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Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2015, Volume 35, Issue 1, pp. 155 - 162
OBJECTIVE—Low-density lipoprotein receptor–related protein 1 (LRP1), a multifunctional protein involved in endocytosis and cell signaling pathways, leads to... 
mouse | aortic aneurysm | Angiotensin II | LRP1 protein | SYSTEM | LRP1 protein, mouse | TISSUE GROWTH-FACTOR | angiotensin II | ATHEROSCLEROSIS | PERIPHERAL VASCULAR DISEASE | VASCULAR WALL | LRP | HEMATOLOGY | EXPRESSION | DEFICIENCY | Aortic Aneurysm, Abdominal - chemically induced | Aortic Aneurysm - metabolism | Muscle, Smooth, Vascular - metabolism | Aneurysm - chemically induced | Myocytes, Smooth Muscle - pathology | Male | Aneurysm - physiopathology | RNA, Messenger - metabolism | Muscle, Smooth, Vascular - physiopathology | Aortic Aneurysm, Abdominal - metabolism | Aortic Aneurysm - chemically induced | Arterial Pressure | Tumor Suppressor Proteins - deficiency | Gene Deletion | Tumor Suppressor Proteins - genetics | Receptors, LDL - deficiency | Aorta, Abdominal - pathology | Aortic Aneurysm - pathology | Mesenteric Artery, Superior - pathology | Aorta, Abdominal - metabolism | Dilatation, Pathologic | Myocytes, Smooth Muscle - metabolism | Aneurysm - metabolism | Aortic Aneurysm, Abdominal - pathology | Disease Models, Animal | Elastin - metabolism | Receptors, LDL - genetics | Matrix Metalloproteinase 2 - metabolism | Aortic Aneurysm - physiopathology | Cells, Cultured | Aortic Aneurysm, Abdominal - genetics | Urokinase-Type Plasminogen Activator - genetics | Aneurysm - pathology | Aneurysm - genetics | Mice, Knockout | Matrix Metalloproteinase 2 - genetics | Macrophages - metabolism | Muscle, Smooth, Vascular - pathology | Animals | Mesenteric Artery, Superior - metabolism | Urokinase-Type Plasminogen Activator - metabolism | Ligands | Norepinephrine | Aortic Aneurysm - genetics
Journal Article
Science, ISSN 0036-8075, 4/2011, Volume 332, Issue 6027, pp. 358 - 361
Transforming growth factor—β (TGFβ) signaling drives aneurysm progression in multiple disorders, including Marfan syndrome (MFS), and therapies that inhibit... 
Transcriptional regulatory elements | Echocardiography | Aortic diseases | Root growth | REPORTS | Placebos | Aneurysms | Aorta | Mice | Aortic aneurysm | Marfan syndrome | PATHOGENESIS | CELLS | ACTIVATION | VALSALVA | MULTIDISCIPLINARY SCIENCES | N-TERMINAL KINASE | MOUSE MODEL | NOONANS-SYNDROME | RECEPTOR | MUTATIONS | INHIBITOR | Diphenylamine - pharmacology | Aortic Aneurysm - metabolism | Mitogen-Activated Protein Kinase 8 - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Anthracenes - therapeutic use | MAP Kinase Signaling System | Diphenylamine - analogs & derivatives | Marfan Syndrome - drug therapy | Smad4 Protein - genetics | Transforming Growth Factor beta - antagonists & inhibitors | Aortic Aneurysm - pathology | Aortic Aneurysm - prevention & control | Disease Models, Animal | Transforming Growth Factor beta - immunology | Diphenylamine - therapeutic use | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Mitogen-Activated Protein Kinase 8 - metabolism | Aortic Aneurysm - physiopathology | Losartan - pharmacology | Smad2 Protein - metabolism | Sulfonamides - pharmacology | Anthracenes - pharmacology | Disease Progression | Aorta - pathology | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Sulfonamides - therapeutic use | Protein Kinase Inhibitors - therapeutic use | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Protein Kinase Inhibitors - pharmacology | Smad4 Protein - deficiency | Enzyme Activation | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article
Science, ISSN 0036-8075, 4/2011, Volume 332, Issue 6027, pp. 361 - 365
Angiotensin II (AngII) mediates progression of aortic aneurysm, but the relative contribution of its type 1 (AT1) and type 2 (AT2) receptors remains unknown.... 
