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Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 01/2013, Volume 33, Issue 1, pp. 121 - 130
OBJECTIVE—Inflammatory activation of valve endothelium is an early phase of aortic valve disease pathogenesis, but subsequent mechanisms are poorly understood... 
calcification | invasion | transforming growth factor-β | smooth muscle actin | tumor necrosis factor-α | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Heart Valve Diseases - pathology | Humans | Transforming Growth Factor beta1 - metabolism | NF-kappa B - metabolism | Epithelial-Mesenchymal Transition - genetics | Heart Valve Diseases - immunology | Aortic Valve - pathology | Inflammation - metabolism | Transfection | Time Factors | Swine | Inflammation Mediators - metabolism | Calcinosis - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Interleukin-6 - metabolism | Protein-Serine-Threonine Kinases - metabolism | Aortic Valve - immunology | Recombinant Proteins - metabolism | Signal Transduction | Endothelial Cells - metabolism | Aortic Valve - embryology | Cells, Cultured | Gene Expression Regulation | Activin Receptors, Type I - metabolism | Inflammation - immunology | Quail | Aortic Valve - metabolism | Chick Embryo | Endothelial Cells - immunology | Animals | NF-kappa B - genetics | Receptors, Transforming Growth Factor beta - metabolism | Heart Valve Diseases - metabolism | Heart Valve Diseases - genetics | Inflammation - genetics | Calcinosis - immunology | Primary Cell Culture | Calcinosis - pathology | Endothelial Cells - pathology | Cell Movement | Index Medicus | transforming growth factor-beta | tumor necrosis factor-alpha
Journal Article
Kidney international, ISSN 0085-2538, 12/2012, Volume 82, Issue 12, pp. 1261 - 1270
Vascular calcification is common in chronic kidney disease, where cardiovascular mortality remains the leading cause of death... 
klotho | chronic kidney disease | osteopontin | vitamin D | vascular calcification | Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Up-Regulation | Muscle, Smooth, Vascular - metabolism | Renal Insufficiency, Chronic - etiology | Glucuronidase - metabolism | Osteopontin - metabolism | Aorta - metabolism | Parathyroid Hormone - blood | Aortic Diseases - metabolism | Renal Insufficiency, Chronic - drug therapy | Renal Insufficiency, Chronic - metabolism | Calcitriol - administration & dosage | Calcium - blood | Vascular Calcification - metabolism | Time Factors | Vascular Calcification - etiology | Aortic Diseases - prevention & control | Phosphates - blood | Female | Mice, Inbred DBA | Myocytes, Smooth Muscle - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Elastin - metabolism | Aorta - drug effects | Mice, Inbred C57BL | Cells, Cultured | Injections, Intraperitoneal | Vascular Calcification - prevention & control | Glucuronidase - blood | Receptors, Calcitriol - metabolism | Vascular Calcification - pathology | Aorta - pathology | Glucuronidase - urine | Animals | Diet | Fibroblast Growth Factors - blood | Receptors, Calcitriol - agonists | Calcitriol - pharmacology | Ergocalciferols - pharmacology | Ergocalciferols - administration & dosage | Mice | Aortic Diseases - etiology | Index Medicus | Calcification (ectopic) | Urine | Cell culture | Animal models | Calcium | Secretion | Elastin | Mortality | Calcitriol | Osteopontin | Diets | Klotho protein | Parathyroid hormone | Phosphate | Hyperphosphatemia | Aorta | Kidney diseases | Vitamin D receptors | Cardiovascular diseases | Vascular calcification
Journal Article
EMBO molecular medicine, ISSN 1757-4676, 03/2013, Volume 5, Issue 3, pp. 471 - 481
We used a novel approach of cytostatically induced leucocyte depletion and subsequent reconstitution with leucocytes deprived of classical (inflammatory/Gr1hi)... 
