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Arthritis & rheumatology (Hoboken, N.J.), ISSN 2326-5191, 2016, Volume 68, Issue 11, pp. 2646 - 2661
Journal Article
PLoS biology, ISSN 1545-7885, 2014, Volume 12, Issue 1, p. e1001762
...RI, TLRs, IL-1R, and IL-33R. As a consequence house dust mite- and IL-33-driven lung inflammation, late phase cutaneous anaphylaxis, and collagen-induced arthritis were aggravated, in contrast to experimental autoimmune... 
CYTOKINE PRODUCTION | DENDRITIC CELLS | AUTOIMMUNITY | ASTHMA MODEL | BIOCHEMISTRY & MOLECULAR BIOLOGY | BIOLOGY | INDUCED ARTHRITIS | INTERLEUKIN-33 | MICE | HOUSE-DUST MITE | IL-33 | A20 | Interleukin-1 Receptor-Like 1 Protein | Serum Albumin, Bovine - administration & dosage | Receptors, Interleukin-1 - genetics | Dinitrophenols - administration & dosage | Encephalomyelitis, Autoimmune, Experimental - immunology | Male | Myelin-Oligodendrocyte Glycoprotein - administration & dosage | Intracellular Signaling Peptides and Proteins - deficiency | Interleukins - genetics | Anaphylaxis - chemically induced | Arthritis, Experimental - chemically induced | Pneumonia - immunology | Tumor Necrosis Factor alpha-Induced Protein 3 | Intracellular Signaling Peptides and Proteins - genetics | Cysteine Endopeptidases | Gene Expression | DNA-Binding Proteins - immunology | Receptors, Interleukin - immunology | Interleukin-33 | Toll-Like Receptors - immunology | Anaphylaxis - pathology | Mice, Transgenic | Receptors, Interleukin - genetics | Toll-Like Receptors - genetics | Mice | Pneumonia - metabolism | Ubiquitin-Protein Ligases - genetics | Encephalomyelitis, Autoimmune, Experimental - metabolism | NF-kappa B - immunology | Pneumonia - pathology | Intracellular Signaling Peptides and Proteins - immunology | DNA-Binding Proteins - deficiency | Arthritis, Experimental - metabolism | Arthritis, Experimental - pathology | Ubiquitin-Protein Ligases - immunology | Immunoglobulin E - immunology | Encephalomyelitis, Autoimmune, Experimental - chemically induced | Immunoglobulin E - genetics | Pneumonia - chemically induced | Interleukins - immunology | Mast Cells - metabolism | Anaphylaxis - immunology | Receptors, IgE - immunology | Lung - metabolism | Collagen Type II - administration & dosage | Mast Cells - immunology | Lung - pathology | Encephalomyelitis, Autoimmune, Experimental - pathology | Receptors, Interleukin-1 - immunology | Peptide Fragments - administration & dosage | Arthritis, Experimental - immunology | Receptors, IgE - genetics | DNA-Binding Proteins - genetics | Animals | NF-kappa B - genetics | Mast Cells - pathology | Ubiquitin-Protein Ligases - deficiency | Anaphylaxis - metabolism | Lung - immunology | Pyroglyphidae - immunology | Cell interaction | Cell research | Immune response | Physiological aspects | Mast cells | Inflammation | Research | Health aspects | Immunological research | Antigens | Cytokines | Disease | Arthritis | Grants | Pattern recognition | Gene expression | Kinases | Experiments | Allergies | Asthma | Studies | Histamine | Rodents
Journal Article
Journal Article
Seminars in immunopathology, ISSN 1863-2300, 2011, Volume 34, Issue 1, pp. 43 - 62
The interaction of coagulation factors with the perivascular environment affects the development of disease in ways that extend beyond their traditional roles... 
