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Circulation Research, ISSN 0009-7330, 06/2013, Volume 113, Issue 1, pp. 72 - 77
Journal Article
Kidney International, ISSN 0085-2538, 12/2012, Volume 82, Issue 12, pp. 1261 - 1270
Vascular calcification is common in chronic kidney disease, where cardiovascular mortality remains the leading cause of death. Patients with kidney disease are... 
klotho | chronic kidney disease | osteopontin | vitamin D | vascular calcification | MORTALITY RISK | SERUM PHOSPHORUS | ARTERIAL MEDIAL CALCIFICATION | HEMODIALYSIS-PATIENTS | CARDIOVASCULAR-DISEASE | UROLOGY & NEPHROLOGY | SMOOTH-MUSCLE-CELLS | 1,25-DIHYDROXYVITAMIN D-3 | FIBROBLAST GROWTH FACTOR-23 | D ANALOGS | Up-Regulation | Muscle, Smooth, Vascular - metabolism | Renal Insufficiency, Chronic - etiology | Glucuronidase - metabolism | Osteopontin - metabolism | Aorta - metabolism | Parathyroid Hormone - blood | Aortic Diseases - metabolism | Renal Insufficiency, Chronic - drug therapy | Renal Insufficiency, Chronic - metabolism | Calcitriol - administration & dosage | Calcium - blood | Vascular Calcification - metabolism | Time Factors | Vascular Calcification - etiology | Aortic Diseases - prevention & control | Phosphates - blood | Female | Mice, Inbred DBA | Myocytes, Smooth Muscle - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Elastin - metabolism | Aorta - drug effects | Mice, Inbred C57BL | Cells, Cultured | Injections, Intraperitoneal | Vascular Calcification - prevention & control | Glucuronidase - blood | Receptors, Calcitriol - metabolism | Vascular Calcification - pathology | Aorta - pathology | Glucuronidase - urine | Animals | Diet | Fibroblast Growth Factors - blood | Receptors, Calcitriol - agonists | Calcitriol - pharmacology | Ergocalciferols - pharmacology | Ergocalciferols - administration & dosage | Mice | Aortic Diseases - etiology | Index Medicus | Calcification (ectopic) | Urine | Cell culture | Animal models | Calcium | Secretion | Elastin | Mortality | Calcitriol | Osteopontin | Diets | Klotho protein | Parathyroid hormone | Phosphate | Hyperphosphatemia | Aorta | Kidney diseases | Vitamin D receptors | Cardiovascular diseases | Vascular calcification
Journal Article
Circulation, ISSN 0009-7322, 04/2009, Volume 119, Issue 13, pp. 1785 - 1794
Background-Clinical studies have demonstrated that 50% of individuals with chronic renal disease (CRD) die of cardiovascular causes, including advanced... 
Calcification | Chronic | Kidney failure | Aortic valve | Elastin | Atherosclerosis | atherosclerosis | kidney failure, chronic | aortic valve | ELASTIN PEPTIDES | CARDIAC & CARDIOVASCULAR SYSTEMS | ATHEROSCLEROTIC PLAQUES | VASCULAR CALCIFICATION | elastin | POTENTIAL MECHANISM | FAILURE | IN-VIVO | CYSTATIN-C | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | calcification | CHRONIC KIDNEY-DISEASE | VULNERABLE PLAQUE | Tunica Media - pathology | Osteogenesis - physiology | Cystatin C - blood | Humans | Carotid Artery Diseases - physiopathology | Aorta - metabolism | Aortic Valve - pathology | Nephrectomy | Carotid Artery Diseases - pathology | Mice, Mutant Strains | Tunica Media - metabolism | Phosphates - blood | Calcinosis - metabolism | Carotid Artery Diseases - metabolism | Phosphates - pharmacology | Muscle, Smooth, Vascular - physiology | Kidney Failure, Chronic - physiopathology | Elastin - metabolism | Mice, Inbred C57BL | Osteogenesis - drug effects | Aortic Valve Stenosis - physiopathology | Cells, Cultured | Elastin - pharmacology | Muscle, Smooth, Vascular - cytology | Aortic Valve - metabolism | Kidney Failure, Chronic - metabolism | Aorta - pathology | Tunica Intima - metabolism | Animals | Aortic Valve Stenosis - pathology | Tunica Intima - pathology | Cathepsins - genetics | Cathepsins - metabolism | Apolipoproteins E - genetics | Creatinine - blood | Aortic Valve Stenosis - metabolism | Mice | Calcinosis - pathology | Cathepsins - deficiency | Calcinosis - physiopathology | Cathepsins | Physiological aspects | Research | Chronic kidney failure | Diseases | Index Medicus | Abridged Index Medicus
Journal Article
Cardiovascular Research, ISSN 0008-6363, 12/2015, Volume 108, Issue 3, pp. 377 - 386
Neointimal hyperplasia is a common feature of fibro-proliferative vascular disease and characterizes initial stages of atherosclerosis. Neointimal lesions... 
