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Nature (London), ISSN 1476-4687, 2012, Volume 485, Issue 7399, pp. 512 - 516
Journal Article
JAMA neurology, ISSN 2168-6149, 08/2013, Volume 70, Issue 8, pp. 972 - 980
Journal Article
Nature reviews. Neurology, ISSN 1759-4766, 2013, Volume 9, Issue 2, pp. 106 - 118
Apolipoprotein E (Apo-E) is a major cholesterol carrier that supports lipid transport and injury repair in the brain... 
E GENOTYPE | CEREBRAL AMYLOID ANGIOPATHY | A-BETA DEPOSITION | GENETIC RISK | APOE EPSILON-4 ALLELE | E TYPE-4 ALLELE | MILD COGNITIVE IMPAIRMENT | HIPPOCAMPAL NEUROGENESIS | CLINICAL NEUROLOGY | DENSITY-LIPOPROTEIN RECEPTOR | GENOME-WIDE ASSOCIATION | Inflammation - pathology | Predictive Value of Tests | Humans | tau Proteins - metabolism | Dementia - physiopathology | Risk | Brain Injuries - physiopathology | Dementia - genetics | Apolipoproteins E - metabolism | Neurogenesis - genetics | Neuronal Plasticity - genetics | Neuronal Plasticity - physiology | Apolipoproteins E - antagonists & inhibitors | Immunotherapy | Amyloid beta-Peptides - metabolism | Cognitive Dysfunction - psychology | Alzheimer Disease - epidemiology | Lipid Metabolism - genetics | Alzheimer Disease - psychology | Alzheimer Disease - therapy | Dendritic Spines - physiology | Vascular Diseases - genetics | Vascular Diseases - physiopathology | Brain Injuries - genetics | Cognitive Dysfunction - pathology | Amyloid beta-Peptides - antagonists & inhibitors | Animals | Neurogenesis - physiology | Apolipoproteins E - genetics | Alzheimer Disease - metabolism | Apolipoproteins E - physiology | Brain - pathology | Inflammation - genetics | Alzheimer Disease - genetics | Lipid Metabolism - physiology | Allelomorphism | Physiological aspects | Genetic aspects | Research | Apolipoproteins | Alzheimer's disease | Risk factors | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology: Journal of the American Heart Association, ISSN 1079-5642, 06/2004, Volume 24, Issue 6, pp. 1006 - 1014
.... Among available models, the apolipoprotein E–deficient (apoE−/−) mouse is particularly popular because of its propensity to spontaneously develop atherosclerotic lesions on a standard chow diet... 
Mouse model | Apolipoprotein E | Knockout | Atherosclerosis | atherosclerosis | knockout | APOE KNOCKOUT MICE | ACTIVATED RECEPTOR-GAMMA | mouse model | LIPID-PEROXIDATION | HYPERLIPIDEMIC MICE | ACYL-COA | REDUCES ATHEROSCLEROSIS | IN-VIVO | apolipoprotein E | PERIPHERAL VASCULAR DISEASE | LESION DEVELOPMENT | RED WINE | HEMATOLOGY | CHOLESTEROL ACYLTRANSFERASE INHIBITOR | Apolipoproteins E - deficiency | Arteriosclerosis - drug therapy | Fatty Acids - therapeutic use | Male | Monocytes - metabolism | Arteriosclerosis - genetics | Fatty Acids - toxicity | Brain - metabolism | Biological Transport | Arteriosclerosis - diet therapy | Female | Foam Cells - pathology | Foam Cells - metabolism | Disease Models, Animal | Hyperlipoproteinemia Type II - complications | Liver - metabolism | Dietary Fats - therapeutic use | Cholesterol - metabolism | Fibrinolysis | Mice, Knockout | Arteriosclerosis - prevention & control | Animals | Receptors, Scavenger | Apolipoproteins E - genetics | Apolipoproteins E - physiology | Arteriosclerosis - metabolism | Hypolipidemic Agents - therapeutic use | Mice | Fatty Acids - pharmacokinetics | Arteriosclerosis - pathology | Dietary Fats - toxicity | Dietary Fats - pharmacokinetics | Receptors, Immunologic - metabolism | Hyperlipoproteinemia Type II - genetics
Journal Article