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American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2007, Volume 293, Issue 4, pp. 2210 - 2218
Targeting cannabinoid-2 (CB2) receptors with selective agonists may represent a novel therapeutic avenue in various inflammatory diseases, but the mechanisms... 
Adhesion molecules | Inflammation | RhoA | Endothelial activation | C-REACTIVE PROTEIN | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | MOLECULE EXPRESSION | ATHEROSCLEROSIS | NECROSIS-FACTOR-ALPHA | KNOCKOUT MICE | endothelial activation | CANNABINOID RECEPTORS | inflammation | adhesion molecules | NITRIC-OXIDE | THERAPEUTIC TARGETS | PERIPHERAL VASCULAR DISEASE | POTENTIAL ROLE | CB2 RECEPTOR ACTIVATION | Inflammation - chemically induced | Leukocyte Rolling - drug effects | Tumor Necrosis Factor-alpha - metabolism | Receptor, Cannabinoid, CB2 - agonists | Humans | Male | Monocytes - metabolism | NF-kappa B - metabolism | rhoA GTP-Binding Protein - metabolism | Aorta - metabolism | RNA, Messenger - metabolism | Receptor, Cannabinoid, CB2 - genetics | Coronary Vessels - metabolism | Lipopolysaccharides | Dose-Response Relationship, Drug | Inflammation - metabolism | Anti-Inflammatory Agents - therapeutic use | Chemokine CCL2 - metabolism | Disease Models, Animal | Coronary Vessels - drug effects | Anti-Inflammatory Agents - pharmacology | Endothelial Cells - metabolism | Aorta - drug effects | Mice, Inbred C57BL | Cells, Cultured | Cannabinoids - pharmacology | Monocytes - drug effects | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Cannabinoids - therapeutic use | Intercellular Adhesion Molecule-1 - metabolism | Animals | Signal Transduction - drug effects | Inflammation - prevention & control | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Endothelial Cells - drug effects | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2015, Volume 35, Issue 4, pp. 960 - 972
Objective-Marfan's syndrome is characterized by the formation of ascending aortic aneurysms resulting from altered assembly of extracellular matrix... 
focal adhesion | actin | aortic stiffness | myocardin | TGF-β | aortic aneurysms | extracellular matrix | RhoA | TRANSCRIPTION FACTORS | SMAD2 PATHWAY | EPIGENETIC CONTROL | THORACIC AORTIC-ANEURYSMS | SIGNAL-TRANSDUCTION | TGF-beta | GROWTH-FACTOR-BETA | MOUSE MODEL | DISEASE | PERIPHERAL VASCULAR DISEASE | EXTRACELLULAR-MATRIX | DIFFERENTIATION | HEMATOLOGY | Vascular Remodeling | Aortic Aneurysm - metabolism | Muscle, Smooth, Vascular - metabolism | Humans | Actins - metabolism | Myocytes, Smooth Muscle - pathology | rhoA GTP-Binding Protein - metabolism | Aorta - metabolism | Aortic Aneurysm - etiology | Case-Control Studies | Muscle Proteins - metabolism | Cytoskeletal Proteins - metabolism | Cell Differentiation | Marfan Syndrome - complications | Microfilament Proteins - metabolism | Aortic Aneurysm - pathology | Dilatation, Pathologic | Myocytes, Smooth Muscle - metabolism | Biomarkers - metabolism | Calcium-Binding Proteins - metabolism | Collagen Type I - metabolism | Signal Transduction | Nuclear Proteins - metabolism | Aorta - pathology | Muscle, Smooth, Vascular - pathology | Phenotype | Stress Fibers - metabolism | Focal Adhesions - metabolism | Cell Line, Tumor | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Trans-Activators - metabolism | Transforming Growth Factor beta - metabolism | Index Medicus | Biological Sciences | Medical and Health Sciences | Medicin och hälsovetenskap | Naturvetenskap | Biologiska vetenskaper | Natural Sciences
Journal Article
Nature, ISSN 0028-0836, 10/2010, Volume 467, Issue 7318, pp. 972 - 976
Reciprocity of inflammation, oxidative stress and neovascularization is emerging as an important mechanism underlying numerous processes from tissue healing... 
