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Nature Medicine, ISSN 1078-8956, 05/2016, Volume 22, Issue 5, pp. 516 - 523
Journal Article
Journal Article
Glia, ISSN 0894-1491, 2005, Volume 51, Issue 4, pp. 297 - 305
Perivascular macrophages (PVM) constitute a subpopulation of resident macrophages in the central nervous system (CNS) that by virtue of their strategic... 
perivascular macrophages | antigen presentation and recognition | DC‐SIGN | multiple sclerosis | mannose receptor | costimulatory molecules | Antigen presentation and recognition | Mannose receptor | Multiple sclerosis | Perivascular macrophages | Costimulatory molecules | DC-SIGN | MULTIPLE-SCLEROSIS LESIONS | HUMAN BRAIN | DENDRITIC CELLS | MICROGLIAL CELLS | NEUROSCIENCES | SCAVENGER RECEPTOR | CENTRAL-NERVOUS-SYSTEM | MENINGEAL MACROPHAGES | RAT-BRAIN | IN-SITU | Mannose-Binding Lectins - metabolism | Antigens, CD - immunology | Microglia - metabolism | Central Nervous System - metabolism | Blood-Brain Barrier - cytology | Humans | Middle Aged | Lectins, C-Type - immunology | Male | Cell Adhesion Molecules - immunology | Central Nervous System - immunology | Lectins, C-Type - metabolism | Lymphocyte Activation - immunology | Encephalitis - physiopathology | Microglia - immunology | Encephalitis - metabolism | Inflammation Mediators - metabolism | Female | Histocompatibility Antigens Class II - metabolism | Microcirculation - immunology | Antigens, Differentiation, Myelomonocytic - immunology | Macrophages - immunology | Cerebral Arteries - cytology | Inflammation Mediators - immunology | Antigen-Presenting Cells - metabolism | Blood-Brain Barrier - immunology | Cerebral Arteries - immunology | Receptors, Cell Surface - metabolism | Encephalitis - immunology | Antigen Presentation - immunology | Antigen-Presenting Cells - immunology | Cell Adhesion Molecules - metabolism | Receptors, Cell Surface - immunology | Microcirculation - cytology | Macrophages - metabolism | Histocompatibility Antigens Class II - immunology | Mannose-Binding Lectins - immunology | T-Lymphocytes - immunology | Aged | Central Nervous System - physiopathology
Journal Article
Science, ISSN 0036-8075, 11/2015, Volume 350, Issue 6262, pp. 830 - 834
In healthy individuals, the intestinal microbiota cannot access the liver, spleen, or other peripheral tissues. Some pathogenic bacteria can reach these sites,... 
ORAL TOLERANCE | DENDRITIC CELLS | BLOOD-BRAIN-BARRIER | MULTIDISCIPLINARY SCIENCES | Genomic Islands - immunology | Spleen - immunology | Humans | Antigens, Bacterial - immunology | Salmonella Infections - immunology | Capillary Permeability - immunology | Antigens, Bacterial - blood | Ileum - immunology | Type III Secretion Systems - genetics | Intestines - immunology | Type III Secretion Systems - immunology | Ileum - blood supply | Celiac Disease - microbiology | Liver - immunology | Celiac Disease - immunology | Intestinal Mucosa - immunology | Celiac Disease - blood | Intestines - blood supply | Wnt Signaling Pathway | Genomic Islands - genetics | Microbiota - immunology | Salmonella typhimurium - genetics | Salmonella typhimurium - immunology | Salmonella typhimurium - pathogenicity | Signal Transduction | Mice, Inbred C57BL | Intestinal Mucosa - microbiology | beta Catenin - metabolism | Animals | Intestines - microbiology | Transaminases - blood | Mice | Ileum - microbiology | Immune response | Research | Microbiota (Symbiotic organisms) | Health aspects | Bacteria | Cellular biology | Liver | Veins & arteries | Salmonella | Barriers | Secretions | Islands | Patients | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Gastroenterology and Hepatology | Gastroenterologi
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 135, Issue 6, pp. 1424 - 1432
Anaphylaxis is an acute, severe, life-threatening multisystem allergic reaction resulting from the sudden systemic release of biochemical mediators and... 
Allergy and Immunology | Allergy | signaling | mediators | receptor antagonists | anaphylaxis | platelet-activating factor | mast cells | acetylhydrolase | platelets | sepsis | PHOSPHOLIPASE A INHIBITOR | CLINICAL-TRIAL | IMMUNOLOGY | FACTOR ACETYLHYDROLASE ACTIVITY | PHASE-II TRIAL | FACTOR PAF | FACTOR RECEPTOR ANTAGONIST | DOUBLE-BLIND | HUMAN NASAL AIRWAY | CORONARY-ARTERY-DISEASE | SEVERE SEPSIS | Blood Platelets - pathology | Blood Platelets - immunology | Humans | Platelet Activation - immunology | Platelet Activating Factor - immunology | Eosinophils - immunology | 1-Alkyl-2-acetylglycerophosphocholine Esterase - immunology | Receptors, G-Protein-Coupled - immunology | 1-Alkyl-2-acetylglycerophosphocholine Esterase - genetics | Sepsis - pathology | Asthma - immunology | Anaphylaxis - immunology | Eosinophils - pathology | Platelet Aggregation - genetics | Neutrophils - pathology | Mast Cells - immunology | Platelet Membrane Glycoproteins - immunology | Sepsis - immunology | Platelet Aggregation - immunology | Signal Transduction | Anaphylaxis - pathology | Gene Expression Regulation | Neutrophils - immunology | Platelet Activating Factor - genetics | Platelet Membrane Glycoproteins - genetics | Asthma - genetics | Sepsis - genetics | Anaphylaxis - genetics | Platelet Activation - genetics | Mast Cells - pathology | Receptors, G-Protein-Coupled - genetics | Asthma - pathology | Enzymes | Immune response | Anaphylaxis | Permeability | Metabolites | Analysis | Proteins | Antigens | Blood platelets | E coli | Rodents | Preeclampsia | Lipids | Smooth muscle | Carbon | Biological activity
Journal Article
Blood, ISSN 0006-4971, 05/2007, Volume 109, Issue 9, pp. 3794 - 3802
IL-17 and its receptor are founding members of a novel family of inflammatory cytokines. IL-17 plays a pathogenic role in rheumatoid arthritis (RA)-associated... 
