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Nature (London), ISSN 1476-4687, 2019, Volume 567, Issue 7747, pp. 262 - 266
Cyclic GMP-AMP (cGAMP) synthase (cGAS) detects infections or tissue damage by binding to microbial or self DNA in the cytoplasm(1). Upon binding DNA, cGAS... 
SYNTHASE | CYCLIC GMP-AMP | MAVS | PROTEIN | ROLES | MULTIDISCIPLINARY SCIENCES | ADAPTER | SENSOR | Nucleotides, Cyclic - metabolism | Interferons - biosynthesis | Beclin-1 - deficiency | Vesicular Transport Proteins - metabolism | Humans | Endoplasmic Reticulum - metabolism | Autophagy-Related Protein 5 - genetics | Autophagy-Related Protein-1 Homolog - metabolism | DNA Viruses - metabolism | Interferons - immunology | Autophagy | DNA Viruses - genetics | Sea Anemones | Membrane Proteins - deficiency | HEK293 Cells | Membrane Proteins - metabolism | Nucleotidyltransferases - metabolism | Protein-Serine-Threonine Kinases - metabolism | Phosphate-Binding Proteins | Signal Transduction | Membrane Proteins - genetics | DNA, Viral - metabolism | Class III Phosphatidylinositol 3-Kinases - metabolism | Autophagosomes - metabolism | Cytosol - virology | Protein Transport | Carrier Proteins - genetics | Animals | Autophagy-Related Protein-1 Homolog - deficiency | Carrier Proteins - metabolism | Monomeric GTP-Binding Proteins - metabolism | Autophagy-Related Protein 5 - deficiency | Autophagy-Related Protein 5 - metabolism | Beclin-1 - genetics | Golgi Apparatus - metabolism | Mice | Beclin-1 - metabolism | Autophagy-Related Protein-1 Homolog - genetics | Nucleotides, Cyclic - immunology | Evolution, Molecular | Biological research | Autophagy (Cytology) | Enzymes | Physiological aspects | Interferon | Research | Cytoplasm | Biology, Experimental
Journal Article
Molecular cell, ISSN 1097-2765, 2016, Volume 63, Issue 5, pp. 781 - 795
Mutations in the human autophagy gene EPG5 cause the multisystem disorder Vici syndrome. Here we demonstrated that EPG5 is a Rab7 effector that determines the... 
RAB effector | LC3 | autophagosome maturation | epg-5 | SNARE | MEMBRANE-FUSION | LYSOSOME FUSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MACHINERY | MECHANISMS | SYNTAXIN 17 | MATURATION | VESICLE | C. ELEGANS | HOPS COMPLEX | CELL BIOLOGY | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Humans | Cataract - pathology | Qb-SNARE Proteins - metabolism | R-SNARE Proteins - metabolism | Caenorhabditis elegans Proteins - metabolism | Qb-SNARE Proteins - genetics | rab GTP-Binding Proteins - genetics | Endosomes - metabolism | Lysosomes - metabolism | Qc-SNARE Proteins - metabolism | Agenesis of Corpus Callosum - genetics | Endosomes - ultrastructure | Qa-SNARE Proteins - genetics | Autophagy - genetics | Synaptosomal-Associated Protein 25 - genetics | rab GTP-Binding Proteins - metabolism | Agenesis of Corpus Callosum - metabolism | Amino Acid Sequence | Caenorhabditis elegans - metabolism | Membrane Fusion | Signal Transduction | Caenorhabditis elegans - genetics | R-SNARE Proteins - genetics | Gene Expression Regulation | Autophagosomes - metabolism | Cataract - metabolism | Agenesis of Corpus Callosum - pathology | Autophagosomes - ultrastructure | Lysosomes - ultrastructure | Proteins - genetics | Sequence Homology, Amino Acid | Sequence Alignment | Animals | Proteins - metabolism | Qa-SNARE Proteins - metabolism | Protein Binding | Qc-SNARE Proteins - genetics | Cataract - genetics | Synaptosomal-Associated Protein 25 - metabolism | HeLa Cells | Caenorhabditis elegans Proteins - genetics | Yuan (China)
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 10, p. e0186615
Background Idiopathic pulmonary fibrosis (IPF) is a chronic, progressive and fatal disease. Histone deacetylase 6 (HDAC6) alters function and fate of various... 
