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1. Full Text The influence of β-amyloid on [18F]AV-1451 in semantic variant of primary progressive aphasia
by Whitwell, Jennifer L and Martin, Peter R and Duffy, Joseph R and Clark, Heather M and Machulda, Mary M and Schwarz, Christopher G and Weigand, Stephen D and Sintini, Irene and Senjem, Matthew L and Ertekin-Taner, Nilufer and Botha, Hugo and Utianski, Rene L and Graff-Radford, Jonathan and Jones, David T and Boeve, Bradley F and Knopman, David S and Petersen, Ronald C and Jack, Clifford R and Lowe, Val J and Josephs, Keith A
Neurology, ISSN 0028-3878, 02/2019, Volume 92, Issue 7, pp. e710 - e722
Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Brain - diagnostic imaging | Carbolines | Aniline Compounds | Humans | Middle Aged | Male | Positron-Emission Tomography | Aphasia, Primary Progressive - diagnostic imaging | Brain - metabolism | Magnetic Resonance Imaging | Contrast Media | Semantics | Aphasia, Primary Progressive - metabolism | Alzheimer Disease - metabolism | Amyloid beta-Peptides - metabolism | Thiazoles | Female | Aged | Alzheimer Disease - diagnostic imaging | Index Medicus | Abridged Index Medicus
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2. Full Text [18F]AV-1451 binding in vivo mirrors the expected distribution of TDP-43 pathology in the semantic variant of primary progressive aphasia
by Bevan-Jones, W R and Cope, Thomas E and Jones, P Simon and Passamonti, Luca and Hong, Young T and Fryer, Tim D and Arnold, Robert and Allinson, Kieren S J and Coles, Jonathan P and Aigbirhio, Franklin I and Patterson, Karalyn and O’Brien, John T and Rowe, James B
Journal of Neurology, Neurosurgery & Psychiatry, ISSN 0022-3050, 10/2018, Volume 89, Issue 10, pp. 1032 - 1037
Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Psychiatry | Surgery | Science & Technology | Medical imaging | Neuropathology | Studies | Atrophy | Pathology | Brain research | Neurodegeneration | Education | Semantics | Biomarkers | Dyslexia | Mutation | Dementia | Index Medicus | 1506
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3. Full Text Distinct [18F]THK5351 binding patterns in primary progressive aphasia variants
by Schaeverbeke, Jolien and Evenepoel, Charlotte and Declercq, Lieven and Gabel, Silvy and Meersmans, Karen and Bruffaerts, Rose and Adamczuk, Kate and Dries, Eva and Van Bouwel, Karen and Sieben, Anne and Pijnenburg, Yolande and Peeters, Ronald and Bormans, Guy and Van Laere, Koen and Koole, Michel and Dupont, Patrick and Vandenberghe, Rik
European journal of nuclear medicine and molecular imaging, ISSN 1619-7070, 12/2018, Volume 45, Issue 13, pp. 2342 - 2357
Nonfluent variant | Medicine & Public Health | Agrammatism | Orthopedics | [ 18 F]THK5351 binding | Oncology | Tau | Nuclear Medicine | Imaging / Radiology | Cardiology | Primary progressive aphasia | Motor speech | F]THK5351 binding | Life Sciences & Biomedicine | Radiology, Nuclear Medicine & Medical Imaging | Science & Technology | Quinolines - metabolism | Radioactive Tracers | Aphasia, Primary Progressive - metabolism | Humans | Middle Aged | Aged, 80 and over | Female | Male | Aged | Positron-Emission Tomography | Aminopyridines - metabolism | Aphasia, Primary Progressive - diagnostic imaging | Cortex (premotor) | Cerebellum | Neuroimaging | Brain | Basal ganglia | Mesencephalon | Impairment | Cortex (motor) | Substantia grisea | Proteins | Cortex (frontal) | Thalamus | Alzheimer's disease | Cortex (temporal) | Deoxyribonucleic acid--DNA | Supplementary motor area | Medical imaging | Statistical analysis | Neurodegenerative diseases | Brain mapping | Fluorine isotopes | Regression analysis | Patients | Postcentral gyrus | Ganglia | Pathology | Magnetic resonance imaging | Tau protein | Correlation analysis | Biomarkers | Aphasia | Positron emission tomography
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4. Full Text Prediction of pathology in primary progressive language and speech disorders
by Deramecourt, V and Lebert, F and Debachy, B and MacKowiak-Cordoliani, M.A and Bombois, S and Kerdraon, O and Buée, L and Maurage, C.-A and Pasquier, F
Neurology, ISSN 0028-3878, 2010, Volume 74, Issue 1, pp. 42 - 49
Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Neurology | Biological and medical sciences | Nervous system as a whole | Medical sciences | Nervous system (semeiology, syndromes) | Frontotemporal Lobar Degeneration - pathology | Predictive Value of Tests | Tomography, Emission-Computed, Single-Photon - methods | Alzheimer Disease - complications | Prospective Studies | Humans | Middle Aged | tau Proteins - metabolism | Male | Statistics as Topic | Aphasia, Primary Progressive - pathology | Alzheimer Disease - pathology | DNA-Binding Proteins - metabolism | Speech Disorders - pathology | Amyloid beta-Peptides - metabolism | Female | Aphasia, Primary Progressive - classification | Retrospective Studies | Aphasia, Primary Progressive - physiopathology | Prions - metabolism | Tomography Scanners, X-Ray Computed | Disease Progression | Frontotemporal Lobar Degeneration - complications | Speech Disorders - physiopathology | Neuropsychological Tests | Language Tests | Brain - pathology | Brain Mapping | Aged | Postmortem Changes | alpha-Synuclein - metabolism | Index Medicus | Abridged Index Medicus
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5. Full Text Cortical cholinergic denervation in primary progressive aphasia with Alzheimer pathology
by Mesulam, M.-Marsel and Lalehzari, Nava and Rahmani, Farzan and Ohm, Daniel and Shahidehpour, Ryan and Kim, Garam and Gefen, Tamar and Weintraub, Sandra and Bigio, Eileen and Geula, Changiz
Neurology, ISSN 0028-3878, 04/2019, Volume 92, Issue 14, pp. e1580 - e1588
Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Frontotemporal Lobar Degeneration - pathology | Cholinergic Neurons - metabolism | Humans | Middle Aged | Basal Forebrain - metabolism | Cerebral Cortex - pathology | Male | Basal Forebrain - pathology | Aphasia, Primary Progressive - pathology | Cerebral Cortex - metabolism | Alzheimer Disease - pathology | Frontotemporal Lobar Degeneration - metabolism | Autopsy | Aphasia, Primary Progressive - metabolism | Alzheimer Disease - metabolism | Female | Aged | Cholinergic Neurons - pathology | Acetylcholinesterase - metabolism | Index Medicus | Abridged Index Medicus
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6. Full Text Flortaucipir tau PET imaging in semantic variant primary progressive aphasia
by Makaretz, Sara J and Quimby, Megan and Collins, Jessica and Makris, Nikos and McGinnis, Scott and Schultz, Aaron and Vasdev, Neil and Johnson, Keith A and Dickerson, Bradford C
Journal of neurology, neurosurgery and psychiatry, ISSN 0022-3050, 10/2018, Volume 89, Issue 10, pp. 1024 - 1031
temporallobe | semantic dementia | brain atrophy | primary progressive aphasia | tau PET | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Psychiatry | Surgery | Science & Technology | Neuroimaging | Brain - diagnostic imaging | Carbolines | Humans | Middle Aged | tau Proteins - metabolism | Male | Positron-Emission Tomography - methods | Aphasia, Primary Progressive - diagnostic imaging | Brain - metabolism | Magnetic Resonance Imaging | Aphasia, Primary Progressive - metabolism | Aged, 80 and over | Female | Aged | Brain | Nuclear magnetic resonance--NMR | Medical imaging | Disease | Neuropathology | Amyotrophic lateral sclerosis | Patients | Atrophy | Pathology | Language | Neurodegeneration | Semantics | Aging | Age | Dementia | Index Medicus | 1506
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7. Full Text Typical and atypical pathology in primary progressive aphasia variants
by Spinelli, Edoardo G and Mandelli, Maria Luisa and Miller, Zachary A and Santos‐Santos, Miguel A and Wilson, Stephen M and Agosta, Federica and Grinberg, Lea T and Huang, Eric J and Trojanowski, John Q and Meyer, Marita and Henry, Maya L and Comi, Giancarlo and Rabinovici, Gil and Rosen, Howard J and Filippi, Massimo and Miller, Bruce L and Seeley, William W and Gorno‐Tempini, Maria Luisa
Annals of neurology, ISSN 0364-5134, 03/2017, Volume 81, Issue 3, pp. 430 - 443
Neurosciences | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Pick Disease of the Brain - physiopathology | Frontotemporal Lobar Degeneration - pathology | Alzheimer Disease - physiopathology | Alzheimer Disease - classification | Humans | Middle Aged | Atrophy - pathology | tau Proteins - metabolism | Male | Primary Progressive Nonfluent Aphasia - pathology | Aphasia, Primary Progressive - pathology | Frontotemporal Lobar Degeneration - physiopathology | Support Vector Machine | Alzheimer Disease - pathology | Gray Matter - diagnostic imaging | Frontotemporal Lobar Degeneration - classification | Pick Disease of the Brain - pathology | Primary Progressive Nonfluent Aphasia - physiopathology | White Matter - diagnostic imaging | Aged, 80 and over | Female | Aged | Aphasia, Primary Progressive - classification | Aphasia, Primary Progressive - physiopathology | Pathology | Neuropathology | Medical imaging | Index Medicus
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8. Full Text Divergent patterns of TDP-43 and tau pathologies in primary progressive aphasia