SYSTEM | TOLL-LIKE RECEPTOR-2 | CELLS | PATTERN-RECOGNITION | MULTIDISCIPLINARY SCIENCES | MACULAR DEGENERATION | CD36 | CHRONIC INFLAMMATION | CANCER | INNATE IMMUNITY | Neovascularization, Physiologic - drug effects | Toll-Like Receptor 2 - agonists | Melanoma - blood supply | Humans | Oxidative Stress - physiology | Vascular Endothelial Growth Factor A - metabolism | Inflammation - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Wound Healing - drug effects | Receptors, Scavenger - metabolism | Melanoma - metabolism | Cell Line | Pyrroles - metabolism | Oxidation-Reduction | Endothelial Cells - metabolism | Aorta - drug effects | Mice, Inbred C57BL | Ischemia - metabolism | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Immunity, Innate - immunology | Pyrroles - pharmacology | Animals | Signal Transduction - drug effects | Propionates | Pyrroles - chemistry | Ligands | Mice | Neovascularization, Pathologic - metabolism | Wound Healing - physiology | Aorta - cytology | Hindlimb - metabolism | Myeloid Differentiation Factor 88 - metabolism | rac1 GTP-Binding Protein - metabolism | Aging - metabolism | Cell Movement | Oxidative stress | Complications and side effects | Cell receptors | Physiological aspects | Genetic aspects | Research | Neovascularization | Vascular endothelial growth factor | Angiogenesis | Wound healing | Rodents | Atherosclerosis | Index Medicus
Journal Article
FASEB Journal, ISSN 0892-6638, 2005, Volume 19, Issue 13, pp. 1890 - 1892
Journal Article
Journal Article
Science, ISSN 0036-8075, 11/2004, Volume 306, Issue 5701, pp. 1558 - 1561
In vitro studies suggest a role for c-Jun N-terminal kinases (JNKs) in proatherogenic cellular processes. We show that atherosclerosis-prone ApoE mice... 
T lymphocytes | Phosphorylation | Lipoproteins | Atherosclerosis | Aorta | Lipids | Reports | Mice | Macrophages | Foam cells | Endothelial cells | ADHESION | ACTIVATION | LDL | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | ATHEROSCLEROSIS | DEFICIENT MICE | DIFFERENTIATION | EXPRESSION | VASCULAR PROLIFERATION | Scavenger Receptors, Class A | Cholesterol, Dietary - administration & dosage | Mitogen-Activated Protein Kinase 9 - genetics | CD36 Antigens - metabolism | Bone Marrow Transplantation | Lipoproteins, LDL - metabolism | Endothelial Cells - physiology | Foam Cells - metabolism | Mitogen-Activated Protein Kinase 9 - metabolism | Macrophages, Peritoneal - physiology | Mice, Inbred C57BL | Mitogen-Activated Protein Kinase 8 - metabolism | Cells, Cultured | Myocytes, Smooth Muscle - physiology | Cholesterol - metabolism | Muscle, Smooth, Vascular - cytology | Mice, Knockout | Aorta - pathology | Diet, Atherogenic | Macrophages - metabolism | Animals | Receptors, Scavenger | Apolipoproteins E - genetics | Aorta - chemistry | T-Lymphocytes - immunology | Arteriosclerosis - metabolism | Receptors, Immunologic - genetics | Arteriosclerosis - pathology | Receptors, Immunologic - metabolism | Genetic aspects | Research | Proteins | Pathology | Enzymes | Biochemistry | Cellular biology | Index Medicus | Apolipoproteins E/genetics | Mitogen-Activated Protein Kinase 9/genetics/metabolism | Vascular/cytology | Endothelial Cells/physiology | Scavenger Receptors | Arteriosclerosis/metabolism/pathology | Macrophages/metabolism | MEDICIN OCH HÄLSOVETENSKAP | Myocytes | Receptors | Mitogen-Activated Protein Kinase 8/metabolism | Smooth | Scavenger | Cultured | Muscle | MEDICAL AND HEALTH SCIENCES | Dietary/administration & dosage | Class A | Antigens | T-Lymphocytes/immunology | Cholesterol/metabolism | Peritoneal/physiology | Foam Cells/metabolism | Aorta/chemistry/pathology | CD36/metabolism | Cells | Cholesterol | Inbred C57BL | Knockout | Immunologic/genetics/metabolism | Diet | Smooth Muscle/physiology | LDL/metabolism | Atherogenic
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2007, Volume 27, Issue 12, pp. 2589 - 2596
OBJECTIVE—Aortic calcification is prevalent in type II diabetes (T2DM), enhancing morbidity and tracking metabolic syndrome parameters. Ldlr mice fed high-fat... 
