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Journal Article
Journal Article
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 07/2017, Volume 37, Issue 11, pp. 2102 - 2102
OBJECTIVE—The role of TGF-β (transforming growth factor-β) signaling in abdominal aortic aneurysm (AAA) formation is controversial. Others reported that... 
aneurysm | smooth muscle | SERVICES TASK-FORCE | ATHEROSCLEROSIS | RECEPTOR | mice | macrophages | TGF-beta | DISRUPTION | angiotensin II | ANEURYSM MOUSE MODEL | PERIPHERAL VASCULAR DISEASE | aorta | SMOOTH-MUSCLE-CELLS | APOE(-/-) MICE | HEMATOLOGY | E-DEFICIENT MICE | PROMOTES | PROGRESSION | Protein-Serine-Threonine Kinases - deficiency | Tunica Media - pathology | Aortic Aneurysm, Abdominal - chemically induced | Receptors, Transforming Growth Factor beta - genetics | Aortic Aneurysm, Abdominal - prevention & control | Muscle, Smooth, Vascular - metabolism | Transforming Growth Factor beta1 - antagonists & inhibitors | Transforming Growth Factor beta2 - metabolism | Aortic Aneurysm, Thoracic - prevention & control | Transforming Growth Factor beta1 - metabolism | Male | Aortic Aneurysm, Thoracic - chemically induced | Transforming Growth Factor beta3 - metabolism | Aorta, Abdominal - drug effects | Aortic Aneurysm, Abdominal - metabolism | Adventitia - metabolism | Receptor, Transforming Growth Factor-beta Type II | Aortic Aneurysm, Thoracic - pathology | Tunica Media - metabolism | Transforming Growth Factor beta - antagonists & inhibitors | Female | Aorta, Abdominal - pathology | Aorta, Thoracic - drug effects | Vascular Remodeling - drug effects | Angiotensin II | Aorta, Abdominal - metabolism | Dilatation, Pathologic | Aorta, Thoracic - pathology | Aortic Aneurysm, Abdominal - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Severity of Illness Index | Genetic Predisposition to Disease | Mice, Inbred C57BL | Protein-Serine-Threonine Kinases - genetics | Aorta, Thoracic - metabolism | Transforming Growth Factor beta3 - antagonists & inhibitors | Mice, Knockout | Antibodies - pharmacology | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Signal Transduction - drug effects | Aortic Aneurysm, Thoracic - metabolism | Transforming Growth Factor beta2 - antagonists & inhibitors | Adventitia - pathology | Transforming Growth Factor beta - metabolism | Receptors, Transforming Growth Factor beta - deficiency | growth factor | Animal Models of Human Disease | Vascular Disease | Genetically Altered and Transgenic Models | Aortic Dissection | genetically altered mice
Journal Article
Nature medicine, ISSN 1546-170X, 2019, Volume 25, Issue 3, pp. 496 - 506
Apolipoprotein-E (ApoE) has been implicated in Alzheimer's disease, atherosclerosis, and other unresolvable inflammatory conditions but a common mechanism of... 
IMMUNE | ALZHEIMERS-DISEASE | A-BETA | NEUROFIBRILLARY PATHOLOGY | ATHEROSCLEROSIS | NEURODEGENERATION | MICE | FACTOR-H BINDS | LASER-CAPTURE MICRODISSECTION | APOLIPOPROTEIN-E | MEDICINE, RESEARCH & EXPERIMENTAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | Complement C5 | Complement C1q - immunology | Humans | Middle Aged | Choroid Plexus - immunology | Male | Leukocytes | Cognitive Dysfunction - immunology | Carotid Artery Diseases - pathology | Aged, 80 and over | Female | Plaque, Amyloid - immunology | Carotid Artery Diseases - immunology | Atherosclerosis - pathology | Atherosclerosis - immunology | Plaque, Amyloid - pathology | Choroid Plexus - pathology | Aorta - immunology | Cognitive Dysfunction - pathology | Mice, Knockout, ApoE | Aorta - pathology | Complement Pathway, Classical - immunology | Animals | Apolipoproteins E - immunology | Carotid Arteries - immunology | Brain - pathology | Amyloid beta-Peptides - immunology | Aged | Carotid Arteries - pathology | RNA, Small Interfering | Antigen-Antibody Complex - immunology | Protein Isoforms - immunology | Brain - immunology | Microscopy, Fluorescence | Cogeneration power plants | Analysis | Atherosclerosis | Lipids | Research | Apolipoproteins | Alzheimer's disease | Choroid plexus | Complex formation | Cognitive ability | Complement | Inflammation | siRNA | Ribonucleic acid--RNA | Arteries | Microglia | White blood cells | Proteins | Complement component C1q | Immune checkpoint | Apolipoprotein E | Arteriosclerosis | Isoforms | Attenuation | Infiltration | Plaques
Journal Article
Journal Article
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2018, Volume 38, Issue 2, pp. 425 - 437
OBJECTIVE—DKK3 (dickkopf 3), a 36-kD secreted glycoprotein, has been shown to be involved in the differentiation of partially reprogrammed cells and embryonic... 
