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Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2015, Volume 35, Issue 8, pp. 1746 - 1755
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2017, Volume 37, Issue 11, pp. 2102 - 2102
OBJECTIVE—The role of TGF-β (transforming growth factor-β) signaling in abdominal aortic aneurysm (AAA) formation is controversial. Others reported that... 
aneurysm | smooth muscle | SERVICES TASK-FORCE | ATHEROSCLEROSIS | RECEPTOR | mice | macrophages | TGF-beta | DISRUPTION | angiotensin II | ANEURYSM MOUSE MODEL | PERIPHERAL VASCULAR DISEASE | aorta | SMOOTH-MUSCLE-CELLS | APOE(-/-) MICE | HEMATOLOGY | E-DEFICIENT MICE | PROMOTES | PROGRESSION | Protein-Serine-Threonine Kinases - deficiency | Tunica Media - pathology | Aortic Aneurysm, Abdominal - chemically induced | Receptors, Transforming Growth Factor beta - genetics | Aortic Aneurysm, Abdominal - prevention & control | Muscle, Smooth, Vascular - metabolism | Transforming Growth Factor beta1 - antagonists & inhibitors | Transforming Growth Factor beta2 - metabolism | Aortic Aneurysm, Thoracic - prevention & control | Transforming Growth Factor beta1 - metabolism | Male | Aortic Aneurysm, Thoracic - chemically induced | Transforming Growth Factor beta3 - metabolism | Aorta, Abdominal - drug effects | Aortic Aneurysm, Abdominal - metabolism | Adventitia - metabolism | Receptor, Transforming Growth Factor-beta Type II | Aortic Aneurysm, Thoracic - pathology | Tunica Media - metabolism | Transforming Growth Factor beta - antagonists & inhibitors | Female | Aorta, Abdominal - pathology | Aorta, Thoracic - drug effects | Vascular Remodeling - drug effects | Angiotensin II | Aorta, Abdominal - metabolism | Dilatation, Pathologic | Aorta, Thoracic - pathology | Aortic Aneurysm, Abdominal - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Severity of Illness Index | Genetic Predisposition to Disease | Mice, Inbred C57BL | Protein-Serine-Threonine Kinases - genetics | Aorta, Thoracic - metabolism | Transforming Growth Factor beta3 - antagonists & inhibitors | Mice, Knockout | Antibodies - pharmacology | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Signal Transduction - drug effects | Aortic Aneurysm, Thoracic - metabolism | Transforming Growth Factor beta2 - antagonists & inhibitors | Adventitia - pathology | Transforming Growth Factor beta - metabolism | Receptors, Transforming Growth Factor beta - deficiency | growth factor | Animal Models of Human Disease | Vascular Disease | Genetically Altered and Transgenic Models | Aortic Dissection | genetically altered mice
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 02/2013, Volume 33, Issue 2, pp. 294 - 304
OBJECTIVE—Abdominal aortic aneurysms (AAAs) are common, but their exact pathogenesis remains unknown and no specific medical therapies are available. We sought... 
aneurysm | vascular inflammation | animal model surgery | interleukin-1 receptor antagonist | aortic disease | RHEUMATOID-ARTHRITIS | MUSCLE PHENOTYPIC MODULATION | ANAKINRA | MODEL | RECEPTOR ANTAGONIST | PATHOPHYSIOLOGY | INFLAMMATION | DISEASE | PERIPHERAL VASCULAR DISEASE | IL-1-BETA | MICE | HEMATOLOGY | Aortic Aneurysm, Abdominal - chemically induced | Aortic Aneurysm, Abdominal - prevention & control | Humans | Receptors, Interleukin-1 - genetics | Male | Interleukin-1beta - genetics | RNA, Messenger - metabolism | Aorta, Abdominal - drug effects | Interleukin-1beta - deficiency | Aortic Aneurysm, Abdominal - metabolism | Dose-Response Relationship, Drug | Time Factors | Receptors, Interleukin-1 - deficiency | Aorta, Abdominal - pathology | Interleukin 1 Receptor Antagonist Protein - pharmacology | Aorta, Abdominal - metabolism | Dilatation, Pathologic | Neutrophils - metabolism | Aortic Aneurysm, Abdominal - pathology | Disease Models, Animal | Interleukin-1beta - antagonists & inhibitors | Neutrophils - pathology | Elastin - metabolism | Macrophages - pathology | Mice, Inbred C57BL | Neutrophils - drug effects | Gene Expression Regulation | Aortic Aneurysm, Abdominal - genetics | Mice, Knockout | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Pancreatic Elastase | Macrophages - drug effects | Mice | Receptors, Interleukin-1 - antagonists & inhibitors | Interleukin-1 Receptor Antagonist | Aortic disease | Vascular Inflammation | Animal model surgery | Aneurysm
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 11/2016, Volume 36, Issue 11, pp. 2176 - 2190
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2017, Volume 37, Issue 11, pp. 2171 - 2181
OBJECTIVE—Current experimental models of abdominal aortic aneurysm (AAA) do not accurately reproduce the major features of human AAA. We hypothesized that... 