Aortic calcification | Diabetes | TNF-α | Metabolic syndrome | Wnt | TNF-alpha | BONE MORPHOGENETIC PROTEIN-2 | MATRIX GLA PROTEIN | VASCULAR CALCIFICATION | aortic calcification | DEFICIENT MICE | metabolic syndrome | INFLAMMATION | IN-VIVO | ARTERIAL CALCIFICATION | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | DIFFERENTIATION | HEMATOLOGY | diabetes | EXPRESSION | Inflammation - pathology | Fibroblasts - enzymology | Homeodomain Proteins - metabolism | Tumor Necrosis Factor-alpha - blood | Tumor Necrosis Factor-alpha - genetics | Antibodies, Monoclonal - therapeutic use | Male | Aorta - metabolism | Haptoglobins - metabolism | RNA, Messenger - metabolism | Wnt Proteins - metabolism | Aortic Diseases - metabolism | Bone Morphogenetic Proteins - metabolism | Inflammation - metabolism | Calcinosis - etiology | Dietary Fats - administration & dosage | Anti-Inflammatory Agents - therapeutic use | Aortic Diseases - prevention & control | Disease Models, Animal | Fibroblasts - metabolism | Anti-Inflammatory Agents - pharmacology | Antibodies, Monoclonal - pharmacology | Receptors, LDL - metabolism | Mice, Transgenic | Mice, Knockout | Aorta - pathology | Muscle Proteins - genetics | Signal Transduction - drug effects | Wnt3 Protein | Calcinosis - prevention & control | Inflammation - prevention & control | Mice | Diabetes Mellitus, Type 2 - pathology | Calcinosis - pathology | Wnt3A Protein | Tumor Necrosis Factor-alpha - metabolism | Alkaline Phosphatase | Diabetes Mellitus, Type 2 - metabolism | DNA-Binding Proteins - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Wnt Proteins - genetics | Infliximab | Receptors, LDL - deficiency | Calcinosis - metabolism | Microfilament Proteins - genetics | Aorta - enzymology | Diabetes Mellitus, Type 2 - complications | Aortic Diseases - pathology | Promoter Regions, Genetic | Receptors, LDL - genetics | Bone Morphogenetic Protein 2 | Mice, Inbred C57BL | Cells, Cultured | DNA-Binding Proteins - genetics | Homeodomain Proteins - genetics | Animals | Diabetes Mellitus, Type 2 - chemically induced | Transforming Growth Factor beta - metabolism | Aortic Diseases - etiology | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 02/2009, Volume 296, Issue 2, pp. 272 - 281
The farnesoid X receptor (FXR) is a member of the nuclear receptor superfamily that functions as an endogenous sensor for bile acids and regulates cholesterol... 
