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Applied Physiology, Nutrition, and Metabolism, ISSN 1715-5312, 2017, Volume 42, Issue 2, pp. 148 - 156
Obesity is a known risk factor for the development of hepatic disease; obesity-induced fatty liver can lead to inflammation, steatosis, and cirrhosis and is... 
diabète | stéatose hépatique non alcoolique | nonalcoholic fatty liver disease | exercice physique | mitochondria | exercise | diabetes | insulin resistance | insulinorésistance | mitochondries | Insulin resistance | Mitochondria | Nonalcoholic fatty liver disease | Exercise | Diabetes | APOPTOSIS | SPORT SCIENCES | PHYSIOLOGY | FATTY LIVER-DISEASE | MITOCHONDRIAL FISSION | PROTECTS | IMPACT | NUTRITION & DIETETICS | METABOLISM | STEATOSIS | Tumor Necrosis Factor-alpha - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - genetics | Up-Regulation | Liver - pathology | Mitochondria, Liver - metabolism | Obesity - immunology | Tumor Necrosis Factor-alpha - genetics | Non-alcoholic Fatty Liver Disease - etiology | Liver - physiopathology | Motor Activity | Autophagy | Liver - immunology | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - metabolism | Interleukin-6 - metabolism | Biomarkers - metabolism | Hepatomegaly - prevention & control | Mitochondria, Liver - pathology | Interleukin-6 - genetics | Liver - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Insulin Resistance | Obesity - physiopathology | Autophagosomes - metabolism | Diet, Western - adverse effects | Autophagosomes - pathology | Mitochondrial Degradation | Obesity - metabolism | Obesity - pathology | Animals | Autophagosomes - immunology | Mitochondria, Liver - immunology | Hepatomegaly - etiology | Physiological aspects | Obesity | Autophagy (Cytology) | Care and treatment | Research
Journal Article
世界胃肠病学杂志:英文版, ISSN 1007-9327, 2016, Volume 22, Issue 47, pp. 10353 - 10363
AIM To investigate the effects of active vitamin D3 on autophagy and interleukin(IL)-1β expression in Salmonella-infected intestinal epithelial... 
Intestinal epithelia | Salmonella | Vitamin D | Autophagy | Interleukin-1β | Atg16L1 | CELLS | PROTEIN | CROHNS-DISEASE | Interleukin-1 beta | INFLAMMATORY-BOWEL-DISEASE | COLITIS | GENE-EXPRESSION | 1,25-DIHYDROXYVITAMIN D-3 | D-RECEPTOR | GASTROENTEROLOGY & HEPATOLOGY | BETA-DEFENSIN 2 | Intestinal Mucosa - metabolism | Epithelial Cells - metabolism | Microtubule-Associated Proteins - metabolism | Epithelial Cells - drug effects | Humans | Nod2 Signaling Adaptor Protein - genetics | Salmonella Infections - metabolism | Interleukin-1beta - genetics | RNA, Messenger - metabolism | Receptors, Calcitriol - genetics | Autophagy - drug effects | Intestinal Mucosa - drug effects | Autophagosomes - drug effects | Transfection | RNA Interference | Time Factors | Interleukin-1beta - metabolism | Autophagy-Related Proteins - genetics | Caco-2 Cells | Salmonella Infections - drug therapy | Salmonella Infections - pathology | Salmonella typhimurium - pathogenicity | Anti-Inflammatory Agents - pharmacology | RNA, Messenger - genetics | Epithelial Cells - pathology | Autophagosomes - metabolism | Intestinal Mucosa - microbiology | Receptors, Calcitriol - metabolism | Autophagosomes - pathology | Nod2 Signaling Adaptor Protein - metabolism | Signal Transduction - drug effects | Receptors, Calcitriol - agonists | Calcitriol - pharmacology | Epithelial Cells - microbiology | Salmonella Infections - microbiology | Anti-Bacterial Agents - pharmacology | Autophagy-Related Proteins - metabolism | Intestinal Mucosa - pathology | Basic Study
Journal Article
Journal Article
The EMBO Journal, ISSN 0261-4189, 04/2018, Volume 37, Issue 7, p. n/a
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 8/2010, Volume 120, Issue 2, pp. 209 - 222
The mechanisms underlying neurodegenerative diseases are the outcome of pathological alterations of evolutionary conserved molecular and cellular cascades. For... 
