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Journal of Biological Chemistry, ISSN 0021-9258, 10/2005, Volume 280, Issue 43, pp. 35983 - 35991
Journal Article
Journal of Steroid Biochemistry and Molecular Biology, ISSN 0960-0760, 2010, Volume 118, Issue 3, pp. 177 - 187
Aldo-keto reductase (AKR) 1C3 (type 5 17β-hydroxysteroid dehydrogenase and prostaglandin F synthase), may stimulate proliferation via steroid hormone and... 
Estrogen receptor | Peroxisome proliferator activated receptor γ | Prostaglandin F synthase | Prostaglandin D 2 | 17β-Hydroxysteroid dehydrogenase | Prostaglandin D | SIGNALING PATHWAYS | 17-BETA-HYDROXYSTEROID DEHYDROGENASES | BIOCHEMISTRY & MOLECULAR BIOLOGY | AKR1C3 | PROLIFERATOR-ACTIVATED RECEPTOR | RISK | Prostaglandin D-2 | CELECOXIB | ENDOCRINOLOGY & METABOLISM | PROGNOSTIC MARKER | F SYNTHASE | INHIBITORS | 17 beta-Hydroxysteroid dehydrogenase | AROMATASE | Peroxisome proliferator activated receptor | gamma | Up-Regulation | Prostaglandins - pharmacology | Humans | Gene Expression Regulation, Neoplastic | Dinoprost - metabolism | Hydroxyprostaglandin Dehydrogenases - metabolism | Androstenedione - metabolism | Breast Neoplasms - metabolism | Testosterone - metabolism | Breast Neoplasms - enzymology | Transfection | Prostaglandin D2 - metabolism | Prostaglandin D2 - analogs & derivatives | 3-Hydroxysteroid Dehydrogenases - metabolism | Female | 5-alpha-Dihydroprogesterone - metabolism | Dinoprost - pharmacology | Etiocholanolone - metabolism | Estradiol - pharmacology | Dihydrotestosterone - metabolism | Recombinant Proteins - metabolism | Estradiol - metabolism | Estrone - pharmacology | Gonadal Steroid Hormones - metabolism | Biocatalysis | Etiocholanolone - analogs & derivatives | Gonadal Steroid Hormones - pharmacology | Progesterone - analogs & derivatives | Recombinant Proteins - genetics | 20-alpha-Dihydroprogesterone - metabolism | Androsterone - metabolism | Hydroxyprostaglandin Dehydrogenases - genetics | Breast Neoplasms - genetics | Aldo-Keto Reductase Family 1 Member C3 | Estrone - metabolism | Progesterone - metabolism | Cell Line, Tumor | Cell Proliferation - drug effects | Prostaglandin D2 - pharmacology | Ketosteroids - metabolism | Kinetics | 3-Hydroxysteroid Dehydrogenases - genetics | Prostaglandins - metabolism | Physiological aspects | Testosterone | Breast cancer | Progesterone | Estradiol | Synthetic prostaglandins F | Index Medicus
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 05/2016, Volume 22, Issue 9, pp. 2283 - 2289
Journal Article
Molecular and Cellular Endocrinology, ISSN 0303-7207, 01/2015, Volume 399, Issue C, pp. 32 - 42
Aromatase deficiency is a rare disorder resulting in estrogen insufficiency in humans. It has been reported in remarkably few men with loss-of-function... 
Insulin resistance | Aromatase deficiency | Metabolic syndrome | CYP19A1 | Aromatase | Estrogen replacement | ARKO MICE | TESTOSTERONE | INSULIN SENSITIVITY | MICE LACKING | FEMALE | CELL BIOLOGY | IMPACT | GENE | ESTRADIOL | ENDOCRINOLOGY & METABOLISM | MOLECULAR-BASIS | EXPRESSION | Estrogen Replacement Therapy | Infertility, Male - drug therapy | Liver - pathology | Gynecomastia - drug therapy | Cricetulus | Aromatase - deficiency | Humans | Aromatase - metabolism | Male | Bone Density - genetics | Mutation, Missense | Estrogens - therapeutic use | Aromatase - genetics | Infertility, Male - genetics | Bone and Bones - metabolism | Gynecomastia - metabolism | Adult | Lipid Metabolism - genetics | CHO Cells | Protein Structure, Tertiary | 46, XX Disorders of Sex Development - genetics | Liver - metabolism | Metabolism, Inborn Errors - metabolism | Models, Molecular | Glucose - genetics | Gynecomastia - pathology | Infertility, Male - pathology | Metabolism, Inborn Errors - genetics | Gynecomastia - genetics | Bone Density - drug effects | 46, XX Disorders of Sex Development - pathology | Animals | Lipid Metabolism - drug effects | Glucose - metabolism | 46, XX Disorders of Sex Development - drug therapy | Infertility, Male - metabolism | 46, XX Disorders of Sex Development - metabolism | Metabolism, Inborn Errors - drug therapy | Metabolism, Inborn Errors - pathology | Amino Acid Substitution | Glucose metabolism | Hormone therapy | Estrogen | Cytochrome P-450 | Physiological aspects | Genetic aspects | Glucose | Dextrose | Index Medicus | Heart | Lipoproteins | High density | Constants | Ovaries | Hormones | Metabolism | Density
Journal Article
Journal of Clinical Endocrinology and Metabolism, ISSN 0021-972X, 05/2017, Volume 102, Issue 5, pp. 1692 - 1701
Context: Most estrogen-dependent breast cancers occur after menopause, despite low levels of circulating estrogens. Breast expression of the... 
