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Circulation Research, ISSN 0009-7330, 04/2015, Volume 116, Issue 8, pp. 1448 - 1461
Thoracic aortic diseases that involve progressive enlargement, acute dissection, or rupture are influenced by the hemodynamic loads and mechanical properties... 
Elastic fibers | Actomyosin | Marfan syndrome | Focal adhesion | focal adhesion | LIGHT-CHAIN KINASE | ASCENDING AORTA | DEVELOPING MOUSE AORTA | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOTENSIN-II | GROWTH-FACTOR-BETA | actomyosin | MARFAN-SYNDROME | ARTERIAL-WALL | elastic fibers | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | EXTRACELLULAR-MATRIX | HEMATOLOGY | EHLERS-DANLOS-SYNDROME | Aneurysm, Dissecting - genetics | Aortic Aneurysm, Thoracic - genetics | Humans | Stress, Mechanical | Aortic Rupture - metabolism | Aneurysm, Dissecting - physiopathology | Aortic Aneurysm, Thoracic - pathology | Aorta, Thoracic - pathology | Extracellular Matrix Proteins - metabolism | Aortic Rupture - physiopathology | Genetic Predisposition to Disease | Aneurysm, Dissecting - metabolism | Extracellular Matrix Proteins - genetics | Aorta, Thoracic - metabolism | Aortic Rupture - pathology | Aneurysm, Dissecting - pathology | Aneurysm, Dissecting - therapy | Aorta, Thoracic - physiopathology | Aortic Aneurysm, Thoracic - physiopathology | Disease Progression | Mechanotransduction, Cellular | Biomechanical Phenomena | Phenotype | Animals | Aortic Aneurysm, Thoracic - metabolism | Aortic Aneurysm, Thoracic - therapy | Aortic Rupture - genetics | Aortic Rupture - therapy | Hemodynamics | familial thoracic aortic aneurysms and dissections | mechanosensing
Journal Article
Circulation Research, ISSN 0009-7330, 02/2015, Volume 116, Issue 4, pp. 612 - 623
RATIONALE:In-hospital outcomes are generally acceptable in patients with type B dissection; however, some patients present with undesirable complications, such... 
Aortic dissection | Inflammation | Angiotensin II | Chemokines | Neutrophils | Interleukin-6 | CHEMOKINE RECEPTOR CXCR2 | ASCENDING AORTA | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTECTION | PORCINE CORONARY-ARTERIES | ANGIOTENSIN-II | INTERNATIONAL REGISTRY | chemokines | interleukin-6 | inflammation | angiotensin II | ANEURYSMS | HISTOLOGIC-CHANGES | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | aortic dissection | neutrophils | Chemotaxis, Leukocyte | Humans | Middle Aged | Male | Adventitia - metabolism | Time Factors | Aortic Rupture - diagnostic imaging | Chemokine CXCL1 - metabolism | Dilatation, Pathologic | Interleukin-6 - metabolism | Neutrophils - metabolism | Granulocyte Colony-Stimulating Factor - metabolism | Neutrophil Infiltration - drug effects | Disease Models, Animal | Aminopropionitrile - analogs & derivatives | Acute Disease | Interleukin-6 - genetics | Signal Transduction | Antibodies, Monoclonal - pharmacology | Neutrophils - drug effects | Aorta, Thoracic - metabolism | Mice, Knockout | Chemokine CXCL12 - metabolism | Aneurysm, Dissecting - diagnostic imaging | Aorta, Thoracic - diagnostic imaging | Aortic Aneurysm, Thoracic - diagnostic imaging | Neutrophil Activation - drug effects | Adventitia - diagnostic imaging | Aortic Aneurysm, Thoracic - drug therapy | Aortic Aneurysm, Thoracic - chemically induced | Aortic Rupture - metabolism | Receptors, Interleukin-8B - antagonists & inhibitors | Interleukin-6 - blood | Aneurysm, Dissecting - chemically induced | Aortic Rupture - prevention & control | Inflammation Mediators - metabolism | Female | Interleukin-8 - blood | Aneurysm, Dissecting - metabolism | Mice, Inbred C57BL | Aneurysm, Dissecting - drug therapy | Animals | Aortic Aneurysm, Thoracic - metabolism | Neutrophils - transplantation | Receptors, Interleukin-8B - metabolism | Aged | Aortic Rupture - chemically induced | Aortography
Journal Article
Journal Article
Journal of Vascular Surgery, ISSN 0741-5214, 12/2018, Volume 68, Issue 6, pp. 82S - 92S.e2
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2015, Volume 35, Issue 4, pp. 911 - 917
Objective-Studies of mice with mild Marfan syndrome (MFS) have correlated the development of thoracic aortic aneurysm (TAA) with improper stimulation of... 
