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Comparative Biochemistry and Physiology, Part C, ISSN 1532-0456, 11/2013, Volume 158, Issue 4, pp. 216 - 224
Journal Article
PLOS ONE, ISSN 1932-6203, 08/2019, Volume 14, Issue 8, pp. e0220920 - e0220920
Journal Article
Journal of Cleaner Production, ISSN 0959-6526, 12/2017, Volume 168, pp. 39 - 50
In the present study, magnetically Fe O /SiO /hydroxyapatite (Fe O /SiO /HAp) nanocomposites were synthesized and modified with 3-aminopropyl triethoxysilane... 
HydroxyApatite | XTT | Drug delivery | Magnetic materials | Nanostructures | Atenolol | GREEN & SUSTAINABLE SCIENCE & TECHNOLOGY | TARGETED DRUG-DELIVERY | MICROSPHERES | MESOPOROUS SILICA NANOPARTICLES | ADSORPTION | COMPOSITE | AQUEOUS-SOLUTION | ENVIRONMENTAL SCIENCES | REMOVAL | ENGINEERING, ENVIRONMENTAL | ACTIVATED CARBON | FABRICATION
Journal Article
Separation and Purification Technology, ISSN 1383-5866, 01/2020, Volume 231, p. 115927
Degradation of atenolol (ATL) was achieved using combination of UV and chlorine treatment (UV/chlorine). Factors affecting ATL degradation, such as pH,... 
UV/chlorine | Water treatment | Atenolol | Influencing factors | Degradation pathways | Acute toxicity | PHARMACEUTICALS | MECHANISM | UV/CHLORINE PROCESS | AQUEOUS-SOLUTION | ENGINEERING, CHEMICAL | DISINFECTION BY-PRODUCTS | TREATMENT PLANTS | BROMIDE | PHOTOLYSIS | RISK-ASSESSMENT | WATER
Journal Article
Ecotoxicology and Environmental Safety, ISSN 0147-6513, 10/2014, Volume 108, pp. 52 - 57
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 2004, Volume 200, Issue 2, pp. 159 - 168
The cardiotoxicity associated with doxorubicin (DOX) therapy limits the total cumulative dose and therapeutic success of active anticancer chemotherapy.... 
Heart | Mitochondria | Oxidative damage | Carvedilol | Mitochondrionopathy | Permeability transition pore | Atenolol | Doxorubicin | tetraphenylphosphonium cation | DMSO | DOX | TPP | mitochondrial transmembrane potential | LIPID-PEROXIDATION | mitochondria | carvedilol | oxidative damage | CARDIOTOXICITY | RAT-HEART MITOCHONDRIA | ANTIHYPERTENSIVE DRUG | INDUCED CARDIOMYOPATHY | heart | FREE-RADICALS | atenolol | NADH DEHYDROGENASE | permeability transition pore | ADRIAMYCIN | PHARMACOLOGY & PHARMACY | TOXICOLOGY | doxorubicin | mitochondrionopathy | DYSFUNCTION | Antibiotics, Antineoplastic - toxicity | Glutathione Reductase - metabolism | Mitochondria, Heart - pathology | Cardiomyopathies - prevention & control | Male | Onium Compounds | Ion Channels - physiology | Mitochondria, Heart - drug effects | Adrenergic beta-Antagonists - pharmacology | Mitochondrial Membrane Transport Proteins | Cardiomyopathies - physiopathology | Calcium - physiology | Superoxide Dismutase - metabolism | Glutathione Peroxidase - metabolism | Organophosphorus Compounds | Rats | Antioxidants - pharmacology | Random Allocation | Microscopy, Electron | Rats, Sprague-Dawley | Doxorubicin - toxicity | Animals | Oxygen Consumption - drug effects | Cell Surface Extensions - drug effects | Carbazoles - pharmacology | Propanolamines - pharmacology | Cardiomyopathies - chemically induced
Journal Article