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1. Full Text Dichloroacetate prevents restenosis in preclinical animal models of vessel injury
by Deuse, Tobias and Hua, Xiaoqin and Wang, Dong and Maegdefessel, Lars and Heeren, Joerg and Scheja, Ludger and Bolaños, Juan P and Rakovic, Aleksandar and Spin, Joshua M and Stubbendorff, Mandy and Ikeno, Fumiaki and Länger, Florian and Zeller, Tanja and Schulte-Uentrop, Leonie and Stoehr, Andrea and Itagaki, Ryo and Haddad, Francois and Eschenhagen, Thomas and Blankenberg, Stefan and Kiefmann, Rainer and Reichenspurner, Hermann and Velden, Joachim and Klein, Christine and Yeung, Alan and Robbins, Robert C and Tsao, Philip S and Schrepfer, Sonja
Nature (London), ISSN 0028-0836, 2014, Volume 509, Issue 7502, pp. 641 - 644
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Iliac Artery - pathology | Protein-Serine-Threonine Kinases - deficiency | Constriction, Pathologic - pathology | Mitochondria, Heart - metabolism | Iliac Artery - drug effects | Apoptosis - drug effects | Humans | Myocytes, Smooth Muscle - pathology | Iliac Artery - injuries | Arteries - injuries | Secondary Prevention | Mitochondria, Heart - drug effects | Angioplasty, Balloon - adverse effects | Membrane Potential, Mitochondrial - drug effects | Dichloroacetic Acid - therapeutic use | Gene Knockdown Techniques | Swine | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Hyperplasia - drug therapy | Mammary Arteries - drug effects | Myocytes, Smooth Muscle - drug effects | Coronary Vessels - injuries | Disease Models, Animal | Constriction, Pathologic - prevention & control | Rabbits | Coronary Vessels - drug effects | Coronary Vessels - pathology | Aorta - drug effects | Protein-Serine-Threonine Kinases - genetics | Rats | Mammary Arteries - injuries | Arteries - drug effects | Aorta - injuries | Arteries - pathology | Enzyme Activation - drug effects | Tunica Intima - injuries | Aorta - pathology | Hyperplasia - pathology | Tunica Intima - drug effects | Animals | Tunica Intima - pathology | Mammary Arteries - pathology | Dichloroacetic Acid - pharmacology | Cell Proliferation - drug effects | Stents - adverse effects | Complications and side effects | Patient outcomes | Genetic aspects | Research | Coronary heart disease | Health aspects | Risk factors | Stent (Surgery) | Apoptosis | Permeability | Angioplasty | Rodents | Binding sites | Veins & arteries | Index Medicus | Medicin och hälsovetenskap
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2. Full Text Acute canagliflozin treatment protects against in vivo myocardial ischemia-reperfusion injury in non-diabetic male rats and enhances endothelium-dependent vasorelaxation
by Sayour, Alex Ali and Korkmaz-Icöz, Sevil and Loganathan, Sivakkanan and Ruppert, Mihály and Sayour, Viktor Nabil and Oláh, Attila and Benke, Kálmán and Brune, Maik and Benko, Rita and Horváth, Eszter Mária and Karck, Matthias and Merkely, Béla and Radovits, Tamás and Szabó, Gábor
Journal of translational medicine, ISSN 1479-5876, 04/2019, Volume 17, Issue 1, pp. 127 - 127
Sodium-glucose cotransporter-2 inhibitor | Cardioprotection | Canagliflozin | Myocardial ischemia-reperfusion injury | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Apoptosis - drug effects | Canagliflozin - pharmacology | Myocardial Reperfusion Injury - complications | Systole - drug effects | Male | Aldehydes - metabolism | Liver - physiopathology | Cardiotonic Agents - therapeutic use | Liver - drug effects | Canagliflozin - therapeutic use | Diastole - drug effects | Aorta - physiopathology | Myocardial Reperfusion Injury - drug therapy | Phosphorylation - drug effects | Kidney - physiopathology | Biomarkers - metabolism | Endothelium - pathology | Glycosuria - physiopathology | Kidney - drug effects | Aorta - drug effects | Ventricular Function, Left - drug effects | Cardiotonic Agents - pharmacology | Rats, Sprague-Dawley | Glycosuria - complications | Myocardial Reperfusion Injury - physiopathology | Endothelium - physiopathology | Aorta - pathology | Endothelium - drug effects | Animals | Signal Transduction - drug effects | Oxidative Stress - drug effects | Vasodilation - drug effects | Blood Glucose - metabolism | Myocardial Reperfusion Injury - prevention & control | Nitrosative Stress - drug effects | Heart failure | Diabetics | Rats as laboratory animals | Analysis | Clinical trials | Research | Drug therapy | Health aspects | Occlusion | Myocardial infarction | Heart | Oxidative stress | Phosphorylation | Drug delivery systems | Intravenous administration | Heart attacks | Bax protein | 4-Hydroxynonenal | Bcl-2 protein | AKT protein | Myocardial ischemia | mRNA | Size determination | Kinases | Vasodilation | Proteins | Reperfusion | Ischemia | Rodents | Calcium-binding protein | Aorta | Heart diseases | Injury analysis | Medical research | Adenosine monophosphate | AMP | Diabetes mellitus | Coronary artery | Gene expression | Nitric-oxide synthase | Endothelium | Sodium | Protein kinase | Adenosine kinase | Nitric oxide | Ventricle | Diabetes | Laboratory animals | Apoptosis | Index Medicus | Myocardial ischemia–reperfusion injury | Sodium–glucose cotransporter-2 inhibitor