CANCER PATIENTS | TUMOR-NECROSIS-FACTOR | METABOLIC SYNDROME | POLYMERASE-CHAIN-REACTION | REVERSE TRANSCRIPTION | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | BODY-MASS INDEX | STROMAL CELLS | POSTMENOPAUSAL WOMEN | ADIPOSE-TISSUE | Multivariate Analysis | Adipose Tissue, White - immunology | Humans | Leptin - metabolism | Middle Aged | Aromatase - metabolism | Breast Neoplasms | RNA, Messenger - metabolism | Breast - metabolism | Aromatase - genetics | Gene Expression Regulation, Developmental | Postmenopause | Adiponectin - metabolism | Adult | Female | Body Mass Index | Premenopause | Cholesterol, HDL - metabolism | Insulin Resistance | Inflammation | Cholesterol - metabolism | Triglycerides - metabolism | Insulin - metabolism | Menopause - metabolism | Interleukin-6 - immunology | Cholesterol, LDL - metabolism | Aged | Blood Glucose - metabolism | C-Reactive Protein - immunology | Adipose tissues | C-reactive protein | Adipose tissue | Menopause | Estrogens | Interleukin | mRNA | Glucose | Adiponectin | Interleukin 6 | Body mass index | Aromatase | Sensitivity analysis | Markers | Health risks | Breast cancer | Triglycerides | Metabolism | Insulin | Cholesterol | Post-menopause | Ribonucleic acids | Body mass | Womens health | Leptin | Body size | Breast | Hormone replacement therapy | Health risk assessment | Cancer | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Steroids, ISSN 0039-128X, 2005, Volume 70, Issue 5, pp. 372 - 381
Extranuclear estrogen receptors may mediate rapid effects of estradiol that communicate with nuclear receptors and contribute to proliferation of human cancers... 
Estrogen receptor | EGF receptor | HER-2 receptor | Breast cancer | Palmitoylation | Lung cancer | ACTIVATION | estrogen receptor | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | breast cancer | ALPHA | OVARIAN-CANCER | PLASMA-MEMBRANE | BETA | lung cancer | PATHWAY | ENDOTHELIAL-CELLS | ENDOCRINOLOGY & METABOLISM | MEMBRANE-BINDING-SITES | CAVEOLAE | palmitoylation | Estradiol - analogs & derivatives | Phosphorylation | Receptors, Estrogen - metabolism | Cell Proliferation | Protein-Tyrosine Kinases - metabolism | Humans | Lung Neoplasms - metabolism | Aromatase - metabolism | Vascular Endothelial Growth Factor A - metabolism | Breast Neoplasms - metabolism | Vascular Endothelial Growth Factor A - secretion | Cell Nucleus - metabolism | Transfection | Cell Membrane - metabolism | Active Transport, Cell Nucleus | Estrogens - metabolism | Estradiol - pharmacology | Estradiol - metabolism | Mutagenesis, Site-Directed | Antineoplastic Agents, Hormonal - pharmacology | Signal Transduction | Cell Survival | Carcinoma, Non-Small-Cell Lung - metabolism | Subcellular Fractions - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Cell Line, Tumor | Protein Binding | Cytosol - metabolism | Genetic Vectors | Microscopy, Fluorescence | Growth Substances - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus
Journal Article
Molecular and Cellular Endocrinology, ISSN 0303-7207, 2008, Volume 284, Issue 1, pp. 38 - 45
Journal Article
Breast Cancer Research, ISSN 1465-5411, 10/2013, Volume 15, Issue 5, pp. R102 - R102
Introduction: Obesity has been associated with increased incidence and mortality of breast cancer. While the precise correlation between obesity and breast... 
INVASION | ADULT WEIGHT CHANGE | ADIPOCYTES | ONCOLOGY | GROWTH | BODY-MASS INDEX | RISK | LEPTIN RECEPTOR | STROMAL CELLS | POSTMENOPAUSAL BREAST-CANCER | EXPRESSION | Receptors, Estrogen - metabolism | Cell Proliferation | Estrogens - pharmacology | Coculture Techniques | Humans | Leptin - metabolism | Aromatase - metabolism | Gene Expression Profiling | Stem Cells - metabolism | Obesity - genetics | Breast Neoplasms - metabolism | Receptors, Progesterone - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | MCF-7 Cells | Estrogen Receptor alpha - metabolism | Female | Membrane Proteins - metabolism | Estrogens - metabolism | Glutathione S-Transferase pi - genetics | Glutathione S-Transferase pi - metabolism | Signal Transduction | Membrane Proteins - genetics | Intercellular Signaling Peptides and Proteins - genetics | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Leptin - genetics | Tumor Burden | Obesity - metabolism | Animals | Breast Neoplasms - genetics | Estrogen Receptor alpha - genetics | Breast Neoplasms - pathology | Cell Transformation, Neoplastic | Adipocytes - metabolism | Cell Line, Tumor | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Breast Neoplasms - mortality | Mice | Women | Adipose tissues | Obesity | United States | Mortality | Antibodies | Development and progression | Breast cancer | Carcinogenesis | Viral antibodies | Body mass index | Leptin | Progesterone | Health aspects | Biotechnology industry | Index Medicus
Journal Article
Journal Article