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Nature Reviews Cancer, ISSN 1474-175X, 01/2017, Volume 17, Issue 2, pp. 93 - 115
Journal Article
Tumor Biology, ISSN 1010-4283, 3/2016, Volume 37, Issue 3, pp. 3071 - 3080
Journal Article
Cancer Cell, ISSN 1535-6108, 04/2016, Volume 29, Issue 4, pp. 536 - 547
amplification and overexpression are common in neuroendocrine prostate cancer (NEPC). However, the impact of aberrant N-Myc expression in prostate... 
INACTIVATION | ORIGIN | ONCOLOGY | ADENOCARCINOMA | C-MYC | STEM-CELL | DIFFERENTIATION | IDENTIFICATION | CARCINOMA | EXPRESSION | BASAL-CELLS | CELL BIOLOGY | Adenocarcinoma - pathology | Epithelial Cells - metabolism | Humans | Neoplasm Proteins - physiology | Gene Expression Regulation, Neoplastic | Laser Capture Microdissection | Male | Antineoplastic Agents - therapeutic use | Genes, myc | Aurora Kinase A - antagonists & inhibitors | Molecular Targeted Therapy | Recombinant Fusion Proteins - metabolism | Exome | Neoplasm Metastasis | Proto-Oncogene Proteins c-myc - physiology | Prostatic Neoplasms - genetics | Aurora Kinase A - physiology | Neoplastic Stem Cells - metabolism | Cell Transformation, Neoplastic - genetics | Neoplastic Stem Cells - pathology | Adenocarcinoma - genetics | Neoplasm Proteins - genetics | Neuroendocrine Tumors - pathology | Prostatic Neoplasms - pathology | Transduction, Genetic | Neoplasm Invasiveness | Phenylurea Compounds - therapeutic use | Epithelial Cells - pathology | Proto-Oncogene Proteins c-akt - physiology | Mice, SCID | Azepines - therapeutic use | Neuroendocrine Tumors - genetics | Orchiectomy | Xenograft Model Antitumor Assays | Animals | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Cell Line, Tumor | Mice, Inbred NOD | Enzyme Activation | Molecular biology | Health aspects | Prostate cancer | Stem cells | Medical genetics | Cell death
Journal Article
Oncogene, ISSN 0950-9232, 01/2019, Volume 38, Issue 1, pp. 73 - 87
Primary glioblastoma is the most frequent human brain tumor in adults and is generally fatal due to tumor recurrence. We previously demonstrated that... 
INITIATING CELLS | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROLIFERATION | TEMOZOLOMIDE | CELL BIOLOGY | SUBVENTRICULAR ZONES | STEM-LIKE CELLS | INHIBITION | ONCOLOGY | GENETICS & HEREDITY | CENTRAL-NERVOUS-SYSTEM | A KINASE | RADIOTHERAPY | Chemokine CXCL12 - physiology | Glioblastoma - enzymology | Lateral Ventricles - pathology | Phosphorylation | Humans | Neoplasm Proteins - physiology | Brain Neoplasms - pathology | LIM Domain Proteins - biosynthesis | MAP Kinase Signaling System | Heterografts | Aurora Kinase A - physiology | Brain Neoplasms - enzymology | Signal Transduction | Cell Survival | Neoplasm Invasiveness | Cell Movement - drug effects | Receptors, CXCR4 - physiology | Animals | Chemokine CXCL12 - pharmacology | Glioblastoma - pathology | Cell Line, Tumor | LIM Domain Proteins - genetics | Mice | Protein Processing, Post-Translational | Enzyme Activation | Enzymes | Care and treatment | Development and progression | Genetic aspects | Regulation | Glioblastoma multiforme | Phosphotransferases | Health aspects | Vimentin | Cdc42 protein | CXCL12 protein | Cell survival | Brain tumors | Glioblastoma | Extracellular signal-regulated kinase | Guanine nucleotide-binding protein | Subventricular zone | CXCR4 protein | Actin | Xenografts | Cytoskeleton | Glioblastoma cells | Cell migration | Guanosinetriphosphatase | Target validation | Cancer stem cells | CNS cancer | Cancer microenvironment
Journal Article
Journal of Pediatric Surgery, ISSN 0022-3468, 2014, Volume 49, Issue 1, pp. 159 - 165
Journal Article
Journal Article