Connective tissues | Receptors | Root growth | Medical treatment | REPORTS | Aneurysms | Placebos | Aorta | Mice | Aortic aneurysm | Marfan syndrome | PATHOGENESIS | ACTIVATION | MECHANISM | MULTIDISCIPLINARY SCIENCES | MARFAN-SYNDROME | MOUSE MODEL | GROWTH | SMOOTH-MUSCLE-CELLS | BLOCKADE | CONTRIBUTES | EXPRESSION | Aortic Aneurysm - metabolism | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Aortic Rupture - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Enalapril - therapeutic use | MAP Kinase Signaling System | Marfan Syndrome - drug therapy | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Aortic Rupture - prevention & control | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Aortic Aneurysm - pathology | Aortic Aneurysm - prevention & control | Disease Models, Animal | Receptor, Angiotensin, Type 2 - genetics | Angiotensin II - metabolism | Signal Transduction | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Losartan - pharmacology | Aortic Rupture - pathology | Disease Progression | Mice, Knockout | Animals | Enalapril - pharmacology | Receptor, Angiotensin, Type 2 - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Aortic Aneurysm - drug therapy | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Aortic aneurysms | Development and progression | Genetic aspects | Health aspects | Angiotensin | Signal transduction | Peptides | Cellular biology | Coronary vessels | Rodents
Journal Article
Circulation Research, ISSN 0009-7330, 01/2012, Volume 110, Issue 2, pp. 312 - 324
RATIONALE:Marfan syndrome (MFS) is a systemic connective tissue disorder notable for the development of aortic root aneurysms and the subsequent... 
aneurysm | apoptosis | microRNA | extracellular matrix | Marfan syndrome | FIBROSIS | KAPPA-B ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | AORTIC-ANEURYSM | KINASE | FAILURE | MICRORNA EXPRESSION SIGNATURE | INFLAMMATION | METALLOPROTEINASE SECRETION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | REVEALS | Up-Regulation | Age Factors | Aortic Aneurysm - metabolism | Male | MicroRNAs - metabolism | NF-kappa B - metabolism | Aorta - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Marfan Syndrome - therapy | Female | Marfan Syndrome - complications | Microfilament Proteins - metabolism | Aortic Aneurysm - pathology | Microfilament Proteins - genetics | Real-Time Polymerase Chain Reaction | Aortic Aneurysm - prevention & control | Disease Models, Animal | Elastin - metabolism | Fibrillin-1 | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | Cells, Cultured | Losartan - pharmacology | Fibrillins | Mice, Transgenic | Reverse Transcriptase Polymerase Chain Reaction | Marfan Syndrome - genetics | Apoptosis Regulatory Proteins - metabolism | Aorta - pathology | Animals | Elastin - genetics | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Oligonucleotides, Antisense - administration & dosage | Mice | MicroRNAs - genetics | Aortic Aneurysm - genetics | Transforming Growth Factor beta - metabolism | Apoptosis | Genetic Therapy - methods
Journal Article
Nature Medicine, ISSN 1078-8956, 02/2017, Volume 23, Issue 2, pp. 200 - 212
Heritable thoracic aortic aneurysms and dissections (TAAD), including Marfan syndrome (MFS), currently lack a cure, and causative mutations have been... 
