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Autonomic & autacoid pharmacology, ISSN 1474-8665, 2002
Journal
1996, 1, The Taylor & Francis series in pharmaceutical sciences., ISBN 0748405569, viii, 612
.... Covering the diverse group of drugs acting on the autonomous nervous system, their actions are reviewed together with their clinical uses, side effects, interactions and subcellular mechanisms of action... 
Autonomic drugs | Autonomic nervous system | Anatomy | Neuroscience | Pharmacology
Book
Journal of the American College of Cardiology, ISSN 0735-1097, 11/2009, Volume 54, Issue 19, pp. 1747 - 1762
.... Among these responses is the activation of the sympathetic nervous system (SNS) that provides inotropic support to the failing heart increasing stroke volume... 
adrenergic receptor | beta-blockers | heart failure | sympathomimetic inotropes | sympatholytics | sympathetic nervous system | CARDIAC-INSUFFICIENCY BISOPROLOL | OBSTRUCTIVE SLEEP-APNEA | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | AUTONOMIC FUNCTION | RANDOMIZED CONTROLLED-TRIAL | DILATED CARDIOMYOPATHY | LEFT-VENTRICULAR DYSFUNCTION | BETA-BLOCKADE | POSITIVE AIRWAY PRESSURE | BETA-ADRENERGIC RECEPTOR | Receptors, Adrenergic, alpha-1 - metabolism | Sympathetic Nervous System - drug effects | Humans | Heart Failure - physiopathology | Adrenergic alpha-Antagonists - therapeutic use | Receptors, Adrenergic, beta-2 - drug effects | Sympathetic Nervous System - physiopathology | Sympatholytics - therapeutic use | Cardiotonic Agents - therapeutic use | Receptors, Adrenergic - genetics | Parasympathetic Nervous System - physiopathology | Digoxin - therapeutic use | Adrenergic beta-Antagonists - therapeutic use | Sympathomimetics - therapeutic use | Receptors, Adrenergic, alpha-1 - drug effects | Heart Failure - genetics | Heart Failure - metabolism | Receptors, Adrenergic, beta-2 - metabolism | Heart Failure - drug therapy | Polymorphism, Genetic | Randomized Controlled Trials as Topic | Parasympathetic Nervous System - drug effects | Exercise Tolerance | Renin-Angiotensin System - drug effects | Hemodynamics - drug effects | Proteins | Confidence intervals | Heart rate | Heart failure | Heart attacks | Rodents | Mortality | Hospitalization | Kinases | Beta blockers
Journal Article
Trends in autonomic pharmacology, ISSN 0197-0569, 1979
Journal
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 7, p. e22682
Oxidative stress in the central nervous system mediates the increase in sympathetic tone that precedes the development of hypertension... 
NADPH OXIDASE | LIFE-SPAN | CARDIOVASCULAR REGULATION | SPONTANEOUSLY HYPERTENSIVE-RATS | SUPEROXIDE-PRODUCTION | SIGNALING PATHWAY | MULTIDISCIPLINARY SCIENCES | II INFUSED RATS | NITRIC-OXIDE | ANGIOTENSIN-CONVERTING ENZYME | BLOOD-PRESSURE | Genetic Therapy | Reactive Oxygen Species - metabolism | Sympathetic Nervous System - drug effects | Autonomic Nervous System - pathology | NADPH Oxidases - metabolism | Brain - enzymology | Central Nervous System - pathology | Vagus Nerve - drug effects | Male | Sympathetic Nervous System - physiopathology | Primary Dysautonomias - physiopathology | Autonomic Nervous System - physiopathology | Primary Dysautonomias - pathology | Vagus Nerve - pathology | Gene Deletion | Peptidyl-Dipeptidase A - metabolism | Blood Pressure - drug effects | Sympathetic Nervous System - pathology | Angiotensin II - pharmacology | Autonomic Nervous System - drug effects | Brain - physiopathology | Myocardium - pathology | Aging - pathology | Vagus Nerve - physiopathology | Animals | Brain - pathology | Cell Line, Tumor | Central Nervous System - drug effects | Mice | Central Nervous System - physiopathology | Oxidative Stress - drug effects | Oxidases | Hypertension | Advertising executives | Oxidative stress | Angiotensin | Central nervous system | Genetic engineering | Negotiation, mediation and arbitration | Gene therapy | Heart | Brain | Health sciences | Reactive oxygen species | ACE2 gene | Oxidase | Smooth muscle | Nervous system | Paraventricular nucleus | Gene deletion | Kinases | Experiments | Angiotensin-converting enzyme 2--ACE2 | NAD(P)H oxidase | Infusion | Reduction | Clonal deletion | Autonomic nervous system | Rodents | Cell cycle | Aging | Physiology | Blood pressure | Angiotensin II | Heart diseases | Genotypes | Age | Enzymes | Transgenic mice | Cardiomyocytes | Pharmacology | Dysautonomia | Sympathetic nervous system | Medicine | Insects | Hypoxia | In vivo methods and tests | Angiotensin-converting enzyme 2 | ACE2
Journal Article
Journal of autonomic pharmacology, ISSN 0144-1795, 1980
Journal
Journal Article
Journal Article
Journal Article
Scientific reports, ISSN 2045-2322, 2012, Volume 2, Issue 1, p. 898
Journal Article
Psychopharmacology (Berlin, Germany), ISSN 1432-2072, 2017, Volume 235, Issue 2, pp. 467 - 479
...), and placebo in a double-blind, cross-over study in 24 healthy participants. Acute drug effects were tested using psychometric scales, the Facial Emotion Recognition Task (FERT... 
