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Journal Article
11/2013
αβ-TCR+CD3+CD4-CD8-NK1.1- double negative (DN) T cells comprise 1-5% of T lymphocytes in mice and humans. Previous studies have demonstrated that DN T cells... 
APC | 0982 | DN T cell
Dissertation
Current Biology, ISSN 0960-9822, 05/2017, Volume 27, Issue 10, pp. 1462 - 1476.e5
In mammalian females, germ cells remain arrested as primordial follicles. Resumption of meiosis is heralded by germinal vesicle breakdown, condensation of... 
aneuploidy | APC/CCdh1 | aurora kinase | shugoshin-2 | cohesin | meiosis | APC/C | Index Medicus | APC | CCdh1
Journal Article
2009, Advances in experimental medicine and biology, ISBN 9781441911445, Volume 656, xvi, 131
The Apc protein is a multifunctional participant in a diverse array of cellular functions. This book provides an overview of the functions performed by the Apc... 
Adenomatous Polyposis Coli | Colorectal Neoplasms | Colon (Anatomy) | metabolism | Adenomatous Polyposis Coli Protein | Tumor suppressor proteins | Genes, APC | Cancer
Book
Deutsche Apotheker Zeitung, ISSN 0011-9857, 2019, Volume 159, Issue 3
Journal Article
Cell Cycle, ISSN 1538-4101, 05/2015, Volume 14, Issue 10, pp. 1517 - 1528
The Myocyte Enhancer Factor 2C (MEF2C) transcription factor plays a critical role in skeletal muscle differentiation, promoting muscle-specific gene... 
MEF2 | APC/C | proliferation | cell cycle | muscle | degradation | splicing | mitosis | phosphorylation | Degradation | Phosphorylation | Splicing | Mitosis | Cell cycle | Muscle | Proliferation | C-JUN PROMOTER | ZEBRAFISH MEF2CA | HDAC, Histone Deacetylases | CHX, Cycloheximide | CELL-PROLIFERATION | CDK, Cyclin Dependent Kinase | Gadd45b, Growth Arrest and DNA Damage b | APC/C, Anaphase Promoting Complex/Cyclosome | CELL BIOLOGY | degron, degradation signal | GENE-EXPRESSION | MADS, Minichromosome maintenance, Agamous, Deficiens, Serum response factor | ENHANCER FACTOR 2C | MYOD | CYCLIN-A | MyHC, Myosin Heavy Chain | SKELETAL-MUSCLE | CRC, ColoRectal Cancer | MEF2, Myocyte Enhancer Factor 2 | DNA | TRANSCRIPTION FACTOR | UPS, Ubiquitin Proteasome System | NIH 3T3 Cells | MEF2 Transcription Factors - genetics | Cell Proliferation | Cadherins - metabolism | Humans | Molecular Sequence Data | Cyclin B1 - metabolism | HEK293 Cells | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Antigens, Differentiation - metabolism | Cdc20 Proteins - antagonists & inhibitors | Cadherins - genetics | Cdc20 Proteins - genetics | Amino Acid Sequence | Down-Regulation | G2 Phase Cell Cycle Checkpoints | 14-3-3 Proteins - metabolism | Sequence Alignment | Animals | Cdc20 Proteins - metabolism | MEF2 Transcription Factors - metabolism | Cadherins - antagonists & inhibitors | Mice | Anaphase-Promoting Complex-Cyclosome - metabolism | Index Medicus
Journal Article
11/2010
Septins are evolutionarily conserved proteins with roles in chromosome congression and segregation, cytokinesis and microtubule destabilization. Septins form... 
knockdown | APC | 0487 | abscission | septins
Dissertation
Gastroenterological Endoscopy, ISSN 0387-1207, 03/2008, Volume 50, Issue 3, pp. 369 - 373
APC
Journal Article
Biochimica et Biophysica Acta - Molecular Basis of Disease, ISSN 0925-4439, 09/2015, Volume 1852, Issue 9, pp. 1846 - 1855
Constitutive Wnt/β-catenin signaling is a key contributor to colorectal cancer (CRC). Although inactivation of the tumor suppressor adenomatous polyposis coli... 
