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PLoS ONE, ISSN 1932-6203, 01/2018, Volume 13, Issue 1, p. e0190987
Background The association between dilated cardiomyopathy (DCM) and low thyroid hormone (TH) levels has been previously described. In these patients abnormal... 
CARDIAC-FUNCTION | CHRONIC HEART-FAILURE | IODIDE TRANSPORT | TASK-FORCE | MULTIDISCIPLINARY SCIENCES | SUBCLINICAL HYPOTHYROIDISM | REPLACEMENT THERAPY | RAT-HEART | DYSFUNCTION | INDUCTION | HEMODYNAMICS | Biomarkers - metabolism | Cardiomyopathy, Dilated - genetics | Cardiomyopathy, Dilated - pathology | Receptors, Thyrotropin - genetics | Autoantigens - metabolism | Thyroid Hormones - biosynthesis | Receptors, Thyrotropin - metabolism | Dual Oxidases - metabolism | Humans | Middle Aged | Gene Expression Regulation | Male | Gene Expression Profiling | Autoantigens - genetics | Iron-Binding Proteins - metabolism | Case-Control Studies | Ventricular Remodeling | Myocardium - metabolism | Iodide Peroxidase - metabolism | Female | Iron-Binding Proteins - genetics | Iodide Peroxidase - genetics | Dual Oxidases - genetics | High-Throughput Nucleotide Sequencing - methods | RNA sequencing | Heart | Care and treatment | Analysis | Thyroxine | Thyroid gland | Transplantation | Health aspects | Cardiomyopathy, Dilated | Biomedical research | Heart attacks | Cardiomyopathy | Genes | Biosynthesis | Machinery | Gene sequencing | Ischemia | Rodents | Triiodothyronine | Thyroid hormones | Cardiology | Heart diseases | Enzyme-linked immunosorbent assay | Thyroid | Heart failure | Medical research | Medical imaging | Mortality | Heart transplantation | Iodide peroxidase | DUOX2 protein | Gene expression | Hypothyroidism | Ribonucleic acid--RNA | Patients | Dilated cardiomyopathy | Myocardium | Sampling methods | Ventricle | Endocrinology | Apoptosis | RNA | Ribonucleic acid
Journal Article
Oncotarget, ISSN 1949-2553, 2016, Volume 7, Issue 29, pp. 45776 - 45788
Journal Article
Immunity, ISSN 1074-7613, 10/2014, Volume 41, Issue 4, pp. 657 - 669
Journal Article
Circulation Research, ISSN 0009-7330, 03/2005, Volume 96, Issue 4, pp. 427 - 434
Atherosclerosis is a complex disease, bearing many of the characteristics of a chronic inflammatory process. Both cellular and humoral immune responses may be... 
Low-density lipoprotein | Antibodies | CD4+ T lymphocytes | Vaccine | Atherosclerosis | atherosclerosis | T lymphocytes | CARDIAC & CARDIOVASCULAR SYSTEMS | LOW-DENSITY-LIPOPROTEIN | OXIDIZED LDL | INDUCTION | E KNOCKOUT MICE | low-density lipoprotein | GERMINAL CENTER FORMATION | B-CELLS | vaccine | CD4 | PERIPHERAL VASCULAR DISEASE | antibodies | HEMATOLOGY | E-DEFICIENT MICE | POTENTIAL ROLE | SMOOTH-MUSCLE CELLS | T-CELLS | Apolipoproteins E - deficiency | Freund's Adjuvant - pharmacology | Immunoglobulin G - blood | Autoantibodies - blood | Cholesterol - blood | Vasculitis - prevention & control | Antigens, CD - genetics | CD4-Positive T-Lymphocytes - immunology | Freund's Adjuvant - therapeutic use | RNA, Messenger - biosynthesis | Interleukin-4 - biosynthesis | Immunoglobulin G - biosynthesis | Aortic Diseases - prevention & control | Arteriosclerosis - immunology | CD4 Antigens - genetics | Arteriosclerosis - etiology | Antigen-Presenting Cells - metabolism | Immunization | Aortic Diseases - immunology | Random