-kappa B - genetics | Carotid Arteries - pathology | Calcinosis - pathology | Osteoblasts - metabolism | Immunohistochemistry | Smooth muscle | Cardiovascular disease | Palmitic acid | Osteoblasts | Proteins | Mineralization | Biocompatibility | Aorta | Inhibition | NF-κB protein | Cytokines | Osteopontin | Metabolism | Gene expression | Fatty acids | Studies | Osteoblastogenesis | Inhibitors | Arteriosclerosis | Calcification | Cardiovascular diseases | Metabolic disorders | Calcification (ectopic) | Health sciences | Oxidative stress | Calcium | Laboratories | Chains | Lipids | Activation | Kinases | Macrophages | Carotid arteries | Arteries | Msx2 protein | Rodents | Atherosclerosis | Tumor necrosis factor-TNF | Carotid artery | Deposition | Plaques | University graduates | Bone morphogenetic protein 2 | Diabetes mellitus | Muscles | Caspase | siRNA | Medicine | Antibiotics | Kidney diseases | Diabetes | Differentiation | Eicosapentaenoic acid | Apoptosis | Pharmaceuticals
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 1522-1539, 2009, Volume 296, Issue 2, pp. H272 - H281
The farnesoid X receptor (FXR) is a member of the nuclear receptor superfamily that functions as an endogenous sensor for bile acids and regulates cholesterol and fatty acid metabolism... 
Nuclear receptor | Bile acids | Macrophages | Liver | ATHEROSCLEROTIC LESIONS | BILE-ACID | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | LIPID-METABOLISM | TRIGLYCERIDE LEVELS | liver | CELLULAR CHOLESTEROL | NUCLEAR RECEPTORS | SCAVENGER RECEPTORS | TANGIER-DISEASE | macrophages | bile acids | GENE-EXPRESSION | nuclear receptor | PERIPHERAL VASCULAR DISEASE | MACROPHAGE DIFFERENTIATION | Liver - pathology | ATP Binding Cassette Transporter, Sub-Family G, Member 1 | Apolipoproteins E - deficiency | Humans | Male | Aorta - metabolism | PPAR gamma - metabolism | RNA, Messenger - metabolism | Liver - drug effects | Interleukin-1beta - metabolism | Thiazolidinediones - pharmacology | Interleukin-6 - metabolism | Disease Models, Animal | Hyperlipidemias - immunology | Atherosclerosis - pathology | Atherosclerosis - immunology | Hyperlipidemias - metabolism | Liver - metabolism | Aorta - immunology | Receptors, Cytoplasmic and Nuclear - genetics | Toll-Like Receptor 4 - metabolism | Atherosclerosis - metabolism | Chenodeoxycholic Acid - analogs & derivatives | Cardiovascular Agents - pharmacology | Mice, Knockout | Aorta - pathology | Macrophages - metabolism | Hyperlipidemias - pathology | Ligands | Mice | Transcription Factors - agonists | Receptors, Cytoplasmic and Nuclear - metabolism | Tumor Necrosis Factor-alpha - metabolism | Receptors, Cytoplasmic and Nuclear - deficiency | Transcription Factors - deficiency | DNA-Binding Proteins - deficiency | CD36 Antigens - metabolism | DNA-Binding Proteins - agonists | Lipids - blood | ATP-Binding Cassette Transporters - metabolism | Chenodeoxycholic Acid - pharmacology | Female | ATP Binding Cassette Transporter 1 | Macrophages - immunology | Sterol Regulatory Element Binding Protein 1 - metabolism | Aorta - drug effects | Hyperlipidemias - drug therapy | Mice, Inbred C57BL | Receptors, Cytoplasmic and Nuclear - agonists | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Animals | Apolipoproteins E - genetics | PPAR gamma - agonists | Macrophages - drug effects | CD11b Antigen - metabolism | Atherosclerosis - prevention & control
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2015, Volume 35, Issue 2, pp. 399 - 408
OBJECTIVE—Vascular and valvular calcifications are pathological processes regulated by resident cells, and depending on a complex interplay between calcification promoters and inhibitors, resembling skeletal metabolism... 