aortic aneurysm | receptor, angiotensin, type 1 | losartan | transforming growth factor β | Marfan syndrome | PATHOGENESIS | TGF-BETA | type 1 | transforming growth factor beta | MOUSE MODEL | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | receptor, angiotensin | Aortic Aneurysm, Thoracic - genetics | Phosphorylation | Aortic Aneurysm, Thoracic - prevention & control | Humans | Aortic Rupture - metabolism | Mice, 129 Strain | Angiotensin II Type 1 Receptor Blockers - pharmacology | Marfan Syndrome - drug therapy | Aortic Aneurysm, Thoracic - pathology | Time Factors | Mice, Mutant Strains | Aortic Rupture - prevention & control | Transforming Growth Factor beta - antagonists & inhibitors | Aorta, Thoracic - drug effects | Microfilament Proteins - genetics | Aorta, Thoracic - pathology | Disease Models, Animal | Transforming Growth Factor beta - immunology | Fibrillin-1 | Mice, Inbred C57BL | Losartan - pharmacology | Smad2 Protein - metabolism | Aorta, Thoracic - metabolism | Antibodies, Neutralizing - pharmacology | Aortic Rupture - pathology | Fibrillins | Disease Progression | Marfan Syndrome - genetics | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Aortic Aneurysm, Thoracic - metabolism | Receptor, Angiotensin, Type 1 - metabolism | Aortic Rupture - genetics | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Mutation | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article
Cardiovascular research, ISSN 0008-6363, 2017, Volume 113, Issue 11, pp. 1364 - 1375
Journal Article
Science, ISSN 0036-8075, 4/2011, Volume 332, Issue 6027, pp. 361 - 365
Angiotensin II (AngII) mediates progression of aortic aneurysm, but the relative contribution of its type 1 (AT1) and type 2 (AT2) receptors remains unknown.... 
Connective tissues | Receptors | Root growth | Medical treatment | REPORTS | Aneurysms | Placebos | Aorta | Mice | Aortic aneurysm | Marfan syndrome | PATHOGENESIS | ACTIVATION | MECHANISM | MULTIDISCIPLINARY SCIENCES | MARFAN-SYNDROME | MOUSE MODEL | GROWTH | SMOOTH-MUSCLE-CELLS | BLOCKADE | CONTRIBUTES | EXPRESSION | Aortic Aneurysm - metabolism | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Aortic Rupture - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Enalapril - therapeutic use | MAP Kinase Signaling System | Marfan Syndrome - drug therapy | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Aortic Rupture - prevention & control | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Aortic Aneurysm - pathology | Aortic Aneurysm - prevention & control | Disease Models, Animal | Receptor, Angiotensin, Type 2 - genetics | Angiotensin II - metabolism | Signal Transduction | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Losartan - pharmacology | Aortic Rupture - pathology | Disease Progression | Mice, Knockout | Animals | Enalapril - pharmacology | Receptor, Angiotensin, Type 2 - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Aortic Aneurysm - drug therapy | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Aortic aneurysms | Development and progression | Genetic aspects | Health aspects | Angiotensin | Signal transduction | Peptides | Cellular biology | Coronary vessels | Rodents
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2014, Volume 34, Issue 12, pp. 2609 - 2616
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 05/2016, Volume 36, Issue 5, pp. 898 - 907
OBJECTIVE—Abdominal aortic aneurysm (AAA) is an important cause of mortality in older adults. Activity of the local kallikrein–kinin system may be important in... 