Shear stress | Endothelial-to-mesenchymal transition | Neointimal hyperplasia | ERK5 | Fibrosis | ARTERIAL INJURY | MYOCARDIN | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | ATHEROSCLEROSIS | MECHANISMS | FLOW | GROWTH | SMOOTH-MUSCLE-CELLS | EXPRESSION | TRANSDIFFERENTIATION | Carotid Arteries - metabolism | Vascular Remodeling | Cell Proliferation | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Carotid Artery Diseases - physiopathology | Stress, Mechanical | Male | Aortic Diseases - metabolism | Neointima | Transfection | RNA Interference | Time Factors | Carotid Artery Diseases - pathology | Swine | MAP Kinase Kinase 5 - metabolism | Epithelial-Mesenchymal Transition | HEK293 Cells | Carotid Artery Diseases - metabolism | Aorta, Thoracic - pathology | Aortic Diseases - pathology | Disease Models, Animal | Endothelial Cells - metabolism | Mice, Inbred C57BL | Aorta, Thoracic - metabolism | Mitogen-Activated Protein Kinase 7 - genetics | MAP Kinase Kinase 5 - genetics | Mitogen-Activated Protein Kinase 7 - metabolism | Aorta, Thoracic - physiopathology | Plaque, Atherosclerotic | Regional Blood Flow | Mechanotransduction, Cellular | Aortic Diseases - genetics | Aortic Diseases - physiopathology | Animals | Carotid Arteries - physiopathology | Carotid Artery Diseases - genetics | Human Umbilical Vein Endothelial Cells - pathology | Carotid Arteries - pathology | Endothelial Cells - pathology | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2010, Volume 30, Issue 4, pp. 749 - 757
OBJECTIVE—Animal and clinical studies have suggested that polyphenols in fruits, red wine, and tea may delay the development of atherosclerosis through their... 
