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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2011, Volume 108, Issue 27, pp. 11193 - 11198
MicroRNA (miRNA) species (miR) regulate mRNA translation and are implicated as mediators of disease pathology via coordinated regulation of molecular effector... 
Myeloid cells | MicroRNA | Messenger RNA | Cytokines | Synovial membrane | Rheumatoid arthritis | Collagens | Arthritis | Inflammation | Macrophages | RHEUMATOID-ARTHRITIS | PATHOGENESIS | COLLAGEN-INDUCED ARTHRITIS | AUTOIMMUNE INFLAMMATION | SYNOVIAL TISSUE | ROLES | MULTIDISCIPLINARY SCIENCES | DEFICIENT MICE | INDUCTION | EXPRESSION | T-CELLS | Arthritis, Experimental - genetics | Humans | Inositol Polyphosphate 5-Phosphatases | Synovial Membrane - immunology | Synovial Membrane - pathology | MicroRNAs - metabolism | Case-Control Studies | Arthritis, Experimental - metabolism | Arthritis, Experimental - pathology | Arthritis, Rheumatoid - metabolism | Base Sequence | Inflammation Mediators - metabolism | Phosphoric Monoester Hydrolases - genetics | Osteoarthritis - immunology | Osteoarthritis - metabolism | Arthritis, Experimental - immunology | Synovial Membrane - metabolism | Osteoarthritis - genetics | Arthritis, Rheumatoid - pathology | Mice, Knockout | Arthritis, Rheumatoid - genetics | Animals | Mice | MicroRNAs - genetics | Arthritis, Rheumatoid - immunology | Phosphatidylinositol-3,4,5-Trisphosphate 5-Phosphatases | Phosphoric Monoester Hydrolases - metabolism | Cytokines - biosynthesis | Physiological aspects | Genetic aspects | Health aspects | Proteins | Ribonucleic acid--RNA | Gene expression | Cells | Biological Sciences
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2012, Volume 109, Issue 50, pp. 20602 - 20607
Osteoarthritis is one of the leading causes of chronic pain, but almost nothing is known about the mechanisms and molecules that mediate... 
Receptors | Pain | Neurons | Mice | Arthritis | Macrophages | Osteoarthritis | Behavioral neuroscience | Chemokines | Chronic pain | SYSTEM | NEUROPATHIC PAIN | CENTRAL SENSITIZATION | NOCICEPTIVE NEURONS | RAT | MULTIDISCIPLINARY SCIENCES | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | DORSAL-ROOT GANGLIA | SPINAL-CORD | MICE | EXPRESSION | Receptors, CCR2 - genetics | Receptors, CCR2 - physiology | Arthritis, Experimental - genetics | Chemokine CCL2 - immunology | Humans | Male | Pain - immunology | Ganglia, Spinal - physiopathology | RNA, Messenger - metabolism | Osteoarthritis - physiopathology | Arthritis, Experimental - pathology | Pain - genetics | Chemokine CCL2 - physiology | Osteoarthritis - pathology | Disease Models, Animal | Ganglia, Spinal - immunology | Arthritis, Experimental - physiopathology | Macrophages - pathology | Signal Transduction | Osteoarthritis - immunology | Mice, Inbred C57BL | RNA, Messenger - genetics | Chemokine CCL2 - genetics | Arthritis, Experimental - immunology | Osteoarthritis - genetics | Hyperalgesia - physiopathology | Mice, Knockout | Hyperalgesia - immunology | Animals | Hyperalgesia - genetics | Pain - physiopathology | Ganglia, Spinal - pathology | Receptors, CCR2 - deficiency | Prevention | Knee | Physiological aspects | Health aspects | Signal transduction | T cell receptors | Rodents | Biological Sciences
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2012, Volume 109, Issue 46, pp. 18647 - 18648
Neutrophil recruitment into the joint is a hallmark of inflammatory arthritides, including rheumatoid arthritis (RA). In a mouse model of autoantibody-induced... 
PNAS PLUS (AUTHOR SUMMARIES) | IMMUNE-COMPLEX ARTHRITIS | STERILE INFLAMMATION | RHEUMATOID SYNOVIAL-FLUIDS | BONE-MARROW-TRANSPLANTATION | MULTIDISCIPLINARY SCIENCES | AUTOANTIBODY-MEDIATED ARTHRITIS | MAST-CELLS | INFL AMMATORY ARTHRITIS | GENE-EXPRESSION | INFLAMMATORY ARTHRITIS | SERUM-INDUCED ARTHRITIS | Receptor, Anaphylatoxin C5a - genetics | Arthritis, Experimental - genetics | Receptors, Leukotriene B4 - genetics | Interleukin-1beta - genetics | Arthritis, Experimental - pathology | Autoantibodies - pharmacology | Signal Transduction - immunology | Receptors, IgG - genetics | Arthritis, Experimental - chemically induced | Receptor, Anaphylatoxin C5a - immunology | Receptors, CCR1 - immunology | Chemokines - immunology | Neutrophil Infiltration - immunology | Neutrophils - pathology | Neutrophils - immunology | Interleukin-1beta - immunology | Joints - pathology | Arthritis, Experimental - immunology | Receptors, Interleukin-8B - immunology | Signal Transduction - genetics | Receptors, IgG - immunology | Leukotriene B4 - immunology | Chemokines - genetics | Neutrophil Infiltration - genetics | Receptors, Leukotriene B4 - immunology | Mice, Knockout | Receptors, CCR1 - genetics | Animals | Joints - immunology | Receptors, Interleukin-8B - genetics | Complement C5a - genetics | Mice | Complement C5a - immunology | Leukotriene B4 - genetics | Autoantibodies - adverse effects | PNAS Plus | Biological Sciences
Journal Article
Journal Article
Genes and Immunity, ISSN 1466-4879, 01/2017, Volume 18, Issue 1, pp. 48 - 56
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 08/2017, Volume 23, Issue 8, pp. 938 - 944
Inflammatory diseases such as arthritis are chronic conditions that fail to resolve spontaneously. While the cytokine and cellular pathways triggering... 