Models | Animals | Humans | Aneurysm | Immune system | aneurysm | models | MANAGEMENT | RECEPTOR | II-INFUSED MICE | MURINE MODEL | DISRUPTION | DISSECTION | ELASTASE | DEFICIENT | immune system | PERIPHERAL VASCULAR DISEASE | animals | HEMATOLOGY | humans | Antibodies, Monoclonal - toxicity | Aortic Aneurysm, Abdominal - chemically induced | Thrombosis - chemically induced | Aortic Aneurysm, Abdominal - immunology | Extracellular Matrix - metabolism | Male | Aortic Rupture - metabolism | Aortic Rupture - immunology | Apolipoproteins E - metabolism | Aorta, Abdominal - drug effects | Aortic Aneurysm, Abdominal - metabolism | Chemotaxis, Leukocyte - drug effects | Interleukin-1beta - metabolism | Synchrotrons | Ultrasonography | Transforming Growth Factor beta - antagonists & inhibitors | Aorta, Abdominal - pathology | Vascular Remodeling - drug effects | Aorta, Abdominal - metabolism | Dilatation, Pathologic | Wound Healing - drug effects | Aortic Aneurysm, Abdominal - pathology | Disease Models, Animal | Transforming Growth Factor beta - immunology | Extracellular Matrix - drug effects | Mice, Inbred C57BL | Aortic Rupture - pathology | Disease Progression | Mice, Knockout | Aorta, Abdominal - immunology | Thrombosis - pathology | Thrombosis - metabolism | Apolipoproteins E - genetics | Pancreatic Elastase | Aortic Rupture - chemically induced | Kinetics | Extracellular Matrix - pathology | Transforming Growth Factor beta - metabolism
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2013, Volume 33, Issue 4, pp. 718 - 726
OBJECTIVE—Macrophages are critical contributors to abdominal aortic aneurysm (AAA) disease. We examined the ability of MKEY, a peptide inhibitor of CXCL4–CCL5... 
chemokines | mice | CCL5 | abdominal aortic aneurysm | CXCL4 | ATHEROSCLEROTIC LESIONS | PROGENITOR CELLS | RECRUITMENT | WALL | RECEPTOR CCR2 | RANTES | BONE-MARROW | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | INFUSION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | E-DEFICIENT MICE | Aortic Aneurysm, Abdominal - chemically induced | Aortic Aneurysm, Abdominal - prevention & control | Injections, Intravenous | Aortic Aneurysm, Abdominal - immunology | Apolipoproteins E - deficiency | Male | Aorta, Abdominal - drug effects | Chemokine CCL5 - antagonists & inhibitors | Aortic Aneurysm, Abdominal - metabolism | Leukocytes - immunology | Matrix Metalloproteinase 9 - metabolism | Receptors, CCR5 - metabolism | Chemotaxis, Leukocyte - drug effects | Time Factors | Chemokine CCL5 - metabolism | Aorta, Abdominal - pathology | Angiotensin II | Myocytes, Smooth Muscle - drug effects | Aorta, Abdominal - metabolism | Aortic Aneurysm, Abdominal - pathology | Macrophages - immunology | Disease Models, Animal | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | Cells, Cultured | Aortic Aneurysm, Abdominal - genetics | Platelet Factor 4 - antagonists & inhibitors | Disease Progression | Mice, Knockout | Aorta, Abdominal - immunology | Animals | Apolipoproteins E - genetics | Myocytes, Smooth Muscle - immunology | Pancreatic Elastase | Leukocytes - drug effects | Macrophages - drug effects | Mice | Oligopeptides - administration & dosage | Platelet Factor 4 - metabolism | Oligopeptides - pharmacology
Journal Article
Cardiovascular Research, ISSN 0008-6363, 12/2013, Volume 100, Issue 3, pp. 501 - 510
Abdominal aortic aneurysm (AAA) is a life-threatening disease affecting almost 10% of the population over the age of 65. Nitrogen-containing bisphosphonates... 