Nuclear receptor | Bile acids | Macrophages | Liver | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | LIPID-METABOLISM | liver | DEFICIENT MICE | ACTIVATED RECEPTOR | TANGIER-DISEASE | macrophages | bile acids | CASSETTE TRANSPORTER 1 | FOAM-CELL-FORMATION | GENE-EXPRESSION | nuclear receptor | PERIPHERAL VASCULAR DISEASE | BILE-ACID BIOSYNTHESIS | MACROPHAGE DIFFERENTIATION | Liver - pathology | ATP Binding Cassette Transporter, Sub-Family G, Member 1 | Apolipoproteins E - deficiency | Humans | Male | Aorta - metabolism | PPAR gamma - metabolism | RNA, Messenger - metabolism | Liver - drug effects | Interleukin-1beta - metabolism | Thiazolidinediones - pharmacology | Interleukin-6 - metabolism | Disease Models, Animal | Hyperlipidemias - immunology | Atherosclerosis - pathology | Atherosclerosis - immunology | Hyperlipidemias - metabolism | Liver - metabolism | Aorta - immunology | Receptors, Cytoplasmic and Nuclear - genetics | Toll-Like Receptor 4 - metabolism | Atherosclerosis - metabolism | Chenodeoxycholic Acid - analogs & derivatives | Cardiovascular Agents - pharmacology | Mice, Knockout | Aorta - pathology | Macrophages - metabolism | Hyperlipidemias - pathology | Ligands | Mice | Transcription Factors - agonists | Receptors, Cytoplasmic and Nuclear - metabolism | Tumor Necrosis Factor-alpha - metabolism | Receptors, Cytoplasmic and Nuclear - deficiency | Transcription Factors - deficiency | DNA-Binding Proteins - deficiency | CD36 Antigens - metabolism | DNA-Binding Proteins - agonists | Lipids - blood | ATP-Binding Cassette Transporters - metabolism | Chenodeoxycholic Acid - pharmacology | Female | ATP Binding Cassette Transporter 1 | Macrophages - immunology | Sterol Regulatory Element Binding Protein 1 - metabolism | Aorta - drug effects | Hyperlipidemias - drug therapy | Mice, Inbred C57BL | Receptors, Cytoplasmic and Nuclear - agonists | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Animals | Apolipoproteins E - genetics | PPAR gamma - agonists | Macrophages - drug effects | CD11b Antigen - metabolism | Atherosclerosis - prevention & control | T cell receptors | Gene expression | Rodents | Coronary vessels | Cells | Bile | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2007, Volume 27, Issue 12, pp. 2684 - 2690
OBJECTIVE—Whereas growth factors, via their ability to stimulate vascular smooth muscle cell (VSMC) proliferation and migration, have been thought to play a... 
Oxidative stress | Inflammatory response | Insulin-like growth factor | Apolipoprotein E | Atherosclerosis | atherosclerosis | PROGENITOR CELLS | SEVERELY BURNED CHILDREN | inflammatory response | LOW-DENSITY-LIPOPROTEIN | DOWN-REGULATION | ENDOTHELIAL NITRIC-OXIDE | INSULIN | MESSENGER-RNA | apolipoprotein E | PERIPHERAL VASCULAR DISEASE | insulin-like growth factor | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | EXPRESSION | oxidative stress | GROWTH-FACTOR-I | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Phosphorylation | Apolipoproteins E - deficiency | Antioxidants - metabolism | Atherosclerosis - genetics | Humans | Aorta - metabolism | RNA, Messenger - metabolism | Stem Cells - metabolism | Anti-Inflammatory Agents - metabolism | Apolipoproteins E - metabolism | Inflammation - metabolism | Dietary Fats - administration & dosage | Insulin-Like Growth Factor I - administration & dosage | Superoxides - metabolism | Anti-Inflammatory Agents - administration & dosage | Dinoprost - analogs & derivatives | Proto-Oncogene Proteins c-akt - metabolism | Interleukin-6 - metabolism | Disease Models, Animal | Recombinant Proteins - metabolism | Atherosclerosis - pathology | Dinoprost - urine | Macrophages - pathology | Endothelial Cells - metabolism | Mice, Inbred C57BL | Cells, Cultured | Nitric Oxide Synthase Type III | Atherosclerosis - metabolism | Disease Progression | Mice, Knockout | Aorta - pathology | Macrophages - metabolism | Animals | Apolipoproteins E - genetics | Antioxidants - administration & dosage | Inflammation - genetics | Stem Cells - pathology | Inflammation - prevention & control | Mice | Oxidative Stress - drug effects | Endothelial Cells - pathology | Atherosclerosis - prevention & control | Insulin-Like Growth Factor I - metabolism | Nitric Oxide Synthase Type II - metabolism | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 12/2016, Volume 311, Issue 6, pp. H1360 - H1366
Anesthesia can affect respiratory, circulatory, and endocrine systems but is necessary for certain experimental procedures such as echocardiography and blood... 
