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1. Full Text The evolution and pathology of frontotemporal dementia
by Kertesz, Andrew and McMonagle, Paul and Blair, Mervin and Davidson, Wilda and Munoz, David G
Brain, ISSN 0006-8950, 2005, Volume 128, Issue 9, pp. 1996 - 2005
This is a clinicopathologic study of a prospective, clinic-based cohort of patients with frontotemporal dementia (FTD)/Pick complex, who were followed to... 
Pick's disease | Frontotemporal dementia | Primary progressive aphasia | Progressive supranuclear palsy | Corticobasal degeneration | CLINICAL-PICTURE | DIAGNOSIS | GANGLIONIC DEGENERATION | corticobasal degeneration | SUPRANUCLEAR PALSY | PICKS-DISEASE | frontotemporal dementia | DISEASE-INCLUSION DEMENTIA | NEUROSCIENCES | primary progressive aphasia | CLINICAL NEUROLOGY | NON-ALZHEIMER TYPE | progressive supranuclear palsy | FRONTAL-LOBE DEGENERATION | Prospective Studies | Supranuclear Palsy, Progressive - pathology | Humans | Middle Aged | Male | Aphasia, Primary Progressive - pathology | Dementia - genetics | Pick Disease of the Brain - genetics | Alzheimer Disease - pathology | Pick Disease of the Brain - pathology | Dementia - metabolism | Adult | Female | Pick Disease of the Brain - metabolism | Supranuclear Palsy, Progressive - genetics | Dementia - pathology | Aphasia, Primary Progressive - genetics | Disease Progression | Syndrome | Aphasia, Primary Progressive - metabolism | Age of Onset | Alzheimer Disease - metabolism | Survival Analysis | Sex Distribution | Supranuclear Palsy, Progressive - metabolism | Aged | tau Proteins - analysis | Alzheimer Disease - genetics
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2. Full Text Tau pathology and neurodegeneration contribute to cognitive impairment in Alzheimer's disease
by Bejanin, Alexandre and Schonhaut, Daniel R and La Joie, Renaud and Kramer, Joel H and Baker, Suzanne L and Sosa, Natasha and Ayakta, Nagehan and Cantwell, Averill and Janabi, Mustafa and Lauriola, Mariella and O'Neil, James P and Gorno-Tempini, Maria L and Miller, Zachary A and Rosen, Howard J and Miller, Bruce L and Jagust, William J and Rabinovici, Gil D
Brain, ISSN 0006-8950, 12/2017, Volume 140, Issue 12, pp. 3286 - 3300
Neuropathological and in vivo studies have revealed a tight relationship between tau pathology and cognitive impairment across the Alzheimer's disease... 
atrophy | tau | cognitive impairment | amyloid | Alzheimer's disease | NEUROFIBRILLARY TANGLES | DEPOSITION | CLINICAL CHARACTERISTICS | NEUROSCIENCES | CLINICAL NEUROLOGY | NATIONAL INSTITUTE | POSITRON-EMISSION-TOMOGRAPHY | DIAGNOSTIC GUIDELINES | NEURONS | HYPOMETABOLISM | PET | ASSOCIATION WORKGROUPS | Brain - diagnostic imaging | Carbolines | Humans | Middle Aged | tau Proteins - metabolism | Male | Positron-Emission Tomography | Case-Control Studies | Aphasia, Primary Progressive - diagnostic imaging | Brain - metabolism | Amyloid - metabolism | Cognitive Dysfunction - psychology | Female | Aphasia, Primary Progressive - psychology | Cognitive Dysfunction - diagnostic imaging | Alzheimer Disease - psychology | Cognitive Dysfunction - metabolism | Neuropsychological Tests | Magnetic Resonance Imaging | Regression Analysis | Aphasia, Primary Progressive - metabolism | Alzheimer Disease - metabolism | Aged | Alzheimer Disease - diagnostic imaging | Fluorine Radioisotopes | Benzothiazoles | Carbon Radioisotopes
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3. Distinct [18F]THK5351 binding patterns in primary progressive aphasia variants
by Schaeverbeke, Jolien and Evenepoel, Charlotte and Declercq, Lieven and Gabel, Silvy and Meersmans, Karen and Bruffaerts, Rose and Adamczuk, Kate and Dries, Eva and Van Bouwel, Karen and Sieben, Anne and Pijnenburg, Yolande and Peeters, Ronald and Bormans, Guy and Van Laere, Koen and Koole, Michel and Dupont, Patrick and Vandenberghe, Rik
European Journal of Nuclear Medicine and Molecular Imaging, ISSN 1619-7070, 12/2018, Volume 45, Issue 13, pp. 2342 - 2357
PurposeTo assess the binding of the PET tracer [F-18]THK5351 in patients with different primary progressive aphasia (PPA) variants and its correlation with... 