MEDICINE, RESEARCH & EXPERIMENTAL | INFLAMMATORY ANEURYSM | BIOCHEMISTRY & MOLECULAR BIOLOGY | THORACIC AORTA | ANGIOTENSIN-II | CELL BIOLOGY | SYNTHASE | DISINTEGRIN | IN-VIVO | DISSECTIONS | ANEURYSM FORMATION | EXPRESSION | PROGRESSION | Aneurysm, Dissecting - genetics | Aortic Aneurysm, Thoracic - genetics | Aortic Aneurysm - metabolism | ADAMTS1 Protein - genetics | Humans | Middle Aged | Immunoblotting | Male | Aorta - metabolism | Gene Knockdown Techniques | Nitric Oxide Synthase Type II - antagonists & inhibitors | Adult | Female | Fibrillin-1 - genetics | Real-Time Polymerase Chain Reaction | ADAMTS1 Protein - metabolism | Disease Models, Animal | NG-Nitroarginine Methyl Ester - pharmacology | Aneurysm, Dissecting - metabolism | Aorta - drug effects | Enzyme Inhibitors - pharmacology | Marfan Syndrome - genetics | Haploinsufficiency | Animals | Nitric Oxide Synthase Type II - genetics | Aortic Aneurysm, Thoracic - metabolism | Marfan Syndrome - metabolism | Aged | Mice | Aortic Aneurysm - genetics | Nitric Oxide - metabolism | Nitric Oxide Synthase Type II - metabolism | Blood circulation disorders | Molecular targeted therapy | Care and treatment | Proteases | Innovations | Development and progression | Genetic aspects | Oxidoreductases | Health aspects | Aneurysms | Cardiovascular disease | Genetic disorders | Rodents | Nitric oxide
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2017, Volume 37, Issue 3, pp. 553 - 566
OBJECTIVE—Sclerostin (SOST) has been identified as an important regulator of bone formation; however, it has not been previously implicated in arterial... 
atherosclerosis | DNA methylation | aortic aneurysm | epigenetics | apolipoprotein E-null mouse | sclerostin | OSTEOPROTEGERIN | BETA-CATENIN | DIFFERENTIAL GENE-EXPRESSION | APOLIPOPROTEIN-E | OSTEOPONTIN | DENSITY-LIPOPROTEIN | PERIPHERAL VASCULAR DISEASE | TRANSFORMING-GROWTH-FACTOR | MATRIX-METALLOPROTEINASE-9 EXPRESSION | HEMATOLOGY | ASSOCIATION | Aortic Aneurysm - metabolism | Apolipoproteins E - deficiency | Muscle, Smooth, Vascular - metabolism | Atherosclerosis - genetics | Humans | Myocytes, Smooth Muscle - pathology | Extracellular Matrix - metabolism | Male | Aorta, Abdominal - drug effects | Aortic Aneurysm - chemically induced | Bone Morphogenetic Proteins - metabolism | Aged, 80 and over | Female | Epigenesis, Genetic - drug effects | Aorta, Abdominal - pathology | Aorta, Thoracic - drug effects | Vascular Remodeling - drug effects | Angiotensin II | Myocytes, Smooth Muscle - drug effects | Aorta, Abdominal - metabolism | Bone Morphogenetic Proteins - genetics | Myocytes, Smooth Muscle - metabolism | Aorta, Thoracic - pathology | Aortic Aneurysm - prevention & control | Muscle, Smooth, Vascular - drug effects | Genetic Predisposition to Disease | Cytokines - metabolism | Atherosclerosis - chemically induced | Mice, Inbred C57BL | Cells, Cultured | Aorta, Thoracic - metabolism | Mice, Transgenic | Atherosclerosis - metabolism | Glycoproteins - administration & dosage | Mice, Knockout | Macrophages - metabolism | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Wnt Signaling Pathway - drug effects | Apolipoproteins E - genetics | Genetic Markers - genetics | Macrophages - drug effects | Aged | Aortic Aneurysm - genetics | Atherosclerosis - prevention & control | Sclerostin | Abdominal aortic aneurysm
Journal Article
Circulation, ISSN 0009-7322, 06/2014, Volume 129, Issue 25, pp. 2661 - 2672
Background-Increased production of reactive oxygen species (ROS) throughout the vascular wall is a feature of cardiovascular disease states, but therapeutic... 
