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1. Full Text Platelet–vessel wall interactions and drug effects
by Twarock, Sören and Bagheri, Sayeh and Bagheri, Saghar and Hohlfeld, Thomas
Pharmacology and Therapeutics, ISSN 0163-7258, 11/2016, Volume 167, pp. 74 - 84
Platelet–vessel wall interaction is necessary for hemostasis and vascular repair, but also plays a fundamental role in the early and late development of... 
Antiplatelet therapy | Platelet-vessel wall interaction | Vascular inflammation | Atherothrombosis | COA REDUCTASE INHIBITOR | GLYCOPROTEIN-VI | INTEGRIN ALPHA-2-BETA-1 | IN-VIVO DEPLETION | HUMAN ATHEROSCLEROTIC PLAQUE | EP3 RECEPTOR | VON-WILLEBRAND-FACTOR | APTAMER ARC1779 | P-SELECTIN | PHARMACOLOGY & PHARMACY | THROMBOXANE A | Coronary Artery Disease - metabolism | Humans | Endothelium, Vascular - drug effects | Coronary Artery Disease - drug therapy | Cell Adhesion - drug effects | Receptors, Prostaglandin - metabolism | Animals | Receptors, Prostaglandin - agonists | Blood Platelets - metabolism | Cell Adhesion - physiology | Endothelium, Vascular - metabolism | Platelet Aggregation Inhibitors - pharmacology | Platelet Aggregation Inhibitors - metabolism | Blood Platelets - drug effects | Platelet Aggregation Inhibitors - therapeutic use | Receptors, Prostaglandin - antagonists & inhibitors | Drug interactions | Cardiovascular agents | COX-2 inhibitors | Aspirin | Proteases | Nitric oxide | Von Willebrand factor | Atherosclerosis | Anticoagulants (Medicine) | Coronary heart disease | Thrombosis | Statins | Blood clot
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2. Full Text Association of Drug Effects on Serum Parathyroid Hormone, Phosphorus, and Calcium Levels With Mortality in CKD: A Meta-analysis
by Palmer, Suetonia C., MBChB, PhD and Teixeira-Pinto, Armando, PhD and Saglimbene, Valeria, MSc and Craig, Jonathan C., MBChB, PhD and Macaskill, Petra, PhD and Tonelli, Marcello, MD and de Berardis, Giorgia, MSc and Ruospo, Marinella, MSc and Strippoli, Giovanni F.M., MD, PhD
American Journal of Kidney Diseases, ISSN 0272-6386, 2015, Volume 66, Issue 6, pp. 962 - 971
Background Serum parathyroid hormone (PTH), phosphorus, and calcium levels are surrogate outcomes that are central to the evaluation of drug treatments in... 
Nephrology | dialysis | calcium | death | calcitonin | chronic kidney disease–mineral and bone disorder (CKD-MBD) | cardiovascular mortality | meta-analysis | drug effect | parathyroid hormone (PTH) | surrogate endpoint | biomarker | phosphate binders | calcimimetic agents | outcomes | Renal failure | all-cause mortality | bisphosphonates | phosphorus | vitamin D compounds | Vitamin D compounds | chronic kidney disease-mineral and bone disorder (CKD-MBD) | CINACALCET HYDROCHLORIDE | SECONDARY HYPERPARATHYROIDISM | DOSE VITAMIN-D | CORONARY-ARTERY CALCIFICATION | DIALYSIS PATIENTS | CLINICAL-TRIALS | DOUBLE-BLIND | UROLOGY & NEPHROLOGY | PLACEBO-CONTROLLED TRIAL | CHRONIC KIDNEY-DISEASE | Renal Insufficiency, Chronic - blood | Calcium - blood | Renal Agents - adverse effects | Phosphorus - blood | Humans | Renal Insufficiency, Chronic - mortality | Treatment Outcome | Mortality - trends | Biomarkers - blood | Parathyroid Hormone - blood | Renal Insufficiency, Chronic - drug therapy
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3. Full Text Biologic Drug Effect and Particulate Embolization of Drug-Eluting Stents versus Drug-Coated Balloons in Healthy Swine Femoropopliteal Arteries
by Torii, Sho and Yahagi, Kazuyuki and Mori, Hiroyoshi and Harari, Emanuel and Romero, Maria E and Kolodgie, Frank D and Young, Brandt and Ragheb, Anthony and Virmani, Renu and Finn, Aloke V
Journal of Vascular and Interventional Radiology, ISSN 1051-0443, 07/2018, Volume 29, Issue 7, pp. 1041 - 1049.e3
Purpose: To compare the drug effect in treated vessels and downstream effects in distal skeletal muscle of drug-coated balloons (DCBs) and drug-eluting stents... 