phenotype | smooth muscle cells | atherosclerosis E | stem cells | fibroblasts | STEM-CELLS | ANGIOGENESIS | ACUTE CORONARY SYNDROMES | PROLIFERATION | VEIN GRAFTS | LESIONS | PATHOPHYSIOLOGY | PATHOGENESIS | APOE-DEFICIENT MICE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | NEOINTIMAL FORMATION | Carotid Arteries - metabolism | Muscle, Smooth, Vascular - metabolism | Atherosclerosis - genetics | Humans | Transforming Growth Factor beta1 - metabolism | Myocytes, Smooth Muscle - pathology | Male | Hemorrhage - prevention & control | Aorta - metabolism | Stem Cells - metabolism | Activating Transcription Factor 6 - genetics | Aortic Diseases - metabolism | Carotid Stenosis - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Carotid Stenosis - genetics | Ataxin-1 - metabolism | Cell Transdifferentiation | Female | Intercellular Signaling Peptides and Proteins - deficiency | Myocytes, Smooth Muscle - metabolism | Wnt Signaling Pathway | Aortic Diseases - pathology | Disease Models, Animal | Fibroblasts - metabolism | Rabbits | Atherosclerosis - pathology | Hemorrhage - genetics | Mice, Inbred C57BL | Cells, Cultured | Intercellular Signaling Peptides and Proteins - genetics | Hemorrhage - metabolism | Carotid Stenosis - pathology | Mice, Knockout, ApoE | Fibroblasts - pathology | Plaque, Atherosclerotic | Atherosclerosis - metabolism | Aorta - pathology | Activating Transcription Factor 6 - metabolism | Aortic Diseases - genetics | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Stem Cells - pathology | Carotid Arteries - pathology | Hemorrhage - pathology
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 6, p. e38998
Background: Kawasaki disease is recognized as the most common cause of acquired heart disease in children in the developed world. Clinical, epidemiologic, and... 
INFANTILE PERIARTERITIS-NODOSA | ADVENTITIAL FIBROBLASTS | YOUNG-ADULTS | INTRAVENOUS GAMMA-GLOBULIN | CYTOPLASMIC INCLUSION-BODIES | CORONARY-ARTERY LESIONS | ANEURYSMS | BIOLOGY | INTRAVASCULAR ULTRASOUND | PATHOLOGY | LONG | Mucocutaneous Lymph Node Syndrome - pathology | Myofibroblasts - pathology | Aneurysm - etiology | Cell Proliferation | Myocarditis - etiology | Humans | Myocytes, Smooth Muscle - pathology | Child, Preschool | Infant | Male | Aneurysm, Ruptured - etiology | Myofibroblasts - metabolism | Lymphocytes - pathology | Mucocutaneous Lymph Node Syndrome - complications | Myofibroblasts - ultrastructure | Thrombosis - etiology | Myocarditis - metabolism | Female | Myocytes, Smooth Muscle - ultrastructure | Myocarditis - pathology | Mucocutaneous Lymph Node Syndrome - etiology | Child | Myocytes, Smooth Muscle - metabolism | Neutrophils - pathology | Heart | Virus diseases | Medical research | Medicine, Experimental | Aneurysms | Transplantation | Heart diseases | Diseases | Syngeneic grafts | Aneurysm | Stenosis | Smooth muscle | Epidemiology | Arteries | Arteritis | Kawasaki disease | Vasculitis | Vascular diseases | Aorta | Children | Lesions | Thromboembolism | Autopsies | Leukocytes (eosinophilic) | Heart transplantation | Muscles | Transplants | Leukocytes (neutrophilic) | Electron microscopy | Thrombosis | Saccule | Coronary artery disease | Fever | Morbidity | Pulmonary artery | Endothelium | Studies | Myocarditis | Pathology | Disease transmission | Transmission electron microscopy | Lungs | Mucocutaneous lymph node syndrome | Veins & arteries
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 07/2011, Volume 26, Issue 7, pp. 1543 - 1553
.... Interestingly, depending on the pathology, different regions of the arterial system can be affected... 
Hyperphosphatemia | Alkaline phosphatase | Smooth muscle cells | Matrix Gla protein | Vascular calcifications | Neural crest | SMOOTH MUSCLE CELLS | MATRIX GLA PROTEIN | FORM | VASCULAR CALCIFICATIONS | PATTERNS | IMPLANTS | ALKALINE PHOSPHATASE | FATE | CANINE ATHEROSCLEROSIS | CARTILAGE | ARTERIES | HYPERPHOSPHATEMIA | AORTIC HOMOGRAFTS | MINERALIZATION | ENDOCRINOLOGY & METABOLISM | NEURAL CREST | Extracellular Matrix Proteins - deficiency | Neural Crest - pathology | Aging - drug effects | Alkaline Phosphatase - metabolism | Mesoderm - drug effects | Myocytes, Smooth Muscle - pathology | Aorta, Abdominal - drug effects | Muscle, Smooth, Vascular - embryology | Aorta, Abdominal - pathology | Aorta, Thoracic - drug effects | Calcinosis - metabolism | Myocytes, Smooth Muscle - drug effects | Aorta, Abdominal - metabolism | Myocytes, Smooth Muscle - metabolism | Phosphates - pharmacology | Aorta, Thoracic - pathology | Extracellular Matrix Proteins - metabolism | Calcium-Binding Proteins - metabolism | Alkaline Phosphatase - genetics | Mesoderm - embryology | Cells, Cultured | Aorta, Thoracic - metabolism | Calcium-Binding Proteins - deficiency | Aging - pathology | Neural Crest - drug effects | Muscle, Smooth, Vascular - pathology | Animals | Neural Crest - embryology | Calcinosis - embryology | Mice | Kinetics | Calcinosis - pathology | Mesoderm - pathology | Life Sciences | Morphogenesis | Human health and pathology | Biochemistry, Molecular Biology | Development Biology | Molecular biology | Cellular Biology | Embryology and Organogenesis | Cardiology and cardiovascular system | Cell Behavior
Journal Article