Aorta | Reactive oxygen species | Angiotensin II | Dissection | NADPH oxidase | Cyclophilin A | CARDIAC & CARDIOVASCULAR SYSTEMS | VASCULAR OXIDATIVE STRESS | cyclophilin A | ATHEROSCLEROSIS | ANGIOTENSIN-II | dissection | SUPPLEMENTATION | angiotensin II | reactive oxygen species | PERIPHERAL VASCULAR DISEASE | aorta | HYPERTENSION | E-DEFICIENT MICE | ANEURYSM FORMATION | NOX1 | TRANSGENIC MICE | Aneurysm, Dissecting - epidemiology | Reactive Oxygen Species - metabolism | Aortic Aneurysm - metabolism | Membrane Glycoproteins - metabolism | Muscle, Smooth, Vascular - metabolism | NADPH Oxidases - metabolism | Disease Susceptibility - etiology | Disease Susceptibility - metabolism | Male | Cyclophilins - metabolism | Aortic Aneurysm - etiology | NADPH Oxidases - genetics | Angiotensin II - adverse effects | Aneurysm, Dissecting - etiology | Disease Models, Animal | Aneurysm, Dissecting - metabolism | Signal Transduction | Aortic Aneurysm - epidemiology | Mice, Inbred C57BL | Mice, Transgenic | Cyclophilins - genetics | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Animals | Disease Susceptibility - epidemiology | Endothelium, Vascular - metabolism | Mice | Matrix Metalloproteinases - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Dissecting aneurysm | Physiological aspects | Disease susceptibility | Research | Endothelium | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2015, Volume 35, Issue 4, pp. 960 - 972
Objective-Marfan's syndrome is characterized by the formation of ascending aortic aneurysms resulting from altered assembly of extracellular matrix... 
focal adhesion | actin | aortic stiffness | myocardin | TGF-β | aortic aneurysms | extracellular matrix | RhoA | TRANSCRIPTION FACTORS | SMAD2 PATHWAY | EPIGENETIC CONTROL | THORACIC AORTIC-ANEURYSMS | SIGNAL-TRANSDUCTION | TGF-beta | GROWTH-FACTOR-BETA | MOUSE MODEL | DISEASE | PERIPHERAL VASCULAR DISEASE | EXTRACELLULAR-MATRIX | DIFFERENTIATION | HEMATOLOGY | Vascular Remodeling | Aortic Aneurysm - metabolism | Muscle, Smooth, Vascular - metabolism | Humans | Actins - metabolism | Myocytes, Smooth Muscle - pathology | rhoA GTP-Binding Protein - metabolism | Aorta - metabolism | Aortic Aneurysm - etiology | Case-Control Studies | Muscle Proteins - metabolism | Cytoskeletal Proteins - metabolism | Cell Differentiation | Marfan Syndrome - complications | Microfilament Proteins - metabolism | Aortic Aneurysm - pathology | Dilatation, Pathologic | Myocytes, Smooth Muscle - metabolism | Biomarkers - metabolism | Calcium-Binding Proteins - metabolism | Collagen Type I - metabolism | Signal Transduction | Nuclear Proteins - metabolism | Aorta - pathology | Muscle, Smooth, Vascular - pathology | Phenotype | Stress Fibers - metabolism | Focal Adhesions - metabolism | Cell Line, Tumor | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Trans-Activators - metabolism | Transforming Growth Factor beta - metabolism | Biological Sciences | Medical and Health Sciences | Medicin och hälsovetenskap | Naturvetenskap | Biologiska vetenskaper | Natural Sciences
Journal Article
Circulation Research, ISSN 0009-7330, 02/2016, Volume 118, Issue 3, pp. 388 - 399
RATIONALE:Aortic aneurysm is a life-threatening cardiovascular disorder caused by the predisposition for dissection and rupture. Genetic studies have proved... 
