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Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 10/2013, Volume 33, Issue 10, pp. 2374 - 2379
Journal Article
Journal Article
Journal Article
Journal of Vascular Surgery, ISSN 0741-5214, 12/2018, Volume 68, Issue 6, pp. 82S - 92S.e2
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 06/2009, Volume 119, Issue 24, pp. 3125 - 3132
Background-Development and rupture of aortic aneurysms involve a combination of complex biological processes. Rosiglitazone, a peroxisome... 
Polymerase chain reaction | Aneurysm, ruptured | Inflammation | PPAR gamma | Aneurysm | Angiotensin | ATHEROSCLEROTIC LESIONS | aneurysm | ISCHEMIA/REPERFUSION INJURY | CARDIAC & CARDIOVASCULAR SYSTEMS | RATS | aneurysm, ruptured | ANGIOTENSIN-II | ENDOTHELIAL FUNCTION | BLOOD-PRESSURE | inflammation | angiotensin | ACTIVATED-RECEPTOR-GAMMA | GENE-EXPRESSION | PPAR-GAMMA | PERIPHERAL VASCULAR DISEASE | E-DEFICIENT MICE | polymerase chain reaction | Aortic Aneurysm, Abdominal - chemically induced | Aortic Rupture - blood | Aortic Aneurysm, Abdominal - prevention & control | Cholesterol - blood | Humans | Tumor Necrosis Factor-alpha - blood | Body Weight - drug effects | E-Selectin - blood | PPAR gamma - metabolism | Aortic Aneurysm, Abdominal - blood | Apolipoproteins E - metabolism | Hypercholesterolemia - pathology | Interleukin-6 - blood | Aortic Rupture - prevention & control | Hypercholesterolemia - prevention & control | Angiotensin II - adverse effects | Thiazolidinediones - pharmacology | Aortic Aneurysm, Abdominal - pathology | Disease Models, Animal | PPAR gamma - genetics | Angiotensin II - pharmacology | Hypercholesterolemia - blood | Blood Glucose - analysis | Receptors, Angiotensin - blood | Aortic Aneurysm, Abdominal - genetics | Hypoglycemic Agents - pharmacology | Mice, Knockout | Animals | Apolipoproteins E - genetics | PPAR gamma - agonists | Aortic Rupture - genetics | Mice | Aortic Rupture - chemically induced | Hypercholesterolemia - chemically induced | Hypercholesterolemia - genetics | Prevention | Usage | Peroxisomes | Animal models in research | Aortic aneurysms | Development and progression | Rosiglitazone maleate | Health aspects | Index Medicus | Abridged Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2015, Volume 35, Issue 4, pp. 911 - 917
Objective-Studies of mice with mild Marfan syndrome (MFS) have correlated the development of thoracic aortic aneurysm (TAA) with improper stimulation of... 
aortic aneurysm | receptor, angiotensin, type 1 | losartan | transforming growth factor β | Marfan syndrome | PATHOGENESIS | TGF-BETA | type 1 | transforming growth factor beta | MOUSE MODEL | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | receptor, angiotensin | Aortic Aneurysm, Thoracic - genetics | Phosphorylation | Aortic Aneurysm, Thoracic - prevention & control | Humans | Aortic Rupture - metabolism | Mice, 129 Strain | Angiotensin II Type 1 Receptor Blockers - pharmacology | Marfan Syndrome - drug therapy | Aortic Aneurysm, Thoracic - pathology | Time Factors | Mice, Mutant Strains | Aortic Rupture - prevention & control | Transforming Growth Factor beta - antagonists & inhibitors | Aorta, Thoracic - drug effects | Microfilament Proteins - genetics | Aorta, Thoracic - pathology | Disease Models, Animal | Transforming Growth Factor beta - immunology | Fibrillin-1 | Mice, Inbred C57BL | Losartan - pharmacology | Smad2 Protein - metabolism | Aorta, Thoracic - metabolism | Antibodies, Neutralizing - pharmacology | Aortic Rupture - pathology | Fibrillins | Disease Progression | Marfan Syndrome - genetics | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Aortic Aneurysm, Thoracic - metabolism | Receptor, Angiotensin, Type 1 - metabolism | Aortic Rupture - genetics | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Mutation | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article
Science, ISSN 0036-8075, 4/2011, Volume 332, Issue 6027, pp. 361 - 365
Angiotensin II (AngII) mediates progression of aortic aneurysm, but the relative contribution of its type 1 (AT1) and type 2 (AT2) receptors remains unknown.... 
Connective tissues | Receptors | Root growth | Medical treatment | REPORTS | Aneurysms | Placebos | Aorta | Mice | Aortic aneurysm | Marfan syndrome | PATHOGENESIS | ACTIVATION | MECHANISM | MULTIDISCIPLINARY SCIENCES | MARFAN-SYNDROME | MOUSE MODEL | GROWTH | SMOOTH-MUSCLE-CELLS | BLOCKADE | CONTRIBUTES | EXPRESSION | Aortic Aneurysm - metabolism | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Aortic Rupture - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Enalapril - therapeutic use | MAP Kinase Signaling System | Marfan Syndrome - drug therapy | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Aortic Rupture - prevention & control | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Aortic Aneurysm - pathology | Aortic Aneurysm - prevention & control | Disease Models, Animal | Receptor, Angiotensin, Type 2 - genetics | Angiotensin II - metabolism | Signal Transduction | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Losartan - pharmacology | Aortic Rupture - pathology | Disease Progression | Mice, Knockout | Animals | Enalapril - pharmacology | Receptor, Angiotensin, Type 2 - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Aortic Aneurysm - drug therapy | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Aortic aneurysms | Development and progression | Genetic aspects | Health aspects | Angiotensin | Signal transduction | Peptides | Cellular biology | Coronary vessels | Rodents
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 05/2016, Volume 36, Issue 5, pp. 898 - 907
OBJECTIVE—Abdominal aortic aneurysm (AAA) is an important cause of mortality in older adults. Activity of the local kallikrein–kinin system may be important in... 
animal model cardiovascular disease | bradykinin | aorta | angiotensin II | abdominal aortic aneurysm | kinins | ACTIVATION | PHENOTYPIC MODULATION | ATHEROSCLEROSIS | OSTEOPROTEGERIN | MODEL | INFLAMMATION | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | ANEURYSM FORMATION | EXPRESSION | Bradykinin - analogs & derivatives | Receptor, Bradykinin B2 - agonists | Aortic Aneurysm, Abdominal - prevention & control | Apolipoproteins E - deficiency | Humans | Neutrophil Activation - drug effects | Male | Osteopontin - metabolism | Aortic Rupture - metabolism | Aorta, Abdominal - drug effects | Aortic Aneurysm, Abdominal - metabolism | Bradykinin B2 Receptor Antagonists - pharmacology | Calcium Phosphates | Matrix Metalloproteinase 9 - metabolism | Time Factors | Aortic Rupture - prevention & control | Aorta, Abdominal - pathology | Angiotensin II | Aorta, Abdominal - metabolism | Dilatation, Pathologic | Aortic Aneurysm, Abdominal - pathology | Bradykinin - pharmacology | Disease Models, Animal | Genetic Predisposition to Disease | Signal Transduction | Matrix Metalloproteinase 2 - metabolism | Tissue Culture Techniques | Aortic Rupture - pathology | Aortic Aneurysm, Abdominal - genetics | Rats, Sprague-Dawley | Mice, Knockout | Phenotype | Animals | Apolipoproteins E - genetics | Aortic Rupture - genetics | Receptor, Bradykinin B2 - metabolism | Osteoprotegerin - metabolism
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Journal Article