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1. Full Text Systemic Delivery of MicroRNA-181b Inhibits Nuclear Factor-κB Activation, Vascular Inflammation, and Atherosclerosis in Apolipoprotein E–Deficient Mice
by Sun, Xinghui and He, Shaolin and Wara, A.K.M and Icli, Basak and Shvartz, Eugenia and Tesmenitsky, Yevgenia and Belkin, Nathan and Li, Dazhu and Blackwell, Timothy S and Sukhova, Galina K and Croce, Kevin and Feinberg, Mark W
Circulation research, ISSN 0009-7330, 01/2014, Volume 114, Issue 1, pp. 32 - 40
atherosclerosis | microRNAs | NF-κB | endothelial cells | karyopherins | inflammation | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | MicroRNAs - therapeutic use | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Diet, High-Fat - adverse effects | MicroRNAs - metabolism | NF-kappa B - metabolism | Aorta - metabolism | Atherosclerosis - etiology | Inflammation - metabolism | Atherosclerosis - therapy | Atherosclerosis - pathology | NF-kappa B - antagonists & inhibitors | MicroRNAs - administration & dosage | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Atherosclerosis - metabolism | Aorta - pathology | Tunica Intima - metabolism | Macrophages - metabolism | Animals | Karyopherins - metabolism | Tunica Intima - pathology | Apolipoproteins E - genetics | Karyopherins - genetics | Mice | MicroRNAs - blood | Index Medicus
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2. Full Text The Cholesterol Metabolite 27-Hydroxycholesterol Promotes Atherosclerosis via Proinflammatory Processes Mediated by Estrogen Receptor Alpha
by Umetani, Michihisa and Ghosh, Pritam and Ishikawa, Tomonori and Umetani, Junko and Ahmed, Mohamed and Mineo, Chieko and Shaul, Philip W
Cell metabolism, ISSN 1550-4131, 07/2014, Volume 20, Issue 1, pp. 172 - 182
Endocrinology & Metabolism | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Steroid Hydroxylases - metabolism | Apolipoproteins E - deficiency | JNK Mitogen-Activated Protein Kinases - metabolism | Male | NF-kappa B - metabolism | I-kappa B Proteins - metabolism | Apolipoproteins E - metabolism | Cytochrome P450 Family 7 | RNA Interference | Estrogen Receptor alpha - metabolism | Female | Cytokines - genetics | Steroid Hydroxylases - genetics | Macrophages - immunology | Cell Line | Atherosclerosis - pathology | NF-KappaB Inhibitor alpha | Cytokines - metabolism | Endothelial Cells - metabolism | Inflammation | Cell Adhesion - drug effects | Cholesterol - metabolism | Hydroxycholesterols - pharmacology | Atherosclerosis - metabolism | Mice, Knockout | Macrophages - metabolism | Animals | Estrogen Receptor alpha - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Endothelial Cells - cytology | Apolipoproteins E - genetics | Steroid Hydroxylases - deficiency | Hydroxycholesterols - metabolism | Macrophages - drug effects | Mice | Endothelial Cells - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | RNA, Small Interfering - metabolism | Metabolites | Atherosclerosis | Estrogen | Cytochrome P-450 | Physiological aspects | Phenols | Cholesterol | Prevention | Blood circulation disorders | Liver | Endothelium | Index Medicus
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3. Full Text Artery Tertiary Lymphoid Organs Control Multilayered Territorialized Atherosclerosis B-Cell Responses in Aged ApoE−/− Mice
by Srikakulapu, Prasad and Hu, Desheng and Yin, Changjun and Mohanta, Sarajo K and Bontha, Sai Vineela and Peng, Li and Beer, Michael and Weber, Christian and McNamara, Coleen A and Grassia, Gianluca and Maffia, Pasquale and Manz, Rudolf A and Habenicht, Andreas J.R
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 06/2016, Volume 36, Issue 6, pp. 1174 - 1185
atherosclerosis | B-lymphocytes | aging | germinal center | inflammation | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Autoimmunity | Tertiary Lymphoid Structures - immunology | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Autoantibodies - blood | Germinal Center - immunology | Transcriptome | Male | Aorta - metabolism | Aortic Diseases - metabolism | Aging - immunology | Aging - genetics | Immunoglobulins - blood | Plasma Cells - metabolism | B-Lymphocytes - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Atherosclerosis - pathology | Genetic Predisposition to Disease | Atherosclerosis - immunology | Cytokines - metabolism | Signal Transduction | Aortic Diseases - immunology | Malondialdehyde - analogs & derivatives | Mice, Inbred C57BL | Gene Expression Regulation | Lymph Nodes - metabolism | Aorta - immunology | Lymph Nodes - immunology | Atherosclerosis - metabolism | Mice, Knockout | Tertiary Lymphoid Structures - pathology | Aorta - pathology | Aortic Diseases - genetics | Phenotype | Animals | B-Lymphocytes - immunology | Germinal Center - metabolism | Malondialdehyde - immunology | Apolipoproteins E - genetics | Tertiary Lymphoid Structures - metabolism | Immunologic Memory | Lipoproteins, LDL - immunology | Aging - metabolism | Plasma Cells - immunology | Index Medicus | 10195 | 10187 | 10188 | Basic Sciences