Pathology | Aplysia | Neurosciences | Autophagosomes | Medicine & Public Health | Alzheimer’s disease | Axoplasmic transport | Lysosomes | Tauopathy
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 12/2016, Volume 55, Issue 6, pp. 837 - 847
Journal Article
Journal Article
Basic & Clinical Pharmacology & Toxicology, ISSN 1742-7835, 05/2018, Volume 122, Issue 5, pp. 489 - 500
Collapse of the mitochondrial membrane potential (MMP) is often considered the initiation of regulated cell death (RCD). Carbonyl cyanide... 
SYNERGISM | CARDIOLIPIN | UNFOLDED PROTEIN RESPONSE | BIOGENESIS | MEMBRANE | ELIMINATION | PHARMACOLOGY & PHARMACY | VALPROIC ACID | PERMEABILIZATION | TOXICOLOGY | QUALITY-CONTROL | AUTOPHAGY | Vincristine - pharmacology | Reactive Oxygen Species - metabolism | Uncoupling Agents - pharmacology | Humans | Membrane Potential, Mitochondrial - drug effects | Autophagy - drug effects | Burkitt Lymphoma - pathology | Dose-Response Relationship, Drug | Autophagosomes - drug effects | Lysosomes - metabolism | Proteasome Endopeptidase Complex - drug effects | Time Factors | Carbonyl Cyanide m-Chlorophenyl Hydrazone - pharmacology | Lysosomes - pathology | Quinazolinones - pharmacology | Burkitt Lymphoma - drug therapy | Lysosomes - drug effects | Cell Survival - drug effects | Unfolded Protein Response - drug effects | Proteasome Inhibitors - pharmacology | Iodoacetamide - pharmacology | Burkitt Lymphoma - metabolism | Mitochondria - metabolism | Autophagosomes - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Autophagosomes - pathology | Leupeptins - pharmacology | Mitochondrial Dynamics - drug effects | Mitochondrial Degradation - drug effects | Cell Line, Tumor | Oxidative Stress - drug effects | Proteasome Endopeptidase Complex - metabolism | Drugs | Evaluation | Divalproex | Glucose metabolism | Cell death | Lymphomas | Biosynthesis | Biochemistry | Valproic acid | Protons | Ubiquitin | Cyanide | Flow cytometry | Oxidative stress | Mitochondrial DNA | Cyanides | Drug delivery | Autophagy | Electron transport chain | Mitochondria | Membrane potential | Quinazolinone | Damage | Drug dosages | Carbonyls | Collapse | Translocation | Cell survival | Mortality | Survival | Vincristine | Lymphoma | Proteasome inhibitors | Burkitt's lymphoma | Cytometry | Inhibitors | Antibiotics | Doxycycline | Glycolysis | Electron transport | Destabilization | Phagocytosis | Cellular stress response | Apoptosis | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2018, Volume 13, Issue 10, p. e0204967
Necrotizing enterocolitis (NEC) remains the leading cause of gastrointestinal morbidity and mortality in premature infants. Human and animal studies suggest a... 
PATHOGENESIS | SEQUENCES | MULTIDISCIPLINARY SCIENCES | SECRETORY AUTOPHAGY | INFANT GUT | PREVENTION | LYSOZYME | MICE | BACTERIAL-INFECTION | ASSOCIATION | KLEBSIELLA-PNEUMONIAE | Animals, Newborn | Paneth Cells - metabolism | Mice, Inbred C57BL | Paneth Cells - pathology | Enterocolitis, Necrotizing - microbiology | Enterobacteriaceae - isolation & purification | Muramidase - metabolism | Autophagosomes - metabolism | Cecum - microbiology | Diphtheria Toxin - toxicity | Autophagosomes - pathology | Animals | Gastrointestinal Microbiome - drug effects | Paneth Cells - drug effects | Klebsiella pneumoniae - physiology | Mice | Dithizone - toxicity | Enterocolitis, Necrotizing - pathology | Enterobacteriaceae - growth & development | Cytokines - blood | Disease Models, Animal | Enterocolitis, Pseudomembranous | Prognosis | Microbiota (Symbiotic organisms) | Enterocolitis, Neonatal necrotizing | Demographic aspects | Infants (Premature) | Physiological aspects | Causes of | Genetic aspects | Research | Diseases | Klebsiella | Neonates | Flow cytometry | Pediatrics | Animal models | Pathogenesis | Microbiomes | Homeostasis | Necrotizing enterocolitis | Infants | Food science | Enterocolitis | Autophagy | Small intestine | Microorganisms | Microbiota | Immunology | Intestine | Tumor necrosis factor-TNF | Bioinformatics | Departments | Bacterial infections | Cecum | Histology | Inflammation | Paneth cells | Electron microscopy | Morbidity | Polymerase chain reaction | Organic chemistry | Depletion
Journal Article
Oncotarget, ISSN 1949-2553, 2016, Volume 7, Issue 49, pp. 80716 - 80734
In the present study, it was found that the ruthenium (II) imidazole complex [ Ru(Im) 4(dppz)](2+) (Ru1) could induce significant growth inhibition and... 