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3. Full Text The Achilles’ heel of senescent cells: from transcriptome to senolytic drugs
by Zhu, Yi and Tchkonia, Tamara and Pirtskhalava, Tamar and Gower, Adam C and Ding, Husheng and Giorgadze, Nino and Palmer, Allyson K and Ikeno, Yuji and Hubbard, Gene B and Lenburg, Marc and O'Hara, Steven P and LaRusso, Nicholas F and Miller, Jordan D and Roos, Carolyn M and Verzosa, Grace C and LeBrasseur, Nathan K and Wren, Jonathan D and Farr, Joshua N and Khosla, Sundeep and Stout, Michael B and McGowan, Sara J and Fuhrmann‐Stroissnigg, Heike and Gurkar, Aditi U and Zhao, Jing and Colangelo, Debora and Dorronsoro, Akaitz and Ling, Yuan Yuan and Barghouthy, Amira S and Navarro, Diana C and Sano, Tokio and Robbins, Paul D and Niedernhofer, Laura J and Kirkland, James L
Aging cell, ISSN 1474-9718, 08/2015, Volume 14, Issue 4, pp. 644 - 658
dasatinib | plasminogen‐activated inhibitor | dependence receptors | quercetin | ephrins | PI3K delta | p21 | Dasatinib | Dependence receptors | Ephrins | Plasminogen-activated inhibitor | Quercetin | P21 | Life Sciences & Biomedicine | Geriatrics & Gerontology | Science & Technology | Cell Biology | Carotid Arteries - drug effects | Endonucleases - genetics | Plasminogen Activator Inhibitor 2 - genetics | Transcriptome | Gene Expression Profiling | Adipocytes - drug effects | Aging - genetics | Osteoporosis - metabolism | Ephrins - metabolism | Plasminogen Activator Inhibitor 2 - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Osteoporosis - genetics | Intervertebral Disc - pathology | Fibroblasts - metabolism | Endothelial Cells - metabolism | Intervertebral Disc - chemistry | Fibroblasts - pathology | Mesenchymal Stem Cells - pathology | Mice, Knockout | Dasatinib - pharmacology | Osteoporosis - pathology | Ephrins - genetics | Fibroblasts - drug effects | Mice | Endothelial Cells - pathology | bcl-X Protein - metabolism | Aging - metabolism | Aging - drug effects | bcl-X Protein - genetics | Cellular Senescence - drug effects | Phosphatidylinositol 3-Kinases - metabolism | Endonucleases - metabolism | DNA-Binding Proteins - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Mesenchymal Stem Cells - drug effects | Mesenchymal Stem Cells - metabolism | Heart - physiopathology | Cellular Senescence - genetics | Osteoporosis - prevention & control | DNA-Binding Proteins - genetics | Adipocytes - pathology | Aging - pathology | Phosphatidylinositol 3-Kinases - genetics | Animals | Quercetin - pharmacology | Adipocytes - metabolism | Heart - drug effects | Carotid Arteries - pathology | Intervertebral Disc - drug effects | Drug Combinations | Endothelial Cells - drug effects | Index Medicus | Original | plasminogen-activated inhibitor
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4. Full Text Effects of blood pressure lowering on outcome incidence in hypertension: 4. Effects of various classes of antihypertensive drugs – Overview and meta-analyses
by Thomopoulos, Costas and Parati, Gianfranco and Zanchetti, Alberto
Journal of hypertension, ISSN 0263-6352, 02/2015, Volume 33, Issue 2, pp. 195 - 211
angiotensin-converting enzyme inhibitors | angiotensin receptor blockers | diuretics | beta-blockers | meta-analysis | calcium antagonists | randomized controlled trials | bloodpressure-lowering treatment | drug class | hypertension | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Blood Pressure | Chlorthalidone - therapeutic use | Stroke - prevention & control | Risk Reduction Behavior | Cardiovascular Diseases - prevention & control | Humans | Risk Factors | Calcium Channel Blockers - therapeutic use | Coronary Artery Disease - prevention & control | Indapamide - therapeutic use | Hypertension - drug therapy | Antihypertensive Agents - therapeutic use | Incidence | Randomized Controlled Trials as Topic | Heart Failure - prevention & control | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Cardiovascular Diseases - epidemiology | Diuretics - therapeutic use | Angiotensin Receptor Antagonists - therapeutic use | Adrenergic beta-Antagonists - therapeutic use | Index Medicus
