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PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, p. e53745
Background: Ipilimumab, a cytotoxic T-lymphocyte antigen-4 (CTLA-4) blocking antibody, has been approved for the treatment of metastatic melanoma and induces... 
REGRESSION | TRIAL | INDUCED SARCOIDOSIS | CTLA-4 BLOCKADE | AUTOIMMUNITY | SAFETY | MULTIDISCIPLINARY SCIENCES | MONOCLONAL-ANTIBODY | PATIENT | HYPOPHYSITIS | CANCER | Skin Neoplasms - drug therapy | Humans | Ipilimumab | Middle Aged | Antibodies, Monoclonal - adverse effects | Antibodies, Monoclonal - therapeutic use | Male | Antineoplastic Agents - therapeutic use | Neoplasm Metastasis | Liver - drug effects | Antineoplastic Agents - adverse effects | Aged, 80 and over | Endocrine System - drug effects | Adult | Female | Retrospective Studies | Antibodies, Monoclonal - immunology | Kidney - drug effects | Antineoplastic Agents - immunology | Pancreas - drug effects | Gastrointestinal Tract - drug effects | Melanoma - pathology | CTLA-4 Antigen - immunology | Nervous System - drug effects | Melanoma - drug therapy | Respiratory System - drug effects | Aged | Skin - drug effects | Antimitotic agents | Antigens | Care and treatment | Algorithms | Lymphocytes | Melanoma | Skin | Metastasis | Antineoplastic agents | Skin cancer | Cancer | Aseptic meningitis | Guillain-Barre syndrome | Exanthema | Central nervous system | Blocking antibodies | Cytotoxicity | Oncology | Lymphocytes T | Metastases | CTLA-4 protein | Intestine | Gastroenterology | Meningitis | Internal medicine | Hematology | Sarcoidosis | Dermatology | Management | Regression analysis | Patients | Eosinophilia | Morbidity | Inflammatory bowel disease | Pathology | Side effects | Hospitals | Perforations | Medical prognosis | Antitumor activity | Autoimmune diseases | Endocrinology
Journal Article
Nature, ISSN 0028-0836, 06/2012, Volume 486, Issue 7404, pp. 554 - 558
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2011, Volume 108, Issue 28, pp. 11692 - 11697
Type 1 diabetes (T1D) is an autoimmune disease characterized by insulitis and islet β-cell loss. Thus, an effective therapy may require β-cell restoration and... 
Depolarization | Antiinflammatories | Cytokines | Diabetes complications | Islet cells | Mice | Diabetes | Type 2 diabetes mellitus | Insulin | Type 1 diabetes mellitus | Inflammation | Regulatory T-cell | AMINOBUTYRIC-ACID GABA | insulin | RAT | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | RECEPTOR | RELEASE | inflammation | AUTOIMMUNITY | regulatory T-cell | MICE | EXPRESSION | T-CELLS | ONSET | Apoptosis - drug effects | Male | Phosphatidylinositol 3-Kinases - metabolism | T-Lymphocytes, Regulatory - immunology | gamma-Aminobutyric Acid - physiology | Inflammation Mediators - metabolism | Female | Diabetes Mellitus, Type 1 - immunology | Proto-Oncogene Proteins c-akt - metabolism | Immunosuppressive Agents - pharmacology | Insulin-Secreting Cells - physiology | Membrane Potentials - drug effects | Hyperglycemia - prevention & control | Diabetes Mellitus, Type 1 - physiopathology | Diabetes Mellitus, Type 1 - pathology | gamma-Aminobutyric Acid - pharmacology | Mice, Transgenic | Insulin-Secreting Cells - immunology | Diabetes Mellitus, Type 1 - drug therapy | Regeneration - drug effects | T-Lymphocytes, Regulatory - drug effects | Animals | Insulin-Secreting Cells - drug effects | Signal Transduction - drug effects | Calcium Signaling - drug effects | Mice, Inbred NOD | Cell Proliferation - drug effects | Insulin-Secreting Cells - pathology | Cytokines - biosynthesis | GABA | Physiological aspects | Pancreatic beta cells | Research | Type 1 diabetes | Biological Sciences
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2014, Volume 111, Issue 36, pp. 13139 - 13144
Journal Article
Journal of Autoimmunity, ISSN 0896-8411, 2012, Volume 39, Issue 3, pp. 222 - 228
Journal Article
Gastroenterology, ISSN 0016-5085, 2017, Volume 152, Issue 8, pp. 2052 - 2062.e2
Background & Aims Interferon-free direct-acting antiviral (DAA) therapies are effective in patients with hepatitis C virus–induced cryoglobulinemia vasculitis... 
