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Current Neurovascular Research, ISSN 1567-2026, 02/2014, Volume 11, Issue 1, pp. 68 - 74
The purpose of the study was to evaluate the expression of CD45, CD34, Sca-1 and CD117 in mouse bone marrow, Lin-ve and Lin+ve population. Bone marrow cells... 
Ataxin
Journal Article
Journal Article
Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics, ISSN 1933-7213, 08/2019, pp. 1 - 24
The effective therapeutic treatment and the disease-modifying therapy for spinocerebellar ataxia type 2 (SCA2) (a progressive hereditary disease caused by an... 
Polyglutamine | Molecular modelling | Ataxin | Therapeutic applications | Spinocerebellar ataxia | Ataxia | Trinucleotide repeat diseases | Hereditary diseases
Journal Article
Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics, ISSN 1933-7213, 07/2019, pp. 1 - 10
The spinocerebellar ataxias (SCAs) are a group of neurodegenerative disorders inherited in an autosomal dominant fashion. The SCAs result in progressive gait... 
Neurological diseases | Molecular modelling | Gait | Ataxin | Neurodegenerative diseases | Therapeutic applications | Spinocerebellar ataxia | Biology
Journal Article
Scientific reports, ISSN 2045-2322, 03/2018, Volume 8, Issue 1, pp. 4285 - 2
A correction to this article has been published and is linked from the HTML and PDF versions of this paper. The error has been fixed in the paper. 
Polyglutamine | Ataxin | Trinucleotide repeat diseases | Heat shock
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 8/2012, Volume 124, Issue 2, pp. 221 - 230
Journal Article
Journal of Molecular Neuroscience, ISSN 0895-8696, 10/2013, Volume 51, Issue 2, pp. 237 - 248
Spinocerebellar ataxia type 2 (SCA2) is caused by triple nucleotide repeat (CAG) expansion in the coding region of the ATAXN2 gene on chromosome 12, which... 
Neurochemistry | Neurology | Neurosciences | Biomedicine | Polyglutamine | Ataxin | Induced pluripotent stem cells | Spinocerebellar ataxia | Proteomics | Cell Biology | CAG REPEAT | DIRECTED DIFFERENTIATION | SCA1 | BIOCHEMISTRY & MOLECULAR BIOLOGY | NEUROSCIENCES | FAMILIAL DYSAUTONOMIA | DISEASE | HUMAN FIBROBLASTS | PATIENT | MICE | EXPRESSION | TRINUCLEOTIDE REPEAT | Humans | Middle Aged | Male | Neural Stem Cells - cytology | Neurogenesis | Cellular Reprogramming | SOXB1 Transcription Factors - metabolism | Octamer Transcription Factor-3 - genetics | Ataxins | Transfection | SOXB1 Transcription Factors - genetics | Kruppel-Like Transcription Factors - metabolism | Adult | Induced Pluripotent Stem Cells - cytology | Fibroblasts - metabolism | Induced Pluripotent Stem Cells - metabolism | Spinocerebellar Ataxias - genetics | Cells, Cultured | Spinocerebellar Ataxias - pathology | Nerve Tissue Proteins - genetics | Proto-Oncogene Proteins c-myc - metabolism | Nerve Tissue Proteins - metabolism | Octamer Transcription Factor-3 - metabolism | Fibroblasts - cytology | Proto-Oncogene Proteins c-myc - genetics | Kruppel-Like Transcription Factors - genetics | Neural Stem Cells - metabolism | Cell Line, Transformed | Genes | Stem cells | Physiological aspects | Genetic aspects | Research | Risk factors | Index Medicus | ataxin- polyglutamine | spinocerebellar ataxia
Journal Article
Acta neuropathologica, ISSN 0001-6322, 07/2019, pp. 1 - 18
Several morphological changes, centered in/around Purkinje cells (PCs), have been identified in the cerebellum of essential tremor (ET) patients. These changes... 
Cerebellum | Parkinson's disease | Neurodegenerative diseases | Tremor | Atrophy | Ataxin | Neurodegeneration | Autopsy | Purkinje cells | Spinocerebellar ataxia | Dystonia | Moveme