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Journal of Leukocyte Biology, ISSN 0741-5400, 12/2013, Volume 94, Issue 6, pp. 1309 - 1315
Adenosine signaling augments IL‐10‐induced STAT‐3 dependent gene expression in macrophages, suggesting its role in M2c polarization. The alternatively... 
TIMP‐1 | arginase | alternative activation | Arginase | TIMP-1 | Alternative activation | CELLS | ACTIVATION | RECEPTOR AGONISTS | IL-10 PRODUCTION | NECROSIS-FACTOR-ALPHA | IMMUNOLOGY | CELL BIOLOGY | TIMP-1 PRODUCTION | A(2B) | INFLAMMATION | TNF-ALPHA | HEMATOLOGY | EXPRESSION | Analgesics - pharmacology | Serum Amyloid A Protein - genetics | Receptor, Adenosine A2B - genetics | Interleukin-13 - genetics | Phosphorylation - genetics | Adenosine A2 Receptor Agonists - pharmacology | Serum Amyloid A Protein - biosynthesis | Phosphorylation - immunology | Phosphorylation - drug effects | Interleukin-6 - metabolism | STAT3 Transcription Factor - genetics | Interleukin-6 - genetics | Macrophage Activation - immunology | Arginase - genetics | Signal Transduction - genetics | Adenosine - pharmacology | Interleukin-13 - metabolism | Receptor, Adenosine A2B - metabolism | Mice, Knockout | Interleukin-4 - immunology | Macrophages - metabolism | Signal Transduction - drug effects | Interleukin-10 - genetics | Interleukin-6 - immunology | Mice | STAT3 Transcription Factor - immunology | Tissue Inhibitor of Metalloproteinase-1 - immunology | Interleukin-10 - immunology | Macrophage Activation - genetics | Tissue Inhibitor of Metalloproteinase-1 - biosynthesis | Interleukin-13 - immunology | Arginase - biosynthesis | Analgesics - immunology | Signal Transduction - immunology | Arginase - immunology | Receptor, Adenosine A2B - immunology | Interleukin-10 - metabolism | Interleukin-4 - genetics | Macrophages - immunology | STAT3 Transcription Factor - metabolism | Cell Line | Adenosine - immunology | Interleukin-4 - metabolism | Gene Expression Regulation | Macrophages - cytology | Serum Amyloid A Protein - immunology | Tissue Inhibitor of Metalloproteinase-1 - genetics | Animals | Aminopyridines - pharmacology | Macrophage Activation - drug effects | Index Medicus | Inflammation, Extracellular Mediators, & Effector Molecules
Journal Article
Journal Article
European Journal of Immunology, ISSN 0014-2980, 06/2011, Volume 41, Issue 6, pp. 1742 - 1753
Alternatively activated macrophages are critical in host defense against parasites and are protective in inflammatory bowel disease, but contribute to... 
IL-4 | Alternatively activated macrophages | SHIP | M2 MACROPHAGES | C/EBP-BETA | ARGINASE | NITRIC-OXIDE SYNTHASE | IMMUNOLOGY | PHOSPHOINOSITIDE 3-KINASE | CUTTING EDGE | SIGNAL-TRANSDUCTION | GENE-EXPRESSION | TUMOR-ASSOCIATED MACROPHAGES | NF-KAPPA-B | RNA, Small Interfering - genetics | Transcriptional Activation - genetics | Macrophage Activation - genetics | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Arginase - metabolism | Inositol Polyphosphate 5-Phosphatases | Phosphatidylinositol 3-Kinases - metabolism | STAT6 Transcription Factor - genetics | src Homology Domains - genetics | Phosphatidylinositol 3-Kinases - immunology | Lectins - metabolism | Phosphoric Monoester Hydrolases - immunology | beta-N-Acetylhexosaminidases - genetics | Macrophages - immunology | Biomarkers - metabolism | Phosphoric Monoester Hydrolases - genetics | Macrophages - pathology | beta-N-Acetylhexosaminidases - metabolism | Interleukin-4 - metabolism | Transgenes - genetics | Cells, Cultured | Arginase - genetics | Signal Transduction - genetics | STAT6 Transcription Factor - metabolism | Mice, Knockout | Neoplasms - drug therapy | Interleukin-4 - immunology | Macrophages - metabolism | Animals | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Neoplasms - immunology | Lectins - genetics | Mice | Neoplasms - pathology | Phosphoric Monoester Hydrolases - metabolism | Kinases | Index Medicus
Journal Article
Oncogene, ISSN 0950-9232, 05/2015, Volume 34, Issue 19, pp. 2471 - 2482
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 10/2017, Volume 106, pp. 279 - 290
Journal Article
Cancer Research, ISSN 0008-5472, 09/2015, Volume 75, Issue 18, pp. 3771 - 3787
Journal Article
PLoS Pathogens, ISSN 1553-7366, 10/2013, Volume 9, Issue 10, pp. e1003731 - e1003731
Journal Article
BBA - Molecular and Cell Biology of Lipids, ISSN 1388-1981, 09/2016, Volume 1861, Issue 9, pp. 1025 - 1035
Resolution of inflammation is an active process involving a novel category of lipid factors known as specialized pro-resolving lipid mediators, which includes... 