EPITHELIAL-MESENCHYMAL TRANSITION | LUNG FIBROSIS | IN-VITRO | TGF-BETA | HISTONE DEACETYLASE INHIBITION | MULTIDISCIPLINARY SCIENCES | TUBULIN ACETYLATION | DISEASE | AUTOPHAGY | MYOFIBROBLAST DIFFERENTIATION | EXPRESSION | Idiopathic Pulmonary Fibrosis - genetics | Ribosomal Protein S6 Kinases - metabolism | TOR Serine-Threonine Kinases - metabolism | Humans | Middle Aged | Phosphoprotein Phosphatases - metabolism | Male | Phosphatidylinositol 3-Kinases - metabolism | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Autophagy - drug effects | Idiopathic Pulmonary Fibrosis - metabolism | Mechanistic Target of Rapamycin Complex 1 | Autophagosomes - drug effects | Multiprotein Complexes - metabolism | Tubulin - metabolism | Bleomycin | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Aged, 80 and over | Female | Indoles - pharmacology | Lung - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Hydroxamic Acids - pharmacology | Fibroblasts - metabolism | Lung - pathology | Collagen Type I - metabolism | Histone Deacetylases - genetics | Histone Deacetylase 6 | RNA, Messenger - genetics | Histone Deacetylases - metabolism | Nuclear Proteins - metabolism | Autophagosomes - metabolism | Mice, Knockout | Transforming Growth Factor beta - pharmacology | Animals | Signal Transduction - drug effects | Fibroblasts - drug effects | Idiopathic Pulmonary Fibrosis - pathology | Hydroxamic Acids - therapeutic use | Indoles - therapeutic use | Aged | Transforming Growth Factor beta - metabolism | Idiopathic Pulmonary Fibrosis - drug therapy | Histone deacetylase | Deregulation | Collagen (type I) | Phosphorylation | Disease | Mesenchyme | Pathogenesis | Critical care | AKT protein | Leucine | Kinases | Autophagy | Proteins | Fibroblasts | Vascular endothelial growth factor | Internal medicine | Lung diseases | Environmental health | 1-Phosphatidylinositol 3-kinase | Medicine | Pulmonary fibrosis | Inhibitors | Lungs | Lysine | Deacetylation | Collagen | Fibrosis | Mice | Protein phosphatase | Phagocytosis
Journal Article
Autophagy, ISSN 1554-8635, 2018, Volume 15, Issue 4, pp. 652 - 667
EBV has been reported to impair monocyte in vitro differentiation into dendritic cells (DCs) and reduce cell survival. In this study, we added another layer of... 
monocytes | SQSTM1/p62 | autophagy | dendritic cells | ROS | STAT3 | EBV | ATG5 | NFE2L2 | ACTIVATION | PHOSPHORYLATION | TRANSCRIPTION | p62 | CANCER | CELL BIOLOGY | NRF2 | BIOGENESIS | INFECTION | SQSTM1 | EPSTEIN-BARR-VIRUS | Reactive Oxygen Species - metabolism | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Humans | Cell Survival - genetics | Apoptosis - genetics | Monocytes - metabolism | rab GTP-Binding Proteins - genetics | Autophagy-Related Protein 5 - genetics | Mitochondrial Proteins - genetics | DNA-Binding Proteins - metabolism | Kelch-Like ECH-Associated Protein 1 - genetics | Cell Differentiation - genetics | Mitochondrial Proteins - metabolism | NF-E2-Related Factor 2 - genetics | Autophagy - genetics | Dendritic Cells - virology | Sequestosome-1 Protein - metabolism | Dendritic Cells - metabolism | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | rab GTP-Binding Proteins - metabolism | Herpesvirus 4, Human - physiology | Interleukin-4 - metabolism | Kelch-Like ECH-Associated Protein 1 - metabolism | Mitochondria - metabolism | Signal Transduction - genetics | Autophagosomes - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Autophagosomes - virology | Transcription Factors - metabolism | Monocytes - virology | Sequestosome-1 Protein - genetics | NF-E2-Related Factor 2 - metabolism | Autophagy-Related Protein 5 - metabolism | RNA, Small Interfering
Journal Article
Autophagy, ISSN 1554-8635, 2016, Volume 12, Issue 3, pp. 547 - 564
Journal Article
Autophagy, ISSN 1554-8635, 2014, Volume 6, Issue 4, pp. 506 - 522
Journal Article
Autophagy, ISSN 1554-8627, 04/2019, Volume 15, Issue 4, pp. 707 - 725
Helicobacter pylori (H. pylori) is a common human pathogenic bacterium. Once infected, it is difficult for the host to clear this organism using the innate... 
PDIA3 | calcium | Helicobacter pylori | Autophagy | lysosome | vitamin D3 | MUCOSA | DISEASE | ELECTRON-MICROSCOPY | CELL BIOLOGY | Epithelial Cells - metabolism | Calcium - metabolism | Protein Disulfide-Isomerases - metabolism | Epithelial Cells - drug effects | Humans | Lysosomes - enzymology | Helicobacter pylori - drug effects | Male | Antimicrobial Cationic Peptides - metabolism | Receptors, Calcitriol - genetics | Autophagy - drug effects | Anti-Bacterial Agents - therapeutic use | Autophagosomes - drug effects | Acid Phosphatase - metabolism | Lysosomes - metabolism | Acetylglucosaminidase - metabolism | Cholecalciferol - therapeutic use | Helicobacter Infections - drug therapy | Autophagy - genetics | Transient Receptor Potential Channels - metabolism | Antimicrobial Cationic Peptides - genetics | Stomach - drug effects | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Autophagosomes - microbiology | Lysosomes - drug effects | Cell Line | Helicobacter pylori - growth & development | Mice, Inbred C57BL | Autophagosomes - metabolism | Receptors, Calcitriol - metabolism | Autophagosomes - ultrastructure | Protein Disulfide-Isomerases - genetics | Animals | Carrier Proteins - metabolism | Lysosomes - microbiology | Transient Receptor Potential Channels - genetics | Epithelial Cells - microbiology | Autophagy-Related Protein 5 - metabolism | Anti-Bacterial Agents - pharmacology | Helicobacter pylori - isolation & purification | Stomach - microbiology | Cholecalciferol - pharmacology
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 2018, Volume 360, Issue 6390, pp. 751 - 758
Journal Article
Journal Article