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1. Full Text Phosphatidylcholine biosynthesis and lipoprotein metabolism
by Cole, Laura K and Vance, Jean E and Vance, Dennis E
Biochimica et biophysica acta. Molecular and cell biology of lipids, ISSN 1388-1981, 05/2012, Volume 1821, Issue 5, pp. 754 - 761
Phosphatidylcholine | Apo B | Lipoprotein | Liver | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Endoplasmic Reticulum-Associated Degradation | Cardiovascular Diseases - etiology | Cardiovascular Diseases - metabolism | Humans | Liver - metabolism | Phosphatidylcholines - biosynthesis | Lipoproteins, HDL - metabolism | Receptors, LDL - metabolism | Phosphatidylcholines - metabolism | Apolipoproteins E - metabolism | Animals | Lipoproteins, VLDL - blood | Metabolic Networks and Pathways | Lipoproteins, VLDL - metabolism | Lipoproteins, VLDL - secretion | Physiological aspects | Enzymes | Phospholipids | Triglycerides | Blood lipids | Low density lipoproteins
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2. Full Text Allergic asthma aggravated atherosclerosis increases cholesterol biosynthesis and foam cell formation in apolipoprotein E-deficient mice
by Gao, Shanshan and Wang, Chen and Li, Wenyuan and Shu, Shan and Zhou, Juan and Yuan, Zuyi and Wang, Lijun
Biochemical and biophysical research communications, ISSN 0006-291X, 11/2019, Volume 519, Issue 4, pp. 861 - 867
Apolipoprotein E deficient mice | Allergic asthma | Foam cell | Cholesterol | Atherosclerosis | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | Index Medicus
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3. Full Text Impaired Phosphatidylcholine Biosynthesis Reduces Atherosclerosis and Prevents Lipotoxic Cardiac Dysfunction in ApoE−/− Mice
by Cole, Laura K and Dolinsky, Vernon W and Dyck, Jason R.B and Vance, Dennis E
Circulation research, ISSN 0009-7330, 03/2011, Volume 108, Issue 6, pp. 686 - 694
atherosclerosis | apolipoprotein E | lipotoxic cardiomyopathy | phosphatidylethanolamine N-methyltransferase | phosphatidylcholine | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Fundamental and applied biological sciences. Psychology | Cardiology. Vascular system | Heart | Atherosclerosis (general aspects, experimental research) | Biological and medical sciences | Myocarditis. Cardiomyopathies | Medical sciences | Vertebrates: cardiovascular system | Blood and lymphatic vessels | Heart - physiopathology | Atherosclerosis - pathology | Apolipoproteins E - deficiency | Atherosclerosis - physiopathology | Heart Diseases - physiopathology | Cholesterol - blood | Liver - metabolism | Phosphatidylcholines - biosynthesis | Heart Diseases - prevention & control | Lipid Metabolism | Myocardium - pathology | Lipoproteins - blood | Mice, Knockout | Atherosclerosis - etiology | Heart Diseases - etiology | Myocardium - enzymology | Animals | Triglycerides - antagonists & inhibitors | Phosphatidylethanolamine N-Methyltransferase - deficiency | Aging | Aorta | Mice | Phosphatidylcholines - deficiency | Atherosclerosis - prevention & control | Index Medicus
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4. Full Text Apolipoprotein E Enhances MicroRNA-146a in Monocytes and Macrophages to Suppress Nuclear Factor-κB–Driven Inflammation and Atherosclerosis
by Li, Kang and Ching, Daniel and Luk, Fu Sang and Raffai, Robert L
Circulation research, ISSN 0009-7330, 06/2015, Volume 117, Issue 1, pp. e1 - e11
