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Nihon rinsho. Japanese journal of clinical medicine, ISSN 0047-1852, 07/2007, Volume 65 Suppl 7, p. 600
Journal Article
Nihon rinsho. Japanese journal of clinical medicine, ISSN 0047-1852, 03/2001, Volume 59 Suppl 3, p. 281
Journal Article
Nihon rinsho. Japanese journal of clinical medicine, ISSN 0047-1852, 12/1999, Volume 57, Issue 12, p. 2795
Apolipoprotein (apo) A-I is the major apolipoprotein of HDL and is a single polypeptide chain with 243 amino acid residues. Familial apo A-I deficiency is a... 
Apolipoprotein A-I - genetics | Apolipoprotein A-I - deficiency | Amyloidosis - genetics | Humans
Journal Article
Rinsho byori. The Japanese journal of clinical pathology, ISSN 0047-1860, 07/1998, Volume 46, Issue 7, p. 684
Mutations in the apo A-I gene which affect apo A-I and HDL cholesterol levels have been described. We characterized two apo A-I deficiencies and 11 apo A-I... 
Coronary Disease - blood | Point Mutation | Apolipoprotein A-I - genetics | Apolipoprotein A-I - deficiency | Humans | Cholesterol, HDL - blood | Structure-Activity Relationship | Apolipoprotein A-I - chemistry
Journal Article
by Kato, H
Nihon rinsho. Japanese journal of clinical medicine, ISSN 0047-1852, 12/1994, Volume 52, Issue 12, p. 3253
Journal Article
Nihon rinsho. Japanese journal of clinical medicine, ISSN 0047-1852, 12/1994, Volume 52, Issue 12, p. 3246
Several epidemiological and clinical studies have shown an inverse correlation between low plasma concentration of high density lipoprotein (HDL) cholesterol... 
Genetic Variation | Apolipoprotein A-I - genetics | Coronary Disease | Apolipoprotein A-I - deficiency | Humans | Cholesterol, HDL - blood | Female | Male | Risk | Cholesterol, HDL - deficiency | Mutation
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 11/2018, Volume 39, Issue 1, pp. 63 - 72
OBJECTIVE—Apo (apolipoprotein) CIII inhibits lipoprotein lipase (LpL)-mediated lipolysis of VLDL (very-low-density lipoprotein) triglyceride (TG) and decreases... 
apolipoprotein C-III | cardiovascular diseases | lipolysis | isotopes | lipoprotein lipase | CLEARANCE | LOW-DENSITY-LIPOPROTEIN | TRIGLYCERIDE-RICH LIPOPROTEINS | RISK | OF-FUNCTION MUTATIONS | INHIBITION | PERIPHERAL VASCULAR DISEASE | LIPASE | HEMATOLOGY | A-I | APOLIPOPROTEIN-C-III | TRANSGENIC MICE
Journal Article
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 2014, Volume 50, Issue 4, pp. 787 - 795
Gata5 is a transcription factor expressed in the lung, but its physiological role is unknown. To test whether and how Gata5 regulates airway constrictor... 
IL-13 | Transcription factor | Apolipoprotein E | Methacholine | Asthma | transcription factor | CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | SUSCEPTIBILITY | asthma | APOLIPOPROTEIN-E | CELL BIOLOGY | GENE | RESPIRATORY SYSTEM | INFLAMMATION | GATA-4,-5 | DISEASE | apolipoprotein E | PROMOTER | methacholine | Asthma - metabolism | Pneumonia - physiopathology | Myocytes, Smooth Muscle - pathology | Apolipoproteins E - metabolism | Bronchial Hyperreactivity - genetics | Bronchoconstriction | GATA5 Transcription Factor - genetics | Interleukin-13 - genetics | Pneumonia - chemically induced | Bronchial Hyperreactivity - physiopathology | Bronchial Hyperreactivity - metabolism | Lung - metabolism | GATA5 Transcription Factor - deficiency | Myocytes, Smooth Muscle - metabolism | Disease Models, Animal | Pneumonia - genetics | Bronchial Hyperreactivity - chemically induced | Ovalbumin | Asthma - physiopathology | Mice, Inbred C57BL | Gene Expression Regulation | Genotype | Lung - physiopathology | Interleukin-13 - metabolism | Goblet Cells - metabolism | Asthma - chemically induced | Asthma - genetics | Mice, Knockout | Metaplasia | Phenotype | Animals | Apolipoproteins E - genetics | Mice | Goblet Cells - pathology | Pneumonia - metabolism | Flow cytometry | Genotype & phenotype | Lungs | Peptides | Lasers | Rodents | Cloning | Smooth muscle | Gene expression | Original Research
Journal Article
Intensive Care Medicine, ISSN 0342-4642, 11/2009, Volume 35, Issue 11, pp. 1877 - 1885
Lipoproteins modulate vascular cell function in inflammation. In this study, we analyzed whether plasma concentrations of lipoproteins and apolipoproteins in... 