Allocation | Disease Progression | Mice, Knockout | Arteriosclerosis - prevention & control | Receptors, Scavenger | Autoantigens - immunology | Lipoproteins, LDL - therapeutic use | T-Cell Antigen Receptor Specificity | Vascular Cell Adhesion Molecule-1 - biosynthesis | Mice | Arteriosclerosis - pathology | Lipoproteins, LDL - immunology | Genes, MHC Class II | Antigens, CD - biosynthesis | Aortic Diseases - blood | Autoantibodies - biosynthesis | Vasculitis - complications | Vasculitis - immunology | Immunoglobulin G - immunology | Female | Vascular Cell Adhesion Molecule-1 - genetics | Interleukin-4 - genetics | Arteriosclerosis - blood | Aortic Diseases - pathology | CD4 Antigens - immunology | Vasculitis - blood | Antigens, Differentiation, Myelomonocytic - biosynthesis | Malondialdehyde - analogs & derivatives | Mice, Inbred C57BL | RNA, Messenger - genetics | Histocompatibility Antigens Class II - biosynthesis | CD4 Antigens - physiology | Antigen-Presenting Cells - immunology | Antigens, Differentiation, Myelomonocytic - genetics | Autoantibodies - immunology | Animals | Malondialdehyde - immunology | Apolipoproteins E - genetics | Triglycerides - blood | Aortic Diseases - etiology | Interferon-gamma - biosynthesis | Receptors, Immunologic - metabolism
Journal Article
Journal Article
Journal Article
PLoS Genetics, ISSN 1553-7390, 12/2016, Volume 12, Issue 12, p. e1006474
Increasing energy expenditure through brown adipocyte recruitment is a promising approach to combat obesity. We report here the comprehensive profiling of the... 
ADIPOSE-TISSUE DEVELOPMENT | SUPER-ENHANCERS | MOUSE GENOME | ADAPTIVE THERMOGENESIS | EMBRYONIC STEM-CELLS | GENETICS & HEREDITY | ADIPOCYTE DIFFERENTIATION | PPAR-GAMMA | ADULT HUMANS | FAT DEVELOPMENT | FACTOR HOTSPOTS | CCAAT-Enhancer-Binding Protein-beta - biosynthesis | Homeodomain Proteins - metabolism | Autoantigens - genetics | Obesity - genetics | Cell Differentiation - genetics | Mesenchymal Stromal Cells | Gene Expression Regulation, Developmental | RNA, Long Noncoding - biosynthesis | Basic Helix-Loop-Helix Transcription Factors - biosynthesis | CCAAT-Enhancer-Binding Proteins - biosynthesis | CCAAT-Enhancer-Binding Proteins - genetics | Energy Metabolism - genetics | Adipogenesis - genetics | Cell Lineage - genetics | CCAAT-Enhancer-Binding Proteins - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | CCAAT-Enhancer-Binding Protein-beta - genetics | Transcription Factors - biosynthesis | Transcriptome - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | CCAAT-Enhancer-Binding Protein-beta - metabolism | Homeodomain Proteins - genetics | Obesity - metabolism | Obesity - pathology | Animals | Adipose Tissue, Brown - growth & development | Adipose Tissue, Brown - metabolism | Mice | DNA-Binding Proteins - biosynthesis | Obesity | Laboratories | Funding | Biology | Adipocytes | Kinases | Metabolism | Gene expression | Weight control | Stem cells | Expenditures | Life sciences | Standard deviation | Respiration | Bioinformatics
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2014, Volume 111, Issue 25, pp. 9157 - 9162
The transcription factor c-MYC is stabilized and activated by phosphorylation at serine 62 (S62) in breast cancer. Protein phosphatase 2A (PP2A) is a critical... 
Tumor cell line | Cell growth | Cell lines