multivesicular bodies | aortic valve, calcification of | gene expression | vascular calcification | aortic valve | calcification of | AORTIC-STENOSIS | MATRIX VESICLES | VALVE | CARTILAGE | DISEASE | VITAMIN-K | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | FETUIN-A | INSIGHTS | Calcinosis - genetics | Aortic Valve Stenosis - genetics | Calcium - metabolism | Humans | Middle Aged | Vascular Calcification - genetics | Actins - metabolism | Osteocalcin - genetics | Male | Aorta - metabolism | Case-Control Studies | Aortic Valve - pathology | Coronary Vessels - metabolism | Aortic Valve Stenosis - prevention & control | Vascular Calcification - metabolism | Coronary Artery Disease - pathology | Aged, 80 and over | Adult | Female | Calcinosis - metabolism | Extracellular Matrix Proteins - metabolism | Calcium-Binding Proteins - metabolism | Coronary Vessels - pathology | Osteocalcin - metabolism | Tissue Culture Techniques | Coronary Artery Disease - metabolism | Extracellular Matrix Proteins - genetics | Coronary Artery Disease - prevention & control | Gene Expression Regulation | Vascular Calcification - prevention & control | Vascular Calcification - pathology | Aortic Valve - metabolism | Aorta - pathology | Proteins - genetics | Aortic Valve Stenosis - pathology | Proteins - metabolism | Coronary Artery Disease - genetics | alpha-2-HS-Glycoprotein - metabolism | Aortic Valve Stenosis - metabolism | Aged | Calcinosis - prevention & control | Calcinosis - pathology | Calcium-Binding Proteins - genetics
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 12/2016, Volume 311, Issue 6, pp. H1360 - H1366
... (K/X), and low-or high-dose isoflurane (ISO)] on hemodynamics, cardiac function, and glucose and lipid metabolism in Sprague-Dawley rats... 
Ketamine-xylazine | Pentobarbital sodium | Isoflurane | pentobarbital sodium | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ketamine-xylazine | MECHANISMS | BODY-TEMPERATURE | COMBINATION | BLOOD-PRESSURE | INSULIN | IMPACT | PERIPHERAL VASCULAR DISEASE | MICE | isoflurane | STRESS | HYPERTENSIVE-RATS | Renin - metabolism | Anesthetics, Dissociative - pharmacology | Isoflurane - pharmacology | Pentobarbital - pharmacology | Male | Fatty Acids, Nonesterified - metabolism | Heart Rate - drug effects | Aldosterone - metabolism | Xylazine - pharmacology | Renin - drug effects | Blood Pressure - drug effects | Dopamine - metabolism | Anesthetics, Inhalation - pharmacology | Angiotensin II - drug effects | Echocardiography | Angiotensin II - metabolism | Cholesterol, HDL - drug effects | Cholesterol, HDL - metabolism | Aorta - drug effects | Insulin Resistance | Rats | Cholesterol, LDL - drug effects | Cholesterol - metabolism | Rats, Sprague-Dawley | Triglycerides - metabolism | Blood Glucose - drug effects | Insulin - metabolism | Animals | Norepinephrine - metabolism | Lipid Metabolism - drug effects | Cholesterol, LDL - metabolism | Glucose - metabolism | Renin-Angiotensin System - drug effects | Hemodynamics - drug effects | Hypnotics and Sedatives - pharmacology | Blood Glucose - metabolism | Ketamine - pharmacology | Epinephrine - metabolism
Journal Article
Atherosclerosis, ISSN 0021-9150, 2012, Volume 224, Issue 2, pp. 340 - 347
Abstract Objective The objective of this study is to develop a method for selective detection of the calcific (hydroxyapatite) component in human aortic smooth... 
Cardiovascular | Molecular imaging | Calcification | Osteocalcin | Hydroxyapatite | Atherosclerosis | ATHEROSCLEROTIC LESIONS | CARDIAC & CARDIOVASCULAR SYSTEMS | VASCULAR CALCIFICATION | DEFICIENT MICE | STEM-CELL DIFFERENTIATION | IN-VIVO | ARTERIAL CALCIFICATION | MINERALIZATION | PERIPHERAL VASCULAR DISEASE | BONE | ENERGY-METABOLISM | Carotid Arteries - metabolism | Heart Valve Diseases - pathology | Muscle, Smooth, Vascular - metabolism | Humans | Fluorescein-5-isothiocyanate - metabolism | Fluorescent Dyes - metabolism | Calcinosis - diagnostic imaging | Carbocyanines - metabolism | Aorta - metabolism | X-Ray Microtomography | Aortic Valve - pathology | Durapatite - metabolism | Vascular Calcification - metabolism | Molecular Probes - metabolism | Time Factors | Calcinosis - metabolism | Biomarkers - metabolism | Atherosclerosis - pathology | Aortic Valve - diagnostic imaging | Osteocalcin - metabolism | Cells, Cultured | Molecular Imaging - methods | Oligopeptides - metabolism | Plaque, Atherosclerotic | Vascular Calcification - pathology | Aortic Valve - metabolism | Atherosclerosis - metabolism | Aorta - pathology | Muscle, Smooth, Vascular - pathology | Heart Valve Diseases - metabolism | Protein Binding | Carotid Arteries - pathology | Kinetics | Magnetic Fields | Calcinosis - pathology | Microscopy, Fluorescence | Osteogenesis | Heart Valve Diseases - diagnostic imaging | Hydroxylapatite | atherosclerosis | hydroxyapatite | osteocalcin | calcification | molecular imaging
Journal Article