atherosclerosis | microRNAs | macrophages | apolipoproteins E | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Transcription, Genetic - drug effects | Hyperlipidemias - genetics | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Diet, High-Fat - adverse effects | Monocytes - metabolism | NF-kappa B - metabolism | Proto-Oncogene Proteins - biosynthesis | Trans-Activators - physiology | Atherosclerosis - etiology | Receptors, LDL - deficiency | Trans-Activators - genetics | MicroRNAs - agonists | Trans-Activators - biosynthesis | Hyperlipidemias - complications | NF-kappa B - antagonists & inhibitors | Lipopolysaccharides - toxicity | Specific Pathogen-Free Organisms | Hyperlipidemias - metabolism | Mice, Inbred C57BL | MicroRNAs - biosynthesis | Proto-Oncogene Proteins - genetics | Inflammation - etiology | Mice, Knockout | Monocytes - drug effects | Macrophages - metabolism | Animals | Apolipoproteins E - genetics | Proto-Oncogene Proteins - physiology | Apolipoproteins E - physiology | Inflammation - genetics | Macrophages - drug effects | Inflammation - prevention & control | Mice | MicroRNAs - genetics | MicroRNAs - physiology | Atherosclerosis - prevention & control | Index Medicus | apoE | miR-146a | microRNA | Atherosclerosis | NF-Kb
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5. Full Text Lack of the antioxidant enzyme glutathione peroxidase-1 accelerates atherosclerosis in diabetic apolipoprotein E-deficient mice
by Lewis, Paul and Stefanovic, Nada and Pete, Josefa and Calkin, Anna C and Giunti, Sara and Thallas-Bonke, Vicki and Jandeleit-Dahm, Karin A and Allen, Terri J and Kola, Ismail and Cooper, Mark E and De Haan, Judy B
Circulation (New York, N.Y.), ISSN 0009-7322, 04/2007, Volume 115, Issue 16, pp. 2178 - 2187
Antioxidants | Aorta | Free radicals | Cardiovascular diseases | Diabetes mellitus | Atherosclerosis | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Diabetes. Impaired glucose tolerance | Diseases of the peripheral vessels. Diseases of the vena cava. Miscellaneous | Atherosclerosis (general aspects, experimental research) | Biological and medical sciences | Endocrinopathies | Medical sciences | Blood and lymphatic vessels | Endocrine pancreas. Apud cells (diseases) | Associated diseases and complications | Superoxide Dismutase - genetics | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Membrane Glycoproteins - biosynthesis | Male | Aorta - metabolism | Vascular Endothelial Growth Factor A - genetics | Tyrosine - analogs & derivatives | NADPH Oxidases - genetics | Diabetes Mellitus, Experimental - complications | Glutathione Peroxidase - deficiency | NADPH Oxidases - biosynthesis | Receptor for Advanced Glycation End Products | Atherosclerosis - pathology | Macrophages - pathology | Oxidation-Reduction | Tyrosine - biosynthesis | Isoenzymes - genetics | Immediate-Early Proteins - biosynthesis | Glutathione Peroxidase - genetics | Mice, Knockout | Aorta - pathology | Aortic Diseases - genetics | Immediate-Early Proteins - genetics | NF-kappa B - biosynthesis | Fibrosis | Glutathione Peroxidase - physiology | Vascular Cell Adhesion Molecule-1 - biosynthesis | Mice | Receptors, Immunologic - genetics | Inflammation - enzymology | Vascular Endothelial Growth Factor A - biosynthesis | Superoxide Dismutase - biosynthesis | Glutathione - metabolism | Streptozocin | Intercellular Signaling Peptides and Proteins - biosynthesis | Atherosclerosis - etiology | Atherosclerosis - enzymology | Receptors, Immunologic - biosynthesis | Diabetic Angiopathies - complications | Diabetic Angiopathies - enzymology | Vascular Cell Adhesion Molecule-1 - genetics | Aortic Diseases - enzymology | Sinus of Valsalva - pathology | Aortic Diseases - pathology | Hyperlipoproteinemia Type II - complications | Mice, Inbred C57BL | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Glutathione Peroxidase - biosynthesis | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Animals | NF-kappa B - genetics | Apolipoproteins E - genetics | Inflammation - genetics | Isoenzymes - biosynthesis | Aortic Diseases - etiology | Connective Tissue Growth Factor | Hyperlipoproteinemia Type II - genetics | Animal models in research | Physiological aspects | Development and progression | Peroxidase | Research | Apolipoproteins | Glutathione | Index Medicus | Abridged Index Medicus