Ketamine-xylazine | Pentobarbital sodium | Isoflurane | pentobarbital sodium | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ketamine-xylazine | MECHANISMS | BODY-TEMPERATURE | COMBINATION | BLOOD-PRESSURE | INSULIN | IMPACT | PERIPHERAL VASCULAR DISEASE | MICE | isoflurane | STRESS | HYPERTENSIVE-RATS | Renin - metabolism | Anesthetics, Dissociative - pharmacology | Isoflurane - pharmacology | Pentobarbital - pharmacology | Male | Fatty Acids, Nonesterified - metabolism | Heart Rate - drug effects | Aldosterone - metabolism | Xylazine - pharmacology | Renin - drug effects | Blood Pressure - drug effects | Dopamine - metabolism | Anesthetics, Inhalation - pharmacology | Angiotensin II - drug effects | Echocardiography | Angiotensin II - metabolism | Cholesterol, HDL - drug effects | Cholesterol, HDL - metabolism | Aorta - drug effects | Insulin Resistance | Rats | Cholesterol, LDL - drug effects | Cholesterol - metabolism | Rats, Sprague-Dawley | Triglycerides - metabolism | Blood Glucose - drug effects | Insulin - metabolism | Animals | Norepinephrine - metabolism | Lipid Metabolism - drug effects | Cholesterol, LDL - metabolism | Glucose - metabolism | Renin-Angiotensin System - drug effects | Hemodynamics - drug effects | Hypnotics and Sedatives - pharmacology | Blood Glucose - metabolism | Ketamine - pharmacology | Epinephrine - metabolism | Lipids | Anesthesia | Ultrasonic imaging | Glucose | Metabolism | Rodents | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, pp. e68197 - e68197
Free fatty acids (FFAs), elevated in metabolic syndrome and diabetes, play a crucial role in the development of atherosclerotic cardiovascular disease, and... 
