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Circulation, ISSN 0009-7322, 06/2015, Volume 131, Issue 24, pp. 2104 - 2113
Background-Data are limited on the presence, distribution, and extent of subclinical atherosclerosis in middle-aged populations. Methods and Results-The PESA... 
Population | Epidemiology | Risk assessment | Atherosclerosis | Ultrasonography | Multidetector computed tomography | atherosclerosis | YOUNG-ADULTS | risk assessment | DESIGN | CARDIAC & CARDIOVASCULAR SYSTEMS | EVENTS | ultrasonography | RISK | PLAQUE | population | epidemiology | multidetector computed tomography | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | HEALTH | CORONARY-HEART-DISEASE | ARTERY CALCIUM | ASSOCIATION | Aortic Diseases - diagnostic imaging | Femoral Artery - pathology | Iliac Artery - pathology | Plaque, Atherosclerotic - diagnostic imaging | Prevalence | Prospective Studies | Age Factors | Follow-Up Studies | Ankle Brachial Index | Humans | Middle Aged | Calcinosis - diagnostic imaging | Male | Plaque, Atherosclerotic - epidemiology | Asymptomatic Diseases | Atherosclerosis - epidemiology | Aorta, Abdominal - diagnostic imaging | Atherosclerosis - diagnostic imaging | Carotid Artery Diseases - diagnostic imaging | Carotid Artery Diseases - pathology | Coronary Artery Disease - pathology | Adult | Female | Aorta, Abdominal - pathology | Aortic Diseases - pathology | Multidetector Computed Tomography | Atherosclerosis - pathology | Aortic Diseases - epidemiology | Comorbidity | Risk Factors | Carotid Artery Diseases - epidemiology | Disease Progression | Calcinosis - epidemiology | Coronary Angiography | Coronary Artery Disease - diagnostic imaging | Femoral Artery - diagnostic imaging | Iliac Artery - diagnostic imaging | Spain - epidemiology | Coronary Artery Disease - epidemiology | Aortography | Calcinosis - pathology | Index Medicus | Abridged Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 02/2014, Volume 129, Issue 6, pp. 673 - 682
Journal Article
British Journal of Dermatology, ISSN 0007-0963, 12/2015, Volume 173, Issue 6, pp. 1505 - 1513
Journal Article
Cardiovascular Research, ISSN 0008-6363, 12/2015, Volume 108, Issue 3, pp. 377 - 386
Neointimal hyperplasia is a common feature of fibro-proliferative vascular disease and characterizes initial stages of atherosclerosis. Neointimal lesions... 
Shear stress | Endothelial-to-mesenchymal transition | Neointimal hyperplasia | ERK5 | Fibrosis | ARTERIAL INJURY | MYOCARDIN | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | ATHEROSCLEROSIS | MECHANISMS | FLOW | GROWTH | SMOOTH-MUSCLE-CELLS | EXPRESSION | TRANSDIFFERENTIATION | Carotid Arteries - metabolism | Vascular Remodeling | Cell Proliferation | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Carotid Artery Diseases - physiopathology | Stress, Mechanical | Male | Aortic Diseases - metabolism | Neointima | Transfection | RNA Interference | Time Factors | Carotid Artery Diseases - pathology | Swine | MAP Kinase Kinase 5 - metabolism | Epithelial-Mesenchymal Transition | HEK293 Cells | Carotid Artery Diseases - metabolism | Aorta, Thoracic - pathology | Aortic Diseases - pathology | Disease Models, Animal | Endothelial Cells - metabolism | Mice, Inbred C57BL | Aorta, Thoracic - metabolism | Mitogen-Activated Protein Kinase 7 - genetics | MAP Kinase Kinase 5 - genetics | Mitogen-Activated Protein Kinase 7 - metabolism | Aorta, Thoracic - physiopathology | Plaque, Atherosclerotic | Regional Blood Flow | Mechanotransduction, Cellular | Aortic Diseases - genetics | Aortic Diseases - physiopathology | Animals | Carotid Arteries - physiopathology | Carotid Artery Diseases - genetics | Human Umbilical Vein Endothelial Cells - pathology | Carotid Arteries - pathology | Endothelial Cells - pathology | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 04/2009, Volume 119, Issue 13, pp. 1785 - 1794
Background-Clinical studies have demonstrated that 50% of individuals with chronic renal disease (CRD) die of cardiovascular causes, including advanced... 