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6. Full Text The genetic architecture of Alzheimer's disease: beyond APP, PSENs and APOE
by Guerreiro, Rita J and Gustafson, Deborah R and Hardy, John and Sahlgrenska akademin and Institutionen för neurovetenskap och fysiologi, sektionen för psykiatri och neurokemi and Göteborgs universitet and Gothenburg University and Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry and Sahlgrenska Academy
Neurobiology of aging, ISSN 0197-4580, 2012, Volume 33, Issue 3, pp. 437 - 456
Neurology | Internal Medicine | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Geriatrics & Gerontology | Science & Technology | Presenilin-2 - biosynthesis | Amyloid beta-Protein Precursor - chemistry | Humans | Presenilin-1 - genetics | Alzheimer Disease - pathology | Apolipoproteins E - chemistry | Amyloid beta-Protein Precursor - genetics | Animals | Presenilin-2 - chemistry | Presenilin-2 - genetics | Apolipoproteins E - genetics | Alzheimer Disease - metabolism | Presenilin-1 - chemistry | Alzheimer Disease - genetics | Presenilin-1 - biosynthesis | Amyloid beta-Protein Precursor - biosynthesis | Apolipoproteins E - biosynthesis | Genes | Genetic research | Genetics | Apolipoproteins | Alzheimer's disease | Risk factors | Index Medicus | Psykiatri | Psychiatry
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7. Full Text Thioesterase Superfamily Member 2 Promotes Hepatic VLDL Secretion by Channeling Fatty Acids Into Triglyceride Biosynthesis
by Alves‐Bezerra, Michele and Li, Yingxia and Acuña, Mariana and Ivanova, Anna A and Corey, Kathleen E and Ortlund, Eric A and Cohen, David E
Hepatology (Baltimore, Md.), ISSN 0270-9139, 08/2019, Volume 70, Issue 2, pp. 496 - 510
Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Obesity | Phenotypes | Thioesterase | Liver diseases | Secretion | Liver | Dyslipidemia | Homeostasis | Triglycerides | Apolipoproteins | Fatty acids | Insulin | Lipoproteins (very low density) | Skeletal muscle | Steatosis | Glucose tolerance | Intolerance | Mitochondria | Fatty liver | Thermogenesis | Coenzyme A | Oxidation | Lipogenesis | Adipose tissue (brown) | Index Medicus | nonalcoholic fatty liver disease | Fatty acid metabolism | very low-density lipoprotein | fatty acyl-CoA | fatty acid oxidation | triglycerides | dyslipidemia | obesity
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8. Full Text Expression of adhesion, attachment and invasion markers in eutopic and ectopic endometrium: a link to the aetiology of endometriosis
by Sundqvist, J and Andersson, K.L and Scarselli, G and Gemzell-Danielsson, K and Lalitkumar, P.G.L
Human reproduction (Oxford), ISSN 0268-1161, 9/2012, Volume 27, Issue 9, pp. 2737 - 2746
endometriosis | endometrium | attachment | invasion | cell adhesion | Life Sciences & Biomedicine | Obstetrics & Gynecology | Reproductive Biology | Science & Technology | Gynecology. Andrology. Obstetrics | Biological and medical sciences | Medical sciences | Cell Proliferation | Cell Adhesion Molecules - biosynthesis | Humans | Integrin beta Chains - biosynthesis | Gene Expression Regulation | Menstrual Cycle | Endometriosis - pathology | Cell Adhesion | Immunohistochemistry - methods | Biopsy | Endometrium - physiopathology | Adult | Female | Receptors, Cell Surface - biosynthesis | Laminin - biosynthesis | CD24 Antigen - biosynthesis | CD18 Antigens - biosynthesis | Apolipoproteins E - biosynthesis | Index Medicus | Medicin och hälsovetenskap
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