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Arthritis & Rheumatology, ISSN 2326-5191, 07/2017, Volume 69, Issue 7, pp. 1480 - 1494
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2010, Volume 107, Issue 33, pp. 14751 - 14756
In several murine models of autoimmune arthritis, Th17 cells are the dominant initiators of inflammation. In human arthritis the majority of IL-17–secreting... 
T lymphocytes | Synovial fluid | Phenotypes | Cytokines | Juvenile rheumatoid arthritis | Cultured cells | Arthritis | Joint diseases | Joints | T cell antigen receptors | Juvenile | CD161 | RORC2 | RHEUMATOID-ARTHRITIS | GAMMA | MULTIDISCIPLINARY SCIENCES | JUVENILE IDIOPATHIC ARTHRITIS | CD4(+) T-CELLS | INDUCTION | MEMORY CELLS | IL-17 | juvenile | INFLAMED JOINTS | T-H-17 CELLS | LINEAGE | Receptors, CCR6 - immunology | Humans | Interleukin-17 - immunology | Molecular Sequence Data | T-Box Domain Proteins - immunology | Interferon-gamma - metabolism | Th1 Cells - immunology | Th1 Cells - metabolism | Flow Cytometry | T-Lymphocytes, Helper-Inducer - immunology | Base Sequence | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Receptors, CCR6 - metabolism | Interferon-gamma - genetics | Child | Cell Lineage - genetics | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Transforming Growth Factor beta - immunology | Amino Acid Sequence | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Gene Expression | T-Lymphocytes, Helper-Inducer - metabolism | NK Cell Lectin-Like Receptor Subfamily B - metabolism | Interleukin-17 - genetics | Interleukin-12 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Arthritis, Juvenile - genetics | NK Cell Lectin-Like Receptor Subfamily B - immunology | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Arthritis, Juvenile - metabolism | Interleukin-17 - metabolism | NK Cell Lectin-Like Receptor Subfamily B - genetics | Arthritis, Juvenile - immunology | Interferon-gamma - immunology | Interleukin-12 - immunology | Receptors, CCR6 - genetics | Transforming Growth Factor beta - metabolism | Cell Lineage - immunology | Autoimmunity | Research | Adaptation (Physiology) | Animal models | Transcription factors | Helper cells | Interleukin 12 | Interleukin 23 | Inflammation | Lymphocytes T | Transforming growth factor- beta | double prime T-cell receptor | CCR6 protein | Interleukin 17 | Plasticity | Children | Biological Sciences
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 05/2011, Volume 70, Issue 5, pp. 747 - 754
Journal Article
Lancet, The, ISSN 0140-6736, 2007, Volume 369, Issue 9563, pp. 767 - 778
Summary Juvenile idiopathic arthritis is a broad term that describes a clinically heterogeneous group of arthritides of unknown cause, which begin before 16... 
Internal Medicine | RADIOGRAPHIC PROGRESSION | ONSET SPONDYLOARTHROPATHIES | MEDICINE, GENERAL & INTERNAL | CHILDHOOD RHEUMATIC-DISEASES | CLINICAL-TRIAL | JOINT INVOLVEMENT | MIGRATION INHIBITORY FACTOR | MACROPHAGE ACTIVATION SYNDROME | REGULATORY T-CELLS | DOUBLE-BLIND | ILAR CLASSIFICATION | Steroids - administration & dosage | Diagnosis, Differential | Prognosis | Humans | Arthritis, Juvenile - diagnosis | Child, Preschool | Infant | Male | Treatment Outcome | Inflammation - immunology | HLA Antigens - genetics | Arthritis, Juvenile - genetics | Disease Progression | Arthritis, Juvenile - classification | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Arthritis, Juvenile - immunology | Arthritis, Juvenile - therapy | Adolescent | Injections, Intra-Articular | Physical Therapy Modalities | Female | Child | Chronic Disease | Development and progression | Care and treatment | Juvenile arthritis | Rheumatology | Rheumatic diseases | Bone density | Arthritis | Joint surgery | Patients | Risk factors | Fever | Children & youth | Chronic illnesses | Drugs | Laboratories | Pathogenesis | Medical services | Drug development | Immunosuppressive agents | Child development | Classification | Age | Immune system | Immune response | Psoriasis | Anemia | Medical treatment | Inflammation | Environmental factors | Disease control | Molecular chains | Antirheumatic agents | Rheumatoid arthritis | Adults
Journal Article
Genes and Immunity, ISSN 1466-4879, 06/2012, Volume 13, Issue 4, pp. 289 - 298
Macrophage activation syndrome (MAS) is a severe, frequently fatal complication of systemic juvenile idiopathic arthritis (sJIA) with features of... 
systemic juvenile idiopathic arthritis | hemophagocytosis | cytolysis | interferongamma | interleukin-1 | Macrophage activation syndrome | RHEUMATOID-ARTHRITIS | INTERLEUKIN-1 RECEPTOR ANTAGONIST | macrophage activation syndrome | FAMILIAL HEMOPHAGOCYTIC LYMPHOHISTIOCYTOSIS | LINKED LYMPHOPROLIFERATIVE DISEASE | IMMUNOLOGY | CD8(+) T-CELLS | INFLAMMATORY DISORDERS | ETANERCEPT TREATMENT | KAWASAKI-DISEASE | GENETICS & HEREDITY | ONSET STILLS-DISEASE | STEM-CELL TRANSPLANTATION | interferon-gamma | Macrophage Activation Syndrome - diagnosis | Macrophage Activation Syndrome - physiopathology | Receptors, Interleukin-1 - drug effects | Cyclosporine - pharmacology | Macrophage Activation Syndrome - drug therapy | Receptors, Interleukin-1 - immunology | Humans | Adrenal Cortex Hormones - pharmacology | Macrophage Activation Syndrome - immunology | Arthritis, Juvenile - genetics | Arthritis, Juvenile - physiopathology | Toll-Like Receptor 9 - immunology | Animals | Arthritis, Juvenile - immunology | Receptors, Interleukin-10 - antagonists & inhibitors | Mice | Antirheumatic Agents - pharmacology | Interleukin 1 Receptor Antagonist Protein - pharmacology | Macrophage Activation Syndrome - genetics | Mutation | Receptors, Interleukin-1 - antagonists & inhibitors | Child | Disease Models, Animal | Receptors, Interleukin-10 - immunology | Complications and side effects | Care and treatment | Syndromes | Development and progression | Rheumatoid arthritis in children | Genetic aspects | Diagnosis | Macrophages | Health aspects | Autoimmunity | Animal models | Liver | Central nervous system | Arthritis | Inflammation | Infection | Cell activation | Pancytopenia | Interleukin 1 | Toll-like receptors | Children
Journal Article