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Basic research in cardiology, ISSN 0300-8428, 7/2018, Volume 113, Issue 4, pp. 1 - 14
Atrial arrhythmopathy | Heart failure | Two-pore-domain (K 2P ) potassium channels | K 2P 3.1 | Atrial cardiomyopathy | Medicine & Public Health | Atrial fibrillation | CREM-IbΔC-X | TASK-1 | Cardiology | Two-pore-domain (K | 3.1 | potassium channels | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Heart Atria - pathology | Atrial Remodeling | Humans | Heart Failure - physiopathology | Male | Atrial Fibrillation - physiopathology | Oocytes | Action Potentials | Cloning, Molecular | Female | Heart Atria - physiopathology | Heart Ventricles - pathology | Disease Models, Animal | Potassium Channels, Tandem Pore Domain - metabolism | Atrial Fibrillation - pathology | Heart Rate | Signal Transduction | Mice, Inbred C57BL | Xenopus laevis | Potassium Channels, Tandem Pore Domain - genetics | Heart Failure - genetics | Mice, Transgenic | Heart Failure - metabolism | Heart Failure - pathology | Atrial Fibrillation - genetics | Nerve Tissue Proteins - genetics | Heart Atria - metabolism | Nerve Tissue Proteins - metabolism | Atrial Fibrillation - metabolism | Animals | Heart Ventricles - physiopathology | Heart Ventricles - metabolism | Sus scrofa | Ventricular Remodeling - drug effects | Genetically modified animals | Heart | Heart beat | Analysis | Genetic engineering | Potassium channels | Cardiac arrhythmia | Animal models | Transgenic mice | Excitability | Action potential | Remodeling | Channels | Molecular chains | Molecular modelling | Fibrillation | Transgenic animals | Rodents | Aorta | Mice | Ventricle | Heart diseases | Potassium | Cyclic AMP response element modulator | Index Medicus
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