TRIAL | ANGIOPLASTY | BARE METAL STENTS | SAFETY | DISEASE | RESTENOSIS | PERIPHERAL VASCULAR DISEASE | MODEL | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | LESIONS | PACLITAXEL | LONG | Femoral Artery - pathology | Muscle, Smooth, Vascular - metabolism | Myocytes, Smooth Muscle - pathology | Angioplasty, Balloon - adverse effects | Popliteal Artery - drug effects | Popliteal Artery - metabolism | Neointima | Paclitaxel - toxicity | Time Factors | Models, Animal | Myocytes, Smooth Muscle - drug effects | Paclitaxel - administration & dosage | Myocytes, Smooth Muscle - metabolism | Muscle, Skeletal - blood supply | Muscle, Smooth, Vascular - drug effects | Cardiovascular Agents - administration & dosage | Drug-Eluting Stents | Proteoglycans - metabolism | Cardiovascular Agents - toxicity | Coated Materials, Biocompatible | Femoral Artery - drug effects | Angioplasty, Balloon - instrumentation | Muscle, Smooth, Vascular - pathology | Animals | Femoral Artery - metabolism | Vascular Access Devices | Popliteal Artery - pathology | Fibrin - metabolism | Sus scrofa | Index Medicus
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4. Full Text Analysis of drug effects on primary human coronary artery endothelial cells activated by serum amyloid a
by Lakota, K and Hrušovar, D and Ogrič, M and Mrak-Poljšak, K and Čučnik, S and Tomšič, M and Božič, B and Žigon, P and Sodin-Semrl, S
Mediators of Inflammation, ISSN 0962-9351, 2018, Volume 2018, pp. 8237209 - 11
Background. RA patients have a higher incidence of cardiovascular diseases compared to the general population. Serum amyloid A (SAA) is an acute-phase protein,... 
C-REACTIVE PROTEIN | RHEUMATOID-ARTHRITIS | PPAR-GAMMA ACTIVATORS | CHOLESTEROL-FED RABBITS | SMOOTH-MUSCLE-CELLS | NECROSIS-FACTOR ANTAGONISTS | ADHESION MOLECULES | IMMUNOLOGY | ANGIOTENSIN-CONVERTING ENZYME | NF-KAPPA-B | HUMAN UMBILICAL VEIN | CELL BIOLOGY | Meloxicam | Dexamethasone | Etoricoxib | Captopril | Atherosclerosis | Methotrexate | Comparative analysis | Cardiovascular diseases | Enzyme-linked immunosorbent assay | Antilipemic agents | Endothelium | Cardiovascular disease | Arthritis | Cell adhesion & migration | Diclofenac | Proteins | Interleukin 6 | Fluvastatin | Etanercept | Physiology | Dihydrofolate reductase | Nonsteroidal anti-inflammatory drugs | Drug therapy | Interleukin 8 | Adenosine | Cytokines | Mortality | Rheumatology | Rheumatic diseases | Coronary artery | Inflammation | Tumor necrosis factor-α | Thrombosis | Endothelial cells | Acute phase substances | Pharmacy | Coronary vessels | Arteriosclerosis | Monoclonal antibodies | Rosiglitazone | Autoimmune diseases | Veins & arteries | Ampules
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5. Full Text The Achilles’ heel of senescent cells: from transcriptome to senolytic drugs
by Zhu, Yi and Tchkonia, Tamara and Pirtskhalava, Tamar and Gower, Adam C and Ding, Husheng and Giorgadze, Nino and Palmer, Allyson K and Ikeno, Yuji and Hubbard, Gene B and Lenburg, Marc and O'Hara, Steven P and LaRusso, Nicholas F and Miller, Jordan D and Roos, Carolyn M and Verzosa, Grace C and LeBrasseur, Nathan K and Wren, Jonathan D and Farr, Joshua N and Khosla, Sundeep and Stout, Michael B and McGowan, Sara J and Fuhrmann‐Stroissnigg, Heike and Gurkar, Aditi U and Zhao, Jing and Colangelo, Debora and Dorronsoro, Akaitz and Ling, Yuan Yuan and Barghouthy, Amira S and Navarro, Diana C and Sano, Tokio and Robbins, Paul D and Niedernhofer, Laura J and Kirkland, James L
Aging Cell, ISSN 1474-9718, 08/2015, Volume 14, Issue 4, pp. 644 - 658
The healthspan of mice is enhanced by killing senescent cells using a transgenic suicide gene. Achieving the same using small molecules would have a tremendous... 
