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Nature Medicine, ISSN 1078-8956, 07/2012, Volume 18, Issue 7, pp. 1069 - 1076
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 12/2015, Volume 74, Issue 12, pp. 2254 - 2258
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2011, Volume 108, Issue 27, pp. 11193 - 11198
MicroRNA (miRNA) species (miR) regulate mRNA translation and are implicated as mediators of disease pathology via coordinated regulation of molecular effector... 
Myeloid cells | MicroRNA | Messenger RNA | Cytokines | Synovial membrane | Rheumatoid arthritis | Collagens | Arthritis | Inflammation | Macrophages | RHEUMATOID-ARTHRITIS | PATHOGENESIS | COLLAGEN-INDUCED ARTHRITIS | AUTOIMMUNE INFLAMMATION | SYNOVIAL TISSUE | ROLES | MULTIDISCIPLINARY SCIENCES | DEFICIENT MICE | INDUCTION | EXPRESSION | T-CELLS | Arthritis, Experimental - genetics | Humans | Inositol Polyphosphate 5-Phosphatases | Synovial Membrane - immunology | Synovial Membrane - pathology | MicroRNAs - metabolism | Case-Control Studies | Arthritis, Experimental - metabolism | Arthritis, Experimental - pathology | Arthritis, Rheumatoid - metabolism | Base Sequence | Inflammation Mediators - metabolism | Phosphoric Monoester Hydrolases - genetics | Osteoarthritis - immunology | Osteoarthritis - metabolism | Arthritis, Experimental - immunology | Synovial Membrane - metabolism | Osteoarthritis - genetics | Arthritis, Rheumatoid - pathology | Mice, Knockout | Arthritis, Rheumatoid - genetics | Animals | Mice | MicroRNAs - genetics | Arthritis, Rheumatoid - immunology | Phosphatidylinositol-3,4,5-Trisphosphate 5-Phosphatases | Phosphoric Monoester Hydrolases - metabolism | Cytokines - biosynthesis | Physiological aspects | Genetic aspects | Health aspects | Proteins | Ribonucleic acid--RNA | Gene expression | Cells | Inositol | Translation | Autoantibodies | Therapeutic applications | Helper cells | Data processing | Homology | Src protein | Synovial fluid | Collagen | miRNA | Osteoarthritis | Tumor necrosis factor- alpha | Index Medicus | Biological Sciences
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 11/2014, Volume 66, Issue 11, pp. 2989 - 3000
Objective. Colony-stimulating factor 1 receptor (CSF-1R) essentially modulates monocyte proliferation, migration, and activation, which are considered... 
RHEUMATOID-ARTHRITIS | COLLAGEN-INDUCED ARTHRITIS | HUMAN SYNOVIAL FIBROBLASTS | TUMOR-NECROSIS-FACTOR | M-CSF | TRANSGENIC EXPRESSION | DENDRITIC CELLS | C-FMS | AUTOIMMUNE ARTHRITIS | INFLAMMATORY ARTHRITIS | RHEUMATOLOGY | Bone and Bones - pathology | Receptor, Macrophage Colony-Stimulating Factor - antagonists & inhibitors | Arthritis, Experimental - drug therapy | Cartilage - pathology | Humans | Middle Aged | Dendritic Cells - pathology | Synovial Membrane - pathology | Antibodies, Monoclonal - therapeutic use | Male | Monocytes - metabolism | Cartilage - drug effects | Synovial Membrane - drug effects | Case-Control Studies | Receptor, Macrophage Colony-Stimulating Factor - metabolism | Arthritis, Experimental - metabolism | Bone and Bones - drug effects | Arthritis, Experimental - pathology | Arthritis, Rheumatoid - metabolism | Bone and Bones - metabolism | Monocytes - pathology | Female | Mice, Inbred DBA | Osteoarthritis - pathology | Antibodies, Monoclonal - immunology | Dendritic Cells - metabolism | Disease Models, Animal | Osteoclasts - pathology | Macrophages - pathology | Antibodies, Monoclonal - pharmacology | Osteoarthritis - metabolism | Cartilage - metabolism | Osteoclasts - metabolism | Synovial Membrane - metabolism | Arthritis, Rheumatoid - pathology | Macrophages - metabolism | Animals | Mice | Receptor, Macrophage Colony-Stimulating Factor - drug effects | Rheumatism | Rodents | Arthritis | Index Medicus | Abridged Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2010, Volume 107, Issue 5, pp. 2159 - 2164
The beneficial effects of probiotics have been described in many diseases, but the mechanism by which they modulate the immune system is poorly understood. In... 