Smooth muscle | Myocytes | Macrophages | Aortic aneurysm | Aortic rupture | Smad4 protein | myocytes, smooth muscle | CARDIAC & CARDIOVASCULAR SYSTEMS | HEREDITARY HEMORRHAGIC TELANGIECTASIA | ENDOTHELIAL SMAD4 | ATHEROSCLEROSIS | RECEPTOR | JUVENILE POLYPOSIS SYNDROME | aortic aneurysm | macrophages | GROWTH-FACTOR-BETA | aortic rupture | PERIPHERAL VASCULAR DISEASE | MUTATIONS | METALLOELASTASE | HEMATOLOGY | EXPRESSION | CONTRIBUTES | Protein-Serine-Threonine Kinases - deficiency | Up-Regulation | Receptors, Transforming Growth Factor beta - genetics | Aortic Aneurysm - metabolism | Muscle, Smooth, Vascular - metabolism | Myocytes, Smooth Muscle - pathology | Male | Aorta - metabolism | Transfection | RNA Interference | Smad4 Protein - genetics | Time Factors | Proteolysis | Female | Aortic Aneurysm - pathology | Myocytes, Smooth Muscle - metabolism | Aortic Aneurysm - prevention & control | Cell Line | Elastin - metabolism | Genetic Predisposition to Disease | Mice, Inbred C57BL | Protein-Serine-Threonine Kinases - genetics | Rats | Matrix Metalloproteinase 12 - metabolism | Chemotaxis | Smad4 Protein - metabolism | Mice, Knockout | Aorta - pathology | Macrophages - metabolism | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Cathepsins - metabolism | Chemokines - metabolism | Smad4 Protein - deficiency | Aortic Aneurysm - genetics | Receptors, Transforming Growth Factor beta - deficiency
Journal Article
Journal of the Royal Society Interface, ISSN 1742-5689, 11/2017, Volume 14, Issue 136, pp. 20170327 - 20170327
The embryonic lineage of intramural cells, microstructural organization of the extracellular matrix, local luminal and wall geometry, and haemodynamic loads... 
Smooth muscle | Inflammatory cells | Angiotensin II | Stress | Aortic stiffness | stress | MULTIDISCIPLINARY SCIENCES | INVOLVEMENT | smooth muscle | ATHEROSCLEROSIS | ANGIOTENSIN-II | MODEL | II-INDUCED HYPERTENSION | aortic stiffness | angiotensin II | inflammatory cells | GROWTH | SMOOTH-MUSCLE-CELLS | EXTRACELLULAR-MATRIX | MICE | MECHANOTRANSDUCTION | Aneurysm, Dissecting - genetics | Aneurysm, Dissecting - metabolism | Aortic Aneurysm - metabolism | Muscle, Smooth, Vascular - metabolism | Models, Cardiovascular | Aortic Aneurysm - physiopathology | Extracellular Matrix - metabolism | Male | Aneurysm, Dissecting - pathology | Muscle, Smooth, Vascular - physiopathology | Neointima - metabolism | Mice, Knockout | Extracellular Matrix - genetics | Neointima - physiopathology | Muscle, Smooth, Vascular - pathology | Neointima - genetics | Animals | Aneurysm, Dissecting - physiopathology | Fibrosis | Neointima - pathology | Mice | Aortic Aneurysm - pathology | Aortic Aneurysm - genetics | Extracellular Matrix - pathology | Hypertension | Animal models | Contractility | Cyclic loads | Aneurysm | Muscles | Mechanical properties | Aneurysms | Stiffening | Muscle contraction | Biomechanics | Arteriosclerosis | Coronary vessels | Collagen | Atherosclerosis | Aorta | Extracellular matrix | Stiffness | Blood pressure | Dissection | Stretching | 1004 | Life Sciences–Engineering interface
Journal Article