Efferocytosis | ALX/N-formyl peptide receptor 2 (FPR2) | Macrophage polarization | Ischemia/reperfusion | Resolvin D1 | Resolution of Inflammation | LIPOXIN A | KINASE PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | ACUTE LUNG INJURY | RECEPTOR | LIPID MEDIATORS | CELL BIOLOGY | ISCHEMIA-REPERFUSION INJURY | BIOPHYSICS | APOPTOTIC CELLS | IN-VIVO | KUPFFER CELLS | INFLAMMATORY RESPONSE | Inflammation - pathology | Kupffer Cells - pathology | Liver - pathology | Humans | Reperfusion Injury - drug therapy | Male | Chemokine CXCL1 - genetics | Lectins, C-Type - genetics | Liver - injuries | Inflammation - metabolism | Inflammation - drug therapy | Chemokine CXCL2 - genetics | Kupffer Cells - metabolism | Phagocytosis - drug effects | Macrophages - pathology | Reperfusion Injury - pathology | Signal Transduction | Arginase - genetics | Docosahexaenoic Acids - administration & dosage | Gene Expression Regulation - drug effects | Animals | Mannose-Binding Lectins - genetics | Receptor Protein-Tyrosine Kinases - genetics | Adaptor Proteins, Signal Transducing - genetics | Macrophages - drug effects | Adaptor Proteins, Signal Transducing - biosynthesis | Mice | Receptors, Formyl Peptide - biosynthesis | Receptors, Formyl Peptide - genetics | Receptors, Cell Surface - genetics | Macrophages | Liver | Genetic engineering | Inflammation | Peptides
Journal Article
Glia, ISSN 0894-1491, 10/2013, Volume 61, Issue 10, pp. 1698 - 1711
Increasing evidence indicates that “functional plasticity” is not solely a neuronal attribute but a hallmark of microglial cells, the main brain resident... 
M2 inflammation | brain macrophages | microglia | endotoxin tolerance | Endotoxin tolerance | Brain macrophages | M1 | Microglia | CELLS | ACTIVATION | BRAIN-INJURY | TOLERANCE | ENDOTOXIN | NEUROSCIENCES | PROSTANOID PRODUCTION | IN-VIVO | MACROPHAGE POLARIZATION | EXPRESSION | Membrane Glycoproteins - metabolism | Arginase - metabolism | Insulin-Like Growth Factor I - genetics | RNA, Messenger - metabolism | Antigens, CD - metabolism | Microglia - physiology | Time Factors | Cyclooxygenase 2 - genetics | Cell Polarity - drug effects | Membrane Proteins - metabolism | Cytokines - genetics | Organ Culture Techniques | Animals, Newborn | Cyclooxygenase 1 - genetics | Phagocytosis - drug effects | Cytokines - metabolism | Enzyme-Linked Immunosorbent Assay | Microglia - drug effects | Membrane Proteins - genetics | Rats | Receptors, Cell Surface - metabolism | Arginase - genetics | Hippocampus - cytology | Membrane Glycoproteins - genetics | Dinoprostone - metabolism | Cell Movement - drug effects | Animals | Nitric Oxide Synthase Type II - genetics | Antigens, Differentiation, Myelomonocytic - metabolism | Cyclooxygenase 2 - metabolism | Lipopolysaccharides - pharmacology | Cell Polarity - physiology | Cyclooxygenase 1 - metabolism | Insulin-Like Growth Factor I - metabolism | Nitric Oxide Synthase Type II - metabolism | Receptors, Cell Surface - genetics | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2016, Volume 11, Issue 1, pp. e0147897 - e0147897
Inflammation plays a fundamental role in the inception and development of atherosclerosis (ATH). Mechanisms of inflammation include the infiltration of... 
CELLS | ACTIVATION | MULTIDISCIPLINARY SCIENCES | MONOCYTE ADHESION | MOUSE | BONE MORPHOGENETIC PROTEIN-7 | ATHEROSCLEROSIS | PROLIFERATION | MODEL | POLARIZATION | EXPRESSION | Tumor Necrosis Factor-alpha - metabolism | Plaque, Atherosclerotic - genetics | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Humans | Tumor Necrosis Factor-alpha - genetics | Arginase - metabolism | Interleukin 1 Receptor Antagonist Protein - genetics | Male | Monocytes - metabolism | Ligation | Protective Agents - pharmacology | Interleukin 1 Receptor Antagonist Protein - metabolism | Monocytes - pathology | Interleukin-10 - metabolism | Female | Cell Differentiation | Chemokine CCL2 - metabolism | Plaque, Atherosclerotic - pathology | Interleukin-6 - metabolism | Disease Models, Animal | Atherosclerosis - pathology | Atherosclerosis - drug therapy | Bone Morphogenetic Protein Receptors, Type II - genetics | Interleukin-6 - genetics | Macrophages - pathology | Plaque, Atherosclerotic - metabolism | Signal Transduction | Gene Expression Regulation | Chemokine CCL2 - genetics | Arginase - genetics | Atherosclerosis - metabolism | Bone Morphogenetic Protein Receptors, Type II - metabolism | Mice, Knockout | Monocytes - drug effects | Macrophages - metabolism | Animals | Apolipoproteins E - genetics | Interleukin-10 - genetics | Carotid Arteries - surgery | Macrophages - drug effects | Carotid Arteries - pathology | Mice | Plaque, Atherosclerotic - drug therapy | Bone Morphogenetic Protein 7 - pharmacology | Blood Flow Velocity - drug effects | Disease | Science | Arginase | Interleukin 1 receptors | Macrophages | Carotid arteries | Interleukin 6 | Proteins | Cell activation | Rodents | Atherosclerosis | Plasticity | Bone morphogenetic proteins | Tumor necrosis factor-TNF | Carotid artery | Immune response | Cytokines | Secretion | Interleukin 1 receptor antagonist | Inflammation | Tumor necrosis factor-α | Metabolism | Velocity | Endothelium | Immune systems | Monocytes | Arteriosclerosis | Interleukin 10 | Infiltration | Plastic properties | Differentiation | Monocyte chemoattractant protein 1 | Apoptosis | Bone morphogenetic protein 7 | Veins & arteries | Plastic foam | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2013, Volume 123, Issue 4, pp. 1580 - 1589
Journal Article