T lymphocytes | Probiotics | Inflammatory bowel diseases | Immune disorders | Population growth | Cytokines | Dendritic cells | B lymphocytes | Drug regulation | Mice | Inflammatory bowel disease | Inflammation | Atopic dermatitis | Rheumatoid arthritis | Regulatory T cell | MIGRATION | BACTERIA | MULTIDISCIPLINARY SCIENCES | RETINOIC-ACID | atopic dermatitis | regulatory T cell | INDUCTION | rheumatoid arthritis | FOXP3 | inflammatory bowel disease | EXPERIMENTAL ARTHRITIS | INFLAMMATORY-BOWEL-DISEASE | inflammation | ORAL TOLERANCE | MICE | EXPRESSION | Probiotics - administration & dosage | T-Lymphocytes, Regulatory - metabolism | Arthritis, Experimental - therapy | Dendritic Cells - immunology | Inflammatory Bowel Diseases - immunology | Dendritic Cells - pathology | Adoptive Transfer | Dermatitis, Atopic - therapy | T-Lymphocytes, Regulatory - pathology | T-Lymphocytes, Regulatory - immunology | Arthritis, Experimental - pathology | CD11c Antigen - metabolism | Forkhead Transcription Factors - metabolism | Inflammatory Bowel Diseases - pathology | Dermatitis, Atopic - immunology | Immune System Diseases - pathology | Disease Models, Animal | Immune System Diseases - immunology | Mice, Transgenic | Arthritis, Experimental - immunology | Dermatitis, Atopic - pathology | Immunosuppression | Animals | Immune System Diseases - therapy | Mice, Inbred BALB C | Inflammatory Bowel Diseases - therapy | T cell receptors | Effects | Autoimmune diseases | Medical treatment | Immune system | Index Medicus | Biological Sciences
Journal Article
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 04/2012, Volume 71, Issue 4, pp. 575 - 581
Objectives Osteoarthritis is associated with cell death and extracellular matrix degradation in articular cartilage. Autophagy is an essential cellular... 
MAMMALIAN TARGET | LIFE-SPAN | AGING-RELATED LOSS | MUTANT HUNTINGTIN | IN-VIVO | DISEASE | MOUSE | MICE | RHEUMATOLOGY | MTOR | CARTILAGE | Arthritis, Experimental - drug therapy | TOR Serine-Threonine Kinases - metabolism | Immunosuppressive Agents - therapeutic use | Male | Osteoarthritis - drug therapy | Autophagy - drug effects | Arthritis, Experimental - metabolism | Arthritis, Experimental - pathology | Anti-Inflammatory Agents - therapeutic use | Osteoarthritis - pathology | Immunosuppressive Agents - pharmacology | Chondrocytes - metabolism | ADAMTS5 Protein | Drug Evaluation, Preclinical - methods | Sirolimus - therapeutic use | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Osteoarthritis - metabolism | Treatment Outcome | Sirolimus - pharmacology | Cartilage, Articular - pathology | ADAM Proteins - metabolism | Animals | Signal Transduction - drug effects | Mice | Autophagy (Cytology) | Care and treatment | Rapamycin | Dosage and administration | Models | Research | Osteoarthritis | Protein kinases | Knee | Antigens | Enzymes | Aging | Homeostasis | Calcification | Arthritis | Inflammation | Kinases | Gene expression | Autophagy | Apoptosis | Immunohistochemistry | TOR protein | Synovitis | Phosphorylation | Cartilage (articular) | Joint diseases | Synovium | Signal transduction | Cell death | ribosomal protein S6 | Interleukin 1 | Immunofluorescence | Ligaments | Phagocytosis | Cartilage diseases | Index Medicus | mTOR | autophagy | osteoarthritis | cartilage | rapamycin
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2012, Volume 109, Issue 50, pp. 20602 - 20607
Osteoarthritis is one of the leading causes of chronic pain, but almost nothing is known about the mechanisms and molecules that mediate... 
Receptors | Pain | Neurons | Mice | Arthritis | Macrophages | Osteoarthritis | Behavioral neuroscience | Chemokines | Chronic pain | SYSTEM | NEUROPATHIC PAIN | CENTRAL SENSITIZATION | NOCICEPTIVE NEURONS | RAT | MULTIDISCIPLINARY SCIENCES | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | DORSAL-ROOT GANGLIA | SPINAL-CORD | MICE | EXPRESSION | Receptors, CCR2 - genetics | Receptors, CCR2 - physiology | Arthritis, Experimental - genetics | Chemokine CCL2 - immunology | Humans | Male | Pain - immunology | Ganglia, Spinal - physiopathology | RNA, Messenger - metabolism | Osteoarthritis - physiopathology | Arthritis, Experimental - pathology | Pain - genetics | Chemokine CCL2 - physiology | Osteoarthritis - pathology | Disease Models, Animal | Ganglia, Spinal - immunology | Arthritis, Experimental - physiopathology | Macrophages - pathology | Signal Transduction | Osteoarthritis - immunology | Mice, Inbred C57BL | RNA, Messenger - genetics | Chemokine CCL2 - genetics | Arthritis, Experimental - immunology | Osteoarthritis - genetics | Hyperalgesia - physiopathology | Mice, Knockout | Hyperalgesia - immunology | Animals | Hyperalgesia - genetics | Pain - physiopathology | Ganglia, Spinal - pathology | Receptors, CCR2 - deficiency | Prevention | Knee | Physiological aspects | Health aspects | Signal transduction | T cell receptors | Rodents | Index Medicus | Biological Sciences
Journal Article