NF-k B | Abdominal aortic aneurysm | Rho | Bisphopsphonate | Angiotensin II | Low-density lipoprotein receptor | MAPK | Zoledronate | mice | Aortic Aneurysm, Abdominal - chemically induced | Phosphorylation | Aortic Aneurysm, Abdominal - prevention & control | Coculture Techniques | Humans | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Monocytes - metabolism | NF-kappa B - metabolism | rhoA GTP-Binding Protein - metabolism | Aorta, Abdominal - drug effects | Dose-Response Relationship, Drug | rho-Associated Kinases - metabolism | Receptors, LDL - deficiency | Aorta, Abdominal - pathology | Dilatation, Pathologic | Aortic Aneurysm, Abdominal - pathology | Disease Models, Animal | Cell Line | Elastin - metabolism | Receptors, LDL - genetics | Anti-Inflammatory Agents - pharmacology | Endothelial Cells - metabolism | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | Aortic Aneurysm, Abdominal - genetics | Imidazoles - pharmacology | Cell Adhesion - drug effects | Aorta, Abdominal - enzymology | Mice, Knockout | Monocytes - drug effects | Macrophages - metabolism | Animals | Aortic Aneurysm, Abdominal - enzymology | Signal Transduction - drug effects | rho GTP-Binding Proteins - metabolism | Macrophages - drug effects | Mice | Enzyme Activation | Vascular Cell Adhesion Molecule-1 - metabolism | Diphosphonates - pharmacology | Endothelial Cells - drug effects
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2017, Volume 37, Issue 3, pp. 553 - 566
OBJECTIVE—Sclerostin (SOST) has been identified as an important regulator of bone formation; however, it has not been previously implicated in arterial... 
atherosclerosis | DNA methylation | aortic aneurysm | epigenetics | apolipoprotein E-null mouse | sclerostin | OSTEOPROTEGERIN | BETA-CATENIN | DIFFERENTIAL GENE-EXPRESSION | APOLIPOPROTEIN-E | OSTEOPONTIN | DENSITY-LIPOPROTEIN | PERIPHERAL VASCULAR DISEASE | TRANSFORMING-GROWTH-FACTOR | MATRIX-METALLOPROTEINASE-9 EXPRESSION | HEMATOLOGY | ASSOCIATION | Aortic Aneurysm - metabolism | Apolipoproteins E - deficiency | Muscle, Smooth, Vascular - metabolism | Atherosclerosis - genetics | Humans | Myocytes, Smooth Muscle - pathology | Extracellular Matrix - metabolism | Male | Aorta, Abdominal - drug effects | Aortic Aneurysm - chemically induced | Bone Morphogenetic Proteins - metabolism | Aged, 80 and over | Female | Epigenesis, Genetic - drug effects | Aorta, Abdominal - pathology | Aorta, Thoracic - drug effects | Vascular Remodeling - drug effects | Angiotensin II | Myocytes, Smooth Muscle - drug effects | Aorta, Abdominal - metabolism | Bone Morphogenetic Proteins - genetics | Myocytes, Smooth Muscle - metabolism | Aorta, Thoracic - pathology | Aortic Aneurysm - prevention & control | Muscle, Smooth, Vascular - drug effects | Genetic Predisposition to Disease | Cytokines - metabolism | Atherosclerosis - chemically induced | Mice, Inbred C57BL | Cells, Cultured | Aorta, Thoracic - metabolism | Mice, Transgenic | Atherosclerosis - metabolism | Glycoproteins - administration & dosage | Mice, Knockout | Macrophages - metabolism | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Wnt Signaling Pathway - drug effects | Apolipoproteins E - genetics | Genetic Markers - genetics | Macrophages - drug effects | Aged | Aortic Aneurysm - genetics | Atherosclerosis - prevention & control | Abdominal aortic aneurysm | Sclerostin | 60 APPLIED LIFE SCIENCES
Journal Article
Atherosclerosis, ISSN 0021-9150, 2011, Volume 217, Issue 2, pp. 350 - 357
Abstract Objective We sought to examine the effect of resveratrol (3,4′,5-trihydroxy-trans-stilbene), a plant-derived polyphenolic compound, on the development... 