animal model cardiovascular disease | bradykinin | aorta | angiotensin II | abdominal aortic aneurysm | kinins | ACTIVATION | PHENOTYPIC MODULATION | ATHEROSCLEROSIS | OSTEOPROTEGERIN | MODEL | INFLAMMATION | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | ANEURYSM FORMATION | EXPRESSION | Bradykinin - analogs & derivatives | Receptor, Bradykinin B2 - agonists | Aortic Aneurysm, Abdominal - prevention & control | Apolipoproteins E - deficiency | Humans | Neutrophil Activation - drug effects | Male | Osteopontin - metabolism | Aortic Rupture - metabolism | Aorta, Abdominal - drug effects | Aortic Aneurysm, Abdominal - metabolism | Bradykinin B2 Receptor Antagonists - pharmacology | Calcium Phosphates | Matrix Metalloproteinase 9 - metabolism | Time Factors | Aortic Rupture - prevention & control | Aorta, Abdominal - pathology | Angiotensin II | Aorta, Abdominal - metabolism | Dilatation, Pathologic | Aortic Aneurysm, Abdominal - pathology | Bradykinin - pharmacology | Disease Models, Animal | Genetic Predisposition to Disease | Signal Transduction | Matrix Metalloproteinase 2 - metabolism | Tissue Culture Techniques | Aortic Rupture - pathology | Aortic Aneurysm, Abdominal - genetics | Rats, Sprague-Dawley | Mice, Knockout | Phenotype | Animals | Apolipoproteins E - genetics | Aortic Rupture - genetics | Receptor, Bradykinin B2 - metabolism | Osteoprotegerin - metabolism
Journal Article
Journal Article
Nature communications, ISSN 2041-1723, 11/2018, Volume 9, Issue 1, pp. 4795 - 16
Aortic intramural hematoma (IMH) can evolve toward reabsorption, dissection or aneurysm. Hypertension is the most common predisposing factor in IMH and... 
INHIBITION | GENE | PEPTIDE IDENTIFICATION | PHOSPHORYLATION | DISSECTION | MULTIDISCIPLINARY SCIENCES | INDUCTION | SYNDROME CRITICAL REGION | EXPRESSION | VASCULAR SMOOTH-MUSCLE | INSIGHTS | Aneurysm, Dissecting - genetics | Antihypertensive Agents - pharmacology | Humans | Hypertension - drug therapy | Male | Aorta - metabolism | Intracellular Signaling Peptides and Proteins - deficiency | rho-Associated Kinases - metabolism | Hematoma - genetics | Gene Deletion | Hypertension - genetics | Myocytes, Smooth Muscle - drug effects | Aneurysm, Dissecting - prevention & control | Intracellular Signaling Peptides and Proteins - genetics | Disease Models, Animal | Genetic Predisposition to Disease | Endothelial Cells - metabolism | Aneurysm, Dissecting - pathology | Hypertension - pathology | Hypertension - metabolism | Mice, Knockout | Aorta - pathology | Muscle Proteins - genetics | Aortic Rupture - genetics | Mice | Endothelial Cells - pathology | Hematoma - prevention & control | Myocytes, Smooth Muscle - pathology | Glycogen Synthase Kinase 3 beta - genetics | Muscle Proteins - deficiency | Aortic Rupture - metabolism | rho-Associated Kinases - antagonists & inhibitors | Aortic Rupture - prevention & control | Protein Isoforms - deficiency | Myocytes, Smooth Muscle - metabolism | Aneurysm, Dissecting - metabolism | Aorta - drug effects | Gene Expression Regulation | rho-Associated Kinases - genetics | Aortic Rupture - pathology | Glycogen Synthase Kinase 3 beta - antagonists & inhibitors | Glycogen Synthase Kinase 3 beta - metabolism | Animals | Hematoma - pathology | Protein Kinase Inhibitors - pharmacology | Primary Cell Culture | Endothelial Cells - drug effects | Hematoma - metabolism | Protein Isoforms - genetics | Hypertension | Phenotypes | Target recognition | Therapeutic applications | Rupture | Aneurysm | Muscles | Smooth muscle | Reabsorption | Endothelial cells | Clonal deletion | Myosin | Isoforms | Angiotensin | Hematoma | Deletion | Aorta | Inhibition | Angiotensin II
Journal Article
Journal of Vascular Surgery, ISSN 0741-5214, 12/2018, Volume 68, Issue 6, pp. 60S - 71S.e3
Journal Article