HDL | Sepsis | Platelet | Apolipoprotein | Monocyte | CHOLESTEROL | DISEASE SEVERITY | HIGH-DENSITY-LIPOPROTEIN | SERUM-LIPIDS | PLASMA | INHIBITION | STATIN THERAPY | LIPOPOLYSACCHARIDE | INFLAMMATORY RESPONSE | A-I | CRITICAL CARE MEDICINE | Predictive Value of Tests | Prognosis | Prospective Studies | Hypolipoproteinemias - immunology | Cholesterol - blood | Humans | Middle Aged | Platelet Activation - immunology | HLA-DR Antigens - blood | Male | Apolipoproteins B - blood | Monocytes - immunology | Hypolipoproteinemias - complications | Apolipoproteins - deficiency | Statistics, Nonparametric | Apolipoproteins B - deficiency | Adult | Cholesterol, LDL - blood | Female | Lipoproteins, HDL - deficiency | Hypolipoproteinemias - blood | Sepsis - immunology | Apolipoprotein A-I - deficiency | Cholesterol - deficiency | Logistic Models | Survival Rate | Apolipoproteins - immunology | Germany - epidemiology | Sepsis - mortality | Lipoproteins, HDL - immunology | Cholesterol, HDL - blood | P-Selectin - blood | ROC Curve | Aged | Cholesterol, HDL - deficiency | Sepsis - blood | APACHE | Apolipoprotein A-I - blood | Physiological aspects | Development and progression | Genetic aspects | Research | Apolipoproteins | Lipoproteins (low density) | Mortality | Inflammation | Regression analysis | Vascular system | SAP protein | Blood | Cholesterol | Lipopolysaccharides | Intensive care units | Cell activation | Lipoproteins | Monocytes | Hospitals | Histocompatibility antigen HLA | Platelets | Lipoproteins (high density)
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 02/2009, Volume 104, Issue 4, pp. 455 - 465
The molecular events linking lipid accumulation in atherosclerotic plaques to complications such as aneurysm formation and plaque disruption are poorly... 
P38 | Toll-like receptor | Cathepsin K | PATHWAYS | CARDIAC & CARDIOVASCULAR SYSTEMS | INTRACELLULAR-TRANSPORT | LOW-DENSITY-LIPOPROTEIN | ATHEROSCLEROSIS | FOAM CELLS | PLAQUE | p38 | DISEASE | APOE-DEFICIENT MICE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | cathepsin K | OSTEOCLAST DIFFERENTIATION | EXPRESSION | p38 Mitogen-Activated Protein Kinases - deficiency | Endosomes - immunology | Phosphorylation | Apolipoproteins E - deficiency | Calgranulin A | Humans | Adaptor Proteins, Vesicular Transport - deficiency | Adaptor Proteins, Vesicular Transport - genetics | Aorta - metabolism | Endosomes - metabolism | Time Factors | Toll-Like Receptor 4 - deficiency | Matrix Metalloproteinase 8 - metabolism | Toll-Like Receptor 3 - deficiency | Cell Membrane - metabolism | Toll-Like Receptor 3 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Receptor Activator of Nuclear Factor-kappa B - metabolism | Macrophages - immunology | rab GTP-Binding Proteins - metabolism | Promoter Regions, Genetic | Microphthalmia-Associated Transcription Factor - metabolism | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Enzyme Induction | Toll-Like Receptor 4 - genetics | p38 Mitogen-Activated Protein Kinases - genetics | S100 Proteins - metabolism | Cholesterol - metabolism | Matrix Metalloproteinase 14 - metabolism | Toll-Like Receptor 3 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Inbred C3H | E-Box Elements | Macrophages - enzymology | Mice, Knockout | Proteins - genetics | Cell Membrane - enzymology | Macrophages - metabolism | Animals | Proteins - metabolism | Apolipoproteins E - genetics | Cell Membrane - immunology | Endosomes - enzymology | Mice | Cathepsins - biosynthesis
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2015, Volume 35, Issue 8, pp. 1826 - 1834
Journal Article
Circulation, ISSN 0009-7322, 09/2017, Volume 136, Issue 12, pp. 1140 - 1154
Journal Article
Journal Article
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