Endothelial dysfunction | Soluble P-selectin | Oxidative stress | Polyphenol | Inflammation | Endothelin-1 | polyphenol | endothelial dysfunction | soluble P-selectin | inflammation | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | BLACK TEA CONSUMPTION | SMOOTH-MUSCLE-CELLS | HEME OXYGENASE-1 | RED WINE | endothelin-1 | HEMATOLOGY | E-DEFICIENT MICE | NF-KAPPA-B | oxidative stress | CORONARY-ARTERY-DISEASE | Inflammation - pathology | Heme Oxygenase-1 - metabolism | Apolipoproteins E - deficiency | Cholesterol - blood | Endothelium, Vascular - drug effects | Male | Aorta - metabolism | F2-Isoprostanes - metabolism | Lignans - pharmacology | Aortic Diseases - metabolism | Inflammation - metabolism | Leukotriene B4 - metabolism | Aortic Diseases - prevention & control | Dioxoles - pharmacology | Polyphenols | Anti-Inflammatory Agents - administration & dosage | Disease Models, Animal | Phenols - pharmacology | Biomarkers - metabolism | Atherosclerosis - pathology | Anti-Inflammatory Agents - pharmacology | Atherosclerosis - physiopathology | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Diet | Endothelium, Vascular - metabolism | Inflammation - prevention & control | Mice | Oxidative Stress - drug effects | Biflavonoids - pharmacology | Chlorogenic Acid - pharmacology | Nitrates - urine | Superoxides - metabolism | Flavonoids - administration & dosage | Flavonoids - pharmacology | Membrane Proteins - metabolism | Aorta - physiopathology | Nitric Oxide Synthase Type III - metabolism | Catechin - pharmacology | Fatty Acids - metabolism | Nitrites - urine | Aorta - drug effects | Mice, Inbred C57BL | Endothelium, Vascular - physiopathology | Aortic Diseases - physiopathology | Animals | Quercetin - pharmacology | Apolipoproteins E - genetics | Endothelin-1 - urine | P-Selectin - blood | Phenols - administration & dosage | Nitric Oxide - metabolism | Atherosclerosis - prevention & control | Inflammation - physiopathology | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2011, Volume 31, Issue 7, pp. 1565 - 1572
OBJECTIVE—Interleukin 17A (IL17A) is involved in many inflammatory processes, but its role in atherosclerosis remains controversial. We examined the role of... 
atherosclerosis | nitric oxide | cytokines | vascular biology | superoxide | IL-17A | INTACT | FOAM CELLS | PLAQUE | MONOCYTE MIGRATION | PERIPHERAL VASCULAR DISEASE | DYSFUNCTION | TISSUES | HEMATOLOGY | SUPEROXIDE-DISMUTASE | T-CELLS | Antibodies, Neutralizing - administration & dosage | Inflammation - pathology | Spleen - immunology | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Dendritic Cells - immunology | Humans | Middle Aged | Interleukin-17 - immunology | Male | Aorta - metabolism | Interferon-gamma - metabolism | Aortic Diseases - metabolism | Inflammation - metabolism | Ligation | Lymphocytes - immunology | Time Factors | Carotid Artery Diseases - pathology | Dietary Fats | Superoxides - metabolism | Female | Angiotensin II | Carotid Artery Diseases - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Carotid Artery Diseases - immunology | Interleukin-17 - blood | Atherosclerosis - pathology | Atherosclerosis - immunology | Aortic Diseases - immunology | Mice, Inbred C57BL | Cells, Cultured | Interleukin-17 - genetics | Aorta - immunology | Inflammation - immunology | Interleukin-17 - deficiency | Atherosclerosis - metabolism | Mice, Knockout | Interleukin-17 - metabolism | Aortic Diseases - genetics | Animals | Apolipoproteins E - genetics | Carotid Artery Diseases - genetics | Inflammation - genetics | Carotid Arteries - surgery | Aged | Mice | Nitric Oxide - metabolism | Index Medicus | Interferon-gamma | Reactive oxygen species | Apolipoprotein E | Interleukin 17 | Atherosclerosis
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2007, Volume 27, Issue 12, pp. 2589 - 2596
OBJECTIVE—Aortic calcification is prevalent in type II diabetes (T2DM), enhancing morbidity and tracking metabolic syndrome parameters. Ldlr mice fed high-fat... 