dasatinib | plasminogen‐activated inhibitor | dependence receptors | quercetin | ephrins | 3K delta | p21 | Dasatinib | Dependence receptors | Ephrins | Plasminogen-activated inhibitor | Quercetin | PI3K delta | P21 | ENDOTHELIAL DYSFUNCTION | PLASMINOGEN-ACTIVATOR INHIBITOR-1 | EXPRESSION PROFILES | plasminogen-activated inhibitor | CELLULAR SENESCENCE | CANCER-THERAPY | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | LUNG-CANCER | SET ENRICHMENT ANALYSIS | GENE-EXPRESSION | IONIZING-RADIATION | TUMOR-GROWTH | Carotid Arteries - drug effects | Endonucleases - genetics | Plasminogen Activator Inhibitor 2 - genetics | Transcriptome | Gene Expression Profiling | Adipocytes - drug effects | Aging - genetics | Osteoporosis - metabolism | Ephrins - metabolism | Plasminogen Activator Inhibitor 2 - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Osteoporosis - genetics | Intervertebral Disc - pathology | Fibroblasts - metabolism | Endothelial Cells - metabolism | Intervertebral Disc - chemistry | Fibroblasts - pathology | Mesenchymal Stem Cells - pathology | Mice, Knockout | Dasatinib - pharmacology | Osteoporosis - pathology | Ephrins - genetics | Fibroblasts - drug effects | Mice | Endothelial Cells - pathology | bcl-X Protein - metabolism | Aging - metabolism | Aging - drug effects | bcl-X Protein - genetics | Cellular Senescence - drug effects | Phosphatidylinositol 3-Kinases - metabolism | Endonucleases - metabolism | DNA-Binding Proteins - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Mesenchymal Stem Cells - drug effects | Mesenchymal Stem Cells - metabolism | Heart - physiopathology | Cellular Senescence - genetics | Osteoporosis - prevention & control | DNA-Binding Proteins - genetics | Adipocytes - pathology | Aging - pathology | Phosphatidylinositol 3-Kinases - genetics | Animals | Quercetin - pharmacology | Adipocytes - metabolism | Heart - drug effects | Carotid Arteries - pathology | Intervertebral Disc - drug effects | Drug Combinations | Endothelial Cells - drug effects | Original
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6. Full Text Quantitative evaluation of drug or device effects on ventricular remodeling as predictors of therapeutic effects on mortality in patients with heart failure and reduced ejection fraction: A meta-analytic approach
by Kramer, Daniel G and Trikalinos, Thomas A and Kent, David M and Antonopoulos, George V and Konstam, Marvin A and Udelson, James E
Journal of the American College of Cardiology, ISSN 0735-1097, 07/2010, Volume 56, Issue 5, pp. 392 - 406
Objectives: The purpose of this study was to quantitatively assess the relationship between therapy-induced changes in left ventricular (LV) remodeling and... 
