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Cell Metabolism, ISSN 1550-4131, 11/2017, Volume 26, Issue 5, pp. 719 - 737.e6
Journal Article
Cardiovascular Research, ISSN 0008-6363, 08/2017, Volume 113, Issue 10, pp. 1208 - 1218
Aims Although surfactant protein-D (SP-D) is a pneumoprotein that is predominantly synthesized by type II epithelial cells in the lung, individuals with... 
Surfactant protein D | Apolipoprotein E | IL-6 | Atherosclerosis | Macrophage | C-REACTIVE PROTEIN | MORTALITY | CARDIAC & CARDIOVASCULAR SYSTEMS | MONOCYTE COUNT | MOUSE | INTERLEUKIN-6 | OBSTRUCTIVE PULMONARY-DISEASE | SMOOTH-MUSCLE-CELLS | INTRAPLAQUE HEMORRHAGE | EXPRESSION | ADIPOSE-TISSUE | Inflammation - pathology | Cell Proliferation | Muscle, Smooth, Vascular - metabolism | Atherosclerosis - genetics | Myocytes, Smooth Muscle - pathology | Male | Aorta - metabolism | Obesity - genetics | Aortic Diseases - metabolism | Inflammation - metabolism | Interleukin-6 - blood | Aortic Diseases - prevention & control | Myocytes, Smooth Muscle - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Pulmonary Surfactant-Associated Protein D - deficiency | Atherosclerosis - pathology | Genetic Predisposition to Disease | Macrophages - pathology | Mice, Inbred C57BL | Mice, Knockout, ApoE | Plaque, Atherosclerotic | Atherosclerosis - metabolism | Pulmonary Surfactant-Associated Protein D - genetics | Macrophage Activation | Obesity - metabolism | Aorta - pathology | Aortic Diseases - genetics | Macrophages - metabolism | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Insulin Resistance - genetics | Inflammation - genetics | Inflammation - prevention & control | Hypercholesterolemia - genetics | Hypercholesterolemia - metabolism | Atherosclerosis - prevention & control | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2013, Volume 110, Issue 42, pp. 17023 - 17028
Mice with homozygous null mutations in the HDL receptor (scavenger receptor class B, type I, or SR-BI) and apolipoprotein E (apoE) genes [SR-BI/apoE double KO... 
Heart | Antioxidants | Ischemia | Adenoviruses | Atherosclerosis | Myocardium | Mice | Gene expression | Coronary artery disease | Cardiovascular agents | Antioxidant | Lipocalin | ANTIOXIDANT ACTIVITY | antioxidant | LIPID-METABOLISM | MYOCARDIAL-INFARCTION | RECEPTOR SR-BI | MULTIDISCIPLINARY SCIENCES | lipocalin | CARDIAC DYSFUNCTION | HYPERCHOLESTEROLEMIA | KNOCKOUT MICE | HEART-DISEASE | PREMATURE DEATH | E-DEFICIENT MICE | Myocardial Infarction - blood | Myocardial Infarction - genetics | Rats, Wistar | Apolipoproteins D - genetics | Antioxidants - metabolism | Humans | Apolipoproteins E - metabolism | Coronary Artery Disease - blood | Myocardial Reperfusion Injury - pathology | Myocardial Infarction - pathology | Myocardium - metabolism | Coronary Artery Disease - pathology | Female | Myocardial Reperfusion Injury - genetics | Myocardial Reperfusion Injury - blood | Disease Models, Animal | Coronary Artery Disease - metabolism | Cells, Cultured | Myocardium - pathology | Scavenger Receptors, Class B - genetics | Mice, Knockout | Myocytes, Cardiac - pathology | Animals | Apolipoproteins E - genetics | Coronary Artery Disease - genetics | Myocytes, Cardiac - metabolism | Scavenger Receptors, Class B - metabolism | Cell Hypoxia - genetics | Apolipoproteins D - blood | Physiological aspects | Genetic aspects | Research | Apolipoproteins | Coronary heart disease | Health aspects | Cardiovascular disease | Oxidation | Mutation | Rodents | Index Medicus | Biological Sciences
Journal Article
PLoS Genetics, ISSN 1553-7390, 02/2017, Volume 13, Issue 2, pp. e1006603 - e1006603
Journal Article
Experimental Gerontology, ISSN 0531-5565, 07/2015, Volume 67, pp. 19 - 47
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, pp. e54625 - e54625
Multiple epidemiological studies link vitamin D deficiency to increased cardiovascular disease (CVD), but causality and possible mechanisms underlying these... 
