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Seminars in immunopathology, ISSN 1863-2300, 2011, Volume 34, Issue 1, pp. 43 - 62
The interaction of coagulation factors with the perivascular environment affects the development of disease in ways that extend beyond their traditional roles... 
Autoimmunity | Complement receptor 3 | Multiple sclerosis | Stroke | Alzheimer’s disease | Internal Medicine | Macrophages | Blood brain barrier | Inflammatory disease | Microglia | Biomedicine | Immunology | Rheumatoid arthritis | CD11b/CD18 | Atherosclerosis | Plasminogen | Anticoagulant therapy | Alzheimer's disease | ALZHEIMERS-DISEASE | IMMUNOLOGY | PATHOLOGY | GAMMA-C-DOMAIN | BINDING-SITE | CENTRAL-NERVOUS-SYSTEM | MULTIPLE-SCLEROSIS LESIONS | CRYSTAL-STRUCTURE | BLOOD-BRAIN-BARRIER | INTEGRIN ALPHA(M)BETA | PROTEASE-ACTIVATED RECEPTORS | PERIPHERAL ARTERIAL-DISEASE | Colitis - genetics | Humans | Brain Injuries - metabolism | Arthritis, Rheumatoid - metabolism | Bacterial Infections - genetics | Inflammation - metabolism | Blood Coagulation - immunology | Muscular Dystrophy, Duchenne - immunology | Alzheimer Disease - immunology | Fibrinogen - immunology | Thromboplastin - immunology | Pulmonary Fibrosis - immunology | Vascular Diseases - genetics | Brain Injuries - genetics | Thrombin - genetics | Kidney Diseases - immunology | Arthritis, Rheumatoid - genetics | Neoplasms - immunology | Colitis - metabolism | Muscular Dystrophy, Duchenne - genetics | Bacterial Infections - metabolism | Neoplasms - metabolism | Pulmonary Fibrosis - genetics | Spinal Cord Injuries - genetics | Kidney Diseases - genetics | Stroke - genetics | Brain Injuries - immunology | Neoplasms - genetics | Bacterial Infections - immunology | Pulmonary Fibrosis - metabolism | Colitis - immunology | Thrombin - immunology | Stroke - immunology | Kidney Diseases - metabolism | Multiple Sclerosis - metabolism | Vascular Diseases - immunology | Spinal Cord Injuries - metabolism | Multiple Sclerosis - genetics | Inflammation - immunology | Fibrinogen - genetics | Stroke - metabolism | Animals | Thromboplastin - genetics | Alzheimer Disease - metabolism | Fibrinogen - metabolism | Inflammation - genetics | Multiple Sclerosis - immunology | Blood Coagulation - genetics | Spinal Cord Injuries - immunology | Muscular Dystrophy, Duchenne - metabolism | Thrombin - metabolism | Thromboplastin - metabolism | Alzheimer Disease - genetics | Arthritis, Rheumatoid - immunology | Vascular Diseases - metabolism | Nervous system diseases | Bacterial infections | Oncology, Experimental | Thrombin | Inflammation | Anticoagulants (Medicine) | Research | Rheumatoid factor | Fibrin | Fibrinogen | Genetic research | Colitis | Cancer | Traumatic brain injury | Coagulation | Tissue factor | hemostasis | Spinal cord injury | Inflammatory diseases | Kidney | Signal transduction | Duchenne's muscular dystrophy | Neurodegenerative diseases | Therapeutic applications | Thrombosis | Coagulation factors | Infection | Molecular modelling | Fibrosis | Brain injury
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 05/2009, Volume 119, Issue 5, pp. 1335 - 1349
Atherosclerosis is a chronic inflammatory disease characterized by the accumulation of oxidized lipoproteins and apoptotic cells. Adaptive immune responses to... 
ATHEROSCLEROTIC LESIONS | MEDICINE, RESEARCH & EXPERIMENTAL | STREPTOCOCCUS-PNEUMONIAE | B-CELL LINEAGES | LIPID-PEROXIDATION PRODUCTS | LINKED-IMMUNOSORBENT-ASSAY | APOPTOTIC CELLS | IN-VIVO | MONOCLONAL AUTOANTIBODIES | LOW-DENSITY-LIPOPROTEIN | OXIDIZED PHOSPHOLIPIDS | Germ-Free Life - immunology | B-Lymphocyte Subsets - transplantation | Antibody Affinity - immunology | Male | Adoptive Transfer | Phosphorylcholine - analogs & derivatives | Immunoglobulin M - immunology | Epitopes - immunology | Phosphorylcholine - immunology | Antibody Specificity - immunology | B-Lymphocyte Subsets - immunology | Female | Fetal Blood - immunology | Receptors, LDL - genetics | Antibody Formation - immunology | Atherosclerosis - pathology | Atherosclerosis - immunology | Oxidation-Reduction | Serum Albumin, Bovine - immunology | Malondialdehyde - analogs & derivatives | Mice, Inbred C57BL | Phagocytosis - immunology | Macrophages, Peritoneal - immunology | Homeodomain Proteins - genetics | Mice, Knockout | Immunity, Innate - immunology | Animals | Apoptosis - immunology | Immunoglobulin M - blood | Malondialdehyde - immunology | Mice | Lipoproteins, LDL - immunology | Proteolipids | Oxidative stress | Care and treatment | Lipoproteins | Hypercholesterolemia | Atherosclerosis | Physiological aspects | Genetic aspects | Research | Blood lipoproteins | Risk factors | Immunoglobulin M | Peritoneal | Antibody Specificity | pathology | Mikrobiologi inom det medicinska området | Macrophages | Immunity | B-Lymphocyte Subsets | Receptors | genetics | LDL | immunology | Bovine | Antibody Affinity | Epitopes | Phosphorylcholine | blood | Malondialdehyde | Inbred C57BL | Knockout | Serum Albumin | Microbiology in the medical area | Homeodomain Proteins | analogs & derivatives | transplantation | Germ-Free Life | Antibody Formation | Innate | Fetal Blood | Phagocytosis | Apoptosis
Journal Article
Journal Article
Trends in immunology, ISSN 1471-4906, 2011, Volume 32, Issue 10, pp. 470 - 477
Environmental signals at the site of inflammation mediate rapid monocyte mobilization and dictate differentiation programs whereby these cells give rise to... 