Calcification | Chronic | Kidney failure | Aortic valve | Elastin | Atherosclerosis | atherosclerosis | kidney failure, chronic | aortic valve | ELASTIN PEPTIDES | CARDIAC & CARDIOVASCULAR SYSTEMS | ATHEROSCLEROTIC PLAQUES | VASCULAR CALCIFICATION | elastin | POTENTIAL MECHANISM | FAILURE | IN-VIVO | CYSTATIN-C | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | calcification | CHRONIC KIDNEY-DISEASE | VULNERABLE PLAQUE | Tunica Media - pathology | Osteogenesis - physiology | Cystatin C - blood | Humans | Carotid Artery Diseases - physiopathology | Aorta - metabolism | Aortic Valve - pathology | Nephrectomy | Carotid Artery Diseases - pathology | Mice, Mutant Strains | Tunica Media - metabolism | Phosphates - blood | Calcinosis - metabolism | Carotid Artery Diseases - metabolism | Phosphates - pharmacology | Muscle, Smooth, Vascular - physiology | Kidney Failure, Chronic - physiopathology | Elastin - metabolism | Mice, Inbred C57BL | Osteogenesis - drug effects | Aortic Valve Stenosis - physiopathology | Cells, Cultured | Elastin - pharmacology | Muscle, Smooth, Vascular - cytology | Aortic Valve - metabolism | Kidney Failure, Chronic - metabolism | Aorta - pathology | Tunica Intima - metabolism | Animals | Aortic Valve Stenosis - pathology | Tunica Intima - pathology | Cathepsins - genetics | Cathepsins - metabolism | Apolipoproteins E - genetics | Creatinine - blood | Aortic Valve Stenosis - metabolism | Mice | Calcinosis - pathology | Cathepsins - deficiency | Calcinosis - physiopathology | Cathepsins | Physiological aspects | Research | Chronic kidney failure | Diseases | Index Medicus | Abridged Index Medicus
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 2014, Volume 34, Issue 8, pp. 1674 - 1680
Atherothrombosis is the main cause of myocardial infarction and ischemic stroke. Although the extrinsic (tissue factor-factor VIIa [FVIIa]) pathway is... 
Blood coagulation | Fibrin | Plaque rupture | Blood platelets | Thrombus | Tissue factor | Factor XII | blood platelets | ACTIVATION | tissue factor | blood coagulation | plaque rupture | thrombus | COAGULATION-FACTOR-XII | INHIBITION | PLASMA | HUMAN ATHEROSCLEROTIC PLAQUES | IN-VIVO | PERIPHERAL VASCULAR DISEASE | PLATELET GLYCOPROTEIN-VI | GENERATION | PROVIDES PROTECTION | fibrin | HEMATOLOGY | factor XII | Carotid Arteries - metabolism | Factor XIIa - metabolism | Apolipoproteins E - deficiency | Factor XII Deficiency - blood | Atherosclerosis - genetics | Humans | Aortic Diseases - blood | Carotid Artery Diseases - complications | Time Factors | Carotid Artery Diseases - pathology | Blood Coagulation | Factor XII Deficiency - genetics | Thrombosis - blood | Aorta, Thoracic - pathology | Aortic Diseases - pathology | Disease Models, Animal | Atherosclerosis - pathology | Factor XII - genetics | Mice, Inbred C57BL | Aorta, Thoracic - metabolism | Atherosclerosis - complications | Plaque, Atherosclerotic | Factor VIIa - metabolism | Mice, Knockout | Atherosclerosis - blood | Thrombosis - pathology | Aortic Diseases - genetics | Animals | Blood Platelets - metabolism | Thrombosis - etiology | Apolipoproteins E - genetics | Carotid Artery Diseases - blood | Factor XII - metabolism | Carotid Artery Diseases - genetics | Aortic Diseases - complications | Thrombosis - genetics | Carotid Arteries - pathology | Mice | Factor XI - metabolism | Thromboplastin - metabolism | Cholesterol, Dietary | Rupture, Spontaneous | Index Medicus
Journal Article
Circulation: Cardiovascular Imaging, ISSN 1941-9651, 07/2013, Volume 6, Issue 4, pp. 499 - 507
Background Ascending aortic dilation is important in bicuspid aortic valve (BAV) disease, with increased risk of aortic dissection. We used cardiovascular MR... 
Aorta | Cardiac MRI | Wall shear stress | Vascular function | Bicuspid aortic valve | vascular function | WALL SHEAR-STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | cardiac MRI | bicuspid aortic valve | ELASTIC PROPERTIES | wall shear stress | aorta | NATURAL-HISTORY | BLOOD-FLOW | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | Predictive Value of Tests | Prognosis | Prospective Studies | Heart Valve Diseases - pathology | Pulse Wave Analysis | Aortic Valve - abnormalities | Humans | Middle Aged | Elasticity | Stress, Mechanical | Male | Aortic Aneurysm - etiology | Case-Control Studies | Aortic Valve - pathology | Young Adult | Aneurysm, Dissecting - physiopathology | Cardiac-Gated Imaging Techniques - methods | Heart Valve Diseases - complications | Adult | Female | Aorta - physiopathology | Aortic Aneurysm - pathology | Imaging, Three-Dimensional | Vascular Stiffness | Dilatation, Pathologic | Aneurysm, Dissecting - etiology | Severity of Illness Index | Heart Valve Diseases - physiopathology | Cross-Sectional Studies | Models, Cardiovascular | Risk Factors | Aortic Aneurysm - physiopathology | Aortic Valve - physiopathology | Aneurysm, Dissecting - pathology | Chi-Square Distribution | Regional Blood Flow | Aorta - pathology | Magnetic Resonance Imaging | Analysis of Variance | Hemodynamics | Index Medicus | cardiac magnetic resonance imaging
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 11/2012, Volume 32, Issue 11, pp. 2580 - 2588
OBJECTIVE—Vascular calcification significantly increases cardiovascular morbidity and mortality. We recently reported that the deficiency of cartilage... 