Cardiovascular | Neoangiogenesis | Oxidative stress | Extracellular matrix | Inflammation | Abdominal aortic aneurysm | CELLS | ACTIVATION | ANGIOGENESIS | TRANSCRIPTION | COMPOUND | FACTOR-KAPPA-B | ANGIOTENSIN-II | INHIBITION | TRANS-RESVERATROL | PERIPHERAL VASCULAR DISEASE | E-DEFICIENT MICE | Inflammation - chemically induced | Neovascularization, Physiologic - drug effects | Aortic Aneurysm, Abdominal - chemically induced | Phosphorylation | Aortic Aneurysm, Abdominal - prevention & control | Reactive Oxygen Species - metabolism | Aortic Aneurysm, Abdominal - immunology | Extracellular Matrix - metabolism | Stilbenes - pharmacology | Aorta, Abdominal - drug effects | Aortic Aneurysm, Abdominal - metabolism | Inflammation - metabolism | Matrix Metalloproteinase 9 - metabolism | Inflammation Mediators - metabolism | Aorta, Abdominal - pathology | Aorta, Abdominal - metabolism | Aortic Aneurysm, Abdominal - pathology | Macrophages - immunology | Disease Models, Animal | Calcium Chloride | Anti-Inflammatory Agents - pharmacology | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | Angiogenesis Inhibitors - pharmacology | Inflammation - immunology | Antioxidants - pharmacology | Aorta, Abdominal - immunology | Animals | Transcription Factor RelA - metabolism | Aortic Aneurysm, Abdominal - physiopathology | Macrophages - drug effects | Inflammation - prevention & control | Mice | Oxidative Stress - drug effects | Inflammation - physiopathology | Prevention | Medical colleges | Atherosclerosis | Resveratrol | Neovascularization | Abdominal aneurysm
Journal Article
Circulation Research, ISSN 0009-7330, 11/2015, Volume 117, Issue 11, pp. e80 - e89
RATIONALE:Matrix metalloproteinases (MMPs)–mediated extracellular matrix destruction is the major cause of development and progression of abdominal aortic... 
drug delivery systems | batimastat | matrix metalloproteinase inhibitors | nanoparticles | cardiovascular diseases | vascular remodeling | abdominal aortic aneurysm | CELLS | PROPRANOLOL | CARDIAC & CARDIOVASCULAR SYSTEMS | BB-94 | CANCER | TRIAL | THERAPY | DISEASE | DOXYCYCLINE | PERIPHERAL VASCULAR DISEASE | MICE | HEMATOLOGY | Aortic Aneurysm, Abdominal - chemically induced | Aortic Aneurysm, Abdominal - immunology | Phenylalanine - analogs & derivatives | Matrix Metalloproteinase Inhibitors - administration & dosage | Male | Thiophenes - metabolism | Thiophenes - administration & dosage | Immunoconjugates - administration & dosage | Aorta, Abdominal - drug effects | Drug Carriers | Nanoparticles | Time Factors | Phenylalanine - chemistry | Proteolysis | Vascular Calcification - enzymology | Aorta, Abdominal - pathology | Aortic Aneurysm, Abdominal - pathology | Disease Models, Animal | Elastin - metabolism | Calcium Chloride | Aortic Aneurysm, Abdominal - drug therapy | Phenylalanine - metabolism | Immunoconjugates - metabolism | Vascular Calcification - prevention & control | Matrix Metalloproteinase Inhibitors - chemistry | Vascular Calcification - pathology | Aorta, Abdominal - enzymology | Immunoconjugates - chemistry | Matrix Metalloproteinase Inhibitors - metabolism | Rats, Sprague-Dawley | Chemistry, Pharmaceutical | Disease Progression | Macrophages - enzymology | Aorta, Abdominal - immunology | Polyesters - chemistry | Animals | Aortic Aneurysm, Abdominal - enzymology | Macrophages - drug effects | RAW 264.7 Cells | Mice | Matrix Metalloproteinases - metabolism | Phenylalanine - administration & dosage | Thiophenes - chemistry | Elastin - immunology | targeted therapy | cardiovascular disease | Animal models of human disease | Treatment | MMP inhibitors | nanoparticle | Pharmacology | drug delivery system | Aneurysm Abdominal aortic aneurysm
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2014, Volume 34, Issue 8, pp. 1747 - 1755
OBJECTIVE—Inflammation plays a critical role in the development of abdominal aortic aneurysms (AAAs). Because stromal cell–derived factor 1 (SDF-1) is known... 