Emotion recognition | Neurosciences | Biomedicine | Modafinil | Sexual arousal | Pharmacology/Toxicology | Psychiatry | Methylphenidate | MDMA | POSTTRAUMATIC-STRESS-DISORDER | PSYCHIATRY | 3,4-METHYLENEDIOXYMETHAMPHETAMINE ECSTASY | INTRANASAL OXYTOCIN | NEUROSCIENCES | MALE-VOLUNTEERS | PLASMA OXYTOCIN | NOREPINEPHRINE TRANSPORTERS | PHARMACOLOGY & PHARMACY | CENTRAL-NERVOUS-SYSTEM | PROSOCIAL FEELINGS | D-AMPHETAMINE | Central Nervous System Stimulants - pharmacology | Facial Recognition - physiology | Humans | Male | Facial Recognition - drug effects | Healthy Volunteers | Libido - physiology | Emotions - drug effects | Arousal - drug effects | Libido - drug effects | Sexual Behavior - psychology | Young Adult | N-Methyl-3,4-methylenedioxyamphetamine - pharmacology | Adult | Female | Sexual Behavior - drug effects | Sexual Behavior - physiology | Emotions - physiology | Hallucinogens - pharmacology | Modafinil - pharmacology | Double-Blind Method | Autonomic Nervous System - drug effects | Autonomic Nervous System - physiology | Methylphenidate - pharmacology | Cross-Over Studies | Arousal - physiology | Drugs | Complications and side effects | Smart drugs and nutrients | Ecstasy (Drug) | Adverse and side effects | Comparative analysis | Drug abuse | Face recognition | Oxytocin | Hyperactivity | Attention deficit hyperactivity disorder | Sexual behavior | Cognitive ability | Psychotherapy | Pattern recognition | Hydrocortisone | Emotions | Sleep disorders | Side effects | Prolactin | Psychopharmacology | Arousal | Narcolepsy | Anxiety | Acute effects | Drug dosages | Original Investigation
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 3, p. e57700
.... Altered autonomic nervous system (ANS) activity is an important physiological contributor to the generation of anxiety... 
NEONATAL EXPOSURE | TYROSINE-HYDROXYLASE PHOSPHORYLATION | ENDOTOXIN EXPOSURE | MESSENGER-RNA EXPRESSION | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | RECEPTOR | RAT HIPPOCAMPUS | STRESS | ALTERS | PITUITARY-ADRENAL AXIS | Animals, Newborn | Tyrosine 3-Monooxygenase - metabolism | Rats, Wistar | Anxiety - blood | Brain - physiopathology | Anxiety - physiopathology | Rats | Male | Phosphoserine - metabolism | Autonomic Nervous System - physiopathology | Anxiety - immunology | Brain - drug effects | Brain - metabolism | Gene Expression Regulation - drug effects | Animals | Respiration - drug effects | Autonomic Nervous System - immunology | Lipopolysaccharides - pharmacology | Brain - pathology | Behavior, Animal - drug effects | Female | Weight Gain - drug effects | Corticosterone - blood | Phosphorylation - drug effects | Infants (Newborn) | Tyrosine | Brain | Corticosteroids | RNA | Physiological aspects | GABA | Mitogens | Protein binding | Neonates | Adrenal glands | γ-Aminobutyric acid A receptors | Glucocorticoids | Nervous system | Mental depression | Lipopolysaccharides | Microorganisms | γ-Aminobutyric acid | Autonomic nervous system | Anxiety | Physiology | Behavior | Psychopathology | Stress response | Medical research | Hydroxylase | Exposure | Long term effects | Gene expression | Light effects | Respiratory rate | Pharmacy | Adults | Stimuli | Phosphorylation | Laboratories | Gene regulation | Glands | Schizophrenia | mRNA | Ontogeny | Hypothalamus | Tyrosine 3-monooxygenase | Rodents | Arousal | Tumor necrosis factor-TNF | Functional programming | Immune system | Hypothalamic-pituitary-adrenal axis | Prefrontal cortex | Stress | Brain research | Anxieties | Pituitary | Protein expression | Respiration | Animal cognition
Journal Article