Smooth muscle cell | Calcification | Animal model of human disease remodeling | MicroRNA | Protease | animal model of human disease remodeling | CELLS | smooth muscle cell | PATHOBIOLOGY | MECHANISMS | OLIGOMERIC MATRIX PROTEIN | protease | PHOSPHATE | IN-VITRO | ROLES | ARTERIAL CALCIFICATION | ANEURYSM DEVELOPMENT | microRNA | PERIPHERAL VASCULAR DISEASE | calcification | CHRONIC KIDNEY-DISEASE | HEMATOLOGY | Up-Regulation | Carotid Artery Diseases - chemically induced | Humans | MicroRNAs - metabolism | Matrilin Proteins | Nephrectomy | RNA Interference | Time Factors | Carotid Artery Diseases - pathology | Transcription, Genetic | 3' Untranslated Regions | Real-Time Polymerase Chain Reaction | Organ Culture Techniques | Disease Models, Animal | Uremia - enzymology | Myocytes, Smooth Muscle - enzymology | Down-Regulation | Computational Biology | Rats | Vascular Calcification - pathology | Rats, Sprague-Dawley | Kidney Failure, Chronic - genetics | ADAM Proteins - metabolism | Aortic Diseases - genetics | Carotid Artery Diseases - enzymology | Carotid Artery, Common - enzymology | Carotid Artery Diseases - genetics | Kidney Failure, Chronic - complications | ADAMTS7 Protein | Carotid Artery, Common - pathology | ADAM Proteins - genetics | Muscle, Smooth, Vascular - enzymology | Vascular Calcification - genetics | Glycoproteins - metabolism | Myocytes, Smooth Muscle - pathology | Aortic Diseases - chemically induced | Transfection | Uremia - pathology | Female | Vascular Calcification - enzymology | Aortic Diseases - enzymology | Aorta, Abdominal - pathology | Kidney Failure, Chronic - enzymology | Extracellular Matrix Proteins - metabolism | Aortic Diseases - pathology | Promoter Regions, Genetic | Calcium Chloride | Cartilage Oligomeric Matrix Protein | Cells, Cultured | Aorta, Abdominal - enzymology | Gene Expression Regulation, Enzymologic | Muscle, Smooth, Vascular - pathology | Animals | Vascular Calcification - chemically induced | Index Medicus
Journal Article
Science, ISSN 0036-8075, 4/2006, Volume 312, Issue 5770, pp. 117 - 121
Aortic aneurysm and dissection are manifestations of Marfan syndrome (MFS), a disorder caused by mutations in the gene that encodes fibrillin-1. Selected... 
Airspace | Receptors | Architecture | Dilatation | Root growth | Placebos | Reports | Mice | Aortic aneurysm | Elastic tissue | Marfan syndrome | PATHOGENESIS | GROWTH-FACTOR-BETA | ACTIVATION | FIBRILLIN | MULTIDISCIPLINARY SCIENCES | RATS | RECEPTOR | SMOOTH-MUSCLE-CELLS | BLOCKADE | CONTRIBUTES | DILATATION | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Propranolol - therapeutic use | Neutralization Tests | Aortic Aneurysm - etiology | Lung Diseases - drug therapy | Marfan Syndrome - drug therapy | Propranolol - administration & dosage | Antibodies - immunology | Transforming Growth Factor beta - antagonists & inhibitors | Female | Marfan Syndrome - complications | Microfilament Proteins - genetics | Adrenergic beta-Antagonists - administration & dosage | Adrenergic beta-Antagonists - therapeutic use | Angiotensin II Type 1 Receptor Blockers - administration & dosage | Aortic Aneurysm - prevention & control | Disease Models, Animal | Transforming Growth Factor beta - immunology | Fibrillin-1 | Lung - pathology | Pulmonary Alveoli - pathology | Signal Transduction | Fibrillins | Pregnancy Complications - drug therapy | Aorta - pathology | Pregnancy | Elastic Tissue - pathology | Animals | Receptor, Angiotensin, Type 1 - metabolism | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Losartan - administration & dosage | Lung Diseases - pathology | Mutation | Transforming Growth Factor beta - metabolism | Complications and side effects | Aortic aneurysms | Genetic aspects | Research | Risk factors | Signal transduction | Pharmacology | Models | Genetic disorders | Antagonist drugs | Rodents | Index Medicus
Journal Article
The American Journal of Human Genetics, ISSN 0002-9297, 05/2009, Volume 84, Issue 5, pp. 617 - 627
Journal Article
Journal Article