heart failure | remodeling | clinical trials | SURROGATE END-POINTS | CORONARY-ARTERY DISEASE | CARDIAC & CARDIOVASCULAR SYSTEMS | IDIOPATHIC DILATED CARDIOMYOPATHY | SUSTAINED-RELEASE MOXONIDINE | ECHOCARDIOGRAPHIC FOLLOW-UP | CONVERTING-ENZYME-INHIBITOR | CARDIAC RESYNCHRONIZATION THERAPY | ACUTE MYOCARDIAL-INFARCTION | PLACEBO-CONTROLLED TRIAL | LONG-TERM PROGRESSION | Humans | Heart Failure - physiopathology | Stroke Volume - drug effects | Treatment Outcome | Ventricular Dysfunction, Left - mortality | Ventricular Dysfunction, Left - physiopathology | Randomized Controlled Trials as Topic | Heart Failure - therapy | Regression Analysis | Ventricular Dysfunction, Left - therapy | Ventricular Remodeling | Algorithms | Placebos | Odds Ratio | Heart-Assist Devices | Heart Ventricles - pathology | Heart Failure - mortality | Studies | Intervention | Confidence intervals | Human subjects | Heart failure | Heart attacks | Mortality | ISDN | Drug therapy | Fractions
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7. Full Text Phosphorylation of VE-cadherin is modulated by haemodynamic forces and contributes to the regulation of vascular permeability in vivo
by Orsenigo, Fabrizio and Giampietro, Costanza and Ferrari, Aldo and Corada, Monica and Galaup, Ariane and Sigismund, Sara and Ristagno, Giuseppe and Maddaluno, Luigi and Koh, Gou Young and Franco, Davide and Kurtcuoglu, Vartan and Poulikakos, Dimos and Baluk, Peter and McDonald, Donald and Grazia Lampugnani, Maria and Dejana, Elisabetta
Nature Communications, ISSN 2041-1723, 2012, Volume 3, Issue 1, p. 1208
Endothelial adherens junctions maintain vascular integrity. Arteries and veins differ in their permeability but whether organization and strength of their... 
ARTERIAL BRANCHING MORPHOGENESIS | TIGHT JUNCTIONS | ENDOTHELIAL-CELL JUNCTIONS | TYROSINE PHOSPHORYLATION | SRC | MULTIDISCIPLINARY SCIENCES | DEPENDENT ENDOCYTOSIS | BARRIER FUNCTION | ANGIOPOIETIN-1 | ADHERENS JUNCTIONS | GROWTH-FACTOR | Antigens, CD - immunology | Cadherins - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Capillary Permeability - drug effects | Molecular Sequence Data | Stress, Mechanical | Antigens, CD - metabolism | Cadherins - immunology | Ubiquitination - drug effects | Arteries - physiology | Antibody Specificity - drug effects | Inflammation Mediators - metabolism | src-Family Kinases - metabolism | Phosphorylation - drug effects | Bradykinin - pharmacology | Amino Acid Sequence | Human Umbilical Vein Endothelial Cells - drug effects | Endocytosis - drug effects | Mice, Inbred C57BL | Arteries - drug effects | Enzyme Activation - drug effects | Veins - drug effects | Animals | Hemodynamics - drug effects | Mice | Veins - physiology
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8. Full Text A biodegradable microneedle cuff for comparison of drug effects through perivascular delivery to balloon-injured arteries
by Kim, Dae-Hyun and Jang, Eui Hwa and Lee, Kang Ju and Lee, Ji Yong and Park, Seung Hyun and Seo, Il Ho and Lee, Kang Woog and Lee, Seung Hyun and Ryu, Won Hyoung and Youn, Young-Nam
Polymers, ISSN 2073-4360, 2017, Volume 9, Issue 2, pp. 56 - 56
Restenosis at a vascular anastomosis site is a major cause of graft failure and is difficult to prevent by conventional treatment. Perivascular drug delivery... 
Restenosis | Peripheral vascular disease | Drug delivery | Microneedle | Anastomosis | RAPAMYCIN | CELLS | restenosis | POLYMER SCIENCE | NEOINTIMAL HYPERPLASIA | drug delivery | MODEL | peripheral vascular disease | PACLITAXEL | microneedle | ELUTING STENTS | INHIBITION | INTIMAL HYPERPLASIA | anastomosis | SIROLIMUS | Drugs | Drug delivery systems | Cuffs | Media | Needles | Wall thickness | Devices | Arteries
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9. Full Text Orthotopic animal model of pseudomyxoma peritonei: An in vivo model to test anti-angiogenic drug effects
by Dohan, Anthony and Lousquy, Ruben and Eveno, Clarisse and Goere, Diane and Broqueres-You, Dong and Kaci, Rachid and Lehmann-Che, Jacqueline and Launay, Jean-Marie and Soyer, Philippe and Bonnin, Philippe and Pocard, Marc
American Journal of Pathology, ISSN 0002-9440, 2014, Volume 184, Issue 7, pp. 1920 - 1929
Pseudomyxoma peritonei (PMP) is an uncommon peritoneal mucinous carcinomatosis confined to the peritoneal cavity. The rarity of PMP in humans makes evaluation... 