Autoimmunity | Complement receptor 3 | Multiple sclerosis | Stroke | Alzheimer’s disease | Internal Medicine | Macrophages | Blood brain barrier | Inflammatory disease | Microglia | Biomedicine | Immunology | Rheumatoid arthritis | CD11b/CD18 | Atherosclerosis | Plasminogen | Anticoagulant therapy | Alzheimer's disease | ALZHEIMERS-DISEASE | IMMUNOLOGY | PATHOLOGY | GAMMA-C-DOMAIN | BINDING-SITE | CENTRAL-NERVOUS-SYSTEM | MULTIPLE-SCLEROSIS LESIONS | CRYSTAL-STRUCTURE | BLOOD-BRAIN-BARRIER | INTEGRIN ALPHA(M)BETA | PROTEASE-ACTIVATED RECEPTORS | PERIPHERAL ARTERIAL-DISEASE | Colitis - genetics | Humans | Brain Injuries - metabolism | Arthritis, Rheumatoid - metabolism | Bacterial Infections - genetics | Inflammation - metabolism | Blood Coagulation - immunology | Muscular Dystrophy, Duchenne - immunology | Alzheimer Disease - immunology | Fibrinogen - immunology | Thromboplastin - immunology | Pulmonary Fibrosis - immunology | Vascular Diseases - genetics | Brain Injuries - genetics | Thrombin - genetics | Kidney Diseases - immunology | Arthritis, Rheumatoid - genetics | Neoplasms - immunology | Colitis - metabolism | Muscular Dystrophy, Duchenne - genetics | Bacterial Infections - metabolism | Neoplasms - metabolism | Pulmonary Fibrosis - genetics | Spinal Cord Injuries - genetics | Kidney Diseases - genetics | Stroke - genetics | Brain Injuries - immunology | Neoplasms - genetics | Bacterial Infections - immunology | Pulmonary Fibrosis - metabolism | Colitis - immunology | Thrombin - immunology | Stroke - immunology | Kidney Diseases - metabolism | Multiple Sclerosis - metabolism | Vascular Diseases - immunology | Spinal Cord Injuries - metabolism | Multiple Sclerosis - genetics | Inflammation - immunology | Fibrinogen - genetics | Stroke - metabolism | Animals | Thromboplastin - genetics | Alzheimer Disease - metabolism | Fibrinogen - metabolism | Inflammation - genetics | Multiple Sclerosis - immunology | Blood Coagulation - genetics | Spinal Cord Injuries - immunology | Muscular Dystrophy, Duchenne - metabolism | Thrombin - metabolism | Thromboplastin - metabolism | Alzheimer Disease - genetics | Arthritis, Rheumatoid - immunology | Vascular Diseases - metabolism | Nervous system diseases | Bacterial infections | Oncology, Experimental | Thrombin | Inflammation | Anticoagulants (Medicine) | Research | Rheumatoid factor | Fibrin | Fibrinogen | Genetic research | Colitis | Cancer | Traumatic brain injury | Coagulation | Tissue factor | hemostasis | Spinal cord injury | Inflammatory diseases | Kidney | Signal transduction | Duchenne's muscular dystrophy | Neurodegenerative diseases | Therapeutic applications | Thrombosis | Coagulation factors | Infection | Molecular modelling | Fibrosis | Brain injury
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 11/2012, Volume 109, Issue 46, pp. 18647 - 18648
Neutrophil recruitment into the joint is a hallmark of inflammatory arthritides, including rheumatoid arthritis (RA). In a mouse model of autoantibody-induced... 
PNAS PLUS (AUTHOR SUMMARIES) | IMMUNE-COMPLEX ARTHRITIS | STERILE INFLAMMATION | RHEUMATOID SYNOVIAL-FLUIDS | BONE-MARROW-TRANSPLANTATION | MULTIDISCIPLINARY SCIENCES | AUTOANTIBODY-MEDIATED ARTHRITIS | MAST-CELLS | INFL AMMATORY ARTHRITIS | GENE-EXPRESSION | INFLAMMATORY ARTHRITIS | SERUM-INDUCED ARTHRITIS | Receptor, Anaphylatoxin C5a - genetics | Arthritis, Experimental - genetics | Receptors, Leukotriene B4 - genetics | Interleukin-1beta - genetics | Arthritis, Experimental - pathology | Autoantibodies - pharmacology | Signal Transduction - immunology | Receptors, IgG - genetics | Arthritis, Experimental - chemically induced | Receptor, Anaphylatoxin C5a - immunology | Receptors, CCR1 - immunology | Chemokines - immunology | Neutrophil Infiltration - immunology | Neutrophils - pathology | Neutrophils - immunology | Interleukin-1beta - immunology | Joints - pathology | Arthritis, Experimental - immunology | Receptors, Interleukin-8B - immunology | Signal Transduction - genetics | Receptors, IgG - immunology | Leukotriene B4 - immunology | Chemokines - genetics | Neutrophil Infiltration - genetics | Receptors, Leukotriene B4 - immunology | Mice, Knockout | Receptors, CCR1 - genetics | Animals | Joints - immunology | Receptors, Interleukin-8B - genetics | Complement C5a - genetics | Mice | Complement C5a - immunology | Leukotriene B4 - genetics | Autoantibodies - adverse effects | PNAS Plus | Biological Sciences
Journal Article
Journal Article