Aortic calcification | Diabetes | TNF-α | Metabolic syndrome | Wnt | TNF-alpha | BONE MORPHOGENETIC PROTEIN-2 | MATRIX GLA PROTEIN | VASCULAR CALCIFICATION | aortic calcification | DEFICIENT MICE | metabolic syndrome | INFLAMMATION | IN-VIVO | ARTERIAL CALCIFICATION | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | DIFFERENTIATION | HEMATOLOGY | diabetes | EXPRESSION | Inflammation - pathology | Fibroblasts - enzymology | Homeodomain Proteins - metabolism | Tumor Necrosis Factor-alpha - blood | Tumor Necrosis Factor-alpha - genetics | Antibodies, Monoclonal - therapeutic use | Male | Aorta - metabolism | Haptoglobins - metabolism | RNA, Messenger - metabolism | Wnt Proteins - metabolism | Aortic Diseases - metabolism | Bone Morphogenetic Proteins - metabolism | Inflammation - metabolism | Calcinosis - etiology | Dietary Fats - administration & dosage | Anti-Inflammatory Agents - therapeutic use | Aortic Diseases - prevention & control | Disease Models, Animal | Fibroblasts - metabolism | Anti-Inflammatory Agents - pharmacology | Antibodies, Monoclonal - pharmacology | Receptors, LDL - metabolism | Mice, Transgenic | Mice, Knockout | Aorta - pathology | Muscle Proteins - genetics | Signal Transduction - drug effects | Wnt3 Protein | Calcinosis - prevention & control | Inflammation - prevention & control | Mice | Diabetes Mellitus, Type 2 - pathology | Calcinosis - pathology | Wnt3A Protein | Tumor Necrosis Factor-alpha - metabolism | Alkaline Phosphatase | Diabetes Mellitus, Type 2 - metabolism | DNA-Binding Proteins - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Wnt Proteins - genetics | Infliximab | Receptors, LDL - deficiency | Calcinosis - metabolism | Microfilament Proteins - genetics | Aorta - enzymology | Diabetes Mellitus, Type 2 - complications | Aortic Diseases - pathology | Promoter Regions, Genetic | Receptors, LDL - genetics | Bone Morphogenetic Protein 2 | Mice, Inbred C57BL | Cells, Cultured | DNA-Binding Proteins - genetics | Homeodomain Proteins - genetics | Animals | Diabetes Mellitus, Type 2 - chemically induced | Transforming Growth Factor beta - metabolism | Aortic Diseases - etiology | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2017, Volume 37, Issue 11, pp. 2053 - 2063
OBJECTIVE—Experimental studies suggest that maternal hypercholesterolemia may be relevant for the early onset of cardiovascular disease in offspring. We... 
Apolipoproteins E | Hypercholesterolemia | Atherosclerosis | Trimethylamine-N-oxide | Bile | atherosclerosis | ATHEROGENESIS | RISK-FACTORS | SUSCEPTIBILITY | LOW-DENSITY-LIPOPROTEIN | DEFICIENT MICE | bile | hypercholesterolemia | PREGNANCY | trimethylamine-N-oxide | MATERNAL-HYPERCHOLESTEROLEMIA | apolipoproteins E | PERIPHERAL VASCULAR DISEASE | ACCUMULATION | DYSLIPIDEMIA | HEMATOLOGY | EXPRESSION | Prenatal Exposure Delayed Effects | Age Factors | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Male | Aorta - metabolism | Aortic Diseases - metabolism | Atherosclerosis - etiology | DNA Methylation | Oxygenases - metabolism | Female | Aortic Diseases - pathology | Disease Models, Animal | Methylamines - metabolism | Animals, Newborn | Promoter Regions, Genetic | Receptors, LDL - genetics | Atherosclerosis - pathology | Genetic Predisposition to Disease | Gallbladder - metabolism | Liver - metabolism | Receptors, LDL - metabolism | Bile Acids and Salts - metabolism | Scavenger Receptors, Class B - genetics | Receptors, Cytoplasmic and Nuclear - genetics | Cholesterol - metabolism | Plaque, Atherosclerotic | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Pregnancy | Cholesterol 7-alpha-Hydroxylase - genetics | Aortic Diseases - genetics | Phenotype | Animals | Cholesterol 7-alpha-Hydroxylase - metabolism | Apolipoproteins E - genetics | Hypercholesterolemia - complications | Hypercholesterolemia - genetics | Oxygenases - genetics | Scavenger Receptors, Class B - metabolism | Hypercholesterolemia - metabolism | Aortic Diseases - etiology | Receptors, Cytoplasmic and Nuclear - metabolism | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 06/2016, Volume 36, Issue 6, pp. 1090 - 1100
OBJECTIVE—Hyperlipidemia-induced endothelial cell (EC) activation is considered as an initial event responsible for monocyte recruitment in atherogenesis.... 
