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PLoS ONE, ISSN 1932-6203, 05/2015, Volume 10, Issue 5, pp. e0126765 - e0126765
4-1BB (CD137), a member of the tumor necrosis factor receptor superfamily (TNFRSF), is primarily expressed on activated T cells and is known to enhance... 
RESPONSES IN-VIVO | INTERLEUKIN-2 | ACTIVATION | IMMUNE-RESPONSES | MULTIDISCIPLINARY SCIENCES | PROLIFERATION | IL-2 | INFECTION | COSTIMULATION | INDUCTION | PROGRESSION | Interleukin-2 - metabolism | Autocrine Communication | Gene Expression | Lymphocyte Activation | CD4-Positive T-Lymphocytes - metabolism | Receptors, Interleukin-2 - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Tumor Necrosis Factor Receptor Superfamily, Member 9 - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Mice, Knockout | CD4-Positive T-Lymphocytes - immunology | Receptors, Interleukin-2 - genetics | Animals | Signal Transduction - drug effects | CD8-Positive T-Lymphocytes - metabolism | Mice | Protein Kinase Inhibitors - pharmacology | CD8-Positive T-Lymphocytes - immunology | Proto-Oncogene Proteins c-akt - metabolism | CD27 antigen | Cell proliferation | CD8 antigen | Lymphocytes T | Biology | Kinases | T-cell receptor | Cell activation | Cell growth | Immunology | CD30 antigen | Interleukin 2 | Lymphocytes | Cell cycle | CD25 antigen | Tumor necrosis factor-TNF | Autocrine signalling | Expansion | Interleukin 2 receptors | Immunological memory | Memory cells | Population growth | CD137 antigen | Cytokines | CD4 antigen | Tumor necrosis factor | Cell death | Monoclonal antibodies | Laboratory animals | Viral infections | Cancer | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 01/2018, Volume 553, Issue 7689, pp. 501 - 505
Journal Article
Nature Communications, ISSN 2041-1723, 2014, Volume 5, Issue 1, pp. 5811 - 5811
Epidermal growth factor receptor (EGFR) overexpression plays an important oncogenic role in cancer. Regular EGFR protein levels are increased in cancer cells... 
AUTOCRINE LOOP | RECEPTOR TYROSINE KINASES | MULTIDISCIPLINARY SCIENCES | EPIDERMAL-GROWTH-FACTOR | GENE-EXPRESSION | HUMAN GLIOMAS | CANCER-THERAPY | TRANSGENIC MICE | INNATE IMMUNITY | BRAIN-TUMORS | FACTOR-ALPHA | Receptor, Epidermal Growth Factor - genetics | Shc Signaling Adaptor Proteins - metabolism | Apoptosis - drug effects | Humans | Brain Neoplasms - pathology | Gene Expression Regulation, Neoplastic | Extracellular Signal-Regulated MAP Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Interferon Regulatory Factor-3 - genetics | Brain Neoplasms - metabolism | Proto-Oncogene Proteins c-akt - genetics | Receptor, Epidermal Growth Factor - metabolism | Glioblastoma - genetics | Glioblastoma - metabolism | Antineoplastic Agents - pharmacology | Membrane Proteins - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Protein-Serine-Threonine Kinases - metabolism | Membrane Proteins - genetics | Protein-Serine-Threonine Kinases - genetics | Brain Neoplasms - genetics | Signal Transduction - genetics | TNF-Related Apoptosis-Inducing Ligand - metabolism | Carrier Proteins - genetics | Carrier Proteins - metabolism | Shc Signaling Adaptor Proteins - genetics | Glioblastoma - pathology | Cell Line, Tumor | Interferon Regulatory Factor-3 - metabolism | Epidermal Growth Factor - pharmacology | Primary Cell Culture | TNF-Related Apoptosis-Inducing Ligand - genetics | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2017, Volume 312, Issue 6, pp. H1163 - H1175
Vascular endothelial growth factor B (VEGFB) is highly expressed in metabolically active tissues, such as the heart and skeletal muscle, suggesting a function... 
Heparanase | Endothelial cell | Streptozotocin diabetes | Vascular endothelial growth factor B | CARDIAC-FUNCTION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | GROWTH-FACTOR-B | endothelial cell | KINASE | heparanase | DILATED CARDIOMYOPATHY | LIPOPROTEIN-LIPASE | ENDOTHELIAL HEPARANASE SECRETION | SULFATE PROTEOGLYCANS | streptozotocin diabetes | FATTY-ACID | DISEASE | PERIPHERAL VASCULAR DISEASE | RAT-HEART | vascular endothelial growth factor B | Autocrine Communication | Diabetic Cardiomyopathies - metabolism | Paracrine Communication | Rats, Wistar | Signal Transduction | Streptozocin | Cells, Cultured | Glucuronidase - metabolism | Male | Myocardium - pathology | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Apoptosis Regulatory Proteins - metabolism | Diabetic Cardiomyopathies - genetics | Animals | Vascular Endothelial Growth Factor B - metabolism | Myocardium - metabolism | Apoptosis Regulatory Proteins - genetics | Diabetic Cardiomyopathies - pathology | Vascular Endothelial Growth Factor B - genetics | Diabetes Mellitus, Experimental - chemically induced | Diabetic Cardiomyopathies - chemically induced | Endothelial Cells - enzymology | Apoptosis | Vascular Endothelial Growth Factor Receptor-1 - genetics | Physiological aspects | Cell death | Vascular endothelial growth factor | Endothelium | Hyperglycemia | Analysis | Muscles | Development and progression | Biochemistry | Gene expression | Heart | Signal transduction | Diabetes | Glucose | Index Medicus
Journal Article
2000, Biomedical and health research, ISBN 4274903303, Volume 37., viii, 239
Book
Science, ISSN 0036-8075, 12/2013, Volume 342, Issue 6163, pp. 1226 - 1230
Journal Article
2001, Progress in molecular and subcellular biology, ISBN 3540417095, Volume 26., x, 186
Book
Journal of Experimental Medicine, ISSN 0022-1007, 03/2012, Volume 209, Issue 3, pp. 507 - 520
Journal Article
Nature, ISSN 0028-0836, 09/2016, Volume 538, Issue 7623, pp. 99 - 103
Journal Article