atherosclerosis | monocyte | chemokine | mobilization | recruitment | Chemokine | Mobilization | Monocyte | Atherosclerosis | Recruitment | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Carotid Arteries - metabolism | Chemotaxis, Leukocyte | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Male | Monocytes - immunology | Adoptive Transfer | Aorta - metabolism | Aortic Diseases - metabolism | Atherosclerosis - etiology | Receptors, CCR5 - metabolism | Monocytes - transplantation | Leukocyte Reduction Procedures | Time Factors | Carotid Artery Diseases - pathology | Diet, High-Fat | Leukocyte Count | Chemokine CXCL1 - metabolism | Monocytes - classification | Receptors, Chemokine - genetics | Carotid Artery Diseases - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Carotid Artery Diseases - immunology | Atherosclerosis - pathology | Atherosclerosis - immunology | Carotid Artery Diseases - etiology | Signal Transduction | Aortic Diseases - immunology | Mice, Inbred C57BL | Receptors, Chemokine - metabolism | Aorta - immunology | Atherosclerosis - metabolism | Disease Progression | Mice, Knockout | Receptors, Chemokine - deficiency | Aorta - pathology | Aortic Diseases - genetics | Animals | Apolipoproteins E - genetics | Receptors, Interleukin-8B - metabolism | Carotid Arteries - immunology | Carotid Artery Diseases - genetics | Receptors, CCR1 - metabolism | Carotid Arteries - pathology | Mice | Aortic Diseases - etiology | Apoptosis | Hypercholesterolemia | Universities and colleges | CC chemokine receptors | Funding | CX3CR1 protein | Leukocytes | Macrophages | Adoptive transfer | Blood | High fat diet | CCR1 protein | CCR2 protein | Apolipoprotein E | Bone marrow | Carotid artery | CCR5 protein | Nutrient deficiency | CXCR2 protein | Emigration | Inflammation | Monocytosis | Studies | Monocytes | Microscopy | Arteriosclerosis | Software | Chemokines | Monocyte chemoattractant protein 1 | Index Medicus
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 12/2007, Volume 27, Issue 12, pp. 2589 - 2596
OBJECTIVE—Aortic calcification is prevalent in type II diabetes (T2DM), enhancing morbidity and tracking metabolic syndrome parameters. Ldlr mice fed high-fat... 
Aortic calcification | Diabetes | TNF-α | Metabolic syndrome | Wnt | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Inflammation - pathology | Fibroblasts - enzymology | Homeodomain Proteins - metabolism | Tumor Necrosis Factor-alpha - blood | Tumor Necrosis Factor-alpha - genetics | Antibodies, Monoclonal - therapeutic use | Male | Aorta - metabolism | Haptoglobins - metabolism | RNA, Messenger - metabolism | Wnt Proteins - metabolism | Aortic Diseases - metabolism | Bone Morphogenetic Proteins - metabolism | Inflammation - metabolism | Calcinosis - etiology | Dietary Fats - administration & dosage | Anti-Inflammatory Agents - therapeutic use | Aortic Diseases - prevention & control | Disease Models, Animal | Fibroblasts - metabolism | Anti-Inflammatory Agents - pharmacology | Antibodies, Monoclonal - pharmacology | Receptors, LDL - metabolism | Mice, Transgenic | Mice, Knockout | Aorta - pathology | Muscle Proteins - genetics | Signal Transduction - drug effects | Wnt3 Protein | Calcinosis - prevention & control | Inflammation - prevention & control | Mice | Diabetes Mellitus, Type 2 - pathology | Calcinosis - pathology | Wnt3A Protein | Tumor Necrosis Factor-alpha - metabolism | Alkaline Phosphatase | Diabetes Mellitus, Type 2 - metabolism | DNA-Binding Proteins - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Wnt Proteins - genetics | Infliximab | Receptors, LDL - deficiency | Calcinosis - metabolism | Microfilament Proteins - genetics | Aorta - enzymology | Diabetes Mellitus, Type 2 - complications | Aortic Diseases - pathology | Promoter Regions, Genetic | Receptors, LDL - genetics | Bone Morphogenetic Protein 2 | Mice, Inbred C57BL | Cells, Cultured | DNA-Binding Proteins - genetics | Homeodomain Proteins - genetics | Animals | Diabetes Mellitus, Type 2 - chemically induced | Transforming Growth Factor beta - metabolism | Aortic Diseases - etiology | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Index Medicus
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 04/2009, Volume 119, Issue 13, pp. 1785 - 1794
Background-Clinical studies have demonstrated that 50% of individuals with chronic renal disease (CRD... 