atherosclerosis | hyperlipidemia | endothelial cell | mitochondria | reactive oxygen species | PROGENITOR CELLS | VARESPLADIB | PHOSPHOLIPASE A INHIBITOR | CARDIOVASCULAR EVENTS | RANDOMIZED CLINICAL-TRIAL | ACUTE CORONARY SYNDROME | HEART-DISEASE | INFLAMMATION | PERIPHERAL VASCULAR DISEASE | MICE | ATHEROSCLEROTIC PLAQUE | HEMATOLOGY | Hyperlipidemias - genetics | Reactive Oxygen Species - metabolism | Apolipoproteins E - deficiency | Transcription Factor AP-1 - genetics | Atherosclerosis - genetics | Humans | Aorta - metabolism | Transcription Factor AP-1 - metabolism | Aortic Diseases - metabolism | Time Factors | Membrane Potential, Mitochondrial | Calcium Signaling | Aortic Diseases - pathology | Disease Models, Animal | Atherosclerosis - pathology | Genetic Predisposition to Disease | Lysophosphatidylcholines - pharmacology | Hyperlipidemias - metabolism | Oxidation-Reduction | Endothelial Cells - metabolism | Aorta - drug effects | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Lysophosphatidylcholines - metabolism | Gene Expression Profiling - methods | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - drug effects | Metabolomics - methods | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Aortic Diseases - genetics | Intercellular Adhesion Molecule-1 - metabolism | Phenotype | Animals | Apolipoproteins E - genetics | Intercellular Adhesion Molecule-1 - genetics | Oxidative Stress - drug effects | Endothelial Cells - drug effects | Index Medicus | vascular inflammation
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2013, Volume 33, Issue 1, pp. 121 - 130
OBJECTIVE—Inflammatory activation of valve endothelium is an early phase of aortic valve disease pathogenesis, but subsequent mechanisms are poorly understood.... 
calcification | invasion | transforming growth factor-β | smooth muscle actin | tumor necrosis factor-α | PROGENITOR CELLS | ENDOCARDIAL CUSHION TISSUE | ATHEROSCLEROSIS | FACTOR-KAPPA-B | INTERSTITIAL-CELLS | transforming growth factor-beta | tumor necrosis factor-alpha | HEART | ARTERIAL | GROWTH | RECEPTOR ACTIVATOR | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Heart Valve Diseases - pathology | Humans | Transforming Growth Factor beta1 - metabolism | NF-kappa B - metabolism | Epithelial-Mesenchymal Transition - genetics | Heart Valve Diseases - immunology | Aortic Valve - pathology | Inflammation - metabolism | Transfection | Time Factors | Swine | Inflammation Mediators - metabolism | Calcinosis - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Interleukin-6 - metabolism | Protein-Serine-Threonine Kinases - metabolism | Aortic Valve - immunology | Recombinant Proteins - metabolism | Signal Transduction | Endothelial Cells - metabolism | Aortic Valve - embryology | Cells, Cultured | Gene Expression Regulation | Activin Receptors, Type I - metabolism | Inflammation - immunology | Quail | Aortic Valve - metabolism | Chick Embryo | Endothelial Cells - immunology | Animals | NF-kappa B - genetics | Receptors, Transforming Growth Factor beta - metabolism | Heart Valve Diseases - metabolism | Heart Valve Diseases - genetics | Inflammation - genetics | Calcinosis - immunology | Primary Cell Culture | Calcinosis - pathology | Endothelial Cells - pathology | Cell Movement | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 05/2010, Volume 30, Issue 5, pp. 968 - 975
OBJECTIVE—To determine if niacin can confer cardiovascular benefit by inhibiting vascular inflammation and improving endothelial function independent of... 