RHEUMATOID-ARTHRITIS | MEDICINE, RESEARCH & EXPERIMENTAL | EFFECTOR T-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION FACTOR PU.1 | IL-9 | CELL BIOLOGY | IMAGE-ANALYSIS | PLASMID DNA | DISEASE | ANTIGEN-INDUCED ARTHRITIS | GROWTH-FACTOR | LUNG INFLAMMATION | Optical Imaging | T-Lymphocytes, Regulatory - metabolism | Arthritis, Experimental - genetics | Humans | Middle Aged | Synovial Membrane - immunology | Male | Arthritis, Experimental - diagnostic imaging | Gene Expression Profiling | X-Ray Microtomography | T-Lymphocytes, Regulatory - immunology | Arthritis, Experimental - pathology | Flow Cytometry | Lymphocytes - immunology | T-Lymphocytes, Helper-Inducer - immunology | Adult | Female | Disease Models, Animal | Lymphocytes - metabolism | Gene Transfer Techniques | Cell Proliferation - genetics | Arthritis, Experimental - immunology | Inflammation | Mice, Knockout | Immunity, Innate - immunology | Arthritis, Rheumatoid - genetics | Animals | Interleukin-9 - genetics | Aged | Mice | Arthritis, Rheumatoid - immunology | In Vitro Techniques | Interleukin-9 - immunology | Synovial Membrane - cytology | Cell proliferation | Lymphoid cells | Cytokines | Interleukin | Activation | Arthritis | Lymphocytes T | Joint diseases | Inflammatory diseases | Cartilage | Chronic conditions | Rheumatoid arthritis | Remission | Bone | Bone loss | Chronic illnesses | Interleukin 9 | innate lymphoid cells | Resolution of inflammation | arthritis | chronic inflammation | bone loss
Journal Article
Immunity, ISSN 1074-7613, 2011, Volume 35, Issue 5, pp. 832 - 844
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2017, Volume 127, Issue 10, pp. 3702 - 3716
Autoreactive CD4 T cells that differentiate into pathogenic Th17 cells can trigger autoimmune diseases. Therefore, investigating the regulatory network that... 
ENCEPHALOMYELITIS | MEDICINE, RESEARCH & EXPERIMENTAL | MYELIN OLIGODENDROCYTE GLYCOPROTEIN | IL-6-DEFICIENT MICE | TGF-BETA | MULTIPLE-SCLEROSIS | EPIGENETIC CONTROL | RHEUMATOID-ARTHRITIS SUSCEPTIBILITY | HELPER T-CELLS | MEDIATED REGULATION | PLASTICITY | Autoimmunity | Myelin-Oligodendrocyte Glycoprotein - immunology | Myelin-Oligodendrocyte Glycoprotein - genetics | Humans | Encephalomyelitis, Autoimmune, Experimental - immunology | Organ Specificity - immunology | Cell Differentiation - genetics | Interleukins - genetics | Organ Specificity - genetics | Autocrine Communication - immunology | Interleukins - immunology | Encephalomyelitis, Autoimmune, Experimental - genetics | Th17 Cells - pathology | Interleukin-6 - genetics | Multiple Sclerosis - genetics | MicroRNAs - immunology | Autocrine Communication - genetics | Mice, Knockout | Cell Differentiation - immunology | Animals | Interleukin-6 - immunology | Multiple Sclerosis - immunology | Th17 Cells - immunology | Mice | MicroRNAs - genetics | Research | Cell differentiation | T cells | Analysis | Multiple sclerosis | Oligodendrocyte-myelin glycoprotein | Encephalomyelitis | Disease | Cytokines | Myelin | Helper cells | Lymphocytes T | Inflammation | T cell receptors | Kinases | Gene expression | Experimental allergic encephalomyelitis | CD4 antigen | Interleukin 6 | T-cell receptor | Lymphocytes | Rheumatoid arthritis | MicroRNAs | Rodents | Stem cells | Interleukin 21 | Autocrine signalling | Autoimmune diseases
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2015, Volume 112, Issue 27, pp. E3564 - E3573
Journal Article
Genome Biology, ISSN 1474-7596, 02/2015, Volume 16, Issue 1, p. 28
Journal Article