by Giannini, Lucia A. A and Xie, Sharon X and McMillan, Corey T and Liang, Mendy and Williams, Anew and Jester, Charles and Rascovsky, Katya and Wolk, David A and Ash, Sharon and Lee, Edward B and Trojanowski, John Q and Grossman, Murray and Irwin, David J
Annals of neurology, ISSN 0364-5134, 05/2019, Volume 85, Issue 5, pp. 630 - 643
LANGUAGE | ASYMMETRY | ATROPHY | ALZHEIMERS-DISEASE | WORDS | FRONTOTEMPORAL DEMENTIA | SEMANTIC VARIANT | TEMPORAL-LOBE | DEGENERATION | NONFLUENT | Neurosciences | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | DNA-Binding Proteins - metabolism | Aphasia, Primary Progressive - metabolism | Humans | Middle Aged | Cerebral Cortex - pathology | tau Proteins - metabolism | Female | Male | Aged | Aphasia, Primary Progressive - pathology | Cerebral Cortex - metabolism | Neuroimaging | Brain | Autopsies | Medical imaging | Identification methods | Regional analysis | Cortex | Standard error | Hemispheric laterality | Pathology | Neurology | Hemispheres | Histopathology | Magnetic resonance imaging | Language | Tau protein | Autopsy | Neurodegeneration | Neural networks | Dementia disorders | Aphasia | Degeneration | Frontotemporal dementia | Index Medicus
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9. Full Text Progranulin-associated PiB-negative logopenic primary progressive aphasia
by Josephs, Keith A and Duffy, Joseph R and Strand, Edythe A and Machulda, Mary M and Vemuri, Prashanthi and Senjem, Matthew L and Perkerson, Ralph B and Baker, Matthew C and Lowe, Val and Jack Jr, Clifford R and Rademakers, Rosa and Whitwell, Jennifer L
Journal of neurology, ISSN 0340-5354, 3/2014, Volume 261, Issue 3, pp. 604 - 614
FDG-PET | Neurology | Logopenic | Neurosciences | Progranulin | Medicine & Public Health | MRI | Primary progressive aphasia | Neuroradiology | β-amyloid | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Thiazoles - metabolism | Age Factors | Aniline Compounds - metabolism | Humans | Middle Aged | Atrophy - pathology | Cerebral Cortex - pathology | Magnetic Resonance Imaging - methods | Male | Tomography, X-Ray Computed | Aphasia, Primary Progressive - pathology | Parietal Lobe - metabolism | Positron-Emission Tomography - instrumentation | Parietal Lobe - physiopathology | Cerebral Cortex - metabolism | Cerebral Cortex - physiopathology | Multimodal Imaging | Amyloid beta-Peptides - metabolism | Female | Temporal Lobe - pathology | Aphasia, Primary Progressive - physiopathology | Atrophy - physiopathology | Intercellular Signaling Peptides and Proteins - genetics | Aphasia, Primary Progressive - genetics | Positron-Emission Tomography - methods | Mutation - genetics | Temporal Lobe - metabolism | Parietal Lobe - pathology | Neuropsychological Tests | Temporal Lobe - physiopathology | Magnetic Resonance Imaging - instrumentation | Language Tests | Aphasia, Primary Progressive - metabolism | Apolipoproteins E - genetics | Fluorodeoxyglucose F18 | Aged | Atrophy - metabolism | Aphasia | Complications and side effects | Development and progression | Research | Patient outcomes | Index Medicus
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10. Full Text Tau pathology and neurodegeneration contribute to cognitive impairment in Alzheimer's disease
by Bejanin, Alexandre and Schonhaut, Daniel R and La Joie, Renaud and Kramer, Joel H and Baker, Suzanne L and Sosa, Natasha and Ayakta, Nagehan and Cantwell, Averill and Janabi, Mustafa and Lauriola, Mariella and O'Neil, James P and Gorno-Tempini, Maria L and Miller, Zachary A and Rosen, Howard J and Miller, Bruce L and Jagust, William J and Rabinovici, Gil D
Brain (London, England : 1878), ISSN 0006-8950, 12/2017, Volume 140, Issue 12, pp. 3286 - 3300
atrophy | tau | cognitive impairment | amyloid | Alzheimer's disease | Neurosciences | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Brain - diagnostic imaging | Carbolines | Humans | Middle Aged | tau Proteins - metabolism | Male | Positron-Emission Tomography | Case-Control Studies | Aphasia, Primary Progressive - diagnostic imaging | Brain - metabolism | Amyloid - metabolism | Cognitive Dysfunction - psychology | Female | Aphasia, Primary Progressive - psychology | Cognitive Dysfunction - diagnostic imaging | Alzheimer Disease - psychology | Cognitive Dysfunction - metabolism | Neuropsychological Tests | Magnetic Resonance Imaging | Regression Analysis | Aphasia, Primary Progressive - metabolism | Alzheimer Disease - metabolism | Aged | Alzheimer Disease - diagnostic imaging | Fluorine Radioisotopes | Benzothiazoles | Carbon Radioisotopes | Index Medicus | Abridged Index Medicus | Alzheimer’s disease | Original
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