SUPPLEMENTATION | ARTERY ENDOTHELIAL-CELLS | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | DISEASE | CALCIUM | CALCIFICATION | RISK | FREE FATTY-ACID | DEPENDENT ACTIVATION | EXPRESSION | Calcinosis - genetics | Carotid Arteries - metabolism | Osteopontin - genetics | Palmitic Acid - metabolism | Coenzyme A Ligases - genetics | Muscle, Smooth, Vascular - metabolism | Calcium - metabolism | Gene Expression - genetics | Homeodomain Proteins - metabolism | Humans | Eicosapentaenoic Acid - genetics | Myocytes, Smooth Muscle - pathology | NF-kappa B - metabolism | Osteopontin - metabolism | Aorta - metabolism | Promoter Regions, Genetic - genetics | Coenzyme A Ligases - metabolism | Cell Differentiation - genetics | Caspases - metabolism | Bone Morphogenetic Protein 2 - metabolism | Calcinosis - metabolism | Plaque, Atherosclerotic - pathology | Cell Differentiation - physiology | Myocytes, Smooth Muscle - metabolism | Bone Morphogenetic Protein 2 - genetics | Macrophages - pathology | Plaque, Atherosclerotic - metabolism | Caspases - genetics | Cells, Cultured | Eicosapentaenoic Acid - metabolism | Homeodomain Proteins - genetics | Aorta - pathology | Osteoblasts - pathology | Macrophages - metabolism | Muscle, Smooth, Vascular - pathology | NF-kappa B - genetics | Carotid Arteries - pathology | Calcinosis - pathology | Osteoblasts - metabolism | Immunohistochemistry | Smooth muscle | Cardiovascular disease | Palmitic acid | Osteoblasts | Proteins | Mineralization | Biocompatibility | Aorta | Inhibition | NF-κB protein | Cytokines | Osteopontin | Metabolism | Gene expression | Fatty acids | Studies | Osteoblastogenesis | Inhibitors | Arteriosclerosis | Calcification | Cardiovascular diseases | Metabolic disorders | Calcification (ectopic) | Health sciences | Oxidative stress | Calcium | Laboratories | Chains | Lipids | Activation | Kinases | Macrophages | Arteries | Carotid arteries | Msx2 protein | Rodents | Atherosclerosis | Bone morphogenetic proteins | Tumor necrosis factor-TNF | Deposition | Plaques | University graduates | Bone morphogenetic protein 2 | Diabetes mellitus | Muscles | Caspase | siRNA | Medicine | Antibiotics | Kidney diseases | Bone | Diabetes | Differentiation | Eicosapentaenoic acid | Apoptosis | Pharmaceuticals | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2015, Volume 35, Issue 12, pp. 2605 - 2616
OBJECTIVE—Kawasaki disease (KD) is the most common cause of acute vasculitis and acquired cardiac disease among US children. We have previously shown that both... 
dendritic cells | interleukin-1 | mucocutaneous lymph node syndrome | MyD88 | Endothelial cells | protein | endothelial cells | ELEMENT-BINDING PROTEIN-2 | ACTIVATION | RECEPTOR | CORONARY-ARTERY ANEURYSMS | NLRP3 INFLAMMASOME | POLYMORPHISM | ACUTE-PHASE | INTRAVENOUS IMMUNOGLOBULIN | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | ASSOCIATION | Lactobacillus casei | Mucocutaneous Lymph Node Syndrome - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Stromal Cells - pathology | Caspase 1 - metabolism | Interleukin-1alpha - metabolism | Aortitis - chemically induced | Aorta - metabolism | Coronary Vessels - metabolism | Cell Wall | DNA-Binding Proteins - metabolism | Receptors, Interleukin-1 Type I - metabolism | Interleukin-1beta - metabolism | Bone Marrow Transplantation | Coronary Artery Disease - pathology | Aortitis - pathology | Dendritic Cells - metabolism | Disease Models, Animal | Mucocutaneous Lymph Node Syndrome - pathology | Coronary Vessels - pathology | Signal Transduction | Coronary Artery Disease - chemically induced | Endothelial Cells - metabolism | Mucocutaneous Lymph Node Syndrome - chemically induced | Coronary Artery Disease - metabolism | Mice, Inbred C57BL | Stromal Cells - metabolism | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Aortitis - genetics | DNA-Binding Proteins - genetics | Transplantation Chimera | Mice, Knockout | Aorta - pathology | Mucocutaneous Lymph Node Syndrome - genetics | Aortitis - metabolism | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Coronary Artery Disease - genetics | Receptors, Interleukin-1 Type I - genetics | Myeloid Differentiation Factor 88 - metabolism | Bone Marrow Cells - metabolism | Index Medicus | coronary artery vasculitis | Kawasaki disease | IL-1
Journal Article