Tau | Nonfluent variant | Primary progressive aphasia | Agrammatism | Motor speech | F]THK5351 binding | MRI | ALZHEIMERS-DISEASE | APRAXIA | PATHOLOGY | POSITRON-EMISSION-TOMOGRAPHY | TRACER | F-18-THK5351 | IN-VIVO | HYPOMETABOLISM | [F-18]THK5351 binding | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | Quinolines - metabolism | Radioactive Tracers | Aphasia, Primary Progressive - metabolism | Humans | Middle Aged | Aged, 80 and over | Female | Male | Aged | Positron-Emission Tomography | Aminopyridines - metabolism | Aphasia, Primary Progressive - diagnostic imaging | Cortex (premotor) | Cerebellum | Neuroimaging | Brain | Basal ganglia | Mesencephalon | Impairment | Cortex (motor) | Substantia grisea | Proteins | Cortex (frontal) | Thalamus | Bioindicators | Alzheimer's disease | Cortex (temporal) | Deoxyribonucleic acid--DNA | Supplementary motor area | Medical imaging | Statistical analysis | Neurodegenerative diseases | Brain mapping | Fluorine isotopes | Regression analysis | Patients | Postcentral gyrus | Ganglia | Pathology | Magnetic resonance imaging | Tau protein | Correlation analysis | Biomarkers | Aphasia | Positron emission tomography
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4. Full Text Tau, amyloid, and hypometabolism in the logopenic variant of primary progressive aphasia
by Pascual, Belen and Masdeu, Joseph C
Neurology, ISSN 0028-3878, 02/2016, Volume 86, Issue 5, pp. 487 - 488
A 57-year-old right-handed woman had an 8-year history of progressive word retrieval and repetition deficits, consistent with the logopenic variant of... 
CLINICAL NEUROLOGY | Aphasia, Primary Progressive - diagnosis | tau Proteins - genetics | Amyloid beta-Peptides - genetics | Aphasia, Primary Progressive - metabolism | Humans | Middle Aged | Aphasia, Primary Progressive - genetics | Female | Genetic Variation - genetics | Cerebral Cortex - metabolism
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5. Full Text Progranulin-associated PiB-negative logopenic primary progressive aphasia
by Josephs, Keith A and Duffy, Joseph R and Strand, Edythe A and Machulda, Mary M and Vemuri, Prashanthi and Senjem, Matthew L and Perkerson, Ralph B and Baker, Matthew C and Lowe, Val and Jack Jr, Clifford R and Rademakers, Rosa and Whitwell, Jennifer L
Journal of Neurology, ISSN 0340-5354, 3/2014, Volume 261, Issue 3, pp. 604 - 614
The logopenic variant of primary progressive aphasia (lvPPA) strongly associates with Alzheimer’s disease, but can also associate with frontotemporal lobar... 
FDG-PET | Neurology | Logopenic | Neurosciences | Progranulin | Medicine & Public Health | MRI | Primary progressive aphasia | Neuroradiology | β-amyloid | CRITERIA | 3 VARIANTS | DEMENTIA | ATROPHY | VALIDATION | ALZHEIMER | APRAXIA | FRONTOTEMPORAL LOBAR DEGENERATION | CLINICAL NEUROLOGY | VOXEL-BASED MORPHOMETRY | beta-amyloid | MUTATIONS | Thiazoles - metabolism | Age Factors | Aniline Compounds - metabolism | Humans | Middle Aged | Atrophy - pathology | Cerebral Cortex - pathology | Magnetic Resonance Imaging - methods | Male | Tomography, X-Ray Computed | Aphasia, Primary Progressive - pathology | Parietal Lobe - metabolism | Positron-Emission Tomography - instrumentation | Parietal Lobe - physiopathology | Cerebral Cortex - metabolism | Cerebral Cortex - physiopathology | Multimodal Imaging | Amyloid beta-Peptides - metabolism | Female | Temporal Lobe - pathology | Aphasia, Primary Progressive - physiopathology | Atrophy - physiopathology | Intercellular Signaling Peptides and Proteins - genetics | Aphasia, Primary Progressive - genetics | Positron-Emission Tomography - methods | Mutation - genetics | Temporal Lobe - metabolism | Parietal Lobe - pathology | Neuropsychological Tests | Temporal Lobe - physiopathology | Magnetic Resonance Imaging - instrumentation | Language Tests | Aphasia, Primary Progressive - metabolism | Apolipoproteins E - genetics | Fluorodeoxyglucose F18 | Aged | Atrophy - metabolism | Aphasia | Complications and side effects | Development and progression | Research | Patient outcomes
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6. Full Text Asymmetric pathology in primary progressive aphasia with progranulin mutations and TDP inclusions
by Kim, Garam and Ahmadian, Saman S and Peterson, Melanie and Parton, Zach and Memon, Rohail and Weintraub, Sandra and Rademaker, Alfred and Bigio, Eileen and Mesulam, M.-Marsel and Geula, Changiz
Neurology, ISSN 0028-3878, 02/2016, Volume 86, Issue 7, pp. 627 - 636
OBJECTIVE:To investigate quantitative regional distribution and hemispheric asymmetry of TDP-43 (TAR DNA-binding protein 43) inclusions, neurons, and activated... 