Allergy and Immunology | URINARY-TRACT-INFECTION | MYCOBACTERIUM-TUBERCULOSIS | CELLS IN-VIVO | ATHEROSCLEROTIC PLAQUES | DENDRITIC CELLS | IMMUNE-RESPONSES | BONE-MARROW | LYMPH-NODES | MYD88-DEPENDENT ACTIVATION | BACTERIAL-INFECTION | IMMUNOLOGY | Inflammation - pathology | Spleen - immunology | Monocytes - cytology | Dendritic Cells - immunology | Humans | Monocytes - immunology | Cell Movement - immunology | Signal Transduction - immunology | Inflammation - metabolism | Listeria - immunology | Listeriosis - immunology | Dendritic Cells - metabolism | Macrophages - immunology | Chemokines - immunology | Acute Disease | Atherosclerosis - pathology | Listeriosis - metabolism | Atherosclerosis - immunology | Receptors, Chemokine - metabolism | Rats | Inflammation - immunology | Spleen - cytology | Receptors, Chemokine - immunology | Immunity, Innate | Cell Differentiation - immunology | Macrophages - metabolism | Animals | Listeriosis - microbiology | Chemokines - metabolism | Mice | Chronic Disease | Development and progression | Inflammation | Macrophages | Analysis | Atherosclerosis | Studies | Heart attacks | Listeria | Ligands | Infections | Gene expression | Chemokines | Immune system | Spleen | Dendritic Cells | Signal Transduction | Listeriosis | Life Sciences | Monocytes | Immunology | Cell Differentiation | Receptors, Chemokine | Cell Movement
Journal Article
Immunology and Cell Biology, ISSN 0818-9641, 2006, Volume 84, Issue 2, pp. 115 - 124
Coronary artery disease, leading to myocardial infarction and ischaemia, affects millions of persons and is one of the leading causes of morbidity and... 
atherosclerosis | coronary artery bypass graft (CABG) | immune mechanism | intimal hyperplasia | cell proliferation | Cell proliferation | Coronary artery bypass graft (CABG) | Immune mechanism | Intimal hyperplasia | Atherosclerosis | COAGULATION-FIBRINOLYSIS SYSTEM | LOW-DENSITY-LIPOPROTEIN | IMMUNOLOGY | FACTOR-B-CHAIN | VASCULAR SMOOTH-MUSCLE | FATTY STREAK FORMATION | CELL BIOLOGY | HUMAN ATHEROSCLEROTIC PLAQUES | NITRIC-OXIDE | FOCAL ADHESION KINASE | TISSUE FACTOR | GROWTH-FACTOR-I | Leukocytes - pathology | Graft Survival - immunology | Myocardial Infarction - mortality | Hyperplasia - immunology | Humans | Myocardial Infarction - immunology | Phosphatidylinositol 3-Kinases - immunology | Leukocytes - immunology | Signal Transduction - immunology | Coronary Artery Bypass - mortality | Growth Substances - immunology | Arterial Occlusive Diseases - pathology | Arterial Occlusive Diseases - mortality | Arterial Occlusive Diseases - immunology | Myocardial Infarction - surgery | Hyperplasia - mortality | Tunica Intima - immunology | Hyperplasia - etiology | Arterial Occlusive Diseases - etiology | Hyperplasia - pathology | Coronary Artery Bypass - adverse effects | Myocardial Infarction - complications | Graft Occlusion, Vascular - etiology | Tunica Intima - pathology | Graft Occlusion, Vascular - pathology | Graft Occlusion, Vascular - immunology | Oncogene Protein v-akt - immunology | Immunity, Cellular | Tunica Intima, pathology | Graft Survival, immunology | Arterial Occlusive Diseases, immunology | Oncogene Protein v-akt, immunology | Graft Occlusion, Vascular, pathology | Coronary Artery Bypass, adverse effects | Growth Substances, immunology | Hyperplasia, etiology | Graft Occlusion, Vascular, immunology | Tunica Intima, immunology | Signal Transduction, immunology | Arterial Occlusive Diseases, mortality | Leukocytes, immunology | Leukocytes, pathology | Myocardial Infarction, immunology | Myocardial Infarction, complications | Arterial Occlusive Diseases, pathology | Coronary Artery Bypass, mortality | Hyperplasia, mortality | Arterial Occlusive Diseases, etiology | Myocardial Infarction, mortality | Hyperplasia, pathology | Hyperplasia, immunology | Coronary Artery Bypass | Graft Occlusion, Vascular, etiology | Myocardial Infarction, surgery | 1-Phosphatidylinositol 3-Kinase, immunology
Journal Article