Calcification | Chronic | Kidney failure | Aortic valve | Elastin | Atherosclerosis | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Tunica Media - pathology | Osteogenesis - physiology | Cystatin C - blood | Humans | Carotid Artery Diseases - physiopathology | Aorta - metabolism | Aortic Valve - pathology | Nephrectomy | Carotid Artery Diseases - pathology | Mice, Mutant Strains | Tunica Media - metabolism | Phosphates - blood | Calcinosis - metabolism | Carotid Artery Diseases - metabolism | Phosphates - pharmacology | Muscle, Smooth, Vascular - physiology | Kidney Failure, Chronic - physiopathology | Elastin - metabolism | Mice, Inbred C57BL | Osteogenesis - drug effects | Aortic Valve Stenosis - physiopathology | Cells, Cultured | Elastin - pharmacology | Muscle, Smooth, Vascular - cytology | Aortic Valve - metabolism | Kidney Failure, Chronic - metabolism | Aorta - pathology | Tunica Intima - metabolism | Animals | Aortic Valve Stenosis - pathology | Tunica Intima - pathology | Cathepsins - genetics | Cathepsins - metabolism | Apolipoproteins E - genetics | Creatinine - blood | Aortic Valve Stenosis - metabolism | Mice | Calcinosis - pathology | Cathepsins - deficiency | Calcinosis - physiopathology | Cathepsins | Physiological aspects | Research | Chronic kidney failure | Diseases | Index Medicus | Abridged Index Medicus | atherosclerosis | kidney failure, chronic | aortic valve | calcification | elastin
Journal Article
Cardiovascular research, ISSN 1755-3245, 12/2015, Volume 108, Issue 3, pp. 377 - 386
Neointimal hyperplasia is a common feature of fibro-proliferative vascular disease and characterizes initial stages of atherosclerosis... 
ARTERIAL INJURY | MYOCARDIN | PULMONARY-HYPERTENSION | Neointimal hyperplasia | ATHEROSCLEROSIS | CADHERIN EXPRESSION | SMOOTH-MUSCLE-CELL | FLOW | Fibrosis | ERK5 | IN-VIVO | GROWTH | Shear stress | GENE-EXPRESSION | Endothelial-to-mesenchymal transition | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Carotid Arteries - metabolism | Vascular Remodeling | Cell Proliferation | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Carotid Artery Diseases - physiopathology | Stress, Mechanical | Male | Aortic Diseases - metabolism | Neointima | Transfection | RNA Interference | Time Factors | Carotid Artery Diseases - pathology | Swine | MAP Kinase Kinase 5 - metabolism | Epithelial-Mesenchymal Transition | HEK293 Cells | Carotid Artery Diseases - metabolism | Aorta, Thoracic - pathology | Aortic Diseases - pathology | Disease Models, Animal | Endothelial Cells - metabolism | Mice, Inbred C57BL | Aorta, Thoracic - metabolism | Mitogen-Activated Protein Kinase 7 - genetics | MAP Kinase Kinase 5 - genetics | Mitogen-Activated Protein Kinase 7 - metabolism | Aorta, Thoracic - physiopathology | Plaque, Atherosclerotic | Regional Blood Flow | Mechanotransduction, Cellular | Aortic Diseases - genetics | Aortic Diseases - physiopathology | Animals | Carotid Arteries - physiopathology | Carotid Artery Diseases - genetics | Human Umbilical Vein Endothelial Cells - pathology | Carotid Arteries - pathology | Endothelial Cells - pathology | Index Medicus
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 02/2015, Volume 35, Issue 2, pp. 399 - 408
OBJECTIVE—Vascular and valvular calcifications are pathological processes regulated by resident cells, and depending on a complex interplay between calcification promoters and inhibitors, resembling skeletal metabolism... 