HETEROGENEITY | NEUROFIBRILLARY TANGLES | DEMENTIA | NEUROINFLAMMATION | ALZHEIMERS-DISEASE | INFLAMMATION | CLASSIFICATION | FRONTOTEMPORAL LOBAR DEGENERATION | EXPRESSION | CLINICAL NEUROLOGY | TRANSGENIC MICE | Neurons - pathology | Humans | Middle Aged | Intercellular Signaling Peptides and Proteins - genetics | Aphasia, Primary Progressive - genetics | Male | Aphasia, Primary Progressive - pathology | Functional Laterality | Brain - metabolism | DNA-Binding Proteins - metabolism | Phenotype | Aphasia, Primary Progressive - metabolism | Brain - pathology | Microglia - pathology | Female | Aged | Mutation
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7. Full Text White matter tract signatures of the progressive aphasias
by Mahoney, Colin J and Malone, Ian B and Ridgway, Gerard R and Buckley, Aisling H and Downey, Laura E and Golden, Hannah L and Ryan, Natalie S and Ourselin, Sebastien and Schott, Jonathan M and Rossor, Martin N and Fox, Nick C and Warren, Jason D
Neurobiology of Aging, ISSN 0197-4580, 2013, Volume 34, Issue 6, pp. 1687 - 1699
Abstract The primary progressive aphasias (PPA) are a heterogeneous group of language-led neurodegenerative diseases resulting from large-scale brain network... 
Neurology | Internal Medicine | DTI | Networks | Primary progressive aphasia | White matter | 3 VARIANTS | ATROPHY | MRI | ALZHEIMERS-DISEASE | PATTERNS | SEMANTIC DEMENTIA | FRONTOTEMPORAL LOBAR DEGENERATION | NEUROSCIENCES | POSTMORTEM | GERIATRICS & GERONTOLOGY | DIFFUSION | BRAIN | Nerve Fibers, Myelinated - metabolism | Cross-Sectional Studies | Humans | Middle Aged | Male | Aphasia, Primary Progressive - pathology | Disease Progression | Diffusion Tensor Imaging | Nerve Fibers, Myelinated - pathology | Aphasia, Primary Progressive - metabolism | Female | Aged | Cohort Studies | Aphasia | Nervous system diseases | Alzheimer's disease | Amyloid beta-protein | Medical imaging equipment | Aging | Regular
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8. Full Text Is in vivo amyloid distribution asymmetric in primary progressive aphasia?
by Martersteck, Adam and Murphy, Christopher and Rademaker, Alfred and Wieneke, Christina and Weintraub, Sandra and Chen, Kewei and Mesulam, M.‐Marsel and Rogalski, Emily and Alzheimer's Dis Neuroimaging Initi and Alzheimer's Disease Neuroimaging Initiative
Annals of Neurology, ISSN 0364-5134, 03/2016, Volume 79, Issue 3, pp. 496 - 501
We aimed to determine whether 18F‐florbetapir amyloid positron emission tomography imaging shows a clinically concordant, left‐hemisphere–dominant pattern of... 