Cytotoxicity | Reactive oxygen species | Ruthenium imidazole complex | Autophagy | Apoptosis | BLADDER-CANCER | DNA-BINDING | OXIDATIVE STRESS | autophagy | apoptosis | CISPLATIN RESISTANCE | SPECTRAL PROPERTIES | CELL BIOLOGY | OXYGEN SPECIES ROS | POLYPYRIDYL COMPLEXES | IN-VITRO | PHASE ARREST | METHYLIMIDAZOLE COMPLEXES | reactive oxygen species | ruthenium imidazole complex | cytotoxicity | Lung Neoplasms - drug therapy | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Microtubule-Associated Proteins - metabolism | Humans | Lung Neoplasms - metabolism | Caspase 3 - metabolism | Lung Neoplasms - pathology | Extracellular Signal-Regulated MAP Kinases - metabolism | Autophagy - drug effects | Dose-Response Relationship, Drug | Autophagosomes - drug effects | Time Factors | Female | Antineoplastic Agents - pharmacology | A549 Cells | Cytochromes c - metabolism | Imidazoles - pharmacology | Mitochondria - metabolism | Autophagosomes - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Ruthenium - pharmacology | Autophagosomes - pathology | Xenograft Model Antitumor Assays | Animals | Signal Transduction - drug effects | Tumor Burden - drug effects | Cell Cycle Checkpoints - drug effects | Mice, Nude | Cell Proliferation - drug effects | Mice, Inbred BALB C | Organometallic Compounds - pharmacology
Journal Article
Cancer Cell, ISSN 1535-6108, 07/2008, Volume 14, Issue 1, pp. 90 - 102
Renal cell carcinomas (RCCs) are refractory to standard therapies. The von Hippel-Lindau ( ) tumor suppressor gene is inactivated in 75% of RCCs. By screening... 
CHEMBIO | CELLCYCLE | HYPOXIA-INDUCIBLE FACTOR | MALIGNANT GLIOMA-CELLS | PROTEIN | CANCER CELLS | ONCOLOGY | PATHWAY | DEATH | ENDOPLASMIC-RETICULUM STRESS | GROWTH-FACTOR | LINDAU TUMOR-SUPPRESSOR | HIF-ALPHA | CELL BIOLOGY | Kidney Neoplasms - genetics | Microtubule-Associated Proteins - metabolism | Humans | Gene Expression Regulation, Neoplastic | Carcinoma, Renal Cell - genetics | Male | Kidney Neoplasms - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Structure-Activity Relationship | Hypoxia-Inducible Factor 1 - metabolism | Vacuoles - drug effects | Vacuoles - pathology | Autophagy - drug effects | Dose-Response Relationship, Drug | Transfection | Basic Helix-Loop-Helix Transcription Factors - metabolism | Time Factors | Inhibitory Concentration 50 | Carcinoma, Renal Cell - enzymology | Antineoplastic Agents - pharmacology | Molecular Structure | Von Hippel-Lindau Tumor Suppressor Protein - genetics | Von Hippel-Lindau Tumor Suppressor Protein - metabolism | Yeasts - metabolism | Cell Survival - drug effects | Carcinoma, Renal Cell - pathology | Gene Silencing | Yeasts - drug effects | Antineoplastic Agents - chemistry | Mice, SCID | Protein Transport | Carcinoma, Renal Cell - metabolism | Animals | Autophagy-Related Protein 5 | Kidney Neoplasms - enzymology | Yeasts - growth & development | Cell Line, Tumor | Vacuoles - metabolism | Golgi Apparatus - metabolism | Kidney Neoplasms - pathology | Cell Proliferation - drug effects | Mice | Pyridines - pharmacology | Thiazoles - pharmacology | Hydrogen-Ion Concentration | Proteins | Cancer | Index Medicus | autophagy | von Hippel-Lindau | cell death | Renal cell carcinoma | autophagosomes | lysosomes | LC3
Journal Article