multivesicular bodies | aortic valve, calcification of | gene expression | vascular calcification | aortic valve | calcification of | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Calcinosis - genetics | Aortic Valve Stenosis - genetics | Calcium - metabolism | Humans | Middle Aged | Vascular Calcification - genetics | Actins - metabolism | Osteocalcin - genetics | Male | Aorta - metabolism | Case-Control Studies | Aortic Valve - pathology | Coronary Vessels - metabolism | Aortic Valve Stenosis - prevention & control | Vascular Calcification - metabolism | Coronary Artery Disease - pathology | Aged, 80 and over | Adult | Female | Calcinosis - metabolism | Extracellular Matrix Proteins - metabolism | Calcium-Binding Proteins - metabolism | Coronary Vessels - pathology | Osteocalcin - metabolism | Tissue Culture Techniques | Coronary Artery Disease - metabolism | Extracellular Matrix Proteins - genetics | Coronary Artery Disease - prevention & control | Gene Expression Regulation | Vascular Calcification - prevention & control | Vascular Calcification - pathology | Aortic Valve - metabolism | Aorta - pathology | Proteins - genetics | Aortic Valve Stenosis - pathology | Proteins - metabolism | Coronary Artery Disease - genetics | alpha-2-HS-Glycoprotein - metabolism | Aortic Valve Stenosis - metabolism | Aged | Calcinosis - prevention & control | Calcinosis - pathology | Calcium-Binding Proteins - genetics | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, pp. e68197 - e68197
Free fatty acids (FFAs), elevated in metabolic syndrome and diabetes, play a crucial role in the development of atherosclerotic cardiovascular disease, and eicosapentaenoic acid (EPA... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Calcinosis - genetics | Carotid Arteries - metabolism | Osteopontin - genetics | Palmitic Acid - metabolism | Coenzyme A Ligases - genetics | Muscle, Smooth, Vascular - metabolism | Calcium - metabolism | Gene Expression - genetics | Homeodomain Proteins - metabolism | Humans | Eicosapentaenoic Acid - genetics | Myocytes, Smooth Muscle - pathology | NF-kappa B - metabolism | Osteopontin - metabolism | Aorta - metabolism | Promoter Regions, Genetic - genetics | Coenzyme A Ligases - metabolism | Cell Differentiation - genetics | Caspases - metabolism | Bone Morphogenetic Protein 2 - metabolism | Calcinosis - metabolism | Plaque, Atherosclerotic - pathology | Cell Differentiation - physiology | Myocytes, Smooth Muscle - metabolism | Bone Morphogenetic Protein 2 - genetics | Macrophages - pathology | Plaque, Atherosclerotic - metabolism | Caspases - genetics | Cells, Cultured | Eicosapentaenoic Acid - metabolism | Homeodomain Proteins - genetics | Aorta - pathology | Osteoblasts - pathology | Macrophages - metabolism | Muscle, Smooth, Vascular - pathology | NF-kappa B - genetics | Carotid Arteries - pathology | Calcinosis - pathology | Osteoblasts - metabolism | Immunohistochemistry | Smooth muscle | Cardiovascular disease | Palmitic acid | Osteoblasts | Proteins | Mineralization | Biocompatibility | Aorta | Inhibition | NF-κB protein | Cytokines | Osteopontin | Metabolism | Gene expression | Fatty acids | Studies | Osteoblastogenesis | Inhibitors | Arteriosclerosis | Calcification | Cardiovascular diseases | Metabolic disorders | Calcification (ectopic) | Health sciences | Oxidative stress | Calcium | Laboratories | Chains | Lipids | Activation | Kinases | Macrophages | Carotid arteries | Arteries | Msx2 protein | Rodents | Atherosclerosis | Tumor necrosis factor-TNF | Carotid artery | Deposition | Plaques | University graduates | Bone morphogenetic protein 2 | Diabetes mellitus | Muscles | Caspase | siRNA | Medicine | Antibiotics | Kidney diseases | Diabetes | Differentiation | Eicosapentaenoic acid | Apoptosis | Pharmaceuticals | Index Medicus
Journal Article