3 VARIANTS | F 18 | POSITRON-EMISSION-TOMOGRAPHY | DEMENTIA | ALZHEIMERS-DISEASE | PATHOLOGY | FLORBETAPIR | NEUROSCIENCES | PITTSBURGH COMPOUND-B | BRAIN | CLINICAL NEUROLOGY | IMPAIRMENT | Heart | Radiopharmaceuticals - pharmacokinetics | Humans | Middle Aged | Male | Molecular Imaging - methods | Ethylene Glycols - pharmacokinetics | Aniline Compounds - pharmacokinetics | Positron-Emission Tomography - methods | Cerebral Cortex - metabolism | Tissue Distribution | Aphasia, Primary Progressive - metabolism | Amyloid beta-Peptides - metabolism | Dominance, Cerebral | Aged, 80 and over | Aged
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9. Full Text The role of single-subject brain metabolic patterns in the early differential diagnosis of primary progressive aphasias and in prediction of progression to dementia
by Cerami, Chiara and Dodich, Alessandra and Greco, Lucia and Iannaccone, Sandro and Magnani, Giuseppe and Marcone, Alessandra and Pelagallo, Elisabetta and Santangelo, Roberto and Cappa, Stefano F and Perani, Daniela
Journal of Alzheimer's Disease, ISSN 1387-2877, 2017, Volume 55, Issue 1, pp. 183 - 197
Background and Objective: Primary progressive aphasia (PPA) is a clinical syndrome due to different neurodegenerative conditions in which an accurate early... 
FDG-PET | semantic variant of primary progressive aphasia | non fluent variant of primary progressive aphasia | positron emission tomography | logopenic variant of primary progressive aphasia | primary progressive aphasia | 3 VARIANTS | ALZHEIMERS-DISEASE | FRONTOTEMPORAL DEMENTIA | SOCIAL COGNITION | SEMANTIC DEMENTIA | NEUROSCIENCES | POSITRON-EMISSION-TOMOGRAPHY | MEMORY | GLUCOSE-METABOLISM | NONFLUENT APHASIA | Dementia - diagnostic imaging | Brain - diagnostic imaging | Diagnosis, Differential | Follow-Up Studies | Humans | Male | Cognition | Positron-Emission Tomography | Disease Progression | Aphasia, Primary Progressive - diagnostic imaging | Brain - metabolism | Neuropsychological Tests | Dementia - metabolism | Aphasia, Primary Progressive - metabolism | Female | Fluorodeoxyglucose F18 | Aged | Retrospective Studies | Radiopharmaceuticals | Early Diagnosis | Precision Medicine | Neuroimaging | Brain | Data acquisition | Brain stem | Positron emission | Metabolism | Progressive supranuclear palsy | Neurodegeneration | Diagnostic software | Predictions | Tomography | Dementia disorders | Biomarkers | Aphasia | Degeneration | Differential diagnosis | Diagnostic systems | Paralysis | Frontotemporal dementia | Positron emission tomography | Dementia
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10. Full Text Aβ amyloid and glucose metabolism in three variants of primary progressive aphasia
by Rabinovici, Gil D and Jagust, William J and Furst, Ansgar J and Ogar, Jennifer M and Racine, Caroline A and Mormino, Elizabeth C and O'Neil, James P and Lal, Rayhan A and Dronkers, Nina F and Miller, Bruce L and Gorno‐Tempini, Maria Luisa
Annals of Neurology, ISSN 0364-5134, 10/2008, Volume 64, Issue 4, pp. 388 - 401
Objective Alzheimer's disease (AD) is found at autopsy in up to one third of patients with primary progressive aphasia (PPA), but clinical features that... 
C-11-PIB PET | ALZHEIMERS-DISEASE | SEMANTIC DEMENTIA | PATHOLOGY | HYPOMETABOLISM | FRONTOTEMPORAL LOBAR DEGENERATION | BINDING | NEUROSCIENCES | PITTSBURGH COMPOUND-B | BRAIN | CLINICAL NEUROLOGY | NONFLUENT APHASIA
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11. Full Text TDP-43 pathology in primary progressive aphasia and frontotemporal dementia with pathologic Alzheimer disease
by Bigio, Eileen H and Mishra, Manjari and Hatanpaa, Kimmo J and White III, Charles L and Johnson, Nancy and Rademaker, Alfred and Weitner, Bing Bing and Deng, Han-Xiang and Dubner, Steven D and Weintraub, Sandra and Mesulam, Marsel
Acta Neuropathologica, ISSN 0001-6322, 07/2010, Volume 120, Issue 1, pp. 43 - 54
The clinical syndrome of primary progressive aphasia (PPA) can be associated with a variety of neuropathologic diagnoses at autopsy. Thirty percent of cases... 