Kaiso | Inflammation | Tumorigenesis | Wnt | Apc
Journal Article
Molecular Carcinogenesis, ISSN 0899-1987, 01/2014, Volume 53, Issue 1, pp. 77 - 84
Mutations in adenomatous polyposis coli (APC) gene are found in more than 80% of colorectal cancer (CRC) patients. The nuclear transcription factor Nrf2 plays... 
min | colon carcinogenesis | Nrf2 | Apc | inflammation | oxidative stress | Oxidative stress | Inflammation | Colon carcinogenesis | CARCINOMA CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | APC(MIN/+) MICE | CHEMOPREVENTION | BETA-CATENIN | SUPPRESSION | ONCOLOGY | INCREASED SUSCEPTIBILITY | COLORECTAL-CANCER | Apc(min) | NITRIC-OXIDE | FAMILIAL ADENOMATOUS POLYPOSIS | COLON-CARCINOMA | Colorectal cancer | Tumors | Oncology | Mutation
Journal Article
Gastroenterology, ISSN 0016-5085, 2010, Volume 138, Issue 3, pp. 1003 - 1011.e5
Background and Aims The transcription factor signal transducer and activator of transcription 3 (Stat3) has been considered to promote progression and... 
Gastroenterology and Hepatology | APC | Colorectal Cancer | CEACAM1
Journal Article
British Dental Journal, ISSN 0007-0610, 07/2014, Volume 33, Issue 1, pp. 3342 - 3350
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 07/2019, Volume 514, Issue 4, pp. 1087 - 1092
Etoposide-induced 2.4 kb transcript ( , also known as ) is a p53 target gene involved in cell growth suppression and apoptosis and known to be frequently... 
EI24 | Colorectal cancer | ApcMin/+mice | Apc | mice
Journal Article
Cancer Research, ISSN 0008-5472, 08/2000, Volume 60, Issue 16, pp. 4366 - 4371
Journal Article
Nature, ISSN 0028-0836, 11/2012, Volume 491, Issue 7423, pp. 254 - 258
Approximately 2% of colorectal cancer is linked to pre-existing inflammation known as colitis-associated cancer, but most develops in patients without... 
COLITIS | PROMOTES COLON TUMORIGENESIS | COLORECTAL-CANCER | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | HELPER-CELLS | MOUSE MODEL | APC(MIN/+) MICE | RECEPTOR | DIFFERENTIATION | CUTTING EDGE | Inflammation - pathology | Adenoma - genetics | Colorectal Neoplasms - genetics | Humans | Colitis - complications | Interleukin-17 - immunology | Interleukin-23 - deficiency | Tumor Microenvironment | Adenoma - immunology | Adenoma - microbiology | Genes, APC | Interleukin-23 - genetics | Myeloid Cells - immunology | Cell Division | Myeloid Differentiation Factor 88 - immunology | Toll-Like Receptors - metabolism | Disease Models, Animal | Bacteria - metabolism | Inflammation - microbiology | Signal Transduction | Mice, Inbred C57BL | Toll-Like Receptors - immunology | Interleukin-17 - genetics | Colorectal Neoplasms - microbiology | Inflammation - immunology | beta Catenin - metabolism | Interleukin-23 - immunology | Disease-Free Survival | Animals | Colorectal Neoplasms - immunology | Adenoma - pathology | Inflammation - genetics | Myeloid Cells - metabolism | Bacteria - pathogenicity | Mice | Cell Transformation, Neoplastic - pathology | Colorectal Neoplasms - pathology | Myeloid Differentiation Factor 88 - metabolism | Development and progression | Cellular signal transduction | Research | Interleukins | Properties | Tumors | Index Medicus
Journal Article