Gastroenterology and Hepatology | Immune Regulation | PDCD1 | Response To Treatment | Liver Disease | BAFF | ENVELOPE PROTEIN | RANDOMIZED CONTROLLED-TRIAL | MIXED CRYOGLOBULINEMIA | DEFICIENCY | AUTOIMMUNITY | RITUXIMAB | EXPANSION | REGULATORY T-CELL | GASTROENTEROLOGY & HEPATOLOGY | LYMPHOCYTES | T-Lymphocyte Subsets - immunology | Prospective Studies | Hepatitis C - drug therapy | Humans | Middle Aged | Cryoglobulinemia - diagnosis | Male | Vasculitis - virology | Cryoglobulinemia - virology | Hepatitis Viruses - drug effects | Simeprevir - therapeutic use | Case-Control Studies | Simeprevir - adverse effects | Viral Load | Vasculitis - immunology | Hepatitis C - immunology | Immunity, Cellular - drug effects | Time Factors | T-Lymphocyte Subsets - drug effects | B-Lymphocyte Subsets - immunology | Female | Hepatitis C - complications | Imidazoles - therapeutic use | Sofosbuvir - adverse effects | Drug Therapy, Combination | B-Lymphocyte Subsets - virology | Cytokines - blood | Immune Tolerance - drug effects | Sofosbuvir - therapeutic use | B-Lymphocyte Subsets - drug effects | Antiviral Agents - therapeutic use | Hepatitis Viruses - immunology | Cryoglobulinemia - drug therapy | Imidazoles - adverse effects | Ribavirin - therapeutic use | Treatment Outcome | Biomarkers - blood | T-Lymphocyte Subsets - virology | Cryoglobulinemia - immunology | Hepatitis C - diagnosis | Phenotype | B-Lymphocyte Subsets - metabolism | Vasculitis - diagnosis | Ribavirin - adverse effects | T-Lymphocyte Subsets - metabolism | Antiviral Agents - adverse effects | Vasculitis - drug therapy | Aged | Care and treatment | Vasculitis | Analysis | Gastrointestinal diseases | Interferon | Biological response modifiers | Hepatitis C virus | Antiviral agents | Medical research | Liver | Medicine, Experimental | Life Sciences | Immunology
Journal Article
Journal Article
Nature, ISSN 0028-0836, 2013, Volume 503, Issue 7474, pp. 126 - 130
In systemic sclerosis (SSc), a common and aetiologically mysterious form of scleroderma (defined as pathological fibrosis of the skin), previously healthy... 
PLASMACYTOID DENDRITIC CELLS | PATHOGENESIS | TGF-BETA | MULTIDISCIPLINARY SCIENCES | DISEASE | MICE | SKIN | SYSTEMIC-SCLEROSIS | EXPRESSION | FIBROBLASTS | I INTERFERON | Fibrosis - drug therapy | Antibodies, Antinuclear - immunology | Scleroderma, Systemic - pathology | Skin Diseases, Genetic - drug therapy | Male | Integrins - metabolism | Mutation, Missense - genetics | Contracture - drug therapy | Plasma Cells - drug effects | T-Lymphocytes, Helper-Inducer - drug effects | Antibodies, Neutralizing - immunology | Autoimmunity - immunology | Antibodies, Neutralizing - therapeutic use | Integrins - drug effects | Transforming Growth Factor beta - antagonists & inhibitors | Dendritic Cells - drug effects | Female | Contracture - prevention & control | Microfilament Proteins - metabolism | Skin Diseases, Genetic - prevention & control | Contracture - immunology | Fibrosis - prevention & control | Microfilament Proteins - genetics | Transforming Growth Factor beta - immunology | Microfilament Proteins - chemistry | Fibrillin-1 | Skin Diseases, Genetic - immunology | Contracture - pathology | Skin Diseases, Genetic - pathology | Antibodies, Neutralizing - pharmacology | Fibrillins | Scleroderma, Systemic - immunology | Animals | Amino Acid Substitution - genetics | Mice | Fibrosis - pathology | Amino Acid Motifs - genetics | Autoimmunity - drug effects | Scleroderma, Systemic - drug therapy | Scleroderma, Systemic - prevention & control | Autoimmunity | Cell interaction | Systemic scleroderma | Scleroderma (Disease) | Research | Prevention | Complications and side effects | Fibrosis | Physiological aspects | Dosage and administration | Drug therapy | Molecular biology | Health aspects | Integrins | Flow cytometry | Cell culture | Cytokines | Rodents | Collagen | Mutation | Scleroderma | Age
Journal Article
New Phytologist, ISSN 0028-646X, 03/2017, Volume 213, Issue 4, pp. 1802 - 1817
Journal Article