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Journal of cellular and molecular medicine, ISSN 1582-1838, 2017, Volume 21, Issue 8, pp. 1545 - 1554
Fibrosis in animal models and human diseases is associated with aberrant activation of the Wnt/β‐catenin pathway. Despite extensive research efforts, effective... 
Smad2/3 | myofibroblast proliferation | pulmonary fibrosis | Wnt/β‐catenin | Wnt/β-catenin | MEDICINE, RESEARCH & EXPERIMENTAL | TGF-BETA | MECHANISMS | BETA-CATENIN | PROTEIN CBP | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | PATHOGENESIS | INHIBITION | PATHWAY | Wnt/beta-catenin | IDIOPATHIC PULMONARY-FIBROSIS | PROMOTE | Cell Proliferation | Cadherins - metabolism | Vimentin - metabolism | Humans | Transforming Growth Factor beta1 - metabolism | Actins - metabolism | Smad3 Protein - metabolism | Wnt Proteins - metabolism | Actins - genetics | Myofibroblasts - metabolism | Smad3 Protein - genetics | Collagen Type I - genetics | Wnt Proteins - genetics | Vimentin - genetics | Smad2 Protein - genetics | Epithelial-Mesenchymal Transition | Cadherins - genetics | Fibroblasts - metabolism | A549 Cells | Cell Line | Myofibroblasts - pathology | Collagen Type I - metabolism | Signal Transduction | Gene Expression Regulation | Smad2 Protein - metabolism | Rats | Transforming Growth Factor beta1 - genetics | Fibroblasts - pathology | Rats, Sprague-Dawley | beta Catenin - metabolism | beta Catenin - genetics | Animals | Fibrosis | Vimentin | Animal models | Collagen (type I) | Wnt protein | Mesenchyme | Pathogenesis | Transforming growth factor-b | Activation | Proteins | β-catenin | Signal transduction | Molecular modelling | Lungs | Smad2 protein | Effectors | Fibroblasts | Extracellular matrix | Autocrine signalling | Aberration | Smad2 | β‐catenin | Wnt | Original
Journal Article
Nature, ISSN 0028-0836, 01/2018, Volume 553, Issue 7689, pp. 501 - 505
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2015, Volume 10, Issue 5, p. e0126765
...(+) T cells was moderately enhanced by direct 4-1BB triggering in the absence of signaling through IL2R alpha/IL-2 interactions, but further promoted in the presence of IL-2R alpha/IL-2 interactions... 
RESPONSES IN-VIVO | INTERLEUKIN-2 | ACTIVATION | IMMUNE-RESPONSES | MULTIDISCIPLINARY SCIENCES | PROLIFERATION | IL-2 | INFECTION | COSTIMULATION | INDUCTION | PROGRESSION | Interleukin-2 - metabolism | Autocrine Communication | Gene Expression | Lymphocyte Activation | CD4-Positive T-Lymphocytes - metabolism | Receptors, Interleukin-2 - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Tumor Necrosis Factor Receptor Superfamily, Member 9 - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Mice, Knockout | CD4-Positive T-Lymphocytes - immunology | Receptors, Interleukin-2 - genetics | Animals | Signal Transduction - drug effects | CD8-Positive T-Lymphocytes - metabolism | Mice | Protein Kinase Inhibitors - pharmacology | CD8-Positive T-Lymphocytes - immunology | Proto-Oncogene Proteins c-akt - metabolism | CD27 antigen | Cell proliferation | CD8 antigen | Lymphocytes T | Biology | Kinases | T-cell receptor | Cell activation | Cell growth | Immunology | CD30 antigen | Interleukin 2 | Lymphocytes | Cell cycle | CD25 antigen | Tumor necrosis factor-TNF | Autocrine signalling | Expansion | Interleukin 2 receptors | Immunological memory | Memory cells | Population growth | CD137 antigen | Cytokines | CD4 antigen | Tumor necrosis factor | Cell death | Monoclonal antibodies | Laboratory animals | Viral infections | Cancer
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 1522-1539, 2017, Volume 312, Issue 6, pp. H1163 - H1175
.... The immediate response to high glucose and the secretion of endothelial heparanase is the release of this surface-bound VEGFB, which triggers signaling pathways and gene... 
Heparanase | Endothelial cell | Streptozotocin diabetes | Vascular endothelial growth factor B | CARDIAC-FUNCTION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | GROWTH-FACTOR-B | endothelial cell | KINASE | heparanase | DILATED CARDIOMYOPATHY | LIPOPROTEIN-LIPASE | ENDOTHELIAL HEPARANASE SECRETION | SULFATE PROTEOGLYCANS | streptozotocin diabetes | FATTY-ACID | DISEASE | PERIPHERAL VASCULAR DISEASE | RAT-HEART | vascular endothelial growth factor B | Autocrine Communication | Diabetic Cardiomyopathies - metabolism | Paracrine Communication | Rats, Wistar | Signal Transduction | Streptozocin | Cells, Cultured | Glucuronidase - metabolism | Male | Myocardium - pathology | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Apoptosis Regulatory Proteins - metabolism | Diabetic Cardiomyopathies - genetics | Animals | Vascular Endothelial Growth Factor B - metabolism | Myocardium - metabolism | Apoptosis Regulatory Proteins - genetics | Diabetic Cardiomyopathies - pathology | Vascular Endothelial Growth Factor B - genetics | Diabetes Mellitus, Experimental - chemically induced | Diabetic Cardiomyopathies - chemically induced | Endothelial Cells - enzymology | Apoptosis | Vascular Endothelial Growth Factor Receptor-1 - genetics | Physiological aspects | Cell death | Vascular endothelial growth factor | Endothelium | Hyperglycemia | Analysis | Muscles | Development and progression | Biochemistry | Gene expression
Journal Article
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 2013, Volume 342, Issue 6163, pp. 1226 - 1230
Journal Article
The Journal of experimental medicine, ISSN 1540-9538, 2012, Volume 209, Issue 3, pp. 507 - 520
Journal Article