CELLS | ACTIVATION | 25-HYDROXYVITAMIN-D | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | CALCIUM | NATIONAL-HEALTH | RISK | ENDOPLASMIC-RETICULUM STRESS | HYPERTENSION | MACROPHAGE DIFFERENTIATION | Receptors, LDL - genetics | Vitamin D Deficiency - complications | Atherosclerosis - physiopathology | Humans | Receptors, LDL - metabolism | Atherosclerosis - complications | Vitamin D Deficiency - physiopathology | Apolipoproteins E - metabolism | Atherosclerosis - metabolism | Hypertension - physiopathology | Hypertension - metabolism | Mice, Knockout | Macrophages - metabolism | Vitamin D - genetics | Animals | Apolipoproteins E - genetics | Diet, High-Fat | Endoplasmic Reticulum Stress | Hypertension - complications | Vitamin D - administration & dosage | Vitamin D - metabolism | Mice | Vitamin D Deficiency - metabolism | Aorta, Abdominal - metabolism | Hypertension | Low density lipoproteins | Heat shock proteins | Calcifediol | Macrophages | Apolipoproteins | Epidemiology | Vitamin D deficiency | Diet therapy | Vitamin D | Analysis | Atherosclerosis | Angiotensin | Blood pressure | Alfacalcidol | Lipoproteins (low density) | Animal models | Cardiovascular disease | Blood | Proteins | Cell activation | Renin | Apolipoprotein E | Rodents | Aorta | Plaques | Excretion | Nutrient deficiency | Mortality | Vitamin deficiency | Thorax | Stress | Sodium | Arteriosclerosis | Aortic arch | Infiltration | Cardiovascular diseases | Stress proteins | Endoplasmic reticulum | Peritoneum | Index Medicus
Journal Article
Breast Cancer Research, ISSN 1465-5411, 03/2016, Volume 18, Issue 1, pp. 31 - 31
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2008, Volume 105, Issue 19, pp. 7088 - 7093
Apolipoprotein D (ApoD) expression increases in several neurological disorders and in spinal cord injury. We provide a report of a physiological role for human... 
Oxidative stress | Up regulation | Messenger RNA | Complementary DNA | Neurons | Drosophila | Neuroglia | Hyperoxia | Lipids | Lipid peroxides | Aging | Alzheimer | β-amyloid | Glaz | OXIDATIVE-STRESS | LIPID-PEROXIDATION | MULTIDISCIPLINARY SCIENCES | HOMOLOG | IDENTIFICATION | PATHOPHYSIOLOGY | beta-amyloid | EVOLUTION | TISSUE DISTRIBUTION | PSYCHIATRIC-DISORDERS | aging | MELANOGASTER | EXPRESSION | oxidative stress | GLaz | Membrane Glycoproteins - metabolism | Apolipoproteins D - genetics | Longevity - drug effects | Peptide Fragments - toxicity | Humans | Aging - drug effects | Lipid Peroxides - metabolism | Drosophila Proteins - metabolism | RNA, Messenger - metabolism | Drosophila melanogaster - genetics | Membrane Transport Proteins - genetics | Glycoproteins - genetics | Neurodegenerative Diseases - pathology | Amyloid beta-Peptides - toxicity | Animals, Genetically Modified | RNA, Messenger - genetics | Drosophila melanogaster - cytology | Neurodegenerative Diseases - genetics | Up-Regulation - genetics | Drosophila melanogaster - drug effects | Membrane Glycoproteins - genetics | Up-Regulation - drug effects | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Drosophila Proteins - genetics | Paraquat - toxicity | Control | Physiological aspects | Nervous system | Degeneration | Research | Apolipoproteins | Properties | Health aspects | Proteins | Parkinsons disease | Spinal cord injuries | Oxidation | Alzheimers disease | Neurological disorders | Cells | Index Medicus | Biological Sciences
Journal Article