Endothelial dysfunction | Niacin | Inflammation | METABOLIC SYNDROME | CHOLESTEROL | NICOTINIC-ACID RECEPTOR | HYPOCHLOROUS ACID | niacin | ATHEROSCLEROSIS | FACTOR-KAPPA-B | endothelial dysfunction | inflammation | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | A-I | EXTENDED-RELEASE NIACIN | HIGH-DENSITY-LIPOPROTEINS | Carotid Artery Diseases - prevention & control | Free Radical Scavengers - pharmacology | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Reactive Oxygen Species - metabolism | Glutathione - metabolism | Carotid Artery Diseases - physiopathology | Endothelium, Vascular - drug effects | Niacin - pharmacology | Aortic Diseases - metabolism | Dose-Response Relationship, Drug | Inflammation - metabolism | Carotid Artery Diseases - pathology | Lipids - blood | Aortic Diseases - prevention & control | Chemokine CCL2 - metabolism | Carotid Artery Diseases - metabolism | Neutrophil Infiltration - drug effects | Aortic Diseases - pathology | Disease Models, Animal | Rabbits | Vasodilator Agents - pharmacology | Oxidation-Reduction | Anti-Inflammatory Agents - pharmacology | Endothelium, Vascular - physiopathology | Aortic Diseases - physiopathology | Intercellular Adhesion Molecule-1 - metabolism | Animals | Cyclic GMP - metabolism | Endothelium, Vascular - metabolism | Endothelium, Vascular - pathology | Inflammation - prevention & control | Vasodilation - drug effects | Vascular Cell Adhesion Molecule-1 - metabolism | Inflammation - physiopathology | Peroxidase - metabolism | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 06/2017, Volume 37, Issue 6, pp. 1068 - 1075
Objective-CETP (cholesteryl ester transfer protein) plays an important role in lipoprotein metabolism; however, whether inhibition of CETP activity can prevent... 
atherosclerosis | apolipoprotein | cholesteryl ester transfer protein genetics | cholesterol reduction | high-density lipoprotein | FAMILIAL HYPERCHOLESTEROLEMIA | NEW-ZEALAND WHITE | TORCETRAPIB | ZINC-FINGER NUCLEASES | HERITABLE HYPERLIPIDEMIC RABBITS | HIGH-DENSITY-LIPOPROTEIN | GENE | PERIPHERAL VASCULAR DISEASE | CETP ACTIVITY | CORONARY-HEART-DISEASE | HEMATOLOGY | TRANSGENIC RABBITS | Lipid Metabolism, Inborn Errors - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Atherosclerosis - genetics | Male | Apolipoproteins B - blood | Aortic Diseases - metabolism | Cholesterol Ester Transfer Proteins - blood | Time Factors | Cholesterol Ester Transfer Proteins - genetics | Coronary Artery Disease - pathology | Aortic Diseases - prevention & control | Female | Aortic Diseases - pathology | Disease Models, Animal | Cell Line | Rabbits | Atherosclerosis - pathology | Cholesterol Ester Transfer Proteins - deficiency | Human Umbilical Vein Endothelial Cells - drug effects | Lipid Metabolism, Inborn Errors - metabolism | Animals, Genetically Modified | Lipid Metabolism, Inborn Errors - blood | Coronary Artery Disease - metabolism | Coronary Artery Disease - prevention & control | E-Selectin - metabolism | Atherosclerosis - metabolism | Gene Editing | Tumor Necrosis Factor-alpha - pharmacology | Aortic Diseases - genetics | Macrophages - metabolism | Animals | Apolipoproteins E - blood | Coronary Artery Disease - genetics | Cholesterol, HDL - blood | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Cholesterol Ester Transfer Proteins - metabolism | Cholesterol, Dietary | Atherosclerosis - prevention & control | Apolipoprotein A-I - blood | Index Medicus
Journal Article