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5. Full Text Phosphorylation of VE-cadherin is modulated by haemodynamic forces and contributes to the regulation of vascular permeability in vivo
by Orsenigo, Fabrizio and Giampietro, Costanza and Ferrari, Aldo and Corada, Monica and Galaup, Ariane and Sigismund, Sara and Ristagno, Giuseppe and Maddaluno, Luigi and Koh, Gou Young and Franco, Davide and Kurtcuoglu, Vartan and Poulikakos, Dimos and Baluk, Peter and McDonald, Donald and Grazia Lampugnani, Maria and Dejana, Elisabetta
Nature communications, ISSN 2041-1723, 2012, Volume 3, Issue 1, pp. 1208 - 1208
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Antigens, CD - immunology | Cadherins - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Capillary Permeability - drug effects | Molecular Sequence Data | Stress, Mechanical | Antigens, CD - metabolism | Cadherins - immunology | Ubiquitination - drug effects | Arteries - physiology | Antibody Specificity - drug effects | Inflammation Mediators - metabolism | src-Family Kinases - metabolism | Phosphorylation - drug effects | Bradykinin - pharmacology | Amino Acid Sequence | Human Umbilical Vein Endothelial Cells - drug effects | Endocytosis - drug effects | Mice, Inbred C57BL | Arteries - drug effects | Enzyme Activation - drug effects | Veins - drug effects | Animals | Hemodynamics - drug effects | Mice | Veins - physiology | Index Medicus
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6. Effect of cocaine on pulmonary vascular remodeling and hemodynamics in human immunodeficiency virus-transgenic rats
by Dalvi, Pranjali and Spikes, Leslie and Allen, Julie and Gupta, Vijayalaxmi G and Sharma, Himanshu and Gillcrist, Marion and De Oca, Jamison Montes and O'Brien-Ladner, Amy and Dhillon, Navneet K
American journal of respiratory cell and molecular biology, ISSN 1044-1549, 08/2016, Volume 55, Issue 2, pp. 201 - 212
Pulmonary hypertension; drug abuse; smooth muscle; bone morphogenetic protein receptor; human immunodeficiency virus-1 | Respiratory System | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | HIV - physiology | Apoptosis - drug effects | Humans | Rats, Inbred F344 | Myocytes, Smooth Muscle - pathology | Systole - drug effects | Male | Viral Proteins - metabolism | HIV - drug effects | Lung - virology | Blood Pressure - drug effects | Lung - metabolism | Vascular Remodeling - drug effects | Cocaine - adverse effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Rats, Transgenic | Cell Separation | Bone Morphogenetic Protein Receptors - metabolism | Lung - physiopathology | Pulmonary Artery - physiopathology | Pulmonary Artery - drug effects | Down-Regulation - drug effects | Rats, Sprague-Dawley | Muscle, Smooth, Vascular - pathology | Animals | Signal Transduction - drug effects | Lung - drug effects | Cell Proliferation - drug effects | Hemodynamics - drug effects | Hypertension | Heart | Pulmonary arteries | Laboratories | Diagnostic tests | Mortality | Smooth muscle | Infections | Proteins | Hepatitis | Rodents | Human immunodeficiency virus--HIV | Protein expression | Cocaine | Intubation | Apoptosis
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7. Full Text Doxorubicin eluting beads − 1: Effects of drug loading on bead characteristics and drug distribution
by Lewis, Andrew L and Gonzalez, M Victoria and Leppard, Simon W and Brown, Joanna E and Stratford, Peter W and Phillips, Gary J and Lloyd, Andrew W
Journal of materials science. Materials in medicine, ISSN 0957-4530, 9/2007, Volume 18, Issue 9, pp. 1691 - 1699
Polymer Sciences | Chemistry | Ceramics, Glass, Composites, Natural Methods | Characterization and Evaluation of Materials | Metallic Materials | Surfaces and Interfaces, Thin Films | Biomaterials | Engineering | Materials Science | Technology | Engineering, Biomedical | Materials Science, Biomaterials | Science & Technology | Microscopy, Electron, Scanning | Chemoembolization, Therapeutic | Humans | Hepatic Artery | Materials Testing | Doxorubicin - chemistry | Liver Neoplasms - therapy | Doxorubicin - pharmacokinetics | Drug Carriers | Drug Delivery Systems | Antibiotics, Antineoplastic - administration & dosage | Particle Size | Microspheres | Polyvinyl Alcohol | Antibiotics, Antineoplastic - chemistry | Liver Neoplasms - metabolism | Carcinoma, Hepatocellular - therapy | Antibiotics, Antineoplastic - pharmacokinetics | Carcinoma, Hepatocellular - metabolism | Doxorubicin - administration & dosage | Drug wholesalers | Analysis | Tumors | Index Medicus
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