Chemokine CXCL12 | Inflammation | Pharmacology | Abdominal | Aortic aneurysm | MATRIX | STEM-CELLS | aortic aneurysm, abdominal | CXCR4 ANTAGONIST | LOCAL OVEREXPRESSION | FACTOR-I | chemokine CXCL12 | HEMATOPOIETIC PROGENITOR | RAT MODEL | PATHOGENESIS | THERAPY | inflammation | pharmacology | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Aortic Aneurysm, Abdominal - chemically induced | Aortic Aneurysm, Abdominal - prevention & control | Aortic Aneurysm, Abdominal - immunology | Humans | Rats, Inbred F344 | Heterocyclic Compounds - pharmacology | Male | Green Fluorescent Proteins - genetics | Aorta, Abdominal - drug effects | Aortic Aneurysm, Abdominal - metabolism | Chemokine CXCL12 - genetics | Heterografts | Time Factors | U937 Cells | Bone Marrow Transplantation | Inflammation Mediators - metabolism | Receptors, CXCR4 - genetics | Aorta, Abdominal - pathology | Myocytes, Smooth Muscle - drug effects | Aorta, Abdominal - metabolism | Myocytes, Smooth Muscle - metabolism | Aortic Aneurysm, Abdominal - pathology | Macrophages - immunology | Disease Models, Animal | Calcium Chloride | Guinea Pigs | Anti-Inflammatory Agents - pharmacology | Receptors, CXCR4 - antagonists & inhibitors | Mice, Inbred C57BL | Rats | Mice, Transgenic | Aortic Aneurysm, Abdominal - genetics | Chemotaxis - drug effects | Receptors, CXCR4 - metabolism | Aorta, Abdominal - immunology | Macrophages - metabolism | Animals | Chemokine CXCL12 - metabolism | Green Fluorescent Proteins - biosynthesis | Myocytes, Smooth Muscle - immunology | Macrophages - drug effects | Mice
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 02/2018, Volume 38, Issue 4, pp. 843 - 853
Journal Article
Journal of Vascular Surgery, ISSN 0741-5214, 12/2018, Volume 68, Issue 6, pp. 48S - 59S.e1
Osteoclastogenic activation of macrophages (OCG) occurs in human abdominal aortic aneurysms (AAAs) and in calcium chloride-induced degenerative AAAs in mice,... 
SYSTEM | SURGERY | PERIPHERAL VASCULAR DISEASE | ANEURYSM | INFLAMMATION | Aortic Aneurysm, Abdominal - chemically induced | Aortic Aneurysm, Abdominal - prevention & control | RANK Ligand - metabolism | Muscle, Smooth, Vascular - metabolism | Myocytes, Smooth Muscle - pathology | Male | Aorta, Abdominal - drug effects | Aortic Aneurysm, Abdominal - metabolism | RANK Ligand - immunology | STAT5 Transcription Factor - metabolism | Matrix Metalloproteinase 9 - metabolism | Aneurysm, Dissecting - chemically induced | Cell Transdifferentiation - drug effects | Janus Kinase 2 - metabolism | Aorta, Abdominal - pathology | Angiotensin II | Myocytes, Smooth Muscle - drug effects | Aneurysm, Dissecting - prevention & control | Aorta, Abdominal - metabolism | Myocytes, Smooth Muscle - metabolism | Aortic Aneurysm, Abdominal - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Osteoclasts - pathology | Aneurysm, Dissecting - metabolism | Macrophages - pathology | Signal Transduction | Osteogenesis - drug effects | Antibodies, Neutralizing - pharmacology | Aneurysm, Dissecting - pathology | Mice, Knockout, ApoE | Osteoclasts - metabolism | RANK Ligand - genetics | Macrophages - metabolism | Muscle, Smooth, Vascular - pathology | Animals | RANK Ligand - antagonists & inhibitors | Macrophages - drug effects | RAW 264.7 Cells | Mice | Osteoclasts - drug effects | Immunohistochemistry | Phosphatases | Analysis | Angiotensin | Aneurysms | Macrophages | Apolipoproteins | Calcium chloride
Journal Article