ANTIANGIOGENIC THERAPY | INTRAPERITONEAL CHEMOTHERAPY | CYTOREDUCTIVE SURGERY | COLORECTAL-CANCER | APPENDICEAL ORIGIN | HEPATOCARCINOMA | PATHOLOGY | GENETIC-EVIDENCE | TUMOR-GROWTH | CARCINOMA | MUCINOUS TUMORS | Antibodies, Monoclonal, Humanized - therapeutic use | Mesenteric Arteries - diagnostic imaging | Humans | Pseudomyxoma Peritonei - drug therapy | Bevacizumab | Peritoneal Neoplasms - drug therapy | Animals | Mice, Nude | Neovascularization, Pathologic - drug therapy | Blood Flow Velocity | Ultrasonography | Angiogenesis Inhibitors - therapeutic use | Female | Disease Models, Animal
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10. Full Text A universal system for highly efficient cardiac differentiation of human induced pluripotent stem cells that eliminates interline variability
by Burridge, Paul W and Thompson, Susan and Millrod, Michal A and Weinberg, Seth and Yuan, Xuan and Peters, Ann and Mahairaki, Vasiliki and Koliatsos, Vassilis E and Tung, Leslie and Zambidis, Elias T
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 4, p. e18293
Background: The production of cardiomyocytes from human induced pluripotent stem cells (hiPSC) holds great promise for patient-specific cardiotoxicity drug... 
DEFINED MEDIUM | MESODERM | MULTIDISCIPLINARY SCIENCES | ENDODERM | CARDIOMYOCYTE DIFFERENTIATION | INDUCTION | LINES | FUNCTIONAL-PROPERTIES | Body Patterning - drug effects | Oxygen - pharmacology | Antigens, CD34 - metabolism | Polyvinyl Alcohol - pharmacology | Humans | Fibroblast Growth Factor 2 - pharmacology | Mesoderm - drug effects | Mesoderm - cytology | Cell Culture Techniques - methods | Embryoid Bodies - metabolism | Embryoid Bodies - cytology | Adult | Induced Pluripotent Stem Cells - cytology | Fibroblasts - metabolism | Induced Pluripotent Stem Cells - metabolism | Cell Line | Insulin - pharmacology | Induced Pluripotent Stem Cells - drug effects | Myocytes, Cardiac - cytology | Transgenes - genetics | Fetal Blood - cytology | Cell Adhesion - drug effects | Bone Morphogenetic Protein 4 - pharmacology | Genetic Vectors - genetics | Embryoid Bodies - drug effects | Animals | Myocytes, Cardiac - drug effects | Cell Differentiation - drug effects | Fibroblasts - drug effects | Culture Media - pharmacology | Myocytes, Cardiac - metabolism | Cell Proliferation - drug effects | Fibroblasts - cytology | Mice | Electrophysiological Phenomena - drug effects | Physiological aspects | Bone morphogenetic proteins | Fibroblast growth factors | Embryonic stem cells | Analysis | Drugs | Neonates | Neuropathology | Variability | Embryo cells | Alcohol | Drug development | Blood | Contraction | Regeneration (physiology) | Engineering | Cord blood | Efficiency | Calcium-binding protein | Fibroblasts | Long QT syndrome | Modelling | Polyvinyl alcohol | Heart diseases | Biomedical engineering | Fibroblast growth factor 2 | CD34 antigen | Bone morphogenetic protein 4 | Anemia | Cardiomyocytes | Insulin | Embryos | Coronary artery disease | Medicine | Regeneration | Troponin | Troponin I | Plasmids | Stem cells | Epigenetics | Mutation | Differentiation | Pluripotency
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