FTLD-TDP | Hippocampal sclerosis | Frontotemporal dementia | Primary progressive aphasia | Alzheimer disease | TDP-43 proteinopathy | LOBAR DEGENERATION | INCLUSIONS | UBIQUITIN | TAR-DNA-BINDING | AMYOTROPHIC-LATERAL-SCLEROSIS | ALPHA-SYNUCLEIN | PATHOLOGY | NEUROSCIENCES | CLINICAL NEUROLOGY | PROTEIN 43 | PHOSPHORYLATED TDP-43 | LEWY BODIES | Neurons - pathology | Apolipoprotein E4 - genetics | Humans | Middle Aged | Male | Aphasia, Primary Progressive - pathology | Sclerosis - metabolism | Alzheimer Disease - pathology | Brain - metabolism | DNA-Binding Proteins - metabolism | Frontotemporal Dementia - metabolism | Gliosis - pathology | TDP-43 Proteinopathies - genetics | Sclerosis - pathology | Female | Neurons - metabolism | TDP-43 Proteinopathies - pathology | Temporal Lobe - pathology | Frontotemporal Dementia - pathology | Frontotemporal Dementia - genetics | Gliosis - genetics | Apolipoprotein E4 - metabolism | Gliosis - metabolism | TDP-43 Proteinopathies - metabolism | Organ Size | Aphasia, Primary Progressive - genetics | Hippocampus - pathology | Temporal Lobe - metabolism | Sclerosis - genetics | Hippocampus - metabolism | Phenotype | Aphasia, Primary Progressive - metabolism | Alzheimer Disease - metabolism | Brain - pathology | Aged | Alzheimer Disease - genetics | Aphasia | Medical colleges | Alzheimer's disease | Medicine, Preventive | Preventive health services | Dementia
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12. Full Text Progressive non-fluent aphasia is associated with hypometabolism centred on the left anterior insula
by Nestor, Peter J and Graham, Naida L and Fryer, Tim D and Williams, Guy B and Patterson, Karalyn and Hodges, John R
Brain, ISSN 0006-8950, 11/2003, Volume 126, Issue 11, pp. 2406 - 2418
Progressive non-fluent aphasia (PNFA) is a syndrome in which patients lose the ability to communicate fluently in the context of relative preservation of... 
Insula | Progressive non-fluent aphasia | PET | Voxel-based morphometry | LANGUAGE | ALZHEIMERS-DISEASE | SPEECH PRODUCTION | PICKS-DISEASE | voxel-based morphometry | MOTOR-NEURON DISEASE | SEMANTIC DEMENTIA | insula | FRONTOTEMPORAL LOBAR DEGENERATION | OROFACIAL DYSPRAXIA | NEUROSCIENCES | CLINICAL NEUROLOGY | CINGULATE CORTEX | progressive non-fluent aphasia | EMISSION-TOMOGRAPHY | Follow-Up Studies | Humans | Middle Aged | Male | Aphasia, Broca - metabolism | Cerebral Cortex - diagnostic imaging | Cerebral Cortex - metabolism | Disease Progression | Aphasia, Primary Progressive - diagnostic imaging | Aphasia, Broca - psychology | Neuropsychological Tests | Magnetic Resonance Imaging | Aphasia, Primary Progressive - metabolism | Alzheimer Disease - metabolism | Glucose - metabolism | Female | Aged | Tomography, Emission-Computed | Aphasia, Primary Progressive - psychology | Alzheimer Disease - psychology
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13. Full Text Amyloid pet in primary progressive aphasia: case series and systematic review of the literature
by Villarejo-Galende, Alberto and Llamas-Velasco, Sara and Gómez-Grande, Adolfo and Puertas-Martín, Verónica and Contador, Israel and Sarandeses, Pilar and González-Sánchez, Marta and Trincado, Rocío and Pilkington, Patrick and Ruiz-Solis, Sebastián and Pérez-Martínez, David A and Herrero-San Martín, Alejandro
Journal of Neurology, ISSN 0340-5354, 1/2017, Volume 264, Issue 1, pp. 121 - 130
Primary progressive aphasia (PPA) is considered a heterogeneous syndrome, with different clinical subtypes and neuropathological causes. Novel PET biomarkers... 
Neurology | Neurosciences | Florbetapir | Medicine & Public Health | PIB | Amyloid | Primary progressive aphasia | Neuroradiology | PET | FDG-PET | 3 VARIANTS | CLINICAL VARIANTS | ALZHEIMERS-DISEASE | FRONTOTEMPORAL PATHOLOGY | DEPOSITION | PIB-PET | APRAXIA | SEMANTIC DEMENTIA | CLINICAL NEUROLOGY | BIOMARKERS | Brain - metabolism | Brain - diagnostic imaging | Amyloid - metabolism | Aphasia, Primary Progressive - metabolism | Humans | Middle Aged | Female | Male | Aged | Positron-Emission Tomography | Aphasia, Primary Progressive - psychology | Aphasia, Primary Progressive - diagnostic imaging | Complications and side effects | Usage | PET imaging | Aphasia | Amyloidosis | Diagnosis | Risk factors
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