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4. Full Text Apolipoprotein B: a clinically important apolipoprotein which assembles atherogenic lipoproteins and promotes the development of atherosclerosis
by OLOFSSON, S.‐O and BORÈN, J and Sahlgrenska akademin and Wallenberg Laboratory and Wallenberglaboratoriet and Göteborgs universitet and Gothenburg University and Sahlgrenska Academy
Journal of internal medicine, ISSN 0954-6820, 11/2005, Volume 258, Issue 5, pp. 395 - 410
atherosclerosis | proteoglycans | apolipoproteins B | very low‐density lipoprotein assembly | very low‐density lipoproteins | Very low-density lipoproteins | Proteoglycans | Apolipoproteins B | Very low-density lipoprotein assembly | Atherosclerosis | Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Cardiology. Vascular system | Biological and medical sciences | General aspects | Medical sciences | Atherosclerosis (general aspects, experimental research) | Blood and lymphatic vessels | Arteriosclerosis - etiology | Lipoproteins, VLDL - chemistry | Apolipoproteins B - metabolism | Hyperlipidemias - metabolism | Glycosaminoglycans - metabolism | Humans | Risk Factors | Proteoglycans - metabolism | Apolipoprotein B-100 | Apolipoprotein B-48 | Hyperlipidemias - prevention & control | Triglycerides - metabolism | Models, Biological | Lipoproteins, VLDL - metabolism | Arteriosclerosis - metabolism | Apolipoproteins B - chemistry | Index Medicus | Proteoglycans/metabolism | Apolipoproteins B/chemistry/metabolism | Triglycerides/metabolism | Arteriosclerosis/etiology/metabolism | MEDICIN OCH HÄLSOVETENSKAP | Biological | Hyperlipidemias/metabolism/prevention & control | Lipoproteins | Glycosaminoglycans/metabolism | Models | VLDL/chemistry/metabolism | MEDICAL AND HEALTH SCIENCES
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5. Full Text Insulin Resistance, Hyperglycemia, and Atherosclerosis
by Bornfeldt, Karin E and Tabas, Ira
Cell metabolism, ISSN 1550-4131, 11/2011, Volume 14, Issue 5, pp. 575 - 585
Endocrinology & Metabolism | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Apolipoproteins B - metabolism | Muscle, Smooth, Vascular - metabolism | Humans | Hyperglycemia - complications | Diabetes Mellitus, Type 2 - metabolism | Coronary Artery Disease - pathology | Hyperglycemia - pathology | Diabetes Mellitus, Type 2 - complications | Atherosclerosis - pathology | Signal Transduction | Endothelial Cells - metabolism | Coronary Artery Disease - metabolism | Risk Factors | Insulin Resistance | Atherosclerosis - complications | Apolipoproteins B - genetics | Macrophages - cytology | Muscle, Smooth, Vascular - cytology | Atherosclerosis - metabolism | Mice, Knockout | Hyperglycemia - metabolism | Insulin - metabolism | Macrophages - metabolism | Animals | Endothelial Cells - cytology | Coronary Artery Disease - complications | Receptor, Insulin - metabolism | Mice | Diabetes Mellitus, Type 2 - pathology | Blood Glucose - metabolism | Type 2 diabetes | Insulin resistance | Hyperglycemia | Coronary heart disease | Atherosclerosis | Index Medicus
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6. Full Text Ectopic lipid storage and insulin resistance: a harmful relationship
by Borén, J and Taskinen, M.‐R and Olofsson, S.‐O and Levin, M and Sahlgrenska akademin and Institute of Medicine, Department of Molecular and Clinical Medicine and Institutionen för medicin, avdelningen för molekylär och klinisk medicin and Göteborgs universitet and Gothenburg University and Sahlgrenska Academy
Journal of internal medicine, ISSN 0954-6820, 07/2013, Volume 274, Issue 1, pp. 25 - 40
lipids | NAFLD | storage | lipid droplets | insulin resistance | lipotoxicity | Insulin resistance | Lipids | Storage | Lipotoxicity | Lipid droplets | Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Humans | Muscle, Skeletal - metabolism | Diabetes Mellitus, Type 2 - metabolism | Phosphoproteins - metabolism | Inflammation - metabolism | Myocardium - metabolism | Lipoproteins - metabolism | Diabetes Mellitus, Type 2 - etiology | Lipogenesis | Fatty Liver - metabolism | Signal Transduction | Obesity - complications | Liver - metabolism | Insulin Resistance | Lipid Metabolism | Heart Failure - metabolism | Immunity, Innate | Toll-Like Receptor 4 - metabolism | Obesity - metabolism | Triglycerides - metabolism | Animals | Carrier Proteins - metabolism | Metabolic Diseases - metabolism | Lipoproteins, VLDL - metabolism | alpha-2-HS-Glycoprotein - metabolism | Perilipin-1 | Type 2 diabetes | Muscles | Development and progression | Index Medicus | pleckstrin homology domain | Klinisk medicin | fatty-acid oxidation | low-density lipoprotein | apolipoprotein-b secretion | triglyceride transfer protein | Clinical Medicine | deletion | tyrosine-phosphatase 1b | endoplasmic-reticulum stress | droplet-associated protein | differentiation-related protein | liver-specific
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