RHEUMATOID-ARTHRITIS | ALVEOLAR BONE | COLONY-STIMULATING FACTOR | T-CELL INTERLEUKIN-17 | PORPHYROMONAS-GINGIVALIS | HOST-DEFENSE | JOINT DESTRUCTION | IL-17-DEFICIENT MICE | PERIODONTAL-DISEASE | HEMATOLOGY | KLEBSIELLA-PNEUMONIAE | Leukopoiesis - genetics | Osteitis - genetics | Humans | Receptors, Interleukin - deficiency | Bacteroidaceae Infections - genetics | Interleukin-17 - immunology | Pulmonary Disease, Chronic Obstructive - pathology | Receptors, Interleukin-8B - deficiency | Signal Transduction - immunology | Coronary Artery Disease - pathology | Porphyromonas gingivalis - immunology | Osteolysis - pathology | Bacteroidaceae Infections - pathology | Chemokines - immunology | Neutrophil Infiltration - immunology | Neutrophils - pathology | Pulmonary Disease, Chronic Obstructive - genetics | Periodontitis - immunology | Leukopoiesis - immunology | Receptors, Interleukin - immunology | Osteolysis - genetics | Neutrophils - immunology | Osteolysis - immunology | Periodontitis - pathology | Receptors, Interleukin-8B - immunology | Signal Transduction - genetics | Periodontitis - genetics | Neutrophil Infiltration - genetics | Mice, Knockout | Animals | Osteitis - immunology | Bacteroidaceae Infections - immunology | Osteitis - pathology | Coronary Artery Disease - genetics | Mice | Coronary Artery Disease - immunology | Pulmonary Disease, Chronic Obstructive - immunology | Immunobiology
Journal Article
Immunology and Cell Biology, ISSN 0818-9641, 04/2006, Volume 84, Issue 2, pp. 115 - 124
Coronary artery disease, leading to myocardial infarction and ischaemia, affects millions of persons and is one of the leading causes of morbidity and... 
atherosclerosis | coronary artery bypass graft (CABG) | immune mechanism | intimal hyperplasia | cell proliferation | Cell proliferation | Coronary artery bypass graft (CABG) | Immune mechanism | Intimal hyperplasia | Atherosclerosis | COAGULATION-FIBRINOLYSIS SYSTEM | LOW-DENSITY-LIPOPROTEIN | IMMUNOLOGY | FACTOR-B-CHAIN | VASCULAR SMOOTH-MUSCLE | FATTY STREAK FORMATION | CELL BIOLOGY | HUMAN ATHEROSCLEROTIC PLAQUES | NITRIC-OXIDE | FOCAL ADHESION KINASE | TISSUE FACTOR | GROWTH-FACTOR-I | Leukocytes - pathology | Graft Survival - immunology | Myocardial Infarction - mortality | Hyperplasia - immunology | Humans | Myocardial Infarction - immunology | Phosphatidylinositol 3-Kinases - immunology | Leukocytes - immunology | Signal Transduction - immunology | Coronary Artery Bypass - mortality | Growth Substances - immunology | Arterial Occlusive Diseases - pathology | Arterial Occlusive Diseases - mortality | Arterial Occlusive Diseases - immunology | Myocardial Infarction - surgery | Hyperplasia - mortality | Tunica Intima - immunology | Hyperplasia - etiology | Arterial Occlusive Diseases - etiology | Hyperplasia - pathology | Coronary Artery Bypass - adverse effects | Myocardial Infarction - complications | Graft Occlusion, Vascular - etiology | Tunica Intima - pathology | Graft Occlusion, Vascular - pathology | Graft Occlusion, Vascular - immunology | Oncogene Protein v-akt - immunology | Immunity, Cellular | Tunica Intima, pathology | Graft Survival, immunology | Arterial Occlusive Diseases, immunology | Oncogene Protein v-akt, immunology | Graft Occlusion, Vascular, pathology | Coronary Artery Bypass, adverse effects | Growth Substances, immunology | Hyperplasia, etiology | Graft Occlusion, Vascular, immunology | Tunica Intima, immunology | Signal Transduction, immunology | Arterial Occlusive Diseases, mortality | Leukocytes, immunology | Leukocytes, pathology | Myocardial Infarction, immunology | Myocardial Infarction, complications | Arterial Occlusive Diseases, pathology | Coronary Artery Bypass, mortality | Hyperplasia, mortality | Arterial Occlusive Diseases, etiology | Myocardial Infarction, mortality | Hyperplasia, pathology | Hyperplasia, immunology | Coronary Artery Bypass | Graft Occlusion, Vascular, etiology | Myocardial Infarction, surgery | 